midterm 2 Flashcards

1
Q

consciousness

A

our awareness of our environment/selves

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2
Q

dual processing

A

brains process info at 2 levels

serial processing (consciousness can only process 1 step at a time
unconscious processes super fast

parallel processing

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3
Q

blindsight

A

consciously, we are blind

unconsciously, we still have some vision

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4
Q

subliminal stimulation

A

we’re exposed to a stimulus and the exposure is super fast
–> info gets to brain but never reaches consciousness

cannot see object consciously but can still influence behaviours

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5
Q

serial processing

A

consciousness can only process one step at a time

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6
Q

parallel processing

A

processes multiple pirces of info simultaneously

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7
Q

subconscious processing (priming)

A

we are exposed to a stimulus and are aware of it, HOWEVER we don’t know that the stimulus is unconsciously activating in our brain memories and the information linked to it
–> can affect behaviour

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8
Q

where does consciousness come from

A

the brain

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9
Q

attention

A

allow us to focus our awareness on a tiny point of stimulation around us

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10
Q

value (attention)

A

ability to pay attention to valuable & essential things for survival

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11
Q

(t/f) attention is an unlimited resourse

A

FALSE

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12
Q

is attention selective

A

YES. focusing attention on a certain stimuli causes us to disregard other stimuli in the environment

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13
Q

inhibition (attention)

A

paying attention to a specific stimuli causes the brain to inhibit us from processing other info
–> sharpens attention

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14
Q

cocktail party effect

A

at a party –> very loud and noisy but can focus attention on an interesting convo

in spite of this, if someone said our name we’re likely to pick it up

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15
Q

dichotic listening

A

used to study selective attention

2 diff msgs in each ear at the same time –> patients asked to pay attention to one ear only

we still pick up info from he other ear

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16
Q

Corteen and Wood Experiment (1972)

A

experiment to examine DEGREE we use dichotic listening

conditioned to associate city names with an electric shock
–> city names were played in unattended ear
–> used GSR (galvanic skin response) measurements ( measures arousal in sympathic nervous system)
–> 38% response rate for recognized city names, 10% for unrelated words

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17
Q

passive (attention attraction)

A

bottom-up process

stimuli in environment grabs our attention bc there is something that stands out about it

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18
Q

active (attention attraction)

A

top-down

we intentionally chose what we are going to pay attention to

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19
Q

inattentional blindness

A

we’re going to fail to detect a significant stimulus in our environment

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20
Q

change blindness

A

we’re engaged with a stimulus but don’t notice the change in the stimulus

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21
Q

intentional change detection

A

we’re told that the stimulus is going to change, but we still have a hard time detecting the change

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22
Q

divided attention (multitasking)

A

consciousness can’t multitask, we tend to slow down and make errors

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23
Q

visual neglect

A

attention disorder

part of the world becomes non existent for us

can happen in the right or left hemisphere depending on what’s dmged
ex. u only shave one side of ur face –> u can see the other side but ur not aware of it

