Musculoskeletal 1 Flashcards
(51 cards)
Commonly missed fractures
- Stress fracture
- Scaphoid, elbow, radial head, midfoot, tibial plateau, hip; supracondylar and torus fractures in kids
- Non-displaced and impacted fractures
- Non-displaced growth plate fractures
- Suspect fracture but negative xray? (Dx: Probable fx. Immobilize, Repeat xray in 2-3 weeks – look for callous, CT, MRI or bone scan next)
Generalized decreased density = osteopenia
Diffuse lucency DDx: (think global process)
Metabolic/Endocrine
- Osteoporosis (late)
- Hyperparathyroidism
Malignancy
- Metastases
- Lung, renal, thyroid, breast CA
Regional
-Reflex Sympathetic Dystrophy Synd (RSDS)
fractures: general concepts
- Radiographs provide confirmation (Hx (mechanism), PE, PMHx, age are key)
- Know anatomy, know views (Order what you need, shoot it once)
- Confirm all “suspected” fractures on >1 view
- Lots of overlap - account for and mentally subtract overlying structures - all views
CPPD arthropathy
- chrondrocalcinosis
- psuedogout
Myositis Ossificans
- Extra-skeletal ossification in soft tissue
- After blunt trauma
- Common in thigh
- Imaging: Xray, CT, MRI
septic arthritis - non-pyogenic
- M. tuberculosis, fungus
- Risks: IVDU, DM, steroids, TB risks
- Indolent, slow
- Monoarticular
- Osteoporosis common
- Spine, knee common
- Red, hot, painful joint. Plain film changes are late, but order one first.
- May see subchondral erosions, periosteal reaction (above)
Bones: decreased density (increased lucency)
Lucent (black) line: suspect fracture
Generalized lucency - Osteopenia
- Osteoporosis
- Endocrine/metabolic disorders, steroids
- Hyperparathyroidism, osteomalacia, rickets
- Multiple Myeloma (disseminated form)
Focal lucency
- Osteolytic metastases, bone cysts, some tumors
- Multiple Myeloma (solitary form)
- Osteomyelitis
Open fracture (compound fx)
- High incidence of infection, osteomyelitis
- Crush injury: vascular complications, infection
- Irrigated and repaired in OR < 8 hrs
- Antibiotics, Tetanus
- Open tuft fx (exception to OR repair)
- Needs to be within 8 hrs!! EVERYONE GETS ABX AND TETANUS
- The only exception is tuft fx
bone tumors
- Where? Joint violated? (tumors don’t cross joints)
- Margin? Sclerotic/thin, well-defined vs. irregular, ragged
- Shape? Longer than wide (w/in medullary) or wider than long (burst through cortex)?
- Characteristics? Geographic, permeative, “moth-eaten”, expansile, “soap bubbly”
- Bony reaction? Lytic, sclerotic
- Periosteal reaction? Codman’s, mixed, onion skin
- Tumors dont cross joints! they stay in the bones they originated in
Bone scans - nuclear med
Indications:
- Occult Fractures
- Stress Fractures
- Bone Infection
- Avascular Necrosis
- Osteomyelitis
- Malignancy
erosive arthritis: gout
- Calcium urate crystals
- Sharp, sclerotic “punched out” or “rat bite” erosions near affected joint
- Acute monoarticular arthritis grt toe: Podagra
- Severe = tophi, periarticular erosions, “punched-out” lesions
- Tophi = soft tissue
- She will not require us to tell apart gout from rheumatoid etc. ON A PICTURE → will need to know the characteristics of them
CT scan: indications
- Complex, intra-articular fx’s (Characteristics, extent)
- Calcaneus, tibial plateau, talus, midfoot
- Spinal column
- Pre-op evaluation
- Occult fracture
- Associated injuries
- Tumors, infection
- Biopsy, interventional procedures
erosive arthritis: psoriatic
- At DIP, spares PIP
- Subchondral erosions
- Terminal phalanges narrow “pencil in cup”
Cartilage (joint space)
- Refers to joint spaces
- Normal appearance on xray views is key
- Widening: disruption, calcification, fluid
- Check if fracture extends into the joint (Intra-articular Fx)
- MRI best for ligamentous injury, disruption
- The joint space should be the same all the way around
- Intraarticular fracture means that the fracture extends into the joint space
- Decreased (Arthritis - most types, Impacted fx, dislocation)
- Increased (Fracture, dislocation, Hemarthrosis, Infection (pus))
- Chondrocalcinosis (Pseudogout, CPPD)
- Hemarthrosis – blood in the joint
Intra articular fractures
- Fracture enters joint
- Important distinction
- Cartilage damage
- Ligamentous injury
- Joint is now at risk for degenerative arthritis
Bones
- Check entire cortex, contour; size, shape of bones; check alignment
- Fractures (Lucent (black) line passes through cortex, Check entire cortical margin for disruption, Check for impaction (bulge, increased density), Acute Fx’s linear, jagged - edges not corticated, Fx’s should be visible on more than one view)
- Decreased density (lucency, osteopenia)?
- Increased density (opaque, sclerosis, impaction)? Generalized process or focal process?
- Break and fracture are the same thing!
- Contour is how the bone should look
- Torus fx is a buckle fracture → compression fracture
- FRACTURES ARE BLACK ON PLAIN FILM
- If you see something that looks white, its not a fracture
Special views: plain xrays
- To highlight particular bones, angles, relationships, non-displaced fx’s, dislocation
- Confirm a questionable abnormality
- “Comparison views” - other side
- “Weight-bearing view” - AC joint, foot
- Perpendicular - axial plane (Patella, calcaneus, shoulder, wrist)
hyperparathyroidism
- Generalized decreased bone density
- Overproduction of PTH (>PTH-> hypercalcemia (plasma), Increase in bone resorption, Stones, bones, abdominal groans)
- Osteoporosis
- Subperiosteal resorption of bone in fingers
- Subchondral resorption in distal clavicles
- “brown tumors”, “salt & pepper skull”
- Brown tumors: not really tumors or malignant. They are radiolucent areas of increased osteoclast activity from abnormal bone metabolism. Rare – associated with hyperparathyroidism
Pagets Disease
- Chronic inflammatory remodeling of bone
- 3 Phases: Early lytic, Mixed, Osteoblastic (Dense, sclerotic bone changes late)
- Pelvis, skull, spine, tibia (spares fibula)
- Thickening of cortex
- Thick, sclerotic traebeculum
- Bone gets bigger overall (“hat don’t fit”)
- Pagets disease: bones get hairy looking! Scalp gets much thicker; IT SPARES THE FIBULA (generally)
Avulsion fracture
-Avulsion fx’s are caused by sudden torque on the ligaments. The ligament stays intact but pulls a piece of bone away. If small piece of bone pulled off, commonly called a “chip fracture”
Pathologic fractures
Fracture occurs because bone is weakened by underlying process:
- Bone tumor
- Bone cyst
- Metastasis
- Lytic or sclerotic changes
erosive arthritis: rheumatoid arthritis
- Narrowed joint spaces
- Periarticular erosions
- Osteopenia
- Subluxation
- Radiocarpal erosion
- Ulnar deviation
Osteoblasts
- Bone (matrix) formers: increase density, sclerotic appearance
- Reparative: heals fx’s,
- Reactive: produce bone in cortex (periosteal lesions) or medullary cavity
Osteoclasts
- Bone (matrix) resorbers: remove, destroy: decrease density, lucent appearance
- Can remove bone at 20x rate of formation
- “make room” for tumors, infection