Paper 2: CP (Endo, Perio, Cons, Paeds, OS) Flashcards
(137 cards)
Aetiology of pulpal and periradiuclar disease
Bacterial entry towards/into pulp/RC due to
- caries
- cracks
- trauma
- resorption
- Perio problems
- microleakage
Compare the changes in pulp seen as bacteria penetrate and the Tx options
Insignificant changes; bacteria within 1.1mm pulp
- reversible
- Tx: remove caries, appropriate restoration
Irreversible damage; bacteria within 0.5mm pulp
- pulp inflamed, areas of necrosis and abscess
- Tx: RCT (pt compliance?)/XLA
- eventually -> pulp death + periradiuclar radiolucency
Discuss the distribution of bacteria in the RC
More bacterial coronal
- why prep. coronal before apical
Coronal: facultative anaerobes
Apical: obligate anaerobes
Within RC NOT in periapical lesion
What is the periapical lesion?
Inflammatory lesion
1st line defence to prevent bacteria entering periapical tissue
If becomes chronic bacteria invade tissues
What is apical periodontitis?
Periapical tissue response to bacteria threat from RC
Compare acute and chronic apical periodontitis
Acute: acute inflammation @ apex
- possibly due to
— response to irritants in healthy periapical tissue
— infection; may develop into 1ry abscess
— acute exacerbation of chronic apical periodontitis
- PMNs restricted to small area = micro-abscess
— if engulf whole periapical area = dento-alveolar abscess
Chronic Apical Periodontitis
- inflammation @ apex of non-vital tooth = periapical granuloma
- granulomatous tissue
- lymphocytes, macrophages, plasma cells
- non/epithelialised
Compare periapical true and pocket cyst
Periapical true cyst
- distinct pathological cavity completely enclosed by epithelial lining
- no communication w/ RC
Periapical pocket cyst
- apical inflammatory cyst
- sad-like epithelial lined cavity
- open to and continuous w/ RC; responds to RCT
Compare microorganisms in 1ry and persistent/2ry infections endodontic infections
1ry
- gram+ and - (prevotella, porphyromonas, fusobacterium)
- polymicrobial
2ry
- monoinfection
- gram+, cocci facultative anaerobes
Discuss yeasts and Enterococcus faecalis in relation to endodontic infection
Yeasts; C. Albicans most common
- found in 1ry and persistent infections
- RF teeth w/ therapy resistant periapical lesions(retreatment)
Enterococcus faecalis
- gram+, extra-oral bacteria
- found in 1ry infection
- predominant species in RF teeth w/ unsuccessful outcome
Ecology of RC
Warm, moist Nutritious Anaerobic Largely protected form host defences Bacteria can communicate w/ each other Produce virulence factors -> tissue damage
Discuss bacterial survival in RC
Planktonic state
- free floating; not attached to surface
- single cell or clumped
Biofilms
- community of microorganisms + EC polymer attached to surface
- resist treatment
— exopolysaccharide (bacteria embedded in) resist diffusion antimicrobial
— different cell layers may act as barrier to diffusion
— lay dormant, more resistant to killing
— specific resistance mechanisms
Mechanical methods of diagnosing periapical disease
Palpation: compare contralateral, -ve doesn’t mean no inflammation Percussion Periodontal probing - narrow pocket — tooth # — RC infection draining creating sinus tract Tooth slooth Transillumination Dentine sensitivity
Possible differential diagnoses for percussion+
Infected pulp O trauma Sinusitis Cuspal # PD disease Apical inflammation
4 soft tissue changes seen in pulpal disease
Reversible pulpitis
Irreversible pulpitis
Hyperplastic pulp
Pulp necrosis
Discuss reversible pulpitis
Possibly due to
- caries
- erosion, attrition, abrasion
- operative procedure
- mild trauma
- scalding
Symptoms
- transient pain
- ceases when stimuli removed
- TTP-
X-ray: normal periradiuclar appearance
Tx
- cover exposed dentine
- remove stimulus
- remove stimulus + dress tooth
Discuss irreversible pulpitis
Due to severe insult on pulp Symptoms - severe, spontaneous pain - lingers; min-hr - Exacerbated: hot liquids; Relieved: cold - PDL involved, pain becomes localised
X-ray: normal periradiuclar appearance; late stage PDL widened
Tx: RCT/XLA
Discuss hyperplastic pulp and pulp necrosis
Hyperplastic pulp
- form of irreversible pulpitis called pulp polyp
- proliferation of chronically inflamed young pulp tissue
- Tx: RCT/XLA
Pulp necrosis
- end of irreversible pulpitis
- Tx: RCT/XLA
Hard tissue changes seen in pulpal disease
Pulp calcification
Internal resorption
Discuss pulp calcification
Physiological 2ry dentine continually deposited post- tooth eruption and root formation
Deposited on pulp chamber floor and ceiling, not walls
W/ T occludes pulp chamber
3ry dentine deposited in response to stimuli
- reactionary: mild
- reparative: string noxious; rapid, irregular, cellular inclusion
Discuss internal resorption
Pulp inflammation may result in resorption of dentine by dentinoclasts
Clinically: pink spot
X-ray: punched out lesion continuous w/ rest of pulp cavity
Tx: RCT, XLA (if lesion too advanced)
Compare cracked tooth in non/vital teeth
Vital - pain — sharp on biting/release, occasionally from cold — difficult to localise - Ix: tooth slooth, staining, transillumination - L6-8, esp. 6 - Tx: ortho/Cu band/temp. crown — progress to cusp coverage restoration
Non-Vital
- pain: dull ache on biting
- Ix: TTP, narrow Perio pocket adjacent to #
- X-ray: halo, J shaped diffuse lesion around root
- Tx: XLA, consider hemisection
Possible Tx options for reversible pulp damage
Indirect pulp capping (stepwise)
- infected softened dentine removed
- layer non-infected dentine left over pulp
- Ca(OH)2 placed, restore
- 6/12 later remove softened dentine, place Ca(OH)2, restore
Direct pulp capping - pulp exposed through non-infected dentine — no recent history spontaneous pain - Ca(OH)2/MTA placed - bacteria tight seal - 12/12 check X-ray + sensibility
Discuss treatment options when pulp damage is irreversible
Pulp amputation - remove part of exposed inflamed pulp; remaining pulp tissue preserved - superficial damage: partial pulpotomy - coronal damage: coronal pulpotomy -
Pulpectomy - total pulp removal followed by RCT - indicated when — pulp irreversibly damaged — pulp cavity req. for retention
Contra/indications for RCT
Indications
- functionally and aesthetically important w/ reasonable prognosis
- irreversibly damaged/necrotic pulp
— w/ or w/o clinical/X-ray finding of apical periodontitis
- elective devitalisation
- dubious pulp prognosis prior to preparation
Contraindications - can’t be made functional or restored - insufficient PD support - poor prognosis: extensive restoration, vertical #s - pt — poor OH; unable to rectify in time — uncooperative — limited opening - complex anatomy: dens in dente