neuroopthalmology Flashcards

(44 cards)

1
Q

what are the muscles of thee upper lid ?

A

superior tarsal
levator palpebral superiors

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2
Q

what is the main retractor of the lower lid ?

A

the inferior rectus

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3
Q

what is the nerve supply of the inferior rectus and the levator muscles ?

A

occulomotor

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4
Q

what is lagopthalmus ?

A

failure to close the eyes due to facial paralysis

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5
Q

what is the most common complication of the cornea that happens as a consequence of lagopthalmos ?

A

exposure keratitis

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6
Q

what are the different causes of ptosis ?

A

congenital
paralytic
neuromuscular
involutional
mechanical

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7
Q

what is the etiology of myasthenia gravis ?

A

formation of auto antibodies against acetylcholine receptors in the neuromuscular junction

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8
Q

what are the clinical tests that can bee used to help in the diagnosis of myasthenia gravis ?

A

increased ptosis with upwards gaze
improved ptosis with icee packs to the lids
IV injection of edrophonium causes rapid improvement of ptosis

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9
Q

what is the mediical therpay for myasthenia gravis ?

A

pyridostigmine
oral steriods

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10
Q

what is the surgical therapy associated with myasthenia gravis ?

A

removal of the thymus gland

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11
Q

what are the causes of paralytic strabismus ?

A

lesions of the motor nerve nucleus
lesions of the nerve trunk
lesions of the muscles

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12
Q

what is paralytic strabismus ?

A

strabismus caused by the affection of the extra occular muscles

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13
Q

what is the nerve supply of the extra occular muscles ?

A

CN3 supplies sphincter pupillae and ciliary muscles
supplies the medial , superior and inferior rectus, inferior and superior oblique
CN4 supplies the superior oblique
CN6 supplies the lateral rectus

all extra occular muscles ar supplied by the occulomottor nerve except for the lateral rectus and supeerrior oblique

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14
Q

what are thee compensatory mechanisms for diplopia ?

A
  1. suppression - more common in children and easier with them due to high brain plasticity
  2. abnormal head posture
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15
Q

suppression as a compensatory mechanism for diplopia increases the risk for ?

A

Amblyopia ( lazy eye)

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16
Q

what is the clinical picture of 3rd CN palsy ?

A

the eye will bee looking downwards and outwards
pupils will be dilated and fixed
lid ptosis
accommodation is completely lost
complete ptosis ? no diplopia

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17
Q

what is the most common cause of isolated CN3 palsy ?

A

posterior communicating artery aneurysm

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18
Q

what is the clinical picture of CN4 palsy ?

A

the head would be tilted away from the lesion to reduce the diplopia
affected eye is deviated upwards
ipsilateral hypertropia

19
Q

what is the function of the superior oblique muscle ?

A

depression
intorsion

20
Q

what is the clinical picture of CN6 palsy ?

A
  • Limitation of abduction with esotropia
21
Q

what are the causes of CN6 palsy ?

A

any increase in intracranial pressure
microvascular disease due to HTN or DM

22
Q

what are the causes of CN6 palsy in children ?

A

secondary to severe otitis media
trauma
post viral disease

23
Q

where is the lesion if RAPD is positive ?

A

asymmetrical lesion that exists before the lateral geniculate body

24
Q

what is the presentation in RAPD ?

A

shine light in normal eye causes constriction in both pupils
shine light in diseased eye then both pupils dilate

25
lesion of the visual field at the optic nerve ?
left or right anopia
26
lesiion of the visual field at the optic chiasm ?
bitemporal hemianopia
27
lesion of the visual field after the optic chiasm ?
homonymous hemianopia ( right or left )
28
lesion of the visual field in the optic tract ?
quandrantopias
29
when should diseease of the optic nerve be suspected ?
vision loss over hours or days visual field loss color vision loss headache or pain pain upon eye movement
30
what is papilledema ?
passive non inflammatory swelling of the optic nerve head (optic disc)
31
case of bilateral papiloedema differential diagnosis ?
intracranial space occupying lesion until proven otherwise
32
what systemic diseases may be associated with papilledema ?
malignant hypertension polycythemia anemia
33
what are thee symptoms of early papilledema ?
may bee asymptomatic amaurosis headache , vomiting due to increased ICP may experience diplopia due to CN6 affection
33
what are the symptoms of early papiloedema ?
may bee asymptomatic amaurosis headache , vomiting due to increased ICP may experience diplopia due to CN6 affection
34
what is the presentation of IIH ?
idiopathic intracranial hypertension is a diagnosis of exclusion , common in young females who are obese and other causes of inc ICP have been excluded always present with papilloedmea
35
what is the differential diagnosis of IHH ?
sleep apnea
36
what is the presentation of a late case of papiloedema ?
gradual painless loss of vision due to secondary optic atrophy
37
what are the investigations required for cases of papilledema ?
CT scan MRI MRV lumabr puncture
38
what are the complications of papilledema ?
post papilledema optic atrophy the optic disc becomes flat and greyish in colour with ill defined borders obscured lamina cribrosa
39
what is the treatment for papilledema ?
treatment of the cause decrease ICP pressure carbonic anhydrase drops weight loss surgery is indicated
40
what surgery is performed in cases of papiloedema ?
optic nerve sheath fenestration CSF shunting procedures
41
what is the difference between papillitis and retrobulbar optic neuritis ?
papillitis - inflammation of the optic nerve head retrobulbar optic neuritis - inflammation of thee optic nerve along with the orbital part of the nerve
42
what is the etiology of optic neuritis ?
infective disease demyelinating disease inflammatory conditions Uveitis and retinitis may cause secondary papillitis
43
what are the signs associated with optic neuritis ?
pain on ocular movement RAPD can be detected fundus examination is normal