Normally asymptomatic With increasing size, pain, swelling and exudate may oocur

Well-defined, smooth, corticated borders Thinning and mild expansion with occasional perforation of cortical plates Displacement of teeth only occasional root resorption (< dentigerous and < radicular cysts) mild B-Li expansion; but extensive antero- posterior extension

Multiloc Radio Flashcards by sophie rosen

skipped
Multilocular Radiolucencies
(7)

Ameloblastoma
Odontogenic Keratocyst
Central Giant Cell Granuloma
Odontogenic Myxoma
Vascular Lesions - Hemangioma
. A-V aneurysm
Familial Fibrous Dysplasia

How well did you know this?

Not at all

Perfectly

Ameloblastoma
(2)

Benign odontogenic neoplasm; one of very few true odontogenic neoplasms
Capable of uncontrolled, unlimited
growth potential

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Classified into:
(2)

Conventional (Multicystic)
Ameloblastoma
Unicystic Ameloblastoma

How well did you know this?

Not at all

Perfectly

Conventional (Multicystic)
Ameloblastoma
* Account for —% of all ameloblastomas
* — histologic sub-types;

85 – 90
Five

How well did you know this?

Not at all

Perfectly

Five histologic sub-types;

follicular - most common
also have plexiform, acanthomatous,
granular, desmoplastic and basaloid

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Conventional
(3)

Usually slow painless swellings
Small lesions only detected by
radiographs
Larger lesions detected clinically

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Conventional
Radiographic
(4)

Small lesions are
unilocular with
corticated borders
Large, aggressive
lesions develop
multilocular patterns
Displace and resorb
teeth
expansive

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Conventional
Age
Site
Gender Predilection

Mainly adults – equal prevalence in 3’rd to 7’th decade
Uncommon in 2’nd decade
Rare in 1’st decade

mandible (85%); maxilla (15%)

none

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Management

Other

Large lesions are aggressive requiring bone
resection

Higher likelihood for recurrence.

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Management

Other

Block or marginal resection; ie resect >1.0cm
past radiographic limits of tumor
15% recurrence

50-90% recurrence if not resected
Rare to be malignant

How well did you know this?

Not at all

Perfectly

Unicystic Ameloblastoma
(3)

arise within a cyst lining; either luminal,
intraluminal or mural
less aggressive form of ameloblastoma
Recurrence rates of 10-20%

How well did you know this?

Not at all

Perfectly

Ameloblastoma
Radiographic
(1)

expansive

How well did you know this?

Not at all

Perfectly

Ameloblastoma
unicystic
Age
Site
Mandible (–%)
Maxilla (–%)

Mean age 23 years

90, 10

How well did you know this?

Not at all

Perfectly

Odontogenic Keratocyst
Pathophysiology
(3)

10-12% of all odontogenic cysts; 3’rd
highest oral cyst frequency
aggressive cysts; behave more like
benign neoplasms
thought to arise from cell rests of dental
lamina

How well did you know this?

Not at all

Perfectly

OKC
Clinical
(2)

Normally asymptomatic
With increasing size, pain, swelling and
exudate may oocur

How well did you know this?

Not at all

Perfectly

OKC
Radiographic
(5)

Well-defined, smooth,
corticated borders
Thinning and mild
expansion with
occasional perforation
of cortical plates
Displacement of teeth
*only occasional
root resorption
(< dentigerous and
< radicular cysts)
*mild B-Li
expansion; but
extensive antero-
posterior extension

Odontogenic Keratocyst
Age
Site
Gender

Majority (i.e., 60%) in 2nd and 4th decade

Majority (60-80%) affect mandible posterior to the
canines

Male predilection

Odontogenic Keratocyst
Management
Other

Enucleation with curettage

High recurrence rate; some rates
reported @ 47 and 62% (probably
parakeratinized variants)

Odontogenic Keratocyst
Other
* When multiple OKCs are found they
may constitute part of the

basal cell
nevus syndrome (a.k.a. nevoid basal
cell carcinoma syndrome)

Nevoid basal cell carcinoma syndrome
(6)

multiple basal CA’s of skin
Palmar and plantar pitting (60%)
> 1 OKC (KOT) of jaws (75%)
Bifid or splayed ribs (60-75%)
Kyphoscoliosis (50%)
Skull anomalies:

Skull anomalies:
(4)

frontal and parietal bossing
hypertelorism
intracranial calcifications;
majority are of falx cerebri

Central Giant Cell
Granuloma
Clinical
(2)

Asymptomatic swelling
Can be aggressive

Central Giant Cell
Granuloma
Radiographic
(4)

Well-defined
borders
Can be multilocular
Thinning and
expansion of cortical
plates
Displacement of
teeth and occasional
root resorption

Central Giant Cell
Granuloma
Age
Site
Gender

Usually < 30 years (60%)

mandible (70%) & frequently between the
molars

Female > male (2:1)

Central Giant Cell Granuloma Management Other

Enucleation with aggressive curretage * Lesion is histologically similar to the Brown tumor of primary hyperparathyroidism

Central Giant Cell Granuloma Other

* Patients with the CGCG should be evaluated to rule out hyperparathyroidism

Screening test in the appropriate blood studies: – serum calcium – alkaline phosphatase – serum phosphorus

increase increase decrease

Odontogenic Myxoma Clinical (2)

* Primarily a lesion of alveolar bone * Basically a fibrous lesion

Odontogenic Myxoma Radiographic

Scalloped and multilocular

Odontogenic Myxoma Age Site

Young to adults (25 – 30 years) Greater prevalence in mandible

Odontogenic Myxoma Management Other

Excision Recurrence up to 25% because the lesions are not encapsulated.

Central Hemangioma Clinical * --- are next most common site after skull and vertebrae * Firm, slow-growing --- expansion * Overlying mucosa is more * Spontaneous * --- on diascopy and --- sensation may be detected * Many require --- to assist diagnosis

Jaws asymmetric erythematous and warm to touch gingival bleeding Bruit, pulsatile needle aspiration

Central Hemangioma Radiographic (2)

* Variable pattern ranging from cyst- like radiolucencies * May have multilocular “soap- bubble” or spoke- like appearance

Central Hemangioma Age Site Sex

Teens and young adults Posterior mandible Female:male 2:1

Central Hemangioma Management (2) Other

* Sclerosing agents, radiation, enucleation * Embolization of major arteries necessary prior to surgery as hemorrhage is a significant and life- threatening complication Etiology is either traumatic/developmental or benign neoplasm

Cherubism Clinical (3)

* Cherubic looking face by 5 years of age due to bilateral bony expansion * Asymptomatic * Bone lesions are more active in younger patients. After age 12, activity usually diminishes and finally lesions become inactive with residual deformity by age 30

Cherubism Radiographic (2)

* Bilateral multilocular cyst-like, expansile lesion, usually affecting the mandible and sometimes the maxilla * Pathologic fracture is not a feature

Cherubism Age Site Sex

Usually detected by age 5 Bilateral mandible, may affect maxilla Male:female 2:1

Cherubism Management Other

Cosmetic osseous contouring at age 12 and later Benign self-limiting condition