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Y2 - CNS > Lecture 16 — Stroke > Flashcards

Lecture 16 — Stroke Flashcards

1
Q

What is the Definiton of a stroke?

A

A serious life threatening condition that occurs when the blood supply to part of the brain is cut off with signs and symptoms persisting for longer than 24hrs

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2
Q

What is a transient ischaemic attack (TIA)?

A

The same clinical features of a stroke but resolves within 24hrs (essentially a stroke resolving in 24hrs)

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3
Q

What are the main types of stroke?

A

Ischaemic stroke (thromboembolism)
Haemorrhagic (rupture of blood vessels)

Arterial dissection
Venous sinus. Thrombosis
Hypoxic brain injury

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4
Q

What occurs in an ischaemic stroke?

A

Occlusion of a blood vessel usually due to the rupture of an atherosclerotic plaque

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5
Q

What are the main types of Haemorrhagic stroke?

A

Subarachnoid haemorrhage
Intracerebral haemorrhage (blood vessel in the brain)

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6
Q

How does venous sinus thrombosis lead to a stroke occurring?

A

Impairs the venous drainage (back pressure) which impairs arterial blood flow to the brain leading to ischaemia

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7
Q

What is the emergency management of stroke?

A

Do CT head to determine if its a bleed

Give thrombolysis if less than 4hrs since stroke and stroke cause is not a bleed

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8
Q

How does blood appear on a CT head?

A

White

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9
Q

How does blood appear on an MRI?

A

High signal area

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10
Q

Slide 6:

What type of haemorrhage is seen on the right CT?

Describe it:

A

Subarachnoid haemorrhage

Blood has filled the basal cisterns producing. A 5 pointed star pattern

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11
Q

How does an acute ischaemic stroke appear on CT?

A

Likely no changes however you may see a hyper dense area indicating a thrombus

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12
Q

Slide 7:

What is visible on the left CT?

A

Hyper dense Middle cerebral artery indicating a thrombus in this area

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13
Q

What type of imaging is very good at identifying ischaemic stroke?

How does it appear?

A

MIR

High signal area (whiter area)

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14
Q

How may a head CT change as a stroke evolves?

A

Can look normal but then weeks area have darker areas indicating neuronal damage and oedema

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15
Q

What makes up the anterior brain circulation?

A

Anterior and middle cerebral arteries

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16
Q

What arteries feed the anterior circulation of the brain?

A

Internal carotid arteries

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17
Q

What arteries supply the posterior circulation of the brain?

A

Vertebral arteries

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18
Q

Go to slide 9 and complete the blood supply to the brain:

A
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19
Q

What part of the brain does the Anterior Cerebral Artery supply? (ACA)

A

Medial aspect of frontal and parietal lobes (motor and sensory homunculus)
Corpus callosum

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20
Q

What part of the brain do the Middle Cerebral Arteries supply?

A

Lateral aspect of frontal and parietal lobes (motor and sensory homunculus)
Lateral aspect of Superior temporal lobe

Deep structures like internal capsule, lentiform nucleus and caudate

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21
Q

What branch from the middle cerebral artery supply the internal capsule?

A

Lenticulostriate arteries

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22
Q

What part of the brain do the posterior cerebral arteries supply?

A

Occipital lobe
Inferior temporal lobe
Thalamus

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23
Q

What is the most commonly affected artery in stroke?

A

Middle cerebral artery

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24
Q

How does an Anterior Cerebral artery infarction present in a patient?

Think about the areas which the anterior cerebral artery supplies

A

Contralateral lower limb weakness
Contralateral lower limb sensory change
Urinary incontinence
Apraxia
Dysarthria/aphasia (rare)
Split brain syndrome (rare)

