OSCE practise Flashcards

1
Q

Adrenaline amount for anaphylaxis?

A

1 in 1000, 5 mg

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2
Q

paeds question I always forget to ask?

A

are they hitting all of their developmental milestones

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3
Q

what makes a febrile seizure complex?

A

when they consist of partial or focal seizures,
last more than 15 minutes
or occur multiple times during the same febrile illness.

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4
Q

risk of epilepsy after febrile seizure

A

Children who have had a febrile seizure have a slightly increased chance of having epilepsy later in life.

1 in 100 - 1 % extra chance

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5
Q

what is coeliacs disease?

A

an autoimmune condition triggered by eating gluten.
It can develop at any age

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6
Q

blood tests for coeliacs

A

total immunoglobulin A levels
anti-TTG and anti-EMA antibodies

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7
Q

what happens in coeliacs if u continue eating gluten even in tiny amounts?

A

It can lead to:

Nutritional deficiencies
Anaemia
Osteoporosis
Hyposplenism (with immunodeficiency, particularly to encapsulated bacteria such as Streptococcus pneumoniae)
Ulcerative jejunitis
Enteropathy-associated T-cell lymphoma (EATL)
Non-Hodgkin lymphoma
Small bowel adenocarcinoma

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8
Q

food with gluten

A

oats, bread, pasta, cereal, and pizza.

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9
Q

GORD managment babies

A

In simple cases some explanation, reassurance and practical advice is all that is needed. Advise:

Small, frequent meals
Burping regularly to help milk settle
Not over-feeding
Keep the baby upright after feeding (i.e. not lying flat)

More problematic cases can justify treatment with

Gaviscon mixed with feeds
Thickened milk or formula (specific anti-reflux formulas are available)
Proton pump inhibitors (e.g., omeprazole) where other methods are inadequate

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10
Q

when does pyloric stenosis present

A

first few weeks of life, with a hungry baby that is thin, pale and generally failing to thrive.

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11
Q

investigation and management of pyloric stenosis

A

Diagnosis is made using an abdominal ultrasound to visualise the thickened pylorus.

Treatment involves a laparoscopic pyloromyotomy (known as “Ramstedt’s operation“). Prognosis is excellent following the operation.

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12
Q

what is the criteria for giving ultrasounds for children with UTIs

A

All children under 6 months with their first UTI should have an abdominal ultrasound within 6 weeks, or during the illness if there are recurrent UTIs or atypical bacteria

Children with recurrent UTIs should have an abdominal ultrasound within 6 weeks

Children with atypical UTIs should have an abdominal ultrasound during the illness

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13
Q

what structural abnormality predisposes children to UTIs

A

Vesicoureteric reflux

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14
Q

investigation for children with Vesicoureteric reflux

A

Micturating Cystourethrogram (MCUG)

Micturating cystourethrogram (MCUG) should be used to investigate atypical or recurrent UTIs in children under 6 months. It is also used where there is a family history of vesico-ureteric reflux, dilatation of the ureter on ultrasound or poor urinary flow. A MCUG is used to diagnose VUR.

It involves catheterising the child, injecting contrast into the bladder and taking a series of xray films to determine whether the contrast is refluxing into the ureters. Children are usually given prophylactic antibiotics for 3 days around the time of the investigation.

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15
Q

management of placental abruption

A

Urgent involvement of a senior obstetrician, midwife and anaesthetist
2 x grey cannula
Bloods include FBC, UE, LFT and coagulation studies
Crossmatch 4 units of blood
Fluid and blood resuscitation as required
CTG monitoring of the fetus

atenatal steroids are offered between 24 and 34 + 6 weeks gestation to mature the fetal lungs in anticipation of preterm delivery.

Rhesus-D negative women require anti-D prophylaxis when bleeding occurs. A Kleihauer test is used to quantify how much fetal blood is mixed with the maternal blood, to determine the dose of anti-D that is required.

