L2 Flashcards

(94 cards)

1
Q

Nasolabial Cyst

A

the Ala of the nose may be elevated

Female to Male 3:1

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2
Q

Nasolabial cyst treatment

A

Biopsy, surgical removal

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3
Q

Incisive canal Cyst develops from

A

Epithelial remnants of the nasopalatine duct

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4
Q

Incisive Canal Cyst occurs

A

At the spices of maxillary central incisors

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5
Q

Incisive Canal Cyst Treatment

A

Simple curettage is curative

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6
Q

Incisive Canal Histopathology

A

Highly variable cyst lining

Cyst wall contains contents of the incisive Canal

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7
Q

Epidermoid Cyst arise from

A

Arise from the hair follicle

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8
Q

Epidermoid cysts usually affect

A

Facial skin, neck or back

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9
Q

Milia

A

Very small epidermoid cysts

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10
Q

epidermoid Cyst treatment

A

Simple excision

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11
Q

Epidermoid Cysts histopathology

A

Cystic lining that resembles the epidermis with production of orthokeratin

Have a very doughy feel to them because they are lined with that keratinized tissue

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12
Q

Milia often occur

A

In clusters/multiples

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13
Q

Milia most common location

A

Perioorbital location

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14
Q

Milia treatment

A

Resolve spontaneously by self marsupialization

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15
Q

Dermoid Cyst lined by

A

Epidermis like epithelium

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16
Q

Dermoid cyst walls

A

Contains adnexal skin structures

-hair follicles, sebaceous glands, sweat glands

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17
Q

Dermoid cysts may be classified as _____

A

Benign cystic form of teratoma composed of tissue derived from multiple germ layers

