L1 Mild TBI Flashcards
(53 cards)
define concussion/mild TBI
- observed/self report of confusion, disorientation, impaired consciousness, memory dysfx around time of injury, loss of consciousness lasting < 30 min
- alteration of consciousness for 24 hrs (GCS)
- post traumatic amnesia <24 hrs
- GCS 13-15 30 min post injury
glasgow coma scale is used when?
after a TBI or head trauma
to assess level of consciousness for inpatient setting and track changes
components of GCS
eye response
motor response
verbal response
GCS scoring: eye
- eyes open spontaneously
- open eyes to verbal command
- eyes open to pain
- no eye opening
GCS scoring: motor
- obeys commands
- localizes pain
- withdraws from pain
- flexion response to pain
- extension response to pain
- no motor response
GCS scoring: verbal
- oriented
- confused
3, inappropriate words - incomprehensible sounds
- no verbal response
structural damage of mild TBI
no structural changes and minimal to no cell death
as opposed to mod/severe which has structural brain injury and cell death
girls vs boys in sports concussions
girls more likely to report
girls more likely to have mental health, severe, or long lasting symptoms
fewer research on girls and concussions
fewer ATs available at games for care of young female athletes
may be more prevalent due to weaker cervical stabilizers in girls
CDC definition of mild TBI
caused by blow/jolt to head, causing brain to move back and forth
creates chemical changes and stretches/damages brain cells in more mod injury
imaging results of mild TBI
normal MRI or CT
does not mean brain function is normal
causes of TBI
direct: local injury
coup/contracoup: acceleration/deceleration injury affecting front and back of brain, stretching in the middle
blast injury with diffuse injury
what age groups are most likely to get concussion
10-17 y/o
risk also increases after 70 y/o due to falls
red flags after a concussion
headache that worsens
drowsiness, can’t be awakened
can’t recognize people/place
repeated vomiting
confusion/irritability
seizures
weak/numb arms and legs
unsteadiness/slurred speech
worsening dizziness
worsening disequilibrium
double vision
all are possible signs of a brain bleed
timeline for improvement in concussion symptoms
7-14 days
maybe 3 weeks
s/s of concussion
irritability
anxiety
sadness
inability to sleep
sensitivity to lights and sound
memory loss
concentration and attention decreased
cognitive fatigue from reading
loss of coord/speed/dexterity
balance, ocular function impairment
metabolic pathology of concussion
- axonal damage
- neuroinflammation
- ionic dysfunction of glutamate
- energy crisis
axonal damage in concussion
may or may not be present
recovers with remyelination over time
generates lots of the recovery seen after TBI
neuroinflammation in concussion
microglia activated by injury, create inflammation
creates excitotoxicity leading to cell damage
ionic dysfunction in concussion
membrane homeostasis is disrupted, leading to excess glutamate release
increased excitation reduces brains ability to inhibit when needed
energy crisis in concussion
excess excitation leads to mitochondrial dysfunction and energy depletion or oxidative stress
hypermetabolism with more brain areas active than needed
creates fatigue
effects of increased brain metabolic activity after concussion
reduced efficiency
reduced information processing
reduced dual tasking
reduced divided attention
increased fMRI brain area activation
increased fatigue
increased sensitivity to environmental stimuli
vestibular system takes what input?
- angular acceleration/rotation
- head position relative to gravity
3 cranial nerves of oculomotor system
3oculomotor
4trochlear
6abducens
lateral vestibular tract gets info from:
primarily LE