LEC 8: Obj Recognition Flashcards
what’s the end of the retino-geniculate-striate pathway?
primary visual cortex - that when higher processing begins!
magnocellular cells
info from RODS but some cones
position and motion /large and fast things/colorblind
parvocellular cells
primarily CONES
detail and texture, small cells,
more layers in LGN than m-cells
small/slow/colorful things
what are the 2 retinoganglion cells
p and m cells
how are you able to see left and right sides of space w one eye?
each eye synapses on both LGNs depending on L and R visual field,
allowing one eye to present both hemispheres w info on L and R visual field
R visual field for each eye maps on L hemisphere
L visual field for each eye maps onto R hemisphere
where is primary visual cortex located?
occipital lobe
indirect pathway
stops btwn LGN and MT
direct pathway
LGN straight to MT
mostly associated w magnocellular pathway (fast to detect motion)
dorsal pathway
the where pathway
- where
- top
ventral pathway
the what pathway
- what
- bottom
obj cue + damage implication
monkeys associate shape of obj w treat (ex: raisins under cube evry time)
damage to temporal lobe causes impairment here
landmark cue + damage implication
monkey finds raisin near wherever certain landmark is
damage to parietal lobe causes impairment here
dual stream hypothesis
as visual info exits occiptal lobe, it follows 2 streams (dorsal and ventral)
receptive fields become ______ further along the ventral stream
larger
ventral pathway steps
retina - LGN - V1- (V2-V3-V4) - PIT - CIT - AIT
visual agnosia
modality specific
gnosis - knowledge
A - without
apperceptive agnosia
cannot copy, cannot recognize objs by shape
can sometimes recognize by other features (texture, color)
some patients have deficit only when noise in pic or non-standard viewpt
associative agnosia
can copy but unaware of what obj is
difficult to read cannot transfer visual inputs into words
can be category specific (ex: prosopagnosia)
normal sensory functions but striped of meaning
apperceptive damage
occipital lobe
associative damage
occipitotemporal cortex damage on both hemisphere
prosopagnosia
inability to recognize faces (face blindness)
type of associative agnosia
population encoding
uses hierarchal feature coding up to a pt
prbs w population encoding
if whole population of neurons required to identify single perception,
how do you link that percept to stored knowledge (there isn’t one cell to connect, there are many)
form-cue invariance
our ability to recognize objects regardless of their dimensionality (diff shapes but same thing)