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24
Q

fatal familial insonmia

A

rare hereditary disease that affects THALAMUS

can die from lack of sleep 12-18 months after symptoms start

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25
electroencephalograms (EEGs)
measure activity across surface of brain
26
electrooculograms
used to measure movements of eyes in sleep
27
electromyograms
used to measure tension and muscles of jaw
28
2 observable patterns when awake
relaxed wakefulness, awake and alert
29
alpha waves
8-12 Hz more regular and predictable than beta waves
30
relaxed wakefulness
alpha waves when person is quietly resting
31
beta waves
13-30 Hz desynchronized and erratic waves --> many neural circuits processing information
32
stages of non-REM sleep
STAGE 1, STAGE 2, slow wave sleep (SWS_
33
STAGE 1 (non-REM sleep)
theta waves transition from relaxed state --> early sleep (very light)
34
theta waves
3.5-7.5Hz
35
awake and alert (stages of sleep)
beta waves
36
STAGE 2 (non-REM sleep)
sleep spindles, K complex
37
sleep spindles
(12-14Hz) occur around 2-5 times/min thought to play a role in memory consolidation higher sleep spindles associated with higher IQ test scores
38
K complex
bursts of energy on EEGs occur around once/min can be triggered by unexpected noises, still wouldn't have a sense of sleep when woken prepares brain to enter SWS
39
slow wave sleep (SWS)
delta waves 15-20 min after STAGE 2 starts deepest stage of sleep, only a strong stimulus will wake u groggy, confused when woken
40
delta waves
less than 4Hz regular, high-amplitude waves
41
REM sleep (rapid eye movement)
theta waves, beta waves 45 min after SWS starts brain is very active with vivid dreams, but easier to wake than SWS
42
REM sleep antonia
generally become paralyzed during REM sleep
43
what happens to blood flow to brain during REM sleep
blood flow reduced, but visual association cortex and prefrontal cortex receive a large proportion of oxygenated blood
44
function of SWS
important for resting brain over body important for explicit memories regions with highest activity during waking hours is resting
45
function of REM
benefits brain's ability to absorb and process information
46
rebound phenomenon (REM)
there is a need for a certain amount of REM sleep --> if brain is deprived from REM for a couple days, brain tries to enter REM more quickly and for a longer period of time increased % of time spent in REM in infants
47
insomnia
inability to fall asleep/inability to remain asleep
48
sleep hygeine
habits and behaviours conducive to sleeping well
49
conditioned insomnia
learned insomnia --> going to bed becomes associated with inability to fall asleep among the most commonly diagnosed forms of primary insomnia
50
idiopathic insomnia
child onset insomnia neurophysiological abnormality in the CNS begins in childhood, more resistant to treatment
51
hypersomnia
excessive sleepiness, caused by poor sleep during the night
52
sleep apnea
intake of oxygen is reduced as person sleeps --> brain sends signal to body as blood oxygen decreases --> sleeper wakes
53
treatment for sleep apnea
CPAP: pressurized air mask htat pushes pressurized air through airway
54
narcolepsy
rare genetic neurodegenerative disorder that has several symptoms: - sudden and extreme need to sleep - cataplectic attacks - paralysis with hallucinations
55
cataplexy/cataplectic attacks (narcolepsy)
paralysis experience during REM initiates and inappropriate times initiated by emotionally engaging events
56
kinds of hallucinations (narcolepsy_
hypnagogic: upon onset of sleep hypnopompic: just before waking
57
circadian rhythym
daily clocks, closer to 25 hours
58
zeitgebers
time givers/cues --> our clock is reset every morning by cues associated with morning activity reliable stimuli in the environment tht provide information about the time of day
59
what resets our daily clock
presence/absence of light
60
suprachiasmatic nucleus (SCN):
your body’s timekeeper sends signals to several regions of the brain → ex. pineal gland pineal gland secretes melatonin
61
psychoactive drugs
drugs that influence/affect function of brain/NS
62
where do drugs produce their effects
synapse --> interfere with communication between neurons
63
dopamine
pleasure molecule
64
what do most street drugs agonize
dopamine
65
tolerance (drugs)
can develop a tolerance if we abuse a drug --> need to consume more and more to get the desired effect
66
neuroadaptation
when we use drugs, our brains adapt to that by changing themselves (plasticity) brain may stop production of certain NT or shut down receptors for certain NT
67
withdrawal
happens when you stop abusing a drug --> can get uncomfortable and painful symptoms
68
drug dependence
physical: need drug to function normally psychological: feel like we can't live without it
69
2 major types of drugs
- depressants - stimulants - hallucinogens
70
71
depressants
reduce, slow down activity of the NS/brain
72
alcohol
depressent at all dosages
73
what area of the brain is affected by alcohol
hippocampus --> part of brain that deals with memories --> memories of a drunk night are hazy