Emergency caesarean section may be required where the mother is unstable, or there is fetal distress.

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16
Q

when to do emergency c section in placental abruption

A

Emergency caesarean section may be required where the mother is unstable, or there is fetal distress.

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17
Q

what are the 3 causes of antepartum haemorrhage to remember

A

The three causes of antepartum haemorrhage to remember are placenta praevia, placental abruption and vasa praevia

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18
Q

causes of spotting in pregnancy

A

Causes of spotting or minor bleeding in pregnancy include cervical ectropion, infection and vaginal abrasions from intercourse or procedures.

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19
Q

risk of placental previa

A

Antepartum haemorrhage
Emergency caesarean section
Emergency hysterectomy
Maternal anaemia and transfusions
Preterm birth and low birth weight
Stillbirth

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20
Q

management of placenta previa

A

ultrasound scan at:

32 weeks gestation
36 weeks gestation (if present on the 32-week scan, to guide decisions about delivery)

Corticosteroids are given between 34 and 35 + 6 weeks (Lungs) given the risk of preterm delivery.

Planned delivery is considered between 36 and 37 weeks gestation. planned early to reduce the risk of spontaneous labour and bleeding. Planned cesarean section is required with placenta praevia and low-lying placenta (<20mm from the internal os).

Emergency caesarean section may be required with premature labour or antenatal bleeding.

-anti-d in rhesus negative mothers

The main complication of placenta praevia is haemorrhage. When this occurs, urgent management is required and may involve:

Emergency caesarean section
Blood transfusions
Intrauterine balloon tamponade
Uterine artery occlusion
Emergency hysterectomy

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21
Q

what is vasa praevia?

A

usually the babies blood vessels are tucked into the placenta or umbilical cord. Vasa praevia is when The fetal vessels travel through the chorioamniotic membranes, and pass across the internal cervical os (the inner opening of the cervix). These exposed vessels are prone to bleeding, particularly when the membranes are ruptured during labour and at birth. This can lead to dramatic fetal blood loss and death.

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22
Q

investigation and management of vasa praevia

A

Ideally, Vasa praevia may be diagnosed by ultrasound during pregnancy. However, ultrasound is not reliable, and it is often not possible to diagnose antenatally.

It may present with antepartum haemorrhage, with bleeding during the second or third trimester of pregnancy.

It may be detected by vaginal examination during labour, when pulsating fetal vessels are seen in the membranes through the dilated cervix.

Finally, it may be detected during labour when fetal distress and dark-red bleeding occur following rupture of the membranes. This carries a very high fetal mortality, even with emergency caesarean section.

management
For asymptomatic women with vasa praevia, the RCOG guidelines (2018) recommend:

Corticosteroids, given from 32 weeks gestation to mature the fetal lungs
Elective caesarean section, planned for 34 – 36 weeks gestation

-anti-d in rhesus negative mothers

Where antepartum haemorrhage occurs, emergency caesarean section is required to deliver the fetus before death occurs.

After stillbirth or unexplained fetal compromise during delivery, the placenta is examined for evidence of vasa praevia as a possible cause.

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23
Q

two main risk factors of GD

A

big baby- shoulder dystocsia
post partem hypogylcaemia

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24
Q

screening test for GD

A

oral glucose tolerance test

An OGTT should be performed in the morning after a fast (they can drink plain water). The patient drinks a 75g glucose drink at the start of the test. The blood sugar level is measured before the sugar drink (fasting) and then at 2 hours.

Normal results are:

Fasting: < 5.6 mmol/l
At 2 hours: < 7.8 mmol/l
Results higher than these values are used to diagnose gestational diabetes.

TOM TIP: It is really easy to remember the cutoff for gestational diabetes as simply 5 – 6 – 7 – 8.

