Lecture 17 Vision II Flashcards

1
Q

What light properties are used to create colour?

How is colour perception formed in the brain?

A

The intensity of the wavelength, the type of wavelength

Retina processes the two properties. Brain recombines this information into colour perception

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2
Q

How are we able to see colours of an object

A

Objects reflect & absorb some colours (wavelengths). The colours we see are reflected wavelengths.

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3
Q

The similarities & differences between rod & cone photoreceptors

A

Similarities
outer & inner segment, synaptic terminal, lots of mitochondria in inner segment (lots of ATP), energetically active tissue, release neurotransmitter

Differences [rod, cone]
Type: 1, 3 Location: periphery, fovea
Vision type: black & white , colour Number: 20X , 1x
Sensitivity: 1000x (1 light photo - activate), 1x (10s - 100s photons - activate )
Recovery time: slow , fast (rapid adapt to illumination change)

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4
Q

Correct this paragraph

Most vertebrae eyes are cone dominant. In humans, the fovea has a mixture of cone and rod cells so it is not involved in acute vision. So, the fovea is no different to the retina

A

Correction

Most vertebrae eyes are rod dominant.
In human, the retina - mainly rod cells
Fovea - only cone cells, mediates acute vision

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5
Q

Scotopic vision

A
  1. Low light/dim conditions
  2. Only rod cells function - very sensitive to light
  3. Cone - no light to respond to
  4. No colour seen
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6
Q

Phototopic vision

A
  1. Daylight conditions
  2. Cones only function - less sensitive to light, adaptable to background light, vision in brighter light
  3. Rod - bleach out quickly (bright light)
  4. 3 cone types - give wide range of bright light colour perception
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7
Q

Mesopic vision (between dim & day light)

A
  1. Moderately low light levels (right after sunset)
  2. Rods & cones function
  3. Cones - enough light to proved colour vision
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8
Q

Colour information processing is carried out by…

What are their features compared to M cells:

A

P cells (parvocellular cells)

  • smaller cell bodies and dendritic fields (midget cells)
  • Sustained fashion response
  • slower conduction velocities
  • Sensitive - light wavelength differences -> convey colour information
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9
Q

Visual pathway for colour

Location in cortex

Outer layers of lateral genicular nucleus

Third distinct LGN anatomical pathway

A

Optic nerve (monocular information) -> optic chiasm (nasal fibres crossover) -> LGN thalamus -> Optic radiation -> V1

Ventral pathway to temporal lobe (optic radiation -> V1 -> V2 -> V4 -> Temporal lobe [object processing: colour, texture, shape, size])

4 layers (P3 -P6) - transmitted necessary information regarding colour perception (especially red & green) to V1

Koniocellular pathway: neurons that reside between LCN layers - ? Involved in relay info from S M L cones

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10
Q

What are the three theories of colour vision

What does trichromatic and colour opponency theory suggest?

A

Trichromatic theory
Opponent colour theory
Stage theory

Trichromatic theory: indicates how retina allow visual system to detect colour with three cone types
Colour opponency theory: considers mechanisms that take and process information from cones

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11
Q

What is the trichromatic theory?

What are the 3 types of cone photoreceptors and their features?

A

All colours can be perceived by a combination of three colours

L-cone - sensitive to long wavelength light -> RED
M-cone - sensitive to medium wavelength light -> GREEN
S-cone - sensitive to short wavelength light -> BLUE a

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12
Q

Trichromatic colour theory

Explain the spectrum of colour absorption and how this allows colour to be perceived by the brain

What is the downside of this theory

A

Each cone overlaps another -> same wavelength trigger different cone response combinations
Difference in signal receive from three cone types -> brain perceive continuous range of colours

Some colour perception phenomenon cannot be explained by this theory

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13
Q

What is the colour opponent theory

A

Some colours can be perceived in ordinary perception simultaneously (purple [red &blue], orange [red&yellow]) while others do not exist (red & green, blue & yellow)

do not relate to paint, this is single visual pigment

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14
Q
  1. What are the three opponent channels
  2. What is their function
  3. Information processing in Red-green opponent channel, Blue-yellow opponent channel & black-white opponent channel
A
  1. Red-green, blue-yellow, black and white (brightness, luminosity/achromatic channel)
  2. A member of the colour pair suppress the other colour. P cell receptive field: center activated by one colour, surround activated by another.
  3. RG: process info -> firing difference - L cone (+ S cone input) & M cone
    BY: process info -> firing difference - S cone & (combine L & M signal). [blue light present: S cone activated, B+Y- cell excitatory input, blue colour perceived. ]
    BW: conduct light intensity infor
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15
Q

Explain colour afterimage

A

Optical illusion - image continuing to appear even after exposure to original image stopped

Prolonged viewing of coloured patch - afterimage with complementary colour.

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16
Q

Colour opponency.

A

Prolonged viewing of a colour stimulus -> neural adaptation of retina : that respective ganglion cell signalling the colour presence reduces its firing rate.

When moved to white light, cells fire very little but the opponent colour ganglion cell fires at a higher rate compared to the first colour so that colour willl be seen

17
Q

What is the stage theory

A

A modern model for normal colour vision that combines trichromatic and opponent colour theory into two stages

  1. Receptor stage : three photoreceptors (trichromatic colour theory) [explain colour vision at photoreceptor level ]
  2. Neural processing: colour opponency [explain colour vision phenomena from way photoreceptors connect neurally]
18
Q

Colour vision deficiencies

Colour blindness (dichromacy)

A
  • inherited colourblindness -> abnormal photoreceptors (colour opponent theory)
  • men more susceptible than women -> most common inherited gene on X chromosome
  • red-green colour blindness most common followed by blue-yellow colour blindness
  • total colour blindness rare (complete colour vision absence)
  • ~ caused by physical/chemical damage to eye, optic nerve, process colour information parts of brain
19
Q

Protanopia

A

-no red cone cells in retina
- most common type of dichromacy
- perception impaired - very long wavelengths (red)

20
Q

Deuteranopia

A
  • no green cone cells in retina
  • perception impaired - medium wavelength (green)
21
Q

Tritanopia

A
  • no blue cone cells in retina
  • rarer colour blindness form
  • perception inability - short wavelengths (blue)
  • confuse green and blue, yellow looks pink