74
barbiturates (depressants)
- more addictive - more powerful - known as downers
75
benzodiazepines (depressants)
- highly addictive - known as tranquilizers - eg. xanax - valium
76
stimulants (drugs)
psychoactive drugs that speed up/increase activity of NS/brain
77
(t/f) depressants have a lethal aditive effect
TRUE
78
nicotine
damages ur DNA 1 cigarette = lose 11/12 min of life
79
why cant u stop smoking
nicotine is highly addictive --> enhances the activity of multiple NTs: - acetylcholine - norepinephrine - dopamine
80
nicotine's dual effect
- sluggish --> smoke --> perk up - anxious --> smoke --> calm down
81
effects of long-term use of nicotine on acetylcholine
reduces levels of acetylcholine
82
cocaine
stimulant that blocks reuptake of some NTS --> enhances: - dopamine - norpinephrine - serotonin can end up with chronic permanent depression that requires meds`
83
formication
feeling as if we have bugs/insects crawling in skin
84
stereotypic behaviours (cocaine)
non sensical behaviours that we repeat while under the influence of cocaine
85
amphetamines
stimulant combats effects of hunger and fatigue agonizes dopamin --> inhibits reuptake of dopamine and stimulates release of it from terminal buttons
86
hallucinogens
directly influence sensory systems and interpretation of reality
87
psychedelics
have most profound effects on consciousness
88
LSD
man-made areas of the brain that usually talk to each other communicate more/stop talking to each other
89
which part of the brain does LSD affect
thalamus --> explains why LSD users experience fusing of senses
90
marijuana
attaches to cannabinoid receptors found all over brain active ingredient: THC increases dopamine release
91
what NTs are inhibited by marijuana
norepinephrine, acetylcholine, glutamate, GABA
92
whats more likely to lead to addiction: inhaled drug, digested drug
INHALED
93
classical conditioning
we learn to associate 2 events/stimuli --> learn that one event signals arrival of another stimulus triggers response
94
respondant behaviour
response that occurs in the presence of a stimulus
95
operant conditioning
learn to associate a behaviour with its consequences
96
operant behaviour
organism initiates the behaviour and it produces consequences
97
fundamental principle (OC)
behaviour is controlled by its consequences --> desired consequence --> likely to repeat it
98
law of effect (OC)
when a behaviour produces an undesirable/desirable consequence, we are more likely to repeat it
99
thorndike
stipulated the law of effect and started research on operant conditioning --> instrumental conditioning
100
skinner
linked and associated with operant conditioning strict behaviourist
101
ABCs (OC)
A = antecedent B = behaviour C = consequence
102
differential vs non-differential consequences
Differential: receiving different consequences --> leads to FASTER LEARNING Non-differential: receiving the same consequence
103
reinforcer
consequence of a behaviour that makes the behaviour more likely to repeat in the future
104
2 types of reinforcers
positive, negative
105
positive reinforcer
produces a consequence where we receive something pleasant
106
negative reinforcer
increases possibility that behaviour will occur again
107
2 forms of negative reinforcers
escape, avoidance
108
primary reinforcers
reinforcers that are naturally reinforcing --> no learning required
109
conditioned reinforcers (secondary reinforcers)
learning is required --> not naturally reinforcing --> learn through experience
110
generalized conditioned reinforcers
objects traded for several other reinforcers --> don't lose their power to reinforce behaviour
111
immediate reinforcers
we do the behaviour and immediately or shortly after are reinforced
112
delayed reinforcers
do the behaviour but wait for the reinforcement
113
2 types of scheduling consequences
continuous, intermittent
114
continuous scheduled consequence
Every single time the behaviour takes place, it is reinforced; without exception good for teaching a new behaviour HOWEVER when the behaviour is learned, it is important to move to intermittent because it helps to maintain the behaviour longer
115
intermittent scheduleing consequence
more resistant to exctinction sometimes the behaviour is reinforced, sometimes is not makes behaviour more likely to be maintained for a longer period PARTIAL EXTINCTION EFFECT
116
categories of partial extinction effect
ratio: number of responses that determine when behaviour is going to be reinforced interval: passage of time that will determine when a behaviour is going to be reinforced
117
2 types of ratio (partial extinction)
ficed ratio: very specific # of behaviours must occur before reinforcement happens variable ratio" # of responses that must occur will vary/change --> PRODUCES THE HIGHEST LEVEL OF RESPONSES
118
2 types of intervals (partial extinction)
fixed interval: very specific amt of time must go by for the desired behaviour to take place variable interval: amt of time that must go by before reinforcing behaviour varies --> produes THIRD LOWEST rate
119
break-and-run pattern of responding
