L10 - Eating Disorders 1 Flashcards

1
Q

What are the growing rates of dieters?

A
  • 42.2% of adolescents trying to lose weight in 2015
  • 26% of people in England and Wales in 2021 are obese
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2
Q

What was a study looking at changes in weight control behaviours and weight perception in adolescents in the UK?

A
  • Methods: examined weight controlled behaviours, weight perception and depression in 3 cohorts of adolescents born in 1970, 1991-2 and 2000-2
  • Results: Increase in weight control behaviours over time
  • Higher in girls but prevalence increased more in boys
  • Results not explained by changes in BMI across cohorts
  • Increase over time in adolescent’s over-estimating their weight
  • Overestimation of weight is associated with higher depression
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3
Q

What is the role of the media

A
  • Media bombard us with a standard of beauty
  • Promotes thin ideal
  • Media forms associations between beautiful (thin) people with happy lives and high moral standards
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4
Q

What was a study on body image in women?

A
  • 146 undergrad women had an ecologically momentary assessment: frequency of appearance comparisons, direction of comparison, context, satisfaction, mood, diet/exercise thoughts and behaviours
  • In person comparisons were most common context
  • Upward comparisons most common and were associated with greatest negative outcomes on social media
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5
Q

What is an Eating Disorder?

A
  • Persistent disturbance of eating, or eating-related behaviour
  • Impaired physical health and psychological wellbeing
  • Examples are Anorexia Nervosa, Bulimia Nervosa and Binge-Eating Disorder
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6
Q

What is Anorexia Nervosa?

A
  • Restriction of energy intake relative to requirements, leading to significantly low body weight
  • Intense fear of gaining weight
  • Disturbance in the way one’s body weight or shape is experiences
  • Results in restricting and binge-eating/purging
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7
Q

What are the Psychological Symptoms of AN? (DIP IN LID)

A
  • Intense fear of weight gain
  • Irritability and anxiety
  • Low self-esteem
  • Pre-occupation with food, calories, weight, size
  • Impaired memory, attention, concentration
  • Not recognising the seriousness of the condition
  • Distorted or unhelpful thinking styles
  • Distorted body image
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8
Q

What are the behavioural symptoms of AN?

A
  • Avoidance or restriction of eating
  • Excessive exercising
  • Lengthy/frequent toilet visits
  • Overt or secret
  • Drinking excessive fluids
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9
Q

What are the physiological symptoms of AN?

A
  • Affects whole body
  • Brain and nerves: can’t think right, fear of gaining weight, moody, bad memory
  • Heart: low bp, slow heart rate, heart palpitations and heart failure
  • Hair: thins and brittles
  • Blood: anemia
  • Muscles: weak, swollen joints, fractures, osteoporosis
  • Kidneys: stones and failures
  • Body fluids: low potassium, magnesium and sodium
  • Intestines: constipation, bloating
  • Skin: bruise easily, dry skin, growth of fine hair all over body, get cold easily, yellow skin, brittle nails
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10
Q

What is Bulimia Nervosa (BN)

A
  • Recurrent episodes of binge eating: feel a lack of control overeating
  • Recurrent inappropriate compensatory behaviours to prevent weight gain
  • Binge-eating and compensatory behaviours occur on average once a week for three weeks
  • Self-evaluation unduly influenced by body shape and weight
  • Not experiencing AN
  • Includes Purging and non-purging
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11
Q

What is Binge-Eating Disorder (BED

A
  • Recurrent episodes of binge eating
  • 3+ of the following: Eating more rapidly than normal, till uncomfortably full, large amounts of food when not hungry, eating alone due to embarrassment, feelings of disgust/depression/guilt
  • Distress
  • At least once a week for 3 months on average
  • No compensatory behaviour: Not AN/BN
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12
Q

What are comorbidities with eating disorders?

A
  • Depression
  • OCD
  • Substance-Use disorders
  • Personality disorders
  • Autism spectrum disorder
  • Eating disorders crossover with each other
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13
Q

What are other un/specified Eds?

A

Using DSM-IV diagnostic criteria, 60% of treatment seeking adolescents and adults are diagnosed with ‘Eating Disorder Not Otherwise Specified’

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14
Q

What is the transdiagnostic CBT-Model?

A
  • Developed to address the high degree of movement within Eds and to support those who have an unspecified ED
  • Life events that affect mood, stress, wellbeing
  • Thoughts include: over-evaluation of eating, perfectionism, low self-esteem
  • Behaviours: restriction of energy/food intake/binge eating, and compensatory weight control behaviours
  • Emotions: Negative emotions and difficulty tolerating these
  • Physiological: physical health changes related to low body weight
  • Apply to both BN and AN
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15
Q

What is the prevalence of ED?

