Neuro Flashcards

(84 cards)

1
Q

most important diagnostic tool in neuro

A

history

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2
Q

what test can see the posterior fossa bets?

A

MRI

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3
Q

what is a good eval for hydro, hemorrhage, gross structures, calcification

A

US

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4
Q

when do HAs typically begin normally?

A

middle school aged

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5
Q

when do you do a CT/ MRI for HA in a kid

A

Concern about sub-arachnoid, subdural hematoma

Concern about increased IC pressure or hemorrhage

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6
Q

Red flags for HA

A
fails to respond to tx
focal neurological findings (first 2-6 months)
progressive frequency/ severity
awake from sleep, worse in morning
AM vomiting
at risk hx or condition
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7
Q

What are some focal neurologic findings

A
CN VI palsy
diplopia 
new onset strabismus
papilledema
hemiparesis
ataxia
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8
Q

can you have photophobia or phonophobia w/ a tension type HA?

A

Yes, but usually only have 1

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9
Q

Constant, aching, tight

Occipital, frontal or constricting band around head

A

Tension type HA

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10
Q

chronic tension HA is often a sign of what

A

depression and/or anxiety

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11
Q

Severe, pulsatile (pounding)
unilateral, can be bilateral
Frontal or temporal regions, retro orbital or cheek

A

Migraine hA

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12
Q

what may be the only symptoms of migraine in a kid

A

vomiting

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13
Q

tx for migraine

A
Ibuprofen APAP early
caffiene + ergot 
triptans and DHE 
rest and quiet 
avoid opioids
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14
Q

Migraine prevention

A
TCA
beta blockers (propranolol) 
Calcium channel blockers (verapamil)
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15
Q

A sudden, transient disturbance of brain function manifested by involuntary motor, sensory, autonomic, or psychic phenomena

A

Seizure

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16
Q

2 or more seizures not provoked by particular event or cause.

A

Epilepsy

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17
Q

a benign condition of childhood with unilateral focal seizures and speech abnormalities, often hereditary.

A

Rolandic epilepsy

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18
Q

when should you do studies w/ a migraine

A

worse on awakening
awakens pt
worse with cough or bending over

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19
Q

Seizure > 30 min

Sequential seizures without regain LOC > 30min

A

Status epilepticus

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20
Q

Onset of seizure begins in one area of one cerebral hemisphere (apparent clinically or via the EEG) with no LOC

A

simple focal seizure

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21
Q

Onset of seizure begins in one area of one cerebral hemisphere (apparent clinically or via the EEG) with altered LOC (staring)

A

complex focal seizure

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22
Q

a simple or complex partial seizure that ends in a generalized convulsion

A

secondary generalized seizure

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23
Q

Seizures arise from both hemispheres, simultaneously

A

generalized seizure

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24
Q

unique to kids 6 month- 6 years. happens with changing temp. doesn’t tend to cause damage or increase risk of epilepsy

