Liver Vasculature Flashcards

(119 cards)

1
Q

what is the primary supplier of oxygen rich blood in the liver

A

Hepatic Artery

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2
Q

what percentage of blood does the hepatic artery supply to the liver

A

30%

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3
Q

where is the prox portion of the hepatic artery visualized best

A

in tranvesre @ the celiac axis level

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4
Q

where is the distal portion of the hepatic artery visualized best

A

intercostally @ the level of the MPV

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5
Q

what kind of flow pattern does the HA have

A

low resistance

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6
Q

why is there a fill-in of the spectral window on the HA

A

because of the small artery diameter

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7
Q

what causes the HA to have variable velocities

A

tortuosity

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8
Q

what is the RI of the HA

A

0.5-0.7

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9
Q

when is the intrahepatic artery evaluated

A

in liver duplex exams
pre and post liver transplant studies
to rule out veno-occlusive disease in bone marrow transplant patients

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10
Q

normal HA blood flow with a celiac artery occlusion would result in what

A

collateralization occurring through the pancreaticduodenal network of vessles

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11
Q

true or false: there are many varients of the HA circulation

A

true

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12
Q

IVC is formed by the union of which vessels

A

common iliac veins

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13
Q

what is the location of the IVC

A

anterior to the spine

to the right of the aorta

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14
Q

where do the HV empty into the IVC

A

just inferior to the diaphragm

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15
Q

what kind of window usually the best to visualize the intrahepatic portion of the IVC

A

intercostal

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16
Q

what kind of waveform does the IVC have

A

spontaneous

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17
Q

what kind of spectral waveform does the prox portion of the IVC have

A

pulsatile

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18
Q

what kind of spectral waveform does the distal portion of the IVC have

A

Phasic

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19
Q

size of the IVC will vary with what factors

A

size of the patient
respiration
right atrial pressure (CHF)

