Lecture 1 Flashcards

(64 cards)

1
Q

three layers of the skin are?

A

epidermis
dermis
subcutaneous tissue/hypodermis

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2
Q

larges portion of the skin, connective tissue between the epidermis and subcutaneous tissue

A

dermis

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3
Q

involved in producing the pigment melanin (brown) which colors the skin and hair?

A

melanocytes

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4
Q

roles as receptors that transmit stimuli to the axon

A

merkel cells

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5
Q

role in cutaneous immune system reactions and protects against microorganism against injury

A

langerhans cells

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6
Q

function of the dermis includes:

A

Provides strength and structure in the form of collagen and elastic fiber
Strength and structure in the form of collagen and elastic fiber

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7
Q

subcutaneous tissue functions

A

innermost layer of the skinprimarily composed of adipose and connective tissue
provides a cushion between the skin layers and the muscles and bones
protects the nerve and vascular structures that transect the layers
subcutaneous tissues and the amount of fat deposited are important factors in body temperature regulation

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8
Q

Hair growth from most rapid:

A
  1. beard
  2. hair on scalp
  3. axillae
  4. thighs
  5. eyebrows
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9
Q

term for excessive hair growth

A

hirsutism

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10
Q

term for hair loss

A

alopecia

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11
Q

glands associated with hair follicleslubricating the hair and rendering the skin soft and pliable

A

sebaceous glands

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12
Q

Thin, watery secretion calledsweatis produced in the basal coiled portion of the eccrine gland and is released into its narrow duct

A

eccrine glands

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13
Q

Regeneration of the nails:

how long for fingernails and toenails?

A

Fingernails – 6 months
Toenails – 18 months

Growth is faster in fingernails than toenails and tends to slow with aging

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14
Q

functions of the skin

A

Protection
Sensation
Receptors in the skin are to sense temperature, pain, light touch, and pressure
Fluid balance
[prevents dehydration] –> Epidermis—has the capacity to absorb water, thereby preventing an excessive loss of water and electrolytes
Severe burn, large quantities of fluids and electrolytes may be lost rapidly
Temperature regulation
Vitamin Production
Immune response function

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15
Q

Bluish discoloration that results from a lack of oxygen in the blood

A

cyanosis

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16
Q

Purple, black which fades to green, yellow, or brown hues over time;most often seen following trauma

A

ecchymosis

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17
Q

Redness of the skin caused by the dilation of capillaries

A

erythema

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18
Q

Yellowing of the skin

A

jaundice

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19
Q

Inflammatory response

A

-A sequential reaction to cell injury.

-Neutralizes and dilutes the inflammatory agent, removes necrotic materials, and establishes an environment suitable for healing and repair.
Inflammationis not similar with infection.

-Inflammation is always present with infection, but infection is NOT always present with inflammation

  • Mechanism of inflammation is basically the same regardless of the injuring agent
  • Intensity of the response depends on the extent and severity of injury and on the injured person’s reactive capacity
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20
Q

Inflammatory response can be divided into

A

Vascular response
Cellular response
Formation of exudate
Healing.

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21
Q

Results from outpouring of fluid. Seen in early stages of inflammation or when injury is mild ex. Skin blisters, pleural effusion

A

serous

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22
Q

Found during the midpoint in healing after surgery or tissue injury
Composed of RBCs and serous fluid, which is semi-clear pink and may have red streaks ex. surgical drain fluid

A

serosanguineous

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23
Q

Occurs with increasing vascular permeability and fibrinogen leakage into interstitial spaces
Excessive amounts of fibrin that coats tissue surfaces may cause them to adhere.

