Movement disorders intro Flashcards

(32 cards)

1
Q

common features of disordered movement

A

impaired postural refelxes that would normally contribute to balance - diminished or slowed movement(hypokinesia/bradykinesia) - excessive involuntary movements (hyperkinesia) -uncoordinated or unsteady movements (ataxia)

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2
Q

Physiological(nonpathological tremor)

A

refelcts low-amplitude oscillatory movement of a , bodily region

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3
Q

Resting tremor

A

common in parkinsons, wanes during voluntary movement of the affected body part, a BASAL GANLIAR dysfunction secondary to mesencephalic degeneration is integral to parkinson’s disease.

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4
Q

Intention (kinetic) termor

A

arises with voluntary movement particularly as the affected body part approaches a target in space (commonly cerebellar lesions yield this)

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5
Q

Chorea

A

brief, purposeless, irregular jerky movements of body parts,(often from Basal gangliar disease. -voluntary movements may be affected as abnormal choreic movements are superimposed. -successive choreic movements resembles dancing. -HALLMARK of Huntington disease and sydenham disease.

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6
Q

athetosis

A

continuous slow writhing body parts, (Often Basal Gangliar) may be seen in athetoid cerebral palsy -can accompany hemiplagia(gate is superimposed.

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7
Q

Ballismus

A

flinging or rotary movements, causal lesions often involve the subthalamus which contributes to basal gangliar function - is expressed unilaterallly with manifestations expressed contralateral to the injury

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8
Q

Lower motor neurons can present hypokinetic disorders presents as

A

flaccid paralysis

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9
Q

hypokinesia is asssociated with

A

upper motor neurons

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10
Q

hypokinesia yields

A

elevated muscular tone and hyperreflexia

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11
Q

dysfunctions involving modulatory and regulator circuits can also produce

A

hypokinesia

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12
Q

sensory and motor systems cooperate to

A

mantian equilibirum

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13
Q

posture is immediately linked to

A

muscel tone which can be adversely affected by a broad array of lesions

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14
Q

aberrations of posturecan

A

reflect tonically increased or decreased muscle tone.

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15
Q

damage to modulatory centers can

A

destabilize muscle tone yielding spontaneous and uncontrolled movements.

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16
Q

Decorticat posture suggests

A

a cereberal lesion affecting the corcio spinal system with sparing of motor centers of the brain stem. including the red nucleus

17
Q

station is sensitive to

A

damage to conscious and unconscious sensory systems and motor systems.

18
Q

ability of patient to stnad steady with feet together may deteriorate when

A

the eyes are close and the visual system isn’t able to compensate for nervous deficits.

19
Q

postural tremor

A

apparent only when a particular posture is maintained may reflect basal gangliar dysfunction

20
Q

Prakinson disease

A

-akinesia/bradykinesia, -facial masking -muscular rigidity -loss of postural refelxes -parkinsonian gait -loss of habituiation to glabellar stimulation -speech is quiet, hoarse and monotonous with accelerated bursts

21
Q

stages of parkinson

A
  1. unilateral 2. bilateral but with preserves postural reflexes, 3. bilateral with loss of postural refelxes 4. severe disability with some movement 5. Akinesia
22
Q

Prodrome

A

hyposomia, autonomic disturbances and REM sleep disorder. reflects degeneration in olfactory and cuadal bulbar centers

23
Q

Cogwhell rigidity

A

recurrent stepwise release and return of resistance to passive movement

24
Q

lead pipe rigidity

A

resistance that is sustained throught a range of passive movement.

25
glabellar reflex involves
noxious stimulation between the eyes to elicit bilateral blinking, normally blinks weaken with repeated stimulation
26
Dystonia
muscle spasms or sustained abnormal postures involving trunk and limbs -post are partial or segmental.
27
spasmodic torticollis
a specific dystonia involving the neck, they contract involuntarily and may become hypertrophic.
28
Tic Syndromes
irregularly occuring sterotyped movements that are usually transient and uncoordinated.
29
tourette syndrome.
multifocal tics, ADHD, vocal tic.
30
Tardive dyskinesia
patients exposed to antipsychotic. oral movements, doesn't necessarly abate with drug withdrawal
31
Dopa induced dyskinesia
often arises during treatment of parkinson disease with precursor to dopamine. -choreic movemnts, facial dystonias
32
drug induced parkinsonism.
suppresion of dopaminergic transmission. may dissipate within weeks