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25
managment for GD
-Fasting glucose less than 7 mmol/l: trial of diet and exercise for 1-2 weeks, followed by metformin, then insulin -Fasting glucose above 7 mmol/l: start insulin ± metformin -Fasting glucose above 6 mmol/l plus macrosomia (or other complications): start insulin ± metformin
26
After how many weeks does pre eclampsia present
20 weeks
27
what can be given as phrophylaxis for pre-eclampsia
Aspirin is used for prophylaxis against the development of pre-eclampsia. It is given from 12 weeks gestation until birth to women with risk factors
28
management for pre eclamsia
Medical management of pre-eclampsia is with: Labetolol is first-line as an antihypertensive Nifedipine (modified-release) is commonly used second-line Blood pressure is monitored closely (at least every 48 hours) Ultrasound monitoring of the fetus, amniotic fluid and dopplers is performed two weekly Treating to aim for a blood pressure below 135/85 mmHg Admission for women with a blood pressure above 160/110 mmHg IV magnesium sulphate is given during labour and in the 24 hours afterwards to prevent seizures Fluid restriction is used during labour in severe pre-eclampsia or eclampsia, to avoid fluid overload Planned early birth may be necessary if the blood pressure cannot be controlled or complications occur. Corticosteroids should be given to women having a premature birth to help mature the fetal lungs.
29
at what blood pressure does someone with pre-eclampsia/hypertension need to be admitted
Treating to aim for a blood pressure below 135/85 mmHg Admission for women with a blood pressure above 160/110 mmHg
30
treatment of Eclampsia
IV magnesium sulphate
31
affects of pre-eclampsia/eclampsia on the baby
pre-eclampsia: Fetal growth restriction eclampsia: poor fetal perfusion/fetal destress
32
Amniocentesis vs cvs
Cvs before 15 weeks
33
downs syndrome testing
The combined test is the first line and the most accurate screening test. It is performed between 11 and 14 weeks gestation and involves combining results from ultrasound and maternal blood tests. Ultrasound measures nuchal translucency, which is the thickness of the back of the neck of the fetus. Down’s syndrome is one cause of a nuchal thickness greater than 6mm. Maternal blood tests: Beta‑human chorionic gonadotrophin (beta-HCG) – a higher result indicates a greater risk Pregnancy‑associated plasma protein‑A (PAPPA) – a lower result indicates a greater risk Triple Test The triple test is performed between 14 and 20 weeks gestation. It only involves maternal blood tests: Beta-HCG – a higher result indicates greater risk Alpha-fetoprotein (AFP) – a lower result indicates a greater risk Serum oestriol (female sex hormone) – a lower result indicates a greater risk Quadruple Test The quadruple test is performed between 14 and 20 weeks gestation. It is identical to the triple test, but also includes maternal blood testing for inhibin-A. A higher inhibin-A indicates a greater risk.
34
Jaundice in the first ??? hours of life is pathological.
24 hrs
35
prolonged jaundice?
21 prem 14 days in term baby
36
When writing out a prescription for a blood transfusion, each unit of blood should be prescribed as
Packed red cells
37
how long should you infuse the blood over
4 hours
38
how to check blood tranfusion
Once the blood transfusion arrives, you will need to check it with a colleague to ensure it is safe for administration: 1. Wash your hands and don appropriate PPE. 2. Request a colleague (nurse or doctor) to assist you with checking the blood transfusion. 3. Ask the patient to tell you their name and date of birth and then compare this to their bracelet, medical notes and blood compatibility report to ensure they all match exactly. 4. Check the blood group and serial number on the blood bag matches the compatibility report. 5. Check the expiry date and time on the unit of blood to ensure it has not expired. 6. Inspect the blood bag for: Signs of tampering Leaks Discolouration Clots Do not administer blood if any of these are noted.