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25
Why does an Anterior Cerebral artery infarct cause Contralateral lower limb weakness and sensory change?
ACA supplies medial aspect of frontal and parietal lobes which include the medial aspect of primary sensory and motor cortex which supplies the lower limb
26
Why does an anterior cerebral artery infarct cause urinary incontinence?
The paracentral lobules which are responsible for supplying the external urinary sphincter are represented medially in the brain
27
Why can an Anterior Cerebral artery infarct cause Split brain syndrome or alien hand syndrome?
Branch of the ACA supplies the corpus callosum which is a white matter tract allowing communication between the 2 hemispheres
28
What is Apraxia?
Difficulty doing every day tasks (motor planing)
29
Why can an Anterior Cerebral artery infarction cause apraxia?
Damage to the left frontal lobe due to the ACA supplying this part of the medial frontal lobe
30
Go to slide 11 and label the arteries:
31
What parts of the brain do the lenticulostriate arteries supply?
Entire internal capsule + Basal ganglia (includes lentiform nucleus, caudate)
32
With middle cerebral artery infarcts, what is worse, the infarction being more proximal or distal?
Proximal Since the more proximal, the worse the damage to the brain so the more oedema that occurs and the higher the ICP becomes
33
Slide 12: Label the Middle Cerebral Artery
34
How does a main stem Middle Cerebral Artery occlusion/infarction present? Think about the areas it supplies
Contralateral hemiparesis of face arm and leg Contralateral sensory loss Contralateral homonymous hemianopia Global aphasia if left sided Left sided neglect if right sided
35
What do the cortical branches supply from the middle cerebral artery?
Lateral aspect of parietal lobe and superior temporal lobe
36
What are the cortical branches of the middle cerebral artery?
Superior division Inferior division
37
What does the superior cortical divison of the middle cerebral artery supply?
Lateral aspect of frontal lobe
38
What does the inferior cortical divison of the middle cerebral artery supply?
Lateral parietal lobe Superior aspect of temporal lobe
39
Why with a main stem Middle Cerebral Artery occlusion/infarct do you get contralateral hemiparesis of face, arm and leg?
MCA gives of Lenticulostriate arteries which supply the internal capsule + basal ganglia The Lenticulostriate arteries contain UMN so UMN are unable to reach the spinal cord due to internal capsule damage
40
Why with a main stem Middle Cerebral Artery occlusion/infarct do you get contralateral sensory loss?
The Lenticulostriate arteries supply the internal capsule and basal ganglia Internal capsule as well as containing UMN also contain ascending sensory fibres
41
Why with a main stem Middle Cerebral Artery occlusion/infarct do you get contralateral homonymous hemianopia?
The white matter of the parietal and temporal lobe contains optic radiations (parietal = superior optic radiations) and (temporal = inferior optic radiations)
42
Why with a main stem Middle Cerebral Artery occlusion/infarct do you get global aphasia if it’s left sided?
Left cerebral hemisphere is responsible for speech Brocas area is in the inferior lateral frontal lobe Wernickes area is in the superior temporal lobe These areas are supplied by the Middle Cerebral Artery
43
What is neglect?
The failure to acknowledge the existence of usually the left side of space, left side of objects or left side of own body
44
What part of the brain is normally damaged with neglect?
Right parietal lobe
45
What signs are seen in neglect?
Tactile extinction (will only acknowledge touch on right side when touched on both left and right side simultaneously) Visual extinction (wave 2 hands will only acknowledge the hand on the right side) Anosognosia
46
What is anosognosia seen in neglect?
Patient doesn’t acknowledge theres a problem They’ll say they just dont want to move their arm even though it’s paralysed
47
How do lacunar infarcts (branch of MCA) present?
Lack cortical features like aphasia or neglect Equally effects sensory and motor function of Face arms and legs
48
What leads to a pure motor stroke affecting the face arms and legs equally in a lacunar infarct?
The infarct affects the internal capsule more anteriorly
49
What leads to a pure sensory stroke affecting the face arms and legs equally in a lacunar infarct?
The infarct affects the internal capsule more posteriorly
50
What leads to a sensorimotor stroke affecting the face arms and legs equally in a lacunar infarct?
Mixture of both UMN and ascending sensory nerves in the internal capsule more in the middle
51
With a main stem middle cerebral artery occlusion/infarct, does the Contralateral homonymous hemianopia have macular sparing?
No macular sparing
52
How does a patient with a Middle Cerebral Artery occlusion at the inferior division present?