FIXED RATIO schedule run: when we produce many responses quickly until we earn a reqrd break: occurs after delivery of reinforcer
120
2 types of punishment
positive punishment: as a consequence, something unpleasant is added negative punishment: as a consequence, something we desire is taken away
121
what did skinner advocate for? why?
positive reinforcement bc it has a longer-lasting effect on behaviour
122
What would reduce/stop an undesired behaviour faster: extinction or punishment?
PUNISHMENT
123
why shouldn't u use punishment
1. Doesn’t teach a person what to do to get reinforcers 2. Involves aversive stimuli, including some that cause pain 3. A person who uses punishment successfully once is more likely to use it again 4. person learns to use punishment to control others’ behaviour 5. only decreases behaviour if the response is punished a) immediately, b) every time, c) with a large aversive stimulus
124
operant extinction
stop reinforcing behaviour of interest --> must identify where is the reinforcer of the behaviour
125
extinction burst
during extinction procedure, this may occur --> situation will get worse before it gets better
126
what happens when exinction alone is used to stop self-injurious behaviours
target behaviour increased before it stopped
127
tolman
father of cognitive psychology --> criticized skinner's ideas, which started the ball rolling --> said sometimes learning can take place without reinforcement
128
behaviourism
dominated psychology for almost 50 years. study of the mind, conscious, etc. that flourishes the study of psychology
129
latent learning
we acquire knowledge without any reinforcement --> knowledge remains hidden until we have a reason to display it
130
cognitive map
mental map u have in ur mind --> skinner said when rats learn to run a maze, they learn via trial and error --> tolman dais they are learning the layout of the map
131
learned helplessness
can experience this when repeatedly exposed to stimulus that is aversive and uncontrollable we give up and stop trying completely
132
bandura
learning by observation --> vicarious learning --> social learning
133
social learning
we observe the social world around us --> what is rewarded/punished and adjust our behaviours accordingly
134
mirror meurons
highly specialized neurons in diff parts of the brain are linked to empathy/learning --> activate when we watch someone or us ourselves perform a behaviour
135
bandura's model of cognitive processes
strongly believed cognitive process is essential for learning in order to imitate behaviour 4 things must be present: 1. attention 2. memory 3. action/motor skills 4. motivation
136
learning
relatively permanent change in how we think, feel and behave as a result of experience
137
3 major ways we learn
classical conditioning operant conditioning observational learning
138
conditioning
we learn to form associations
139
pavlov
classical conditioning, dog and bell
140
higher order conditioning
We trained the dog to salivate at the sound of the bell, and dog does very well.
141
factors associated with classical conditioning
- frequency - timing - order of presentation
142
stimulus generalization
Ex. ONE dog bites you, and you become afraid of ALL dogs
143
stimulus disctimination
One dog bites you, and you're ONLY afraid of that dog; you're NOT afraid of other dogs.
144
pavlov vs. modern researchers
modern researchers say u MUST take cognitive processes into consideration
145
introspection
subjects will observe their own mental processes and observe them
146
watson
believed psychology should only study observable behaviours
147
CT scan
Computerized Tomography Uses x-rays that pas through body, can generate images of "slices" of the body ex. detect changes in structure to to disease
148
pros/cons of CT scan
pro: - Fast, cheap, non-invasive con: - radiation exposure - only less us see the structures of the brain, not the brain in action
149
MRI
Magnetic Resonance Imaging Uses magnetic fields to image alignments of H+ ions (diff tissues have diff amts of water ex. can detect changes in structure due to disease
150
pros/cons of MRI
pro: - noninvasive, great precision, no radiation con: - very expensive - cannot have biomedical devices or metal in patients - just gives an image of the brain, not the brain in action
151
fMRI
functional MRI uses magnetic fields to image alignments of H+ ions. exposed to magnetic field --> tracks oxygenated blood More active parts of brain will consume more oxygenated blood ex. can measure activation during task/stimulation
152
DTI
Diffusion Tensor Imaging tracks and images water movement alone neural pathways, can measure density of neural tracts (bundles of axons). Tracks nerves of the brain and the connections between different areas ex. study white matter degeneration in disease
153
pros/cons of DTI
pro: - noninvasive - no radiation - no injections con: - interpretation can be difficult in tracts with diff kinds of fibers
154
pros/cons of fMRI
pro: - noninvasive - no radiation - no injections con: - cardiovascular disease/compromise function can make measurements unreliable --> elay between stimulus/output - shows the brain in action
