Mouth&Esophagus Flashcards

(44 cards)

1
Q

Main types of Mandibular and maxilla fractures

A

Incisive/ rostral region
Interdental space
Caudal horizontal ramus
Vertical ramus

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2
Q

What causes fractures in the incisive and interdental space?

A

Thin bones and minimal soft tissue coverage
Direct trauma from other horses or objects

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3
Q

Why are fractures of the caudal horizontal ramus (molars/ temporomandibular joint) less common?

A

Greater bone and muscle mass (masseter muscle)
Higher morbidity

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4
Q

Acute clinical signs of mandibular and maxilla fractures

A

Displacements
Pytalism
Swelling
Prehensile difficulties
Dysphasia
Oral/ cutaneous wounds

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5
Q

Chronic clinical signs of mandibular and maxilla fractures

A

Debilitation
Difficult prehension
Malodorous breath
Loss of normal occlusal alignment of dental structures

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6
Q

Diagnosing mandibular and maxilla fractures

A

Clinical signs
Radiographs
Cross bite (cd. mandible, vertical ramus, temporomandibular joint fractures/ luxations)

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7
Q

Treatment for incisive fractures

A

Orthopedic wires
Interdigitate on reduction
Eat normal diet
Heal within 4-8 weeks

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8
Q

Treatments for interdental space fractures

A

Interdigated: orthopedic wires
Long oblique fractures: cortical screws

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9
Q

Treatments for unstable fractures of the interdental space

A

Acrylic splints
Dynamic compression plates
Kirschner- Ehmer appliances

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10
Q

Treatment for unilateral non-displaced ramification fractures

A

Non surgical
Soft diets
Analgesics (non steroidal anti inflammatory drugs)
4-6 weeks (good prog)

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11
Q

Where are caudal angle mandibles fractured?

A

From the junction of the vertical and horizontal ramus as a free fragment (uncommon)

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12
Q

Treatment of caudal angle mandible

A

Excision of fragments
With molar involvement: internal fixation with plates/ screws

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13
Q

Treatment for the temporomandibular area fracture

A

Rare and difficult to repair
Mandibular condylectomy

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14
Q

Treatment for the coronoid process

A

Excision (preferred if dental abnormalities result) or conservative management

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15
Q

Prognosis for mandibular and maxilla fractures

A

Incisive region/ interdental area: good
Horizontal and vertical rami: guarded to fair
Temporomandibular and coronoid: guarded

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16
Q

What are the predisposing causes of esophageal impaction (choke)

A

Communal feeding
Post exercise (after feeding)
Esophageal lesions
Dental problems

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17
Q

What is the most common site of esophageal obstruction?

A

Second cervical vertebra

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18
Q

Causes of esophageal impaction

A

Feeds, pellets, sugar beet pulp, wood shavings, rough hay

19
Q

Clinical signs of esophageal impaction

A

Food, mucous, saliva from nostrils
Retching
Nasal cough
Aspiration pneumonia

20
Q

How do you dx esophageal impaction?

A

CS
Radiographs (radiodense or solid fluid and gas proximal to site of impaction)
Endoscopy (obstruction and esophageal mucosa)

21
Q

When are contrast esophagrams necessary?

A

Chronic recurrent choke

22
Q

Treatment of esophageal impaction

A

Nasogastric tube
Tranquilization
Anesthesia and endotracheal intubation (lateral recumbency)

23
Q

How does nasogatric tube for impaction work?

A

Blow air into esophagus to induce secondary peristalsis then advance obstruction
If fails then sedate with xylazine

24
Q

What is considered a risky tx for impaction?

A

Esophagostomy
Longitudinal incision —> feed through incision —> 4-6w recovery

25
Aftercare of impaction
Confirm if entire esophageal lumen is patent with nasogastric tube Endoscopic evaluation Scurried diet for a few days (pelleted feeds, bran mashes)
26
Prognosis for esophageal impaction
Acute: good Chronic: guarded to good
27
Eophageal ulceration?
Any trauma , chemical or infectious origin Longitudinal scars expand well, circular scars don’t
28
What causes esophageal ulceration
Protraction esophageal impaction (acute, most common) External trauma to neck (chronic)
29
__________ and __________ are the most common causes of recurrent choke
Ulceration and stricture
30
Conservative tx for mild cases of esophageal ulceration
Slurries diet for a few days then pelleted feed and bran mashes Constriction worse at 30 days then normal function at 60
31
Surgical tx for esophageal ulceration
Esophagostomy/ Esophagplasty Esophagomyotomy Patch grafting Resection and anastomosis
32
Why isn’t resection and anastomosis the best option for tx of esophageal ulceration?
Stricture Doesn’t hold together (lacks serosa) Long
33
Esophageal wounds
Longitudinal slits along the ventral and dorsal esophageal walls Closed and open wounds
34
Why are closed esophageal wounds more dangerous?
Secretions and food material trapped in deep fascial planes
35
What causes esophageal wounds?
Closed: ingested wire, impacted foreign bodies, pre-existing defects Open: blunt trauma (cervical vertebrae)
36
CS of open esophageal wounds
Swollen wound Salivary discharge Food drainage
37
CS of closed esophageal wounds
Cellulitis causing ventral neck swelling Anorexia Febrile and depression Shock from toxic products that accumulate in tissues
38
What causes mediastinitis and pleuritis in closed esophageal wounds?
Cellulitis and necrosis in the neck —> causing infection and gas dissect along fascial planes to mediastinum
39
Dx of esophageal wounds
Acute swelling ventral neck SQ emphysema over neck and shoulders Endoscopy Rads (gas through facial planes and extra luminal food)
40
Conservative tx for esophageal wounds
Good ventral drainage: esophagostomy and excise necrotic tissue Second intention healing Abx and anti- inflammatories Tetanus prophylaxis 3-4 weeks healing
41
What does saliva lost via wounds and fistulas causes?
Hyponatremia, hypochloremia Metabolic acidosis then metabolic alkalosis
42
Sx tx for esophageal wounds
Resection and anastomosis Esophagostomy
43
Sx complications from esophageal wounds
Esophagus has no serosa surface Constant movement Neurological complications
44
What results from esophageal wounds?
Cellulitis Laryngeal hemiplegia (left recurrent laryngeal nerve) Horners syndrome (ptosis, miosis, enophyhalmos) Chronic esophagocutaneous fistula Strictures are rare