A

fibrinous

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24
Q

Results from rupture or necrosis of blood vessel walls

A

hemorrhagic

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25
Consists of WBCs, microorganisms (dead and alive), liquefied dead cells, and other debris
purulent (pus)
26
Found in tissues where cells produce mucus | Mucus production is accelerated by inflammatory response
catarrhal
27
cause of manifestation of redness
Hyperemia from vasodilation
28
cause of manifestation heat
Increased metabolism at inflammatory site | ex. vasodilation
29
cause of manifestation of pain
Change in pH. Nerve stimulation by chemicals (e.g., histamine, prostaglandins). Pressure from fluid exudate
30
cause of manifestation of swelling
Fluid shift to interstitial spaces. Fluid exudate accumulation
31
loss of function
swelling and pain ex. calf enlarged -> compartment syndrome
32
localized area of necrotic soft tissue that occurs when pressure applied to the skin usually a bony prominence
pressure ulcer
33
Braden Scale used for Predicting Pressure Injury Risk
Sensory Perception, Moisture, Activity, Mobility, Nutrition
34
Most common site for pressure ulcers
1. sacrum | 2. heels
35
Intact skin with non-blanchable redness of a localized area | Area may be painful, firm, soft, warmer, or cooler as compared to adjacent tissue.
pressure ulcer stage 1
36
Partial-thickness loss of dermis presenting as a shallow open ulcer with a red-pink wound bed, without slough May also present as an intact or open/ruptured serum-filled or serosanguineous-filled blister Presents as a shiny or dry shallow ulcer without slough or bruising
pressure ulcer stage II
37
Full-thickness tissue loss. Subcutaneous fat may be visible but bone, tendon, or muscle are not exposed May include undermining and tunneling
stage III
38
Full-thickness tissue loss with exposed bone, tendon, or muscle. Slough or eschar may be present. Often includes undermining and tunneling
stage IV
39
Full-thickness tissue loss in which actual depth of ulcer is completely obscured by slough (yellow, tan, gray, green, or brown) and/or eschar (tan, brown, or black) in wound bed
unstageable
40
Purple or maroon localized area of discolored intact skin or blood-filled blister due to damage of underlying soft tissue from pressure and/or shear.
suspected deep tissue injury
41
nursing interventions for pressure ulcers:
``` Relieve Pressure Positioning the patient Use pressure-relieving devices Improve mobility Improve sensory perception ``` ``` Improve tissue perception Improve nutritional status Reduce friction and shear Minimize moisture Promote pressure injury healing ```
42
nutritional deficiences for delaying wound healing
- vitamin c - protein - zinc - inadequate blood suppy - corticosteroid drugs
43
factors that delay wound healing include:
- infection - smoking - mechanical friction on wound - advanced age - obesity - diabetes mellitus - poor general health - anemia
44
dressing generally composed of polyurethane. transparency allows visualization of the wound. used for dry, uninfected wounds or wounds with minimal drainage
transparent films
45
Film-coated gel or polyurethane. Able to hold large amounts of exudate; used for Wounds with moderate to heavy drainage. Often used on new wounds
foam dressing
46
Gelatin, pectin, or carboxymethylcellulose bonded to a film or sheet. Produce a flat occlusive dressing that forms a gel on wound surface; used for wounds with light to moderate drainage
hydrocolloids dressing
47
gels, gel-covered gauze, or sheets. Donate moisture to a dry wound and maintain a moist environment. Can rehydrate wound tissue Dry wounds. Wounds with minimal drainage. Necrotic wounds
hydrogels **used to soften hard scabs that will help it fall off
48
Derived from seaweed or kelp. Form a non-sticky gel on contact with draining wound. Easy to use over irregular-shaped wounds
alginates
49
Wound covers that deliver agents such as silver and iodine, and polyhexamethylene biguanide (PHMB), which have antibacterial properties.
antimicrobials dressing
50
complications of wound healing
``` Adhesion Contractions Dehiscence Evisceration Excess Granulation Tissue (Proud Flesh) Fistula Formation Infection Hemorrhage Hypertrophic Scars Keloid Formation ```
51
A chronic suppurative folliculitis of the perianal, axillary, and genital areas or under the breasts
Hidradenitis Suppurativa
52
Management of Hidradenitis Suppurativa
Warm compresses Loose-fitting clothes over the nodules or lesions NSAIDs to relieve the pain Oral antibiotic Incision and drainage of large suppurating areas
53
a common disorder affecting hair follicles and sebaceous glands. 
acne vulgaris
54
mangement of acne vulgaris
``` Nutrition Avoid sugary food products Hygiene Washing twice a day with soap and water Phototherapy Surgical Management ``` ``` Medication Benzoyl peroxide Topical retinoids Topical antibiotics Oral isotretinoin + oral ATB ```
55
Contagious bacterial infection of superficial layers of skin of honey colored crusts; Lesions common on face, hands, neck, extremities, perineum
impetigo
56
1-2 mm erythematous papules or pustules, progress to vesicles or bullae which rupture
nonbullous
57
Contagious bacterial infection of superficial layers of skin of honey colored crusts; Lesions common on face, hands, neck, extremities, perineum
impetigo
58
management of impetigo
Topical antibiotics if superficial, nonbullous, localized Oral antibiotics for multiple lesions, spread of infection to family members Bullous impetigo in infant – parenteral beta-lactamase-resistant antistaphylococcal penicillin Obtain culture if no response in 7 days Educate about hygiene Exclude from day care until treated for 24 hours
59
clinical findings folliculitis and furuncle
Discrete, erythematous 1-2 mm papules or pustules on inflamed base near follicle Face, scalp, extremities, buttocks, back Nodules with furuncles Pruritus papules, pustules, deep red/purple nodules in areas under swimsuit
60
HSV 1
Gingivostomatitis Herpes labialis Herpetic whitlow
61
HSV 2
Grouped vesicopustules/ulceration Vaginal mucosa, labia, perineum, cervix in females; penile shaft and perineum in males Regional lymphadenopathy
62
HSV type 1
oral mucosa, pharynx, lips
63
HSV type 2
neonatal infection, vulvovaginitis or genital infection
64
Herpetic whitlow
fingers, thumb are swollen and painful