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20
Q

typically how many major hepatic veins are there

A

3

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21
Q

true or false: accessory HV are common

A

true

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22
Q

HV drain into where

A

IVC

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23
Q

what happens to HV as they approach the IVC

A

they enlarge

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24
Q

how does the RHV run

A

coronally between the anterior and posterior segments of the right lobe

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25
how does the MHV lie
between the right and left lobes
26
how does the LHV run
between the medial and lateral segments of the left lobe
27
does the caudate lobe gave its own drainage into the IVC
yes
28
commonly what happens the left and middle HV before they enter the IVC
they join together
29
which HV is usually absent with agensis
RHV
30
to visualize all 3 HV what kind of scanning plane needs to be optimized
transverse in a subxiphoid scan plane
31
what kind of spectral tracing does the HV have
mutli-phasic | pulsatile flow pattern
32
are the walls more or less defined then the PV
less, because of no echogenic sheath
33
what are patency and distension a indication of
Budd Chiari or CHF
34
PV drain what kind of blood from the bowl and spleen to the liver
nutrient rich blood
35
what percentage of blood does the PV bring to the liver
70%
36
how is the PV formed
by the confluence of the splenic vein and SMV
37
what also forms the PV
IMV | coronary vein tributaries
38
where does the PV enter the liver
@ the porta hepatis
39
what are the branches of the PV
right | left
40
how is the PV visualized
sub or intercostal approach
41
what kind of velocity does the PV have
low
42
what is the range of PV velocity
15-40 cm/sec
43
what is the mean velocity of the PV
15-18 cm/sec
44
what kidn of signal does a PV spectral tracing produce
continuous
45
true or false: PV will have subtle phasic variations (slight undulations)
true
46
what kind of flow does the PV have
hepatopetal
47
what have to PV flow post-prandially
increases
48
with thrombosis in the PV what will be seen in the porta hepatis
numerous collaterals
49
with thrombosis what does the PV get termed
cavernous transformation
50
with the presence of thrombosis, sonographically what is the appearance of the PV
multiple small tortuous vessels in the porta hepatis with the MPV not visualized
51
what does a liver duplex exam determine
flow direction | normal waveform
52
a liver duplex exam includes the interrogation of which veins
hepatic veins portal veins splenic vein
53
what do you look for when looking for the cause of portal hypertension
collateral routes and indications
54
doppler assessment is assed during what kind of respiration
quiet
55
what are you looking for in the waveform prior to the alteration of flow direction
subtle changes
56
what position should the patient be in for a liver duplex exam
supine
57
the anterior subxiphoid window assesses what
LPV LHV SV MPV
58
coronal (intercostal) assesses what
``` RHV MHV RPV MPV HA ```
59
colour and spectral doppler assesses what
flow direction | quality of flow
60
the hepatic veins should have what kind of flow and waveform
hepatofugal flow | multiphasic waveform
61
the portal veins should have what kind of flow and waveform
hepatopetal | subtle phasicity
62
when meausring the MPV where is the measurment taken
ap diameter
63
the MPV is located what to the IVC
anterior
64
how do you elongate the MPV
rotating the transducer counter clockwise | > towards the patients right shoulder
65
MPV 2D picture is taken in what window
anterior subcostal
66
the patient should have what kind of respiration when assessing the MPV
quiet
67
the normal measurement of the MPV is
less than 13mm
68
the colour and spectral tracing of the MPV should be taken in what window
coronal intercostal
69
the MPV should be sampled where
outside the liver
70
the coronal intercostal approch for assessing the MPV gives what kind of angle
zero angle
71
the anterior subcostal approach for assessing the MPV gives what kind of angle
60 degree angle
72
how is the peak velocity of the MPV assessed
with a generic caliper
73
what is the normal velocity range of the MPV
15-40 cm/sec
74
the splenic vein protocol consits of what
colour and spectral | long axis assessment
75
the LHV protocol consits of what
colour and spectral | long axis assessment
76
what window is the SV and the LHV assessed in
anterior subcostal
77
LPV protocol consists of what
colour and sepectral long axis assessment taken in the subxiphoid window
78
RHV protocol consists of what
colour and spectral long axis assesment taken from a right intercostal window
79
MHV protocol consists of what
colour and spectral long axis assessment taken from either a midline subxiphoid or intercostal at the anterior axillary line
80
RPV protocol consists of what
colour and spectral taken from a coronal intercostal > same window and area as MPV
81
HA protocol consists of what
colour and spectral taken from a coronal intercostal window > same window and as MPV colour and spectral image
82
can the patient hold there breath for the assessment of the HA
yes
83
why does the scale have to be adjusted accordingly when assessing portal venous flow whem compared to the hepatic vein flow
because it is slower
84
sweep speed should be should be adjusted to what
medium
85
for portal veins what should be done to the baseline
moved down slightly
86
for hepatic veins what should be done to the baseline
moved up slightly
87
all the images have quiet respiration except which one
HA
88
for intercostal scanning what should be done to the probe
rotate the transducer to go between the ribs | point transducer indicator towards patients right scapula
89
what is portal hypertension
elevated pressure in the portal venous system which impedes blood flow through the liver
90
what does PHT cause
volume overload | increased resistance to flow
91
what are the three classifications
pre-hepatic intrahepatic post-hepatic
92
what is the most common clssification of PHT
intrahepatic
93
which classifcations are extrahepatic PHT
pre-hepatic | post-hepatic
94
what are causes Pre-hepatic PHT
``` portal thrombosis splenic thrombosis portal vein invasion splenic vein invasion inflammation of the pancreas (pancreatitis) extrinsic compression by a tumor ```
95
what is pre-hepatic PHT
pathology that occurs to blood flow before it enters the liver
96
what is intraheptic PHT caused from
damage to the liver sinusoids hepatocytes
97
what is intrahepatic PHT
hepatocellular disease
98
what are the four main causes of intrahepatic PHT
cirrhosis hepatitis fatty infiltration tumor invasion
99
can hepatitis, fatty infiltration anf tumor invasion lead to cirrohis
yes
100
what are the 3 steps of cirrhosis
1. normal liver tissue becomes inflammed 2. regeneration and scarring of liver tissue (fibrosis) 3. increased risistance to blood flow at the sinusoid level
101
what are the cause of post-hepatic PHT
thrombosis of the hepatic veins thrombosis of the IVC CHF right sided heart disease
102
what is post-hepatic PHT effect
drainage of blood from the liver (outflow)
103
what is Budd Chiari
thrombosis of the hepatic veins
104
can tumor invade into the portal vein
yes
105
what are the risk factors of chronic liver disease that proceeds fibrosis or cirrhosis
viral hepatitis: chronic hep B or C alcoholic liver disease autoimmune disorders: primary biliary cirrhoosis, primary sclerosing cholangitis metabolic & geetic disorders: hemochromatosis, Wilson's disease schistosomiasis non-alcoholic steatohepatitis (NASH) sarcoidosis
106
what are the risk factors of heart diease that results in increased right sided heart pressure
tricuspid regurgitation CHF constrictive pericarditis
107
if these people have these risk factor are they at a higher risk to have PHT
yes, but it does not mean that they will get PHT
108
what are the clinical signs that are related to PHT
``` ascites: abdominal distension splenomagaly GI bleed jaundice abnormal LFT ```
109
what is jaundice common sign of
cirrhosis
110
can patients be asymptomatic if they have vascular liver disease
yes
111
manifestations of signs and symptoms associated with underlying disease is most common with which 2 diseases
pancreatitis | liver disease
112
what is hematemesis
blood in the vomit
113
what is melena
blood in the stool
114
what are other patient symtoms for PHT/vascular liver disease
variceal hemorrhage | bacterial peritonitis
115
what are the fidings of a physical examination when assessing for PHT
jaudice (if liver is sufficiently impaired) splenomegaly (could lead to low platlet count) dialted abdominal wall veins hepatic encephalopathy (confusion due to poor liver function)
116
2-D sonographic evaluation includes documentation of what structures
portal vein diameter greater than 13mm cavernous transformation (intra-abdominal collaterals) portocaval anastomoses dilation/recanalization of the umbilical vein splenomegaly greater than 13cm
117
duplex evaluation of the patency of vessels including collaterals inculdes what kind of documentation
colour doppler assessinf for flow or the lack of flow | power doppler looking tortuous vessels and collaterals
118
duplex evaluation of the direction of flow inculdes what kind of documentation
colour and spectral there is a possility of reverse flow in portal/splenic veins assessing hepatofugal and hepatopedal flow
119
duplex evaluation of the loss of phasic variations inculdes what kind of documentation
spectral tracing