Lecture 6 4/15/25 Flashcards

(36 cards)

1
Q

Which oral diseases can both cats and dogs get?

A

-periodontal dz
-endodontic dz
-neoplasia
-autoimmune conditions
-infectious oral dz

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2
Q

What are the specific feline dental diseases?

A

-tooth resorption
-feline gingivitis/stomatitis syndrome

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3
Q

What is tooth resorption?

A

resorption of the hard tissue of the tooth due to odontoclastic activity

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4
Q

What is the signalment for tooth resorption?

A

-affects 25 to 72% of cats
-typically cats over 2 years of age
-prevalence increases with age
-no reported breed or sex predisposition

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5
Q

What causes tooth resorption?

A

-excessive activation of odontoclasts
-cause of odontoclast activation is unknown

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6
Q

Where are tooth resorption lesions located?

A

-at or below the cemento-enamel junction
-typically on the buccal surface
-most commonly on the premolar and molar teeth
-often multiple lesions

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7
Q

What are the two classifications for tooth resorption?

A

-stage
-type

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8
Q

What are the characteristics of tooth resorption staging?

A

-condition is progressive
-stage of tooth resorption is a marker of disease progression
-need to take rads to stage

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9
Q

What is stage 1 tooth resorption?

A

lesion confined to enamel or cementum

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10
Q

What is stage 2 tooth resorption?

A

lesion extends into dentin

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11
Q

What is stage 3 tooth resorption?

A

lesion extends into pulp

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12
Q

What is stage 4 tooth resorption?

A

majority of tooth structure lost:
A: crown and root equally affected
B: crown > root
C: root > crown

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13
Q

What is stage 5 tooth resorption?

A

only remnants of tooth are visible/”ghost tooth”

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14
Q

What are the potential treatment options for tooth resorption?

A

-crown amputation
-surgical extraction

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15
Q

What is type 1 tooth resorption?

A

-focal or multifocal radiolucency
-normal root opacity
-normal PDL

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16
Q

What is the only treatment option for type 1 tooth resorption?

A

surgical extraction of all tooth material

17
Q

What is type 2 tooth resorption?

A

-radiolucency of part of the tooth (crown and/or root)
-loss of PDL space

18
Q

What are the characteristics of crown amputation treatment for type 2 tooth resorption?

A

-remove the crown (part causing pain)
-allow resorption of the roots to continue

19
Q

What are the contraindications to crown amputations in type 2 tooth resorption?

A

-stomatitis
-endodontic dz

20
Q

What is type 3 tooth resorption?

A

-type 1 and type 2 present in the same tooth
-one root has normal PDL
-the other root has loss of PDL

21
Q

What is the treatment for type 3 tooth resorption?

A

-surgical extraction of the type 1 root
-crown amputation on the type 2 root

22
Q

What is the recommended follow up for cats with tooth resorption?

A

COHAT, oral charting, and rads every 9 to 12 months; cats with tooth resorption in one tooth are predisposed to developing more lesions

23
Q

What are the characteristics of stomatitis?

A

-generalized inflammation of the mucous membranes of the mouth
-characterized by varying degrees of excessive immune response leading to inflammation in the oral cavity of cats

24
Q

What are the clinical signs of FCGS (feline chronic gingivostomatitis)?

A

-oral discomfort
-preference for soft food
-halitosis
-unkempt coat
-ptyalism
-head shy
-decreased appetite/anorexia

25
What are the possible lesions seen in FCGS?
*range from mild inflammation to severely inflamed, proliferative, and ulcerative lesions of the: -caudal mucosa -alveolar mucosa -soft palate -dorsal tongue -sublingual mucosa -labial mucosa -hard palate
26
What is the signalment for FCGS?
-affects 0.7 to 12% of cats -median age of 6 to 7 years -no sex predilection -more common in siamese, abyssinian, persian, himalayan, and burmese breeds
27
What causes FCGS?
-exaggerated immunologic processes to an antigenic stimulus is suspected -current theories on potential stimuli include infectious dz, dental plaque, and tooth material/PDL
28
Which infectious diseases are thought to be stimuli for FCGS?
-calicivirus -herpesvirus-1 -FELV/FIV -Bartonella henselae
29
What are the characteristics of dental plaque vs tooth material/PDL as a possible stimuli for FCGS?
*dental plaque: -clinical signs improve in some cats with professional cleaning and removal of plaque-retentive surfaces *tooth material/PDL: -clinical signs persist with retained root material
30
What are the differentials when cats present with signs similar to FCGS?
-uremia -chemical exposure -electrical cord injury -eosinophilic granuloma complex -neoplasia -vesiculobullous dz
31
Which diagnostics are done in cats with suspected FCGS?
-CBC: shows chronic inflammation -chem: shows hyperglobulinemia -FeLV/FIV testing -full mouth rads: looking for periodontal dz and tooth resorption -biopsy: histopath. shows lymphocytes, plasmacytes, and PMNs
32
What is the goal of FCGS treatment?
decrease antigenic stimulation and modulate abnormal immune response
33
What are the components of medical treatment for FCGS?
*decrease antigenic stimulation -professional cleaning every 4 to 6 months -home care *immunomodulation/anti-inflammatory -pred. -cyclosporine -doxycycline -NSAIDs *analgesia -buprenorphine -gabapentin -fentanyl patch -NSAIDs
34
What are the surgical options for FCGS treatment?
-full mouth extractions -extractions of all premolars and molars only (caudal to canines) *must remove all tooth material*
35
What are potential treatments for refractory FCGS?
-immune-modulating drugs -laser ablation -essential fatty acids -interferon -stem cells
36
Why is it important to avoid oversized mouth gags in cats?
excessive opening and tension on the maxillary artery can lead to blindness