Lecture 9: Vision Flashcards

1
Q

Reception

A
  • you see things
  • going to receive information —> activate those sensor receptors
  • sensor receptors for vision —> in your eyeballs
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2
Q

Transduction

A
  • making that action potential + synaptic communication via those chemicals —> communication between the neurons
  • changing that stimulus energy, taking into consideration for vision
  • stimulus energy —> photons, light
  • change it into action potentials and chemicals to help ourselves out with regards to neurons talking to each other
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3
Q

Coding

A
  • primarily takes place for your sense of vision in your occipital lobe
  • all about photons of light —> the stimulus energy in the first place
  • However you are perceiving your visual world —> all about light + many wavelengths of light that are going to be bouncing off particular objects
  • As you’re looking at me —> light from outside, light from above that’s hitting me —> projecting into your eyeball
  • mostly trying to get the photo receptors —> back wall of your eyeball on retina —> to become activated
  • if we expand or magnify the wall of the retina —> have to get light to hit it at a 90 degree angle/ a perpendicular angle in order to activate those photoreceptors successfully
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4
Q

Cornea

A
  • most superficial layer out the front here
    • protection
    • special almost skin like cells —> able to regenerate and heal in that way —> just like your skin helping you out with protecting your eyeball
    • transparent —> light can come on through
    • We have light usually coming in at this particular coloured pot of your eye —> Iris
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5
Q

Pupil

A
  • Middle of your iris
  • no anatomical structure —> just a hole
  • However big your iris —> what you perceive as being your pupil
  • Size of your pupil
    • Go into a dark room —> larger —> pupil dilation
  • function —> controlling the amount of light coming into your eye
    • brightly lit room —> pupil get smaller —> Pupil constriction —> letting less lighting in —> helping you to see
  • get past the pupil —> controlling the amount of light
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6
Q

Glasses

A
  • Near sighted —> eyeball is too long, an eyeball growth —> even though your lens does what it can, it incorrectly refracts the light into the vitreous humour space (gel like substance that makes up the majority of your eyeball)
    • in advance or well before the actual photoreceptors
    • near sightedness —> bring things closer to us so that our lens doesn’t have to work quite so hard
    • When people squint —> can see better
      • squishing their eyeballs —> your eyeball is too long —> eyeball has a lot of muscles around it —> able to be in order to improve it
    • Near sightedness —> you can see with the addition of glasses —> ends up refracting it in slightly different ways —> help you out with regards to getting it onto the back wall —> onto your retina in turn
    • interocular pressure
  • Far sighted —> eyeball a little bit short
    • Instead of getting projected onto your retina —> gets well past the point
    • 25 to 30 —> eyeball will eventually stop growing
    • As you get older again —> eyeball starts to get a little bit stiff
    • Older people have bifocals on their glasses —> both nearsightedness and farsightedness on their glasses
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7
Q

Lens

A
  • in charge of refracting the light appropriately onto your back wall —> can change shape
  • Look on either side of it —> has a couple of types of muscle that can contract/relax depending on how far away an object is
  • Change in the shape of the lens is what happens when we’re trying to actually focus on a particular object
  • Your lens gets a little bit thinner when looking at things further away —> bc the muscle contracts and it changes the shape
  • Closer to you —> lens gets a little bit fatter to accommodate
  • Whole accommodation of the lens is helping to focus appropriately that bit of light onto your back wall
  • Actually changes the shape in order to focus the light
  • Refract the light onto the back wall —> onto the retina of your eyeball
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8
Q

Astigmatism

A
  • eyeball is lumpy
    • Not too long, not too short —> bumpy
    • projecting onto that back wall retina has a bit of trouble because it’s going to be projecting at different spots on there
    • Usually we can have the circumstances of corrective lenses to help us out so it can refract, do the job a little bit better compared to our lens
    • Refracting it in multiple different locations because of that essentially lumpy aspect
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9
Q

Visual field

A
  • Right visual field —> gets projected to your back left wall
  • Left visual field —> gets projected to your back right wall —> right retina
  • Works bc your eyeball also has a top and a bottom
  • Auditorium here is actually getting a projector to the ceiling of eyeball —> getting projected to your bottom
  • How your visual field comes into your retina: gets projected in an opposing manner —> everything gets switched —> your brain does a whole bunch of stuff in order to compensate
  • Never believe anything coming into your eye because it’s already getting projected in opposing fashion and yet you don’t perceive it as that
  • When it activates your visual receptors —> have transduction happen
  • Photo receptors are at retina —> quite large (yellow area)
  • As it goes towards like what your cheek area —> your lateral wall goes all the way
  • there’s the back wall, but also on this medial side closest to your nose —> where all our visual receptors are at
  • A big piece of area with this whole vitreous humour —> gel-like substance that fills the space between the lens and the retina + makes up the majority of your eyeball room
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10
Q

Three main layers in Retina

A
  • photo receptors —> our visual receptors
    • furthest away from how light was coming into your eye
  • bipolar cells —> two processes coming off of it —> talk to some ganglion cells
  • Ganglion cells —> true neurons of our visual pathway
  • If we’re following light —> goes through the cornea —> goes through the pupil —> goes through the lens —> it goes through your vitreous humour —> go through your ganglion cells and your bipolar cells before it can activate the photoreceptors
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11
Q

Optic

A
  • Retina —> where we have all those photo receptors
  • Lens —> no photo receptors
  • Optic disc —> no photo receptors
    • Optic nerve coming out of that location
    • Also see blood vessels coming in and out of that location
    • Optic disc —> bright area on the right —> convergence of all those blood vessels helping us out with brain nutrients + gases to that area
  • If location where there are no photo receptors —> if light were to shine on your optic disc —> would not perceive it —> your natural blind spot
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12
Q

Glaucoma

A
  • usually observed with regards to high correlation with cardiovascular disease, high blood pressure
  • optic disc —> place where blood vessels coming in out of that location
  • increased pressure —> increased pressure around this area —> increased pressure on the entire eyeball, including actually physically encroaching onto your optic nerve
  • Outside peripheral of your visual field start to disappear
  • Damage visual field usually can’t be recovered because it is actual damage to axons of neurons
  • Axons getting damaged —> if surrounded by Glia —> can have a degree of recovery but not always the case
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13
Q

Fovea

A
  • the very central part of your retina
  • little divot —> a little bit thinner in that location
  • If you want to see something in detail —> you will always try to get light to hit this particular part of your eyeball
  • Light reflecting off me —> going to go straight to your fovea when you’re doing that
  • it’s a little divot —> bc the ratio of communication between your photo receptors, your bipolar cells, and your ganglion cells —> one to one to one
  • Have a direct line of communication between your fovea and your brain —> why you can see things in a greater amount of detail
  • Should have two —> all at the back wall and that central part of your retina
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14
Q
A
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