Pain Theory Flashcards

1
Q

What is pain transmitted via through the body

A

Nocioceptors

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2
Q

When was the pain gate theory established
When did pain become more of a holistic model

A

1965

1990

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3
Q

How do nocioceptors work

A

Nocioception
They are sensory nerves that respond to damaging stimuli by sending signals to spinal cord and brain

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4
Q

What are the different types of pain the body can feel

A

Thermal - harmful heat or cold
Mechanical - excess pressure or deformation
Chemical - wide variety of chemicals

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5
Q

How is the duration of a stimulus / how long pain lasts conveyed

A

By the firing pattern of receptors, after a harmful stimuli, the mechanoreceptors may continue firing action potentials

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6
Q

What does the pain gate control theory suggest happens during the body when experiencing pain
(1965 - melzack and wall)

A

Pain is not a straightforward transmission of sensory information from damaged tissue to brain

There are gates on the nerve fibres between peripheral nerves and the brain, these gates help control how pain messages flow from the PNS to the CNS - gate opens and closes in spinal cord based on the interaction between sensory and emotional factors
If something hyper-stimulates the A-beta fibres in the area experiencing pain, a reaction is caused from nearby inhibitory neurones which will mute the pain signals before they reach the spinal cord and brain ultimately putting a damper on pain

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7
Q

What are the 2 types of fibres that cause either a sharp pain or a dull ache

A

A-delta = myelinated fast nerve fibres = sharp pain
C nerve = non-myelinated slow nerve fibre = dull ache

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8
Q

What came about from melzacks pain gate theory and hyper-stimulisation of a-beta nerve fibres

A

The medic community looked for treatments that could help create hyper-stimulation to inhibit pain signals
They found that massage, acupuncture and electotherapy affect the A-beta nerve fibres

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9
Q

What are the 2 components of pain in patients

A

Individualised loudness of pain
Cognitive-behavioural affects which relates to BPS model

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10
Q

What is the nueromatrix model of pain (Melzack 1990)
How does this relate to our roles as GSR

A

It recognised 3 broad dimensions of pain
Cognitive evaluative - expectations, beliefs, coping
Sensory-discriminative - location, intensity, duration
Affective-motivational - thoughts, feelings of stress

Help patients manage the perceived threat of activities/exercise through the use of, for example, progressive exercise programmes and patient education on pain.

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11
Q

How is the BPS integrated into individualised perceptions of pain and exmanination of our clients

A

Describes how our biology relates to our psychology and social interactions which can affect our pain
In subjective examination, yellow flags should be established that are barriers to recovery
The model is an accepted part of management for low back pain.
Its important to ask patients questions related to their thoughts, expectations, understanding of condition and what impact it’s having on their life

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12
Q

Key points for using the BPS model in sport rehabilitation.

A

Always carry out a diagnostic triage first to exclude any serious pathology and red flags
Isolated behavioural signs don’t mean anything - multiple findings are significant
Presence of behavioural signs doesn’t mean the patients problem is just psychological, presence of pathology can significantly impact a persons behaviour
Illness behaviour isn’t a diagnosis, they alert the potential of poor recovery and are prognostic indicators

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13
Q

What is predictive processing by Jolly and Thacker 2012

A

Pain is an embodied element of suffering encapsulated by an experience of the person within the society and culture in which they live

Our consciousness is predictive rather than reactive, sensory info that comes in is used by the CNS to compare with its predictions which is then used to generate our reality

Synapses now has 3 types of cells that can signal across cells = multipartate

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14
Q

What is the differences between acute and chronic pain

A

Acute =
Sudden onset, nociocpetove, temporary, somatic/visceral/referred
Increase RR, HR, BP
MEDS ARE AFFECTIVE

chronic =
Persistent pain over 3 months
Nocioceotive and neuropathic
Unknown cause
Less obvious physiological responses
Limited effects of meds

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15
Q

What is neuropathic pain and what are the types of neuropathic pains in the body

A

A pain initiated or caused by a primary lesion or dysfunction in the nervous system

MSK = entrapment nueropathy
Trauma = stretch related injuries
Nuero = guillian-barre, multiple sclerosis
Other - diabetes, alcoholism

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16
Q

Where are types of entrapment neuropathy most common in the spine
What are the main causes

A

In L spine the most common are L5 and S1
In C spine the most common in C6 and C7

Disk herniation or spondylosis

17
Q

What are the other types of pain

A

Somatic pain - stimulation of receptors in the skin
Visceral pain - internal organs
Referred pain - at point distant from origin
Somatogenic pain - pain originating from an actual physical cause e,g trauma
Psychogenic pain - no physical cause

18
Q

What are pain thresholds affected by

A

Emotional status
Fatigue
Age
Sex
Fear and apprehension like anxiety
Medication
BPS

19
Q

What are the main RED flags for lower back pain patients

A

Hx of cancer
5 Ds
3 Ns
Unilateral or bilateral paresthesia
Cauda equina symptoms
Sudden weight loss / gain
Sleep disturbance
Medical conditions
Hx of concussions
Hx of trauma
Recent infection
Fever / chills
Recent fracture

20
Q

What are the 5 Ds

A

Dizziness
Dysarthria - difficulty in speech
Dysphagia - difficulty swallowing
Diplopia - double vision
Drop attacks - fainting

21
Q

What are the 3 Ns

A

Nystagmus - dancing eyes
Nausea
Neurological symptoms

22
Q

What are the questions to check for Cauda equina

A

Bladder / bowel symptoms
Saddle anaesthesia
Proprioception / altered gait
Sexual function