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Oral Path 1 > L4 > Flashcards

L4 Flashcards

(61 cards)

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1
Q

Localized Juvenile spongiotic gingival hyperplasia Clinical

A

Bright red, velvety or papillary plaque

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2
Q

Localized Juvenile spongiotic gingival hyperplasia location

A

Facial gingiva; maxillary predilection

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3
Q

Localized Juvenile spongiotic gingival hyperplasia Tx

A

No response to improved oral hygiene

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4
Q

Necrotizing Ulcerative gingivitis

A

Mixed bacterial infection

Stress, poor oral hygiene, poor diet, immune suppression, smoking

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5
Q

Necrotizing Ulcerative gingivitis Clinical

A

Punched out interdental papillae

Localized or diffuse gingival involvement

Severe pain, oral malodor, spontaneous hemorrhage

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6
Q

Necrotizing Ulcerative gingivitis occasionally the process spreads

A

To adjacent soft tissues

Necrotizing ulcerative mucositis, stomatitis

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7
Q

Necrotizing Ulcerative gingivitis if infection extends through mucosa to cutaneous surface of face termed

A

noma (cancrum oris)

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8
Q

NUG Treatment

A

Debridement
Mild salt water rinse or chlorhexidine
Improve oral hygiene and diet

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9
Q

Desquamative Gingivitis

A

Sloughing of the gingival epithelium

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10
Q

Desquamative Gingivitis associated with

A

Several different immune mediate vesiculobullous diseases

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11
Q

Desquamative Gingivitis is not

A

A diagnosis

Clinical description

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12
Q

Desquamative Gingivitis patient management

A

Incisional biopsy is necessary for definitive diagnosis

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13
Q

Drugs-Related Gingival Hypeplasia -Degree of clinical enlargement related to patients susceptibility and level or oral hygiene

A

Abnormal growth of gingival tissues secondary to use of systematic medication

-diffuse involvement

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14
Q

Drugs-Related Gingival Hypeplasia -Degree of clinical enlargement related to

A

patients susceptibility and level or oral hygiene

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15
Q

First drug associated with drug related gingival hyperplasia

A

Dilantin (phenytoin)

Then came

Nifedipine
Cyclosporin

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16
Q

Drug-related gingival hyperplasia Tx

A

Removal of the offending medication may result in cessation and some regession of the gingival enlargement

home plaque control

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17
Q

Gingival Fibromatosis.

A

Slowly progressive collagenous overgrowth of the gingiva

Isolated or familial; localize or generalized

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18
Q

Gingival Fibromatosis gingiva is

A

Firm and normal color

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19
Q

Gingival Fibromatosis other findings sometimes observed

A

Hypertichosis
Epilepsy
Intellectual disability

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20
Q

Gingival Fibromatosis Tex

A

Oral hygiene instruction

Gingivectomy

Selective tooth extraction sometimes necessary in severe cases

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21
Q

Gingival Fibromatosis gingivectomy

A

Ideally delayed until after complete eruption of permanent dentition;
Reduced tendency for recurrence

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22
Q

Impetigo

A

Superficial infection of the skin causes but Staph aureus or Strep pyogenes

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23
Q

Impetigo is easily

A

Spread in crowded unsanitary living conditions

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24
Q

Impetigo peak occurrence

A

During summer or early fall in hot moist climates

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25
Impetigo is most common in
School aged children
26
Impetigo clinical presentation
Facial lesions often around nose and mouth Erythema with superficial vesicles that quickly rupture and become covered in a thick amber crusts
27
Impetigo cases may arise in areas
Of damaged skin; predicting dermatitis, cuts, scratches, insect bites
28
Impetigo diagnosis
Presumptive diagnosis based on clinical presentation | Definitive diagnosis requires isolation of causative organisms in culture of skin
29
Impetigo treatment
Topical or systemic antibiotics
30
Tonsillolithiasis
Calcified structures that develops in enlarged Tonsillar crypts
31
Tonsilar confections
Convoluted crypts of the tonsils are commonly filled with desaquamted cells, foreign debris and bacteria
32
Tonsilloliths
Aggregates of desquamated cells that undergo calcification
33
Tonsillolithiasis clinical presentation
Enlarged crypts filled with yellowish debris varies from soft to fully calcified Variable size Foul smelling Solitary or multiple
34
Tonsillolithiasis Tx
No treatment necessary unless associated with clinical symptoms
35
Syphilis
Chronic infection caused by the spirochete treponema pallidum
36
Syphilis spread by
Intimate sexual contact Transplacental transmission Contaminated blood exposure
37
Syphilis is highly infectious during ________
First two stages
38
Primary Syphilis
Chancre Resolves spontaneously in 3+ weeks
39
Secondary Syphilis
Develops 4-10 weeks after initial infection - Lymphadenopathy - Erythematous maculopapular cutaneous eruption - Mucous patches and condylomata lata or ora mucosa - Split papules at angles of mouth
40
Untreated secondary patients
Will enter latent period
41
Tertiary Syphilis
Develops 1-30 years after latency period May affect any tissue Gumma Formation Oral involvement may produce palatal perforation
42
Congenital Syphilis
Saddle nose deformity Saber Shins Hutchinson’s triad
43
Hutchinson’s Triad
Malformed incisors and molars Ocular interstitial keratitis Eight nerve deafness
44
Primary and secondary lesions show
Intense plasmacytic infiltrate
45
Tertiary histopath
Granulomatous inflammation
46
Spirochete can be identified using
Warthin-Starry Stain
47
Syphilis serology
Screening Tests Specific antibody tests Dark Filed Microscopy for non-oral lesions
48
Syphilis Tx
Penicillin remains the drug of choice
49
Tuberculosis caused by
Mycobacterium tuberculosis
50
TB transmission
Droplet
51
TB Clinical features
Low grade fever, night sweats, fatigue Weight loss Chronic bloody cough
52
TB oral lesions
Rather uncommon Solitary chronic painless ulcer or granular lesion Most common on gingiva and tongue May be due to hematogenous or Direct implantation of organims
53
TB diagnosis
Positive skin test with PPD Chest radiograph Identification of organims in biopsy material or sputum Culture Molecular testing
54
TB histopath
Usually necrotizing granulomatous inflammation Multinucleated giant cells Organisms stain suing the acid fast method
55
TB Tx
Combination of antibiotics Isoniazid (INH) rifampin pyrazimide and ehtybutol Then INH and rifampin for 4motnsh
56
Actinomycosis
Caused by several actinomyces species that normally inhabit the mouth Often associated with local trauma
57
Cervicofacial Actinomycosis may follow
May follow dental extraction or untreated dental disease
58
Cervicofacial Actinomycosis
Diffuse swelling erythema Diffuse swelling and erythema Draining sinus tracts
59
Sulfur granules
Colonies of origins in purulent exudate
60
Actinomycosis histo
Filamenotus bacteria that form colonies bacterial colonies surrounded by neutrophils Adjacent tissue may show granulomatous inflammation or granulation tissue
61
Actinomycosis
Removal of offending tooth High dose antibiotics usually IV PCN for 2 weeks then oral PCN for 2 weeks Periapical actino usually responds to less aggressive treatment Good prognosis with appropriate therapy

Oral Path 1 (4 decks)

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