Contralateral sensory change in face and arm Wernickes/receptive aphasia if left sided Contralateral homonymous hemianopia
53
Why does a patient get Contralateral sensory change in the face and arm with a Middle Cerebral Artery occlusion at the inferior division?
Since lateral parietal lobe supplied by this and this is the part of the sensory homunculus for the face and arms
54
Why does a patient get Wernickes/receptive aphasia with a Middle Cerebral Artery occlusion at the inferior division if it’s on the left side?
Wernickes area is in the superior temporal lobe which is supplied by the inferior division of the MCA
55
Why does a patient get Contralateral homonymous hemianopia with a Middle Cerebral Artery occlusion at the inferior division?
Both superior and inferior optic radiations in the parietal and superior temporal lobe respectively are destroyed
56
How does a patient with an occlusion//infarction in the superior division of the middle cerebral artery present?
Contralateral face and arm weakness Brocas/expressivee aphasia if left sided
57
Why does a patient get Contralateral motor change/weakness in the face and arm with a Middle Cerebral Artery occlusion at the superior division?
Lateral frontal lobe supplied by this and this is where the face and arm are represented on the motor homunculus
58
Why does a patient get Brocas/expressive aphasia with a Middle Cerebral Artery occlusion at the superior division if it’s left sided?
Broca’s area located in the inferior lateral frontal lobe
59
How does a patient present if you get occlusion of all the cortical branches of the middle cerebral artery?
Just the same affects of the superior + inferior division occlusion Contralateral sensory and motor weakness in face and arm Wernickes + Brocas aphasia = global aphasia if its left sided Contralateral homonymous hemianopia without macular sparing
60
How does a patient with a Posterior Cerebral Artery occlusion/infarction present?
Contralateral homonymous hemianopia with macular sparing Complete Contralateral sensory loss or change
61
Why do patients with a posterior cerebral artery occlusion/infacrtion have contralateral homonymous hemianopia with macular sparing?
Supplies occipital lobe Macule sparing since has dual blood supply from the Middle Cerebral Artery (MCA))
62
Why do patients with a posterior cerebral artery occlusion/infacrtion have contralateral sensory loss?
Posterior Cerebral Artery (PCA) supplies the thalamus via thalamoperforator/thalamogeniculate branches So if PCA blocked thalamus not receiving blood supply and its essential in both sensory tracts
63
How do the Contralateral homonymous hemianopias occurring with MCA and PCA occlusions differ?
PCA occlusion = macular sparring since collateral supply from MCA MCA occlusion = NON macular sparring
64
How do patients with brainstem strokes present?
Crossed deficit: -Ipsilateral cranial nerve deficits (brainstem signs) -contralateral sensory and motor signs (above decusstion of pyramids)
65
What are the symptoms of a cerebellar stroke?
Nausea Vomiting Vertigo/dizziness Headache
66
What are the sings of a cerebellar stroke?
Ipsilateral cerebellar signs (DANISH) Ipsilateral cranial nerve signs Possible Contralateral sensory deficient Possible Ipsilateral HORNERS syndrome
67
What are the Ipsilateral cerebellar signs?
Dysdiadochokinesia Ataxia Nystagmus Intention tremor Slurred speech Hypotonia
68
How does a patient present if they have an occlusion in the distal/superior portion of the basilar artery (tip of the basilar)?
Visual and oculomotor deficits Behavioural abnormalities Somnolence, hallucinations and dreamlike behaviour
69
Why can some visual deficits be rescued in a tip of basilar artery occlusion?
Occipital lobes also supplied by the posterior communicating artery
70
Why is motor dysfunction usually absents in tip of basilar artery occlusion?
Cerebral peduncles reduced by posterior communicating artery
71
Why can you get dreamlike behaviour in tip of basilar artery occlusion?
Basilar normally gives of branches that go to the reticular formation that controls sleep in teh mid brain
72
How does a patient present with a full width occlusion of the proximal basilar artery that’s over the pontine artery branches ?
LOCKED IN SYNDROME (can still move eyes)
73
Why does locked in syndrome occur with a basilar artery occlusion over the pontine branches? Why can the eyes still be moved?
Pontine arteries supply the corticospinal tracts of the body, occlusion = full body paralysis Eyes can still be moved because the midbrain is still being supplied by the Posterior Cerebral arteris Still conscious since midbrain’s reticular formation still in tact
74
What system do we use to classify strokes?
Bamford/oxford stroke classification

Y2 - CNS (21 decks)

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