Lp 30 Flashcards

(38 cards)

1
Q

-compex, private experience
-difficult to explain or describe
-physiological perspective: protective mechanism signalling danger

A

Pain

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2
Q

Pain has various aspects? (6)

A

-physiological
-psychological
-motivational
-cognitive
-social & cultural
-gender

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3
Q

Due to stimulation of nociceptors

A

Nociceptive pain

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4
Q

Due to injury to nerves (sensory disturbance)

A

Neuropathic pain

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5
Q

Pain is a separate sentry modality evoked by stimulation of specific receptors that transmit information to pin centres in the brain

A

Specificity theory

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6
Q

Pain receptors share receptors & pathways with other sensory modalities but different patterns of activity of the same neurons signal painful or non-painful stimuli
-gate control theory is a modification of specificity theory

A

Pattern theory

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7
Q

Theory proposes that the brain contains a neural network called?

A

Body-self neuromatrix

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8
Q

Integrates multiple sources of inputs that ultimately yields sensory, emotional & cognitive components of pain experience & behaviour

A

Neuromatrix

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9
Q

Sensitivity to pain is variable?

A

-one person to another
-one circumstance to another
-type & or location of affected tissue

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10
Q

Stimulation of nociceptive nerve endings can be due to?

A

-chemical changes
-hypoxia
-mechanical distortion

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11
Q

Irritation of nerve ending often results in hyperexcitability experienced in several ways?

A

-hypersthesia
-hyperalgesia

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12
Q

Suggested causes of hyperalgesia

A

-decreased threshold
-high intensity stimulation

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13
Q

Unlike other sensory receptors, nocicptors respond to several forms of stimulation?

A

-mechanical
-thermal
-chemical

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14
Q

Increased sensitivity to sensory stimuli (touch), unpleasant sensations (tingling)

A

Hyperesthesia

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15
Q

Increased sensitivity to pain

A

Hyperalgesia

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16
Q

Muscle (extramural fibres {EFF})

17
Q

Tactile

18
Q

Muscle spindle (intrafusal fibres {IFF})

19
Q

Tactile, pain

20
Q

-altered pain sensation (reactions to otherwise non-noxious stimuli)
-hypersensitivity may be due to inflammation, or facilitation of nerve segments

21
Q

No sensation

22
Q

No pain

23
Q

Reduced sensitivity

24
Q

Reduced pain sensation

25
Increased pain sensitivity
Hyperalgesia
26
Increased sensitivity
Hyperesthesia
27
changes in sensory sensitivity
Paresthesia
28
2 types of sensory neurons transmit pain signals to SC
-a nerve fibres -c fibres
29
-yell at Ed -conduct “fast pain” (first pain) -pain from mechanical or thermal stimuli -transmit impulses at a rate of 10-30 metres per second
A nerve fibres
30
Change ins tumuli in sensitivity and consequent nerve impulse generation in the presence of a constant stimulus
Adaptation
31
Provide a sustained response with little or no adaptation
Nociceptors
32
Why are nociceptors not fast adaptive?
Due to their protective function
33
-unmyelinated -conduct “slow-wave pain” (second pain) -pain from chemical or persistent mechanical/ thermal stimuli -transmit impulses at a rate of 0.5-2.0 metres per seconds
C-fibres
34
Nociceptive stimulation that activates C fibres can case a response known as
Neurogenic
35
Inflammation that produces vasodilation & an increased release of chemical mediators to which nociceptors respond
Neurogenic
36
From intense pressure applied to skin or from violent contraction/extreme stretch of muscle -extremes of hot or of cold
Mechanical stimuli
37
From tissue trauma, ischemia, inflammation -released from injured & inflamed tissues
Chemical stimuli
38
Spinothalamic pathways have 2 subdivisions
-one Carrie’s sharp, fast pain -other ravels more slowly