155
PET/SPECT
Single Photon Emission Computed Tomography Uses ingested radioactive compound to track molecular changes, person is injected with radioactive substance ex. visualize the activity of specific neurotransmitters
156
What substance are patients usually injected with for a PET scan?
Glucose
157
Pros/cons of PET/SPECT
pro: - can see molecular changes in real time - determine which part of brain is more active - can see brain in action con: - radiation exposure
158
endocrine system
major communication system that consists of all glands in the body
159
3 types of hormones
homeostasis, reproductive, stress
160
pituitary gland
master gland of the endocrine system, boss of almost all glands of system
161
hypothalamus
controls pituitary gland
162
how does the NS affect the endocrine system
hypothalamus
163
transduction
brain only understands electrochemical messages, so physical energy must be translated into a message the brain can understand
164
transmission
message must be transmitted to brain for processing
165
sensory receptors
respond to physical energy/stimulation from natural world the ones that detect, transduce, and transmit
166
bottom-up processing
collect raw data from world and sent it to brain
167
top-down processing
brain uses excisting knowledge, memories, beliefs, in order to interpret information
168
prosopagnosia
when the eyes work, but no perception
169
psychophysics
scientific study of how physical characteristics of the physical world trsnlate into psychological experiences
170
absolute threshold
minimal amt of energy that must be there for us to detect it 50% of the time
171
difference threshold (JND)
minimum amount of change in stimulation for us to detect it 50% of the times
172
weber's law
ability to notice the difference between 2 stimuli is proportional to the intensity or size of the stimulus
173
signal detection theory
ability to detect a stimulation doesn't depends only on how the stimulation is --> large number of factors ex. how healthy we are, fatigue, motivation, mood
174
perception
brain taking raw sensory data and interpreting it in a meaningful way
175
what do you need to see
light
176
light is a form of ???
electromagnetic radiation
177
what is the range of visible light
400-700nm
178
what colour are long waves
red
179
amplitude
height of wavelength
180
rods and cones (retina)
sensory receptors for vision, connected to bipolar cells
181
bipolar cells
connected to ganglion cells
182
ganglion cells
axons bunch up together to form the optic nerve
183
optic nerve
carry visual information to brain
184
blind psot
where optic nerve leaves the eye --> no rods/cones in that area so nothing to detect light
185
fovea
center of retina, responsible for visual activity allows us to see fine detail
186
where are rods found
in the periphery
187
where are cones found
heavily concentrated in fovea
188
what is the connction for cones - bipolar cells
1-1 ** multiple rods for one bipolar
189
cone function (retina)
needs lots of light to activate allows us to see colour
190
rods function (retina)
activated by little light, used when dark involved in peripheral vision
191
simple cell vs. complex cell
simple cell: respond to small stationary bars of light oriented at specific angles complex cell: respond to lines of particular orientation moving in specific directions
192
parietal lobe
WHERE pathway dorsal stream, lets us know where an object is in space, whether it is moving or not
193
temporal lobe
WHAT pathway ventral stream
194
limbic system
responsible for emotional reactions
195
trichromatic theory
3 primary light colours, combining them allows us to see more colours
196
compl. afterimages
continue to perceive an objecte ven though we aren't looking at it anymore
197
4 RGBY
4 primary light colours --> red, green, blue, yellow 3 antagonistic colour system
198
3 antagonistic colour system
red and green RG blue and yellow BY black and white BW
199
RG
neurons respond to RG light, but respond in opposite ways --> red excites neuron, green inhibits
200
principle of figure-ground
certain information is given priority over the background
201
principle of proximity
objects close to one another will be grouped together
202
principle of similarity
objects physically similar will be grouped together
203
principle of closure
tend to perceive whole objects, even when part of that information is missing
204
principle of good continutation
if lines cross each other/are interrupted, people tend to still see continuously flowing lines
205
principle of common fate
objects moving together will be grouped together
206
depth perception
brain uses bottom-up and top-down processing to understand
207
2 kinds of depth cues
- monocular (1 eye) - binocular (2 eyes)
208
3 characteristics of sound waves
frequency(Hz), --> pitch amplitude(Db) --> loudness, complexity --> timbre (what makes a sound unique)
209
pinna
captures and funnels sound waves into auditory canal
210
auditory canal
soundwaves travel until it reaches the eardrum
211
ossicles (eardrum)
tiniest bone in body, vibration cauyses oval window to vibrate