A

febrile seizure

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25
if a second seizure is going to occur, when is it likely?
within 6 months after 1st seizure
26
if a second seizure is going to occur, when is it likely?
within 6 months after 1st seizure
27
what type seizures are frequently associated w/ underlying structural brain dz?
generalized seizures
28
what type of partial seizure may have an aura/ automatisms?
Complex partial
29
what type seizure will a patient be confused for generalized?
generalized tonic-clonic
30
symptoms in infants that occur in clusters when drowsy. Have severely abnormal EEG pattern. Due to brian injury at birth and are hard to control.
Infantile spasm (west syndrome)
31
if a patient has infantile spasms with no hx of birth problems or MRI or meatbolic origin is there a better or worse outcome.
Better
32
usually 3-13 years old. Normal IQ and MRI. will have twitching and tingling when awake and grand mal when asleep. often runs in families
Benign rolandic epilepsy (benign focal epilepsy of childhood)
33
when do you Tx for benign rolandic epilepsy
only if seizures are frequent, problem in school or anxiety
34
effective txs for benign rolandic epilspey
avoidance of sleep deprivatoin carbamazepine, oxcarbamazepine will usually outgrow
35
when is status epilepticus common
children under 5, especially <12 months
36
can status epilepticus be the first seizures?
yes, often happens
37
most common cause of status epilepticus in children
Infection and metabolic disorders
38
too much potassium can lead to what?
arrhythmia
39
do you tx febrile szs?
no
40
what is a complex febrile seizure?
one side of body shaking, staring prolonged (>15-20 minutes) multiple in 24 hours more likely to lead to epilepsy
41
If you do have to tx a febrile seizure what do you give?
rectal diazepam
42
can syncope have rhythmic jerking?
Yes, can have some
43
can syncope have rhythmic jerking?
Yes, can have some
44
Transient LOC and postural tone due to cerebral ischemia or anoxia.
syncope
45
can syncope have a prodrome?
Yes Dizziness, lightheadedness, nausea, sweating, pallor
46
Neurally mediated Transient hypotension from vasodilation and/or decreased heart rate Arousal 1-2 min up to 1h
Vaso-vagal or neurocardiogenic syncope
47
tx for syncope
Direct management of any cardiac cause | Reassurance and avoidance of triggers for vaso-vagal stimulation
48
causes of increased ICP
Cerebral edema | Mass lesion
49
children will have HA, diplopia/ strabismus, papilledema, herniation syndromes
increased ICP
50
management for hydrocephalus caused by obstruction that will get better as kid ages
shunt that drains into peritoneal cavity
51
where is pain with supratentorial elevated ICP
eye forehead temple
52
where pain with infratentorial increased ICP
occiput | neck
53
when are HA from elevated ICp worse
morning awakening standing up at night
54
Increased intracranial pressure without identifiable mass or hydrocephalus. Obese teenage girl with HA tinnitus, papilledema, visual loss. normal MRI>
pseudotumor cerebri
55
most common causes or childhood stroke
cyanotic heart dz sickle cell anemia meningitis hypercoaguable state
56
symptoms of childhood stroke
hemiplegia, unilateral weakness, seizures.
57
most common cause of concussion in children
fall
58
Brief loss of consciousness or stunned for minutes to hours. Amnesia is common and transient
Head Injury -Concussion
59
what kids with a concussion should be evaluated
All children with amnesia or who were unconscious should be evaluated in ER.
60
what will a meningomyelocele often have on prenatal screen?
elevated alpha fetoprotein
61
displaced cerebellum through foramen magnum into spinal canal – posterior laminectomy. Sx will hve progressive ataxia or vertigo. low lying tonsils alone
Arnold Chiari I
62
displaced cerebellum plus meningomyelocele – surgical repair and shunt. low lying tonsils + hydrocephalus
Arnold Chiari II
63
occipital encephalocele – surgical repair | Hydrocephalus common
Arnold Chiari III (anencephaly)
64
smooth brain Severe delay, seizures. Associated with syndromes Most common disorder of neuronal migration
Lissencephaly
65
Premature closure of sutures | Sporadic and idiopathic
Craniosynostosis
66
what type Craniosynostosis will have a short wide head (brachycephalic)
coronal sutures
67
Tx for craniosynostosis
Treatment is surgical excision of fused suture line – best done prior to 6 months of age
68
Autosomal Dominant | Café au lait spots - >6 of 5mm prepubertal pt
Neurofibromatosis
69
AD, rare , characterized by benign tumors in vital organs such as the brain, eyes, kidneys, heart and skin. will have ash leaf spots, Adenoma Sebaceum Shagreen Patch
Tuberous sclerosis
70
Diagnostics for tuberous sclerosis
CT for calcified nodules | MRI for demyelination
71
Unilateral port wine stain over upper face | Follows cranial nerve V. Can have leptomeningeal vascular anomaly and calcifications can lead to seizures.
Sturge Weber
72
Impairment of coordination and balance of voluntary movement | Cerebellar problem
ataxia
73
most common cause of ataxia in children
post-infectious or drug intoxication
74
Sudden onset of ataxia,staggering, frequent falls Nystagmus, vomiting, irritaility, lethargy possible Sensory and reflexes preserved No evidence increased ICP will recover in 1-4 weeks
post infectious acute cerebellar ataxia
75
do you need to tx post infectious acute cerebellar ataxai
No, most get between in 1-4 weeks by themselves
76
kids uses hands to walk up legs to standing position
Gower sign
77
what are 2 disorders of the anterior horn cells?
Spinal muscular atrophy (SMA) | poliomyelitis
78
If you have a lower motor neuron lesions will be it be high tone or low tone?
low tone
79
Neuromucular junction problem
Myasthenia gravis
80
muscle weakness, which may manifest with floppiness, delayed motor milestones, unsteady gait or muscle fatiguability. Progressive with normal mental development but body fails.
SMA (spinal muscular atrophy)
81
>10 years old, ophthalmoplegia and ptosis, loss of facial expression and difficulty chewing
Juvenile myasthenia gravis
82
Similar to Duchenne except later, less severe | Death usually in adulthood
Becker muscular dystrophy
83
``` X-Linked recessive Onset at 2-6 yrs Proximal muscles affected before distal Waddling gait, difficulty with stairs pseudohypertrophic calf Gower Sign – pelvic weakness Elevated CPK 75% mortality by age 20 ```
Duchenne muscular dystrophy
84
what is used to tx spastic cerebral palsy?
botox