Posterior Pituitary Hormones Flashcards

(30 cards)

1
Q

Oxytocin is produced in _______ and ______.

A

supraoptic nucleus and paraventricular nuclei of the hypothalamus

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2
Q

Primary functions of oxytocin

A

stimulate milk release, stimulate parturition, and stimulate uterine shrinkage after birth

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3
Q

Secondary functions of oxytocin

A

increase renal reabsorption of water and decrease memory

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4
Q

What effect does oxytocin have on the uterus?

A

myometrium OT stimulates smooth muscle contraction. (neurological)

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5
Q

According to Martin, oxytocin is more than just a “love” hormone. it may also do what?

A

May improve social functioning in children with autism spectrum disorders. Findings were mixed.

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6
Q

Oxytocin was found to _____ the intake of high sugar snacks in men.

A

reduce

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7
Q

In bulimia nervosa oxytocin is implicated in the ____________.

A

modulation of reward

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8
Q

Epigenetic modification of oxytocin receptor gene influences ___________.

A

social behavior (anger and fear)

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9
Q

Antidiuretic Hormone (ADH) is also known as:

A

Vasopressin, arginine vasopressin and argipressin

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10
Q

Site of Synthesis of ADH

A

Supra-optic nuclei - hypothalamus - secreted through posterior pituitary.

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11
Q

Primary Function of ADH

A

regulate bodys retention of water

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12
Q

Physiological control of secretion of ADH

A

blood pressure and blood volume

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13
Q

The potential use of ADH is to ________.

A

limit cerebral edema in patients after acute stroke

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14
Q

central diabetes is the ______ output of ADH.

A

decreased

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15
Q

Effects of ADH on central diabetes insipidus is ____ and ______.

A

Excessive urine output (polyuria) and excessive thirst.

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16
Q

Central diabets insipid us can be caused by _____________ by hypothalamus or by pituitary release ADH into the _________.

A

insufficient release of ADH; bloodstream

17
Q

Central diabetes insipidus is caused by ________.

A

deficiency of ADH

18
Q

Physical Characteristics of central diabetes insipidus:

A

polyuria, dilute urine (decreased urine concentration), hypotension (decreased plasma volume), and increased plasma concentration .

19
Q

Compensation for central diabetes insipid us

A

increased aldosterone secretion and increased renal reabsorption of sodium excretion of hydrogen and potassium ions which brings blood pressure back toward normal.

20
Q

Aldosterone is produced by the _______.

21
Q

Function of aldosterone

A

act on the distal tubules and collecting ducts of the kidney, increase reabsorption of water and electrolytes in kidneys, and increases blood volume and blood pressure.

22
Q

Etiology of nephrogenic diabetes insipid us

A

abnormalities in the kidneys

23
Q

Physical characteristics of nephrogenic diabetes insipidus

A

pollyuria, dilute urine (decreased urine concentration), hypotension (decreased plasma volume), and increased plasma concentration.

24
Q

Physical characteristics of nephrogenic diabetes insipidus

A

polyuria, dilute urine (decreased urine concentration), hypotension (decreased plasma volume), and increased plasma concentration.

25
In nephrogenic diabetes inspidus the kidneys fail to respond to ___ and _____ concentrate urine.
ADH; can't
26
In nephrogenic diabetes insipid us the pituitary gland releases ______ but the kidneys ____ respond to the signal.
ADH; don't
27
Etiology of nephrogenic diabetes insipidus
abnormalities in the kidneys
28
Nephrogenic diabets insipid us can be caused by drugs
lithium
29
Syndrome of Inappropriate antidiuretic hormone secretion (SIADH) makes it difficult for your body to get rid of excess water. This causes a buildup of fluids as well as ___________, a condition known as ___________.
abnormally low sodium levels; hyponatremia.
30
Syndrome of Inappropriate antidiuretic hormone secretion (SIADH) causes fluid overload in patients with:
strokes and head trauma