PPQs Flashcards
(331 cards)
optimal dose of fluoride in drinking water
1ppm = 1mg/L
2 foods/drinks other than fluoridated water that are good sources of fluoride & have not have fluoride added by manufacturer
fluoridated milk
fluoridated salt
4 methods of topical fluoride application for an 8 year old
- fluoride varnish 22,600ppm 2x yearly
- fluoride toothpaste 1500ppm
- fluoride tablets 1mg fluoride daily
- fluoride mouthwash 225ppm
how does topical fluoride help prevent cavities
F slows down development of decay by stopping demineralisation of dentine, it makes enamel more resistant to attack from plaque bacteria, speeds up remineralisation & can stop bacterial metabolism at high concentrations to produce less acid. the fluoride that enters the tooth produces fluorapatite which makes the tooth stronger once remineralisation occurs
what daily dose of fluoride tablet would you give a 4 year old at high risk of caries who lives in an area with <0.3mm fluoride in water suply
0.5mg/day
rationale for use of antibiotics for perio caused by cancer
perio is a side effect of cancer treatment and when ptx undergoing cancer tx become immunocompromised they may require antibiotic prophylaxis to ensure that any bacteria present in the mouth that is causing the perio does not upset the ptx systemically
what problems limit the usefulness of antibiotics in tx of perio in cancer ptx
- antibiotics may be inactivated or degraded by non target organisms
- biofilm may resist antibiotics
- allergic reactions may occur
- antibiotic resistance can occur
- super infection can result where broad spectrum antibiotics are not suitable
when is it appropriate to prescribe a systemic antibiotic (5)
- ptx colonised with A.actinomycetemcomitans need systemic antibiotics as local delivery will not kill or inhibit bacteria
- when ptx presents with periodontal abscess of ANUG where systemic symptoms occur
- indication for use when there is ongoing disease despite mechanical therapy and good OH
- if ptx medically compromised
- if aggressive periodontitis & severe recurrent cases
advantages of systemic antibiotics
- delivered via serum to tissues & reaches reservoirs such as tonsils & tongue
- less costly than time required for tx
- must be accompanied by mechanical therapy to reduce bacterial load & disrupt biofilm
types of systemic antibiotics
- amoxicillin 500mg & metronidazole 400mg
- metronidazole alone
- azithromycin
- doxycycline
- tetracycline
3 reasons for carrying out obturation of prepared root canal
- inhibits bacterial growth
- can be easily removed
- seals the canals laterally & apically
describe steps involved in obturating root canal in upper central incisor
- apply dental dam & disinfect access area
- remove provisional restoration using sterile round bur
- irrigate using sodium hypochlorite to remove CaOH medicament; starting with size 10 or 15 introduce files sequentially to confirm access to working length & prepared apical stop
- dry canal with narrow bore aspiration & correct size and length of matching paper points in locking tweezers
- select master gutta percha point which will fill canal at working length & give tug back
- mix root canal sealer (AH+) & coat walls of apical part of root canal thinly using master point itself
- coat tip of master point with sealer & reinsert slowly to working length held in sterile locking tweezers & seat point firmly to the apical stop
cold lateral compaction
- take size A finger spreader, set a silicone stop at 1-2mm from CWL and place into canal alongside master point for 20secs
- coat an extra fine ‘A’ accessory point with sealer, gently remove finger spreader by rotating & immediately insert A point into space left by sealer
- repeat until 3-4 A points have been used or use B spreaders & points if coronal area still wide
- use a heated plugger to remove excess gutta percha within pulp chamber
which part of the root canal filling is the most important in ensuring long term success
good coronal seal is most important for ensuring higher success rate & preventing infection
what is meant by Watt & Macgregor’s biometric guidelines
biometric guide is one of the methods of designing complete dentures
uses remnant of the lingual gingival margin in the buccolingual placement of prosthetic teeth
when applying biometric guides what anatomical feature is used as fixed reference point
positional relation to the central incisors which are about 8-10mm anterior to the incisive papilla
the biometric guide gives you information about the location of the maxillary canines - a perpendicular drawn posterior to the centre of the incisive papilla to the sagittal plane that passes through the canines
