Lecture Six: Neurologic Impairments: Constraints on Motor Control Flashcards
(41 cards)
Motor Cortex Pathology Deficits
- Motor paresis/paralysis
- Abnormal muscle tone
- Loss of selected muscle activation
Paresis
Mild or partial loss of muscle control( weakness)
Paralysis/-plegia
Total or severe loss of muscle activity
Paresis/Paralysis
- Hallmark of UMN injury/lesion in descending motor system
- Prolonged paresis leads to secondary effect of muscle structure changes and further weakness
Abnormal Muscle Tone
Muscle tone- “ stiffness” if muscle; resistance to passive stretch
- Flaccidity, Hypotonia, Normal, Hypertonia,Rigidity, Spasticity, Clonus
Flaccidity
Lack of muscle tone
Hypotonia
Low muscle tone
Normal
that there is the right amount of “tension” inside the muscle at rest, and that the muscle is inherently able to contract on command.
Hypertonia
abnormally increased resistance to externally imposed movement about a joint (increase resistance to passive stretch)
Rigidity
increased muscle tone, means stiffness or inflexibility of the muscles. In rigidity, the muscle tone of an affected limb is always stiff and does not relax, sometimes resulting in a decreased range of motion.
Spasticity
- Velocity dependent, moving the limb faster will show spasticity
- abnormal muscle tightness due to prolonged muscle contraction
Clonus
muscular spasm involving repeated, often rhythmic, contractions.
Loss of selected muscle activation
- Individual/ fractionation of movement
- Skilled, efficient movement
- Impaired Individual
Individual/Fractionation of movement
ability to selectively activate a muscle for isolated joint motion
Skilled, efficient movement
Activate only muscle necessary for the task
Impaired Individual
Abnormal coupling of muscle= Abnormal synergies
- During voluntary movement, an attempt to activate one muscle results in activation of abnormal coupled muscles
- Stereotypical movement patterns that cannot be adapted to task/environment
- Strong linkages; movement outside of fixed pattern is minimal or not possible
Abnormal synergies result from increased recruitment of descending pathways from the brainstem
Abnormal Synergies
UE Flexor Synergy Pattern
LE Extensor Synergy Pattern
UE Flexor Synergy Pattern
Scapular retraction, shoulder abduction/ER, elbow flexion, forearm supination, wrist/ finger flexion
LE Extensor Synergy Pattern
Hip Extension/adduction/IR,knee extension, ankle PF/inversion, toe flexion
Loss of Selected Muscle Activation
Coactivation : Simultaneous activation of agonist/antagonist
Typical
Atypical
Cerebellar Pathology Deficit
Hypotonia
Ataxia/Coordination problems
Hypotonia
Reduction in stiffness to passive lengthen of a muscle
May be associated with cerebellar pathology ot other disorders
Children: development delay; Ex: Down Syndrome
Normal Coordination
Synergistic nature of muscles working together to produce smooth movement
Ataxia/Coordination Problems
Problems with smoothness, sequencing, timing, grading, and accuracy