memory disorders Flashcards

1
Q

brain structures

A
  • different types of memory involve different structures
  • damage to hippocampus, poor episodic
  • damage to para hippocampal cortex, poor semantic memory
  • damage to both regions, poor episodic and semantic
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2
Q

causes of amnesia

A
  • damage to different structures of the brain cause different syndromes
  • causes:surgery, chronic alcohol abuse, brain tumour, dementia, bilateral stroke, closed head injury.
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3
Q

retrograde amnesia

A
  • poor recall for memories formed before onset of amnesia
  • greater for episodic than semantic
  • temporal gradient, older memories less impaired
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4
Q

consolidation theory

A
  • physiological process in the hippocampus leads to formation of long lasting memories
  • consolidated memories stored elsewhere, protecting them from effects of hippocampal damage
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5
Q

semanticization

A

episodic memories become more like semantic memories over time= protected from the effects of damage

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6
Q

reduced learning opportunity

A
  • episodic on a single learning experience= explains amnesia
  • semantic memories depend of several learning experiences
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7
Q

anterograde amnesia

A
  • loss of ability to form new memories after onset of amnesia
  • results from damage to areas of the brain that are involved in forming new memories
  • damage to hippocampus the main cause in most instances
  • mammillary bodies and fornix also commonly involved
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8
Q

global amnesia

A
  • moderate retrograde and severe antergrade
  • results from lesions in medial temporal lobe, hippocampus
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9
Q

patient hm

A
  • amnesic
  • suffered from severe epilepsy from age 10
  • at age 27 had surgery to remove entire medial temporal lobe
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10
Q

korsakoffs syndrome

A
  • also known as diencephalic amnesia
  • vitmain b1 deficiency from chronic alcoholism
  • damage to mammillary bodies in hypothalamus
  • poor ability to remember events before or after onset amnesia
  • some new learning ability, motor skills
  • slight impairment of stm, digit span
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11
Q

symptomology

A
  • typically has a gradual onset
    -Brain damage widespread (hippocampus and frontal)
  • a precise pattern of damage varies across
    -brain plasticity and learning of compensatory strategies
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12
Q

issues

A
  • events happened before or after amnesia onset?
    -damage to frontal lobe = other cognitive deficits
    difficult to generalise across patients
    does not provide a direct assessment of the impact of brain damage on ltm
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13
Q

semantic dementia

A
  • severe problems with semantic memory but intact episodic
  • severe loss of info about meanings of words and concepts
  • difficulty naming pictures/pbjects, single comprehension, categorising and knowing uses and features of objects
  • episodic memory and most executive functions reasonably intact in the early stages.
  • patient differ in symptoms
  • always involves degeneration of anterior temporal lobe
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14
Q

double dissociation

A
  • amnesia and semantic dementia point to a double dissociation in ltm
  • identify that brain damage to one structure disrupts one cognitive process (x) but not another (y)
  • also identify brain damage to different structure disrupts cognitive process y but not x
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15
Q

amnesia=hippocampus

A
  • poor episodic (x)
  • intact semantic (y)
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16
Q

semantic dementia= anterior temporal lobe

A
  • poor semantic
  • intact episodic
17
Q

dissociation

A

identification of a brain region responsible for cognitive process

17
Q

single dissociation

A

brain damage to one structure disrupts one cognitive processes (x) but not (Y)

18
Q

eyewitness testimony

A
  • accuracy of an individuals memory is sometimes of enormous importance
  • you are sole witness to a serious crime
  • you have to identify perpertrator in a line up,used as evidence
19
Q

Misinformation effect

A

Refers to the finding that memories are easily distorted by misleading information presented afterward.

20
Q

confirmation bias

A

Tendency to recall information in a way that confirms pre-existing beliefs.