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ABOS Part I > Misc > Flashcards

Misc Flashcards

1
Q

Treatment of symptomatic Watanabe I and II discoid menisci.

A

Saucerization only.

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2
Q

MSK malignancy associated with Rothmund-Thompson syndrome

A

Osteosarcoma.

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3
Q

Indication for ORIF for ASIS avulsion fractures.

A

Displacement greater than 3cm or painful nonunion.

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4
Q

What factor most significantly contributes to development of pseudotumor formation for MoM implants?

A

Edge loading.

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5
Q

Antoni A.

A

Cellular area in benign schwannoma.

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6
Q

Antoni B.

A

Less cellular area in benign schwannoma.

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7
Q

Serratus anterior innervation.

A

Long thoracic.

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8
Q

Rhomboids innervation.

A

Dorsal scapular.

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9
Q

Supraspinatus innervation.

A

Suprascapular n.

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10
Q

Infraspinatus innervation.

A

Suprascapular n.

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11
Q

Horner syndrome indicates preganglionic injury to these nerve roots.

A

C8, T1.

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12
Q

CC ligaments.

A

Conoid and trapezoid.

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13
Q

Most medial CC ligament.

A

Conoid.

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14
Q

Most lateral CC ligament.

A

Trapezoid.

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15
Q

These ligaments are torn with type 3 AC separation.

A

CC ligaments.

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16
Q

This ligament prevents anterosuperior escape.

A

Coracoacromial.

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17
Q

Hornblower’s sign tests this

A

Teres minor.

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18
Q

Teres minor innervation.

A

Axillary nerve.

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19
Q

Delayed infection after pediatric PSF.

A

p. acnes.

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20
Q

Pediatric spondylolisthesis indications.

A

Progression, grade 3+

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21
Q

Type of surgery for pediatric spondylolisthesis.

A

PSF L4-S1

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22
Q

Highest risk of progression of congenital scoliosis.

A

Hemivertebra with contralateral bar.

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23
Q

EOS progression risk (3).

A
  1. RVAD > 20
  2. Phase 2 ribs
  3. Curve > 30 deg
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24
Q

Phase I versus phase II ribs in infantile scoliosis.

A

Phase I – no rib overlap with vertebral body

Phase – rib overlap with vertebral body

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25
Palpable mass in SCM.
Congenital muscular torticollis.
26
Algorightm for C1-C2 pediatric rotator subluxation.
CT scan, traction.
27
Most important risk factor for DDH.
Breech presentation.
28
Ortolani positive.
Reducible.
29
Barlow positive.
Dislocatable.
30
This column intact with PAO.
Posterior.
31
Boys age maturity.
16
32
Female age maturity.
14
33
Perthes more common in boys or girls.
Boys.
34
Bilateral perthes in ___%.
12-15%.
35
Congital dislocation of knee associated with ____.
DDH.
36
Treat DDH or CDK first?
Treat knee dislocation first with casting.
37
For long bone fx, OI treated with.
IMN.
38
Medical treatment for OI.
Pamidronate.
39
CH17Q21.
Neurofibromatosis.
40
Achondroplasia physeal defect in this region of physis.
Proliferative zone.
41
DTD gene.
Diastrophic dwarfism.
42
Aperts syndrome gene mutation.
FGFR2.
43
FGFR3 encodes this type of protein.
Tyrosine kinase receptor.
44
Camptomelic dysplasia gene mutation.
Sox9.
45
Spondyloepiphyseal dysplasia gene mutation.
COL2A1
46
Multiple epiphyseal dysplasia type I mutation.
COMP.
47
Diastophic dysplasia gene mutation.
DTDST.
48
Pynknodysostosis gene mutation.
CTSK.
49
Gene product disrupted in pynknodysostosis.
Cathepsin K.
50
Urinary excretion in Morquio's syndrome.
Keratan sulfate.
51
Sprengel's deformity is a failure of this.
Descent.
52
Congenital pseudoarthrosis of the clavicle occurs in this clavicle.
Right side.
53
Congenital speudoarthrosis of the clavicle is associated with neurofibromatosis true or false.
FALSE.
54
This must be functional in radial deficiency to perform centralization.
Biceps.
55
Treatment of thumb hypoplasia dependent on this.
CMC stability.
56
Congential radioulnar synostosis is a failure of this.
Separation.
57
Fibrous dysplasia with soft tissue myxomas.
Mazabraud syndrome.
58
Polyostotic fibrous dysplasia and cafe au lait spots.
McCune-Albright syndrome.
59
Multiple non-ossifiying fibromas with cafe au lait skin patches.
Jaffe-Campanacci syndrome.
60
Penicillin mechanism of action.
Cell wall synthesis inhibitor.
61
Cephalosporin mechanism of action.
Cell wall synthesis inhibitor.
62
Vancomycin mechanism of action.
Cell wall synthesis inhibitor.
63
Aminoglycoside mechanism of action.
Protein synthesis inhibition (30S ribosome).
64
Clindamycin/erythromycin mechanism of action.
Protein synthesis inhibition (50S ribosome).
65
Tetracycline mechanism of action.
Protein synthesis inhibition (tRNA).
66
Rifampin mechanism of action.
RNA synthesis inhibition (RNA polymerase).
67
Cipro/quinolone mechanism of action.
Inhibit DNA gyrase.
68
Amphotericin mechanism of action.
Fungal cell membrane disruption.
69
Polymyxin mechanism of action.
Cell membrane disruptor.
70
Diastophic dysplasia site of physis defect.
Reserve zone.
71
Psuedoachondroplasia site of physis defect.
Reserve zone.
72
Multiple hereditary exostoses site of physis defect.
Proliferative zone.
73
Gigantism (HGH) site of physis action.
Proliferative zone.
74
Achondroplasia site of physis defect.
Proliferative zone.
75
Rickets physis defect site.
Provisional calcification zone fo hypertrophic zone of physis.
76
Site of physeal defect of endchondromas.
Hypertrophic zone.
77
Mucopolysacharide diseases site of physis defects.
Hypertrophic zone.
78
Zones of the physis from proximal to distal.
1. Reserve zone. 2. Proliferative zone 3. Hypertrophic zone 4. Primary spongiosa 5. Secondary spongiosa (metaphysis)
79
Three phases of hypertrophic zone of physis.
1. maturation zone 2. degenerative zone 3. provisional calcification zone
80
Non-renal SCFE occurs in this physeal zone.
Hypertrophic.
81
Renal SCFE occurs in this physeal zone.
Secondary spongiosa
82
Metaphyseal corner fractures occur in this physeal zone.
Primary spongiosa.
83
Scurvy has effects in this physeal zone.
Primary spongiosa.
84
Most abundant non-collagenous protein in bone matrix.
Osteocalcin.
85
Function of osteocalcin.
Promotes mineralization.
86
Osteocalcin is produced by these cells.
Osteoblasts.

ABOS Part I (16 decks)

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