average horizontal bone loss for the fixed reference point in the following maxillary tooth positions
central incisors - 6.3mm
canine - 8.5mm
premolar - 10mm
molar - 12.8mm
minimum data set that should be recorded onto occlusal record block
OVD - distance between jaws with teeth in occlusion
centre line - centre of ptx mouth symmetrical with face
occlusal plane - central occlusal plane indicates where incisal level of tooth will be
high lip line
canine line - line extended from inner canthus of eye
arch form - width:lip support
give history, symptoms & presentation of periodontal abscess
localised acute exacerbation of pre existing pocket (chronic perio) caused by trauma to pocket epithelium, or obstruction of pocket entrance
symptoms - pain on biting/constant pain, swelling, discharge release causing halitosis
O/E - swelling adjacent to periodontal pocket, tooth may be TTP, suppuration (discharge through sinus or pocket), tooth mobility, more likely to have pain on lateral movement, more likely to have generalised horizontal bone loss
tx - drainage via incision or via pocket with instrumentation to dilate, gentle sub gingival debridement, hot saline mouthwash, XLA of teeth with poor prognosis, antibiotic use if systemic involvement
follow up - HPT, surgery if required & maintenance
history, symptoms & presentation of periapical abscess
can be chronic / acute but both due to inflammatory reaction to pulpal infection where there is localised collection of pus around apex of non-vital tooth as a result of necrosis
chronic characteristics - gradual onset, little or not discomfort, intermittent discharge of pus through associated sinus tract
chronic radiography - sign of osseous destruction i.e. radiolucency at apex
acute characteristics - rapid onset, spontaneous pain, extreme tenderness to pressure, pus formation, swelling of associated tissues, systemic signs of malaise, fever, lymphadenopathy
acute radiography - may be no signs
tooth can be mobile, TTP vertically, non vital tooth, loss of lamina dura
history, symptoms & presentation of occlusal trauma
causes tooth mobility which is progressively increasing & associated symptoms, radiographic evidence of increased PDL width; in combination with plaque induced inflammation this may exacerbate loss of attachment
tx - control of plaque induced inflammation, correction of occlusal relations, splinting required when:
- mobility is due to advanced LoA
- mobility causing discomfort or difficulty when chewing
- when teeth need stabilised for debridement
history, symptoms & presentation of periapical periodontitis
symptomatic / asymptomatic
symptomatic:
- represents inflammation of apical periodontium that extends beyond root canals
- causes resorption of alveolar bone & LoA
- painful response to biting, palpation, percussion
- can be accompanied by radiographic changes i.e. periapical radiolucency
- severe TTP indicative of degenerating pulp & RCT required
asymptomatic:
- inflammation & destruction of periapical periodontium of pulpal origin
- apical migration of junctional epithelium
- appears as apical radiolucency & does not present clinical symptoms i.e. TTP or palpation
risk factors = plaque accumulation, diabetes, stress
history, symptoms & presentation of chronic gingivitis
inflammation confined to gingival tissue characterised by redness & swelling of marginal gingival tissue
swelling leads to formation of gingival pocket which manifests as increase in probing depth where epithelium lining of pocket is friable & easily traumatised
there is altered microbial colonisation, increased flow of GCF, influx of neutrophils, lymphocytes, monocytes & plasma cell infiltrate
proliferation & ulceration of junctional epithelium, dilated vessels, vascular proliferation, increased collagen loss & very few plasma cells present
chronic can be localised (<30%) or generalised (>30%)
symptoms = bleeding, swollen, red gingivae, false pocketing with no LoA
risk factors = pregnancy, leukaemia, puberty related, poor OH
tx = OH, HPT if required for removal of plaque / calculus
branches of maxillary nerve & where they pass through
trigeminal nerve has 3 branches:
1. ophthalmic - exits through superior orbital fissure
2. maxillary - exits through foramen rotunda
3. mandibular - exits through foramen ovale
maxillary nerve then has branches from the pterygopalatine fossa - zygomatic through zygomatic foramen, nasopalatine through sphenopalatine foramen, posterior superior alveolar nerve, greater & lesser palatine nerve, pharyngeal nerve
maxillary nerve also has branches from infraorbital canal - middle superior alveolar, anterior superior alveolar, infraorbital nerve