Lecture 38 4/29/25 Flashcards

(35 cards)

1
Q

What is cellulitis?

A

subcutaneous infection/inflammation

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2
Q

What is the presentation of cellulitis?

A

-acute grade 4/5 lameness and diffuse swelling/pitting edema
-usually one limb but can be multiple
-usually precipitated by a small skin scrape/wound that allows bacteria to colonize
-typically normal commensals; Staph, Strep, E. coli

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3
Q

What are the treatment steps for cellulitis?

A

-broad spectrum antibiotics
-pain management/NSAIDs
-compressive bandage

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4
Q

What is the prognosis for cellulitis?

A

-excellent for life
-good to excellent for athletics with early management
-prognosis decreases with lymphedema

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5
Q

What are the characteristics of lymphangitis/lymphedema?

A

-almost always concurrent with cellulitis or develops during cellulitis progression
-signs include progressive swelling of proximal limb, sheath, and ventral edema

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6
Q

What are the characteristics of chronic progressive lymphedema?

A

-occurs with repeat episodes of cellulitis/lymphangitis
-scar tissue builds up
-every episode/flair is typically worse than previous one

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7
Q

What are the treatment options for chronic progressive lymphedema?

A

-hyperbaric oxygen therapy
-mitigate flairs
-culture/treat intermittent infection
-therapeutic exercise

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8
Q

What is the presentation of medial meniscus injury?

A

-grade 3+/5 lameness
-effusion of medial femorotibial joint
-poor healing capacity due to minimal blood flow
-injured most commonly at caudal pole; least accessible area via arthroscopy

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9
Q

What are the treatment options for medial meniscus injuries?

A

*orthobiologics
-intralesional stem cells
-synovial stem cells
-protein-rich plasma/APS
*rest
*controlled return to work

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10
Q

What is the prognosis for medial meniscus injury?

A

-good for life
-guarded for athletics

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11
Q

What are the characteristics of cranial medial meniscotibial ligament injury?

A

-usually diagnosed via arthroscopy
-not typically a “stand alone” injury
-often in conjunction with medial meniscal tear or synovitis/arthritis

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12
Q

What are the treatment options for cranial medial meniscotibial ligament injury?

A

orthobiologics, including intralesional and intrasynovial injections

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13
Q

What is the prognosis for cranial medial meniscotibial ligament injury?

A

-good for life
-guarded for athletics

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14
Q

What are the characteristics of patellar ligament, lateral meniscus, and lateral collateral ligament injuries?

A

-usually secondary to trauma
-treated with intralesional orthobiologics
-good prognosis for life
-guarded prognosis for athletics

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15
Q

What are the characteristics of cranial cruciate lig. injury?

A

-present with grade 4/5 lameness
-effusion seen in medial femorotibial joint and femoropatellar joint
-direct drawer sign positive; very painful
-definitive diagnosis via arthroscopy
-guarded prognosis for life
-grave prognosis for athletics

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16
Q

What are the characteristics of the reciprocal apparatus?

A

-large medial patellar ligament
-“hooks” over proximal aspect of medial femoral trochlea
-prevents stifle flexion and allows for muscle relaxation
-to “unhook,” quadriceps activate to pull patella dorsally, allowing stifle to flex

17
Q

What is the typical signalment of upward fixation of the patella?

A

-young, underdeveloped but fast-growing breeds
-older muscle-wasted, emaciated animals +/- PPID
-ponies and minis over-represented

18
Q

What is the presentation of upward fixation of the patella?

A

-horse cannot flex stifle and advance limb
-typically intermittent but can be constant
-tests to set it off include backing, sudden stops, standing for a period before walking off, and tail pulls to the side with release

19
Q

What are the treatment options for intermittent upward fixation of the patella?

A

*exercise to develop the quadriceps
*medial patellar ligament fenestration
-skin block
-insert needle into ligament and split tendon by raising and lowering hub at several sites
-promote scar tissue and shortening/strengthening of ligament

20
Q

What are the treatment options for constant upward fixation of the patella?

A

*medial patellar ligament desmotomy
-salvage procedure only
-induces patellar instability via fragmentation and arthritic changes

21
Q

What is the prognosis for upward fixation of the patella?

A

*intermittent:
-excellent for life
-good to excellent for athletics
*constant:
-good for life
-guarded to good for athletics dependent on desmotomy outcome

22
Q

What are the characteristics of cervical facet osteoarthritis?

A

-osteoarthritis of cervical facet joints
-history may include reluctance to bend or turn, refusal to jump fences, and general poor performance

23
Q

What is the presentation of cervical facet osteoarthritis?

A

-reduced range of motion to carrot stretch
-may have palpable pain over joint
-muscle atrophy or hypertonicity
-can have mild forelimb lameness
-can have concurrent neurologic dz
-most common around c4 to c7

24
Q

What are the treatment steps for cervical facet osteoarthritis?

A

-typical OA approach
-corticosteroid injections
-shockwave
-mobility exercises
-chiropractic

25
What is the prognosis for cervical facet osteoarthritis?
-depends on degree of spinal cord compression -decreases with concurrent neurologic deficits -good to excellent for life -guarded to good with aggressive management for athletics
26
What are the characteristics of topline dysfunction?
-traditionally "back pain," but does not actually describe clinical dz or help with treatment -history may include noticed pain or just poor performance -present with concurrent hind limb lameness and some degree of weakness, stiffness, and/or pain on MSK exam
27
What is assessed during a hands-on exam when looking for topline dysfunction?
-muscle hypertonicity -muscle/soft tissue pain -lateral perturbations
28
What should be done in areas of concern during a hands-on exam when looking for topline dysfunction?
-detailed dorsal spinous process palpation and mobilization -try to repeat the findings of concern
29
What are signs of generalized pain from topline dysfunction?
-muscle spasm -splinting -avoidance
30
What are the treatment steps for topline dysfunction?
-treat underlying cause(s) -therapeutic exercise -radiofrequency -electroacupuncture -chiropractic -possibly NSAIDs -possibly thoracolumbar facet injections
31
What is the prognosis for topline dysfunction?
-depends on severity and discipline -excellent for life -guarded to good for athletics depending on commitment of owner
32
What are the characteristics of overriding dorsal spinous processes?
-usually present with same symptoms as topline dysfunction -should have a focal area of pain on palpation of DSP or interspinous ligaments
33
What are the diagnostics for overriding DSPs?
-improvement with local analgesia -possible bone scan/nuclear scintigraphy -radiographs; important to note rads alone do not correlate with clinical dz
34
What is the treatment for overriding DSPs?
-conservative treatment is the same as topline dysfunction treatment -surgical treatment can include interspinous ligament desmotomy or wedge resection of DSP
35
What is the prognosis for overriding DSPs?
-excellent for life -variable/unknown for athletics