39
when to check blood tranfusion
The patient’s baseline observations should be checked at 0, 15 and 30 minutes from the onset of the transfusion.
40
What to remember for hand and wrist
Pulse Cap refill Palpate anatomical snuff box Potentially sensation in tips of fingers, anatomical snuff box, hypthenar and hypo eminence
41
Back of the hand including anatomical snuff box is supplied by
Radial nerve
42
Osteoarthritis nodes
Heberden’s nodes (in the DIP joints) Bouchard’s nodes (in the PIP joints)
43
X ray changes seen in rheumatoid vs osteoarthritis
Loss of joint space Subchondral sclerosis Subchondral cysts Osteophytes forming at joint margins Loss of joint space Juxta- articular osteoporosis Periarticular erosions Subluxation
44
Management for carpal tunnel syndrome
Management options for carpal tunnel syndrome are: Rest and altered activities Wrist splints that maintain a neutral position of the wrist can be worn at night (for a minimum of 4 weeks) Steroid injections Surgery
45
What’s the one thing not to forget in lumbar spine
Reflexes
46
Apparent vs true length
Apparent leg length Scoliosis Pelvic tilt True leg length Fracture Perthes SUFE Congenital difference
47
Postive Thomas test
Fixed flexion deformity Ilioposas stiffness
48
Cystic fibrosis management
Chest physio 2 x times a day Deep breathing exercises High calorie diet Pancreatic enzymes if insufficiency Advice on refraining from other children with cystic fibrosis Prophylactic antibiotics Genetic counselling Vaccinations
49
Rubella
Start face and moves down to chest Pro drone low grade fever Blanching manucolopaular rash 5 days off school
50
Measels
Koplik spots Starts behind the ear associated with otitis media Pneumonia most common cause of death NOTIFIABLE disease Supportive management
51
Mumps
Ear ache pain Pain on eating Swelling of face glands No rash Supportive management NOTIFIABLE disease
52
Kawasaki
Crash and burn Conjunctivitis Red eyes Adenopathy S- strawberry tongue H- hands and feet desqumanation Burn- High dose aspirin Iv immunoglobulins Regular echos - risk of coronary aneurysm
53
Pathological 24 hrs jaundice
Within 2 ABO haemolytic reaction Resus haemolytic disease G6PD deficiency Hereditary spherocytosis
54
pulmonary odema, hypertension
TACO Furosemide/oxygen
55
hypoxia, pulmonary infiltrates on chest x-ray, fever, hypotension
TRALI O2 and supportive care
56
fever abdominal pain hypotension
ABO incompatible, acute haemolytic reaction stop transfusion confirm diagnosis -check identity of patient/name on blood product -send blood for direct coombs test, repeat typing and cross matching supportive care - fluid resuscitation
57
fever chills
Non-haemolytic febrile reaction slow or stop the transfusion paracetamol monitor
58
pruritis, urticaria
minor allergic reaction Temporarily stop the tranfusion antihistamine monitor
59
hypotension, dysponea, wheezing, angiodema
Anaphylaxis hypotension, dyponea, wheezing, angiodema
60
Treatment of gbs in pregnancy
Antibiotics
61
Investigations for ankolysing spondylitis
Investigations Key investigations include: Inflammatory markers (e.g., CRP and ESR) may rise with disease activity HLA B27 genetic testing X-ray of the spine and sacrum MRI of the spine can show bone marrow oedema early in the disease before there are any xray changes
62
Anyokokysing spondylitis management
The rheumatology multidisciplinary team will manage patients. Treatment aims to control symptoms and preserve function. Medical management may involve: Non-steroidal anti-inflammatory drugs (NSAIDs) are first-line Anti-TNF medications are second-line (e.g., adalimumab, etanercept or infliximab) Secukinumab or ixekizumab are third-line (monoclonal antibodies against interleukin-17) Upadacitinib is another third-line option (JAK inhibitor) Intra-articular steroid injections may be considered for specific joints. Additional management: Physiotherapy Exercise and mobilisation Avoiding smoking Bisphosphonates for osteoporosis Surgery is occasionally required for severe joint deformity