A
  • Binge Eating Disorder - 2%
  • BN - 1%
  • AN - 0.9% women, 0.3% men
  • Other: most common
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16
Q

Is the prevalence of ED changing over time?

A
  • Mistakenly thought to be a modern condition
  • AN stable for past decades, incidence among those <15 yo has increased
  • BN increase 1970-1990, but now in decline
17
Q

What was a study looking at who would have an ED?

A
  • Searched movie and TV characters depicting individuals with an ED and coded demographics of each character
  • Heterosexual: 75.56%
  • White: 84.85%
  • Women: 89.39%
  • Under 30yo: 84.85%
18
Q

What is the Average age of onset of Eds?

A
  • AN: 16-20yo
  • BN: 21-24
  • BED: 30-50
19
Q

What is the difference in gender in Eds?

A
  • Gender ratio previously thought to be 10:1, but recent estimates suggest 3:1
  • Men experience more sociocultural pressure to have toned/muscular bodies
  • Gay and Bisexual men
20
Q

Why are Eds underdiagnosed in men?

A
  • Seen as a women’s disorder
  • Bias in diagnostic criteria: emphasis on desire to be thin vs lean/muscular, and methods of weight control
  • Leads to risk of misdiagnosis and less likely to receive appropriate, specialist treatment
21
Q

What is the diversity of EDs?

A
  • Lack of cohesion and sense of belonging in identity
  • ED serves a purpose: gain control and to affirm gender identity
  • Discrimination
  • EDs are experienced differently by people who are gender diverse or neurodivergent
  • Perception that different identities are intertwined and interact
22
Q

How are Eating disorders are multi-determined?

A
  • Biological factors
  • Individual risk factors
  • Family influences
  • Sociocultural factors
23
Q

What are the genetics of ED?

A
  • Start by looking at family rates since one would predict higher rates in relatives
  • Found ED at 3x more common among relatives of individuals with AN or BN than among relatives of individuals without AN/BN
  • Indicative of some element of familial transmissibility
  • AN > 10 times more common in relatives In AN sufferers
  • Twin studies show there is a strong but not 100% genetic contribution
  • If there is genetic mechanism underlying the familial pattern, it is not an all-or-nothing mechanism, more consistent with increased susceptibility
24
Q

How is the genetic influence mediated?

A
  • Three obvious ways:
  • Determination of body weight/space/size
  • Determination of brain chemistry
  • Determination of personality traits
  • Not mutually exclusive
25
Q

What brain structures are involved in EDs?

A
  • Hypothalamus: Ventromedial hypothalamus and lateral hypothalamus are involved in satiety and appetite
  • Frontal Cortex: Temporal cortex is involved in body image perception & Orbitofrontal cortex involved in monitoring the stimuli pleasantness
26
Q

What neurotransmitters are involved in Eds?

A
  • Serotonin: Involved in mood, obsessions, appetite regulation and impulse control
  • Patients with AN have low levels of serotonin metabolites
  • After recovery, BN/AN patients have higher levels of 5-HIAA than controls
  • Dopamine: Recent interest given its role in reward systems
27
Q

What is Set Point Theory?

A
  • Hormones involved in appetite and weight regulation
  • Hormones: Leptin (satiety) and Grehlin (hunger)
  • Leptin reduces and grehlin increases food intake
  • Leptin secretion is profoundly disturbed in AN and the degree of hypo-leptinaemia in acute AN is an indicator of the severity of the disorder.
28
Q

Puberty and the role of Sex Hormones:

A
  • Puberty is a key risk period for the development of Eds
  • Most theories focus on the risks arising from the psychosocial effects of pubertal development in girls
  • Before puberty, genetic influences account for 0% of the variability in disordered eating, but genetic factors account for over 50% during and after puberty
  • Oestradiol is potent regulator of gene transcription in CNS and recent theories propose that ovarian hormones may activate genetic risk for disordered eating in girls during puberty
29
Q

Study looking at support for Eds?

A
  • Facebook micro-intervention to improve body image
  • Randomly assigned to one of 1. body positive facebook group, 2. appearance neutral facebook 3. facebook as usual
  • Body positive and neutral groups experienced decreased body dissatisfaction (small to medium)
  • Body positive group experienced decreased appearance comparisons (small to medium effects)
30
Q

Activities to Promote Positivity Body Image and Body Acceptance?

A
  • Curate body positive and inclusive messages
  • Exercise for pleasure, strength and accomplishment
  • Make a gratitude list
  • Challenge negative body talk
  • Practise self-kindness