212
oval window (eardrum)
membrane in ear
213
cochlea(eardrum)
fluid inside cochlea, vibration from oval window causes fluid in cochlea to move in waves
214
basilar membrane (eardrum)
waves of cochlear fluid cause basilar membrane to vibrate
215
cilia (hair cells) (eardrum)
vibration of basilar membrane causes cilia to sway and bend --> they fire sensory receptors of the ear
216
auditory nerve(eardrum)
hair cells detect, transduce, transmit information from brain via auditory nerve carries info to brain
217
thalamus MGN
info goes from auditory nerv to here
218
auditory cortex (temporal lobes)
stops here after going to thalamus MGN
219
tonotopic organization
auditory systems maintains this from basilar membrane to auditory cortex
220
simple sounds are processed in ???
lower regions
221
why do some neurons that process auditory info have faster action potential and larger terminal buttons
timing is critical to understand
222
place theory
sound waves of difference frequencies will activate different areas of the basilar membrane
223
high-frequency sound waves
activate hair cells located at BEGINNING of membrane
224
LF sound waves
activate hair cells located at the END of the basilar membrane
225
frequency theory
soundwave of different frequencies will affect rate of firing
226
2 binural cues
time of arrival, loudness
227
cutaneous senses
sense more than just touch (wetness of our skin, a cut)
228
mechanoreceptors
top layer: - merkel receptor (pressure) - meissner receptor (pressure) deeper layers - ruffini cylinder (stretching of skin) - pacinian corpuscle (vibration, texture)
229
what are the merkel and meissner receptors for
pressure
230
nociceptors
sensory receptors that detect, transduce, and transmit info about pain
231
neurological gate
state of gate is linked ans associated whether we experience pain or not
232
small nerve fibers (S-fibers)
mostly carry pain info --> gate is opened, likely to feel pain
233
large nerve fibers (L-fibers)
mostly carry info NOT related to pain --> gate is closed, likely to feel little pain
234
T-cells
middle men between fibers and gate --> must be activated for gate to open Small nerve fibers activate, which activate t-cells, when then open the gate, and we are likely to feel pain. When large nerve fibers are activated, t-cells are inhibited, the gate doesn’t open, and we are likely to feel little to no pain as a result.
235
olfactory receptors (smell)
scent must reach our nasal cavities and olfactory receptors for us to smell
236
olfactory epithelium(smell)
membrane filled with mucus
237
glomeruli (smell)
located in olfactory bulb, sends info to different parts of the brain, including limbic system
238
regeneration of olfactory cells
cells regenerate every few weeks, but lose numbers as we age
239
filiform papillae (tongue)
entire surface of tongue, doesn't have taste buds
240
fungiform papillae (tongue)
tips and sides of tongue
241
foliate papillae
back of tongue
242
circumvallate
back of tongue
243
4 basic senses
SSSB: sweet, sour, salty, bitter
244
umami
japanese 5th sense linked with savory foods
245
bimodal neurons
neurons that respond to more than one sense
246
kinesthesis
allows us to know where are body is sensory receptors: proprioceptors
247
vestibular sense
balance
248
2 organs linked with sense of balance
- semicircular canals: sense rotation of head - vestibular sacs: respond to cues of balance and posture
249
gustatory cells
sensory receptors for taste found in taste buds
250
papillae
bumps on tongue
251
experimental group vs control group
experimental: expose to IV control: not exposed to IV
252
biological constraints of classical conditioning
you MUST take cognitive processes and take them into considerations. Animals are not stupid learners; they assess and evaluate the information. If the information gives them protective power and it is reliable, they WILL learn it. If it doesn't give them prodective power and it isn't reliable, they WILL NOT learn it.
253
biologically predisposed
we are biologically prepared to learn via observation
254
(t/f) babies start to imitate facial expressions very early in life
T
255
Tolman and Honzik's experiment
hypothesis: Said there is no change in our performance until we receive a reward FOOD GROUP rats: received food when they reached the end of the maze Made fewer errors NO FOOD GROUP: did not receive food for the first 10 days 11th day → received food Made fewer errors and ran faster than previous days as well as the FOOD group Suggested that they were actively constructing cognitive maps of the maze
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(t/f) learned helplessness can be specific and generalized
T
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split brain
we have a Rt visual field & a LVF Info —> RVF —> LH Info —> LVF —> RH LH —> Language, Rt body RH —> Left body
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gestalt principles of organization
we were born with specific, predisposed, ways of organizing information so that it has utility
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synonym for olfaction
smell