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18
Q

Cysts of the incise papilla

A

No bony involvement

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19
Q

Dermoid cysts most commonly

A

Occurs as fluctuate swelling midline floor of mouth

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20
Q

Dermoid above geniohyoid

A

Displaces tongue superiors

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21
Q

Dermoid below geniohyoid

A

Submental swelling

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22
Q

Dermoid cysts treatment

A

Surgical excision

Rare malignant transformation

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23
Q

Thyroglobulin Duct Cyst arise

A

From remnants of thyroglassal tract

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24
Q

Thyroglobulin Duct Cyst may be

A

Attached to hyoid bone or tongue and may elevate on swelling

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25
Thyroglobulin Duct Cyst
Midline; foramen cecum to suprasternal notch
26
Thyroglobulin Duct Cyst treatment
High recurrence rate unless treated by Sistrunk Procedure | -Excision with a portion of hyoid bone and musucural tissue
27
Thyroglobulin Duct Cyst Histopathology
Lined by columnar or statrified squamous epithets with thyroid tissue in cyst wall
28
Bronchial Cleft Cyst
Cervical variant of lymphoepithelial cyst
29
Bronchial Cleft Cyst arise from
Remnants of the branchial arches; 2nd arch
30
Bronchial Cleft Cyst flucutuant
Swelling of upper lateral neck
31
Bronchial Cleft Cyst is usually _______ to SCM
Anterior
32
Bronchial Cleft Cyst may develop
Fistula tract
33
Bronchial Cleft Cyst treatment
Surgical removal - minimal recurrence - questionable malignant transformation
34
Bronchial Cleft Cyst Histopathology
Line by stratified squamous epithelium with lymphoid tissue in cyst wall
35
Oral Lymphoepithelial Cyst Counter part
Oral counterpart of branchial cleft cyst
36
Oral Lymphoepithelial Cyst arise
Arises from epithelial rests trapped in oral lymphoid tissue
37
Oral Lymphoepithelial Cyst
Soft to firm, yellowish white nodule
38
Oral Lymphoepithelial Cyst treatment
Surgical excision is curative
39
Oral Lymphoepithelial Cyst histopathology
Lined by stratified squamous epithelium with lymphoid tissue in cyst wall
40
Enamel Hypoplasia
Incomplete or defective enamel formation
41
Enamel Hypoplasia causes
Variety of environmental influences - Turners Hypoplasia - Antineoplastic agents - Flurosis - Syphilis
42
Attrition
Physiological wear due to tooth to tooth contact during occlusion
43
Abrasion
Tooth structure loss secondary to external agent
44
Erosion
Cupped out depression of occlusal surfaces of cusp tips associated with regurgitated gastric acid or dietary acid
45
Abfraction
Wedge shaped defect limited to cervical area Bruxism associated
46
Internal Resorption
Destruction of tooth structure accomplished by cells located in the dental pulp
47
Teeth may appear pink
Internal resorption
48
Internal resorption therapy
Endo therapy prior to perforation | -once communication with PDL
49
External Resorption
Destruction of tooth structure accomplished by cells located in the PDL
50
External resorption causes
``` Chronic inflammation Cysts NEoplasms Trauma Re-implantation of avlused tooth Impaction Ortho Idiopathic ```
51
External Resorption treatment
Depends on extent Extraction may be necessary
52
Extrinsic Stains
Surface accumulation of exogenous pigment which can typically be removed by prophylaxis
53
Intrinsic stains
Endogenous material is incorporated into developing teeth | -deposition of circulation substances including drugs and blood pigments
54
Hypodontia
reduced number of teeth
55
Oligodontia
Lack of 6 or more teeth
56
Anodontia
Total lack of teeth development
57
Hypodontia most affected
Third molars, second premolars, Lateral incisors
58
Hyperdontia
Presence of supernumerary teeth
59
Microdotnia
Small teeth may also affect shape
60
Microdontia most commonly affect
Maxilllary laterals and third molars
61
Macrodontia
Large teeth Uncommon Associated with various syndromes and systemic disease
62
Germination
Partial division of single tooth buds; Crown appears split Tooth count is normal Anterior teeth
63
Fusion
2 tooth buds merge to from single large tooth Separate root canals but share cementum and dentin Reduced number of teeth in arch
64
Concrescence
Characterized by fusion of two teeth by cementum alone More frequent in posterior maxilla
65
Talon Cusp
Affects maxillary incisors An exaggerated cingulum
66
Dens Evaginatus
Extra cusp in central developmental groove Premolar teeth most commonly affected; mandibular predominance
67
Dens Invaginatus
Maxillary lateral incisors Tooth within a tooth perhaps due to invagination of tooth bud Quite variable in severity Affected tooth becomes non vital shortly after eruption
68
Enamel Pearl
Droplets of ectopic enamel Furcation of maxillary or mandibular molars
69
Enamel pearls may have
Dentin and pulp horns
70
Enamel pearls detected by
Probing if mistaken for calculus can lead to exposure
71
Taurodontism
Enlargement of the body and pulp chamber of a multi roots tooth Apical displacement of pulpal floor and furcation
72
Tarudontism results
Chromosomal lacerations Associated with various syndromes
73
Hypercementosis
Asymptomatic deposition of excessive cementum
74
Hypercementosis most cases
Are sporatic Strong association with pagers disease of bone
75
Dilaceration
Curvature/bend of tooth roots
76
Dilaceration etiology
Trauma to developing tooth
77
Dilaceration complication s
Extraction or RCT may be difficult
78
Amelogenisis Imperfecta
Group of uncommon genetic disorders affecting enamel of teeth
79
Amelogenisis Imperfecta affects
Both primary and permanent teeth
80
Amelogenisis Imperfecta Hypoplastic
Inadequate deposition of enamel matrix
81
Amelogenisis Imperfecta hypomaturation
Incomplete mineralization
82
Amelogenisis Imperfecta hypocalcified
No significant degree of mineralization
83
Clinical Amelogenisis Imperfecta
Rough smooth pitted pigmented or snow capped
84
Amelogenisis Imperfecta radiographic
Thin enamel of normal or decreased density; normal root and pulp morphology
85
Dentinogenesis Imperfecta inheritance
Autosomal dominant
86
Dentinogenesis Imperfecta affects
Both primary and secondary just like AI
87
Dentinogenesis Imperfecta results
In abnormal dentin formation
88
Dentinogenesis Imperfecta clinically
Teeth appear translucent “Opalescent teeth” Enamel is normal but poorly supported rapid attrition of dentition is often seen
89
Dentinogenesis Imperfecta radiographic
Bulbous crowns and obliteration of pulps
90
Dentinogenesis Imperfecta due to
Mutation of DSPP gene
91
DSPP gene
Dentin sialophosphorprotein | -one of the major non collagenous proteins of dentin
92
Dentin Dysplasia
Autosomal Dominant inheritance
93
Type I Dentin Dysplasia
Radicular dentin dysplasia -radicular roots are very short “rootless teeth” obliteration of pulp, periapical radiolucencies
94
Type II Dentin Dysplasia
Coronal Dentin Dysplasia Thought to be related to dentinogenesis imperfect Enlarged pulps with thistle tube appearance, pulp stones