PSY240 3. Anxiety Flashcards

(205 cards)

1
Q

Anxiety

A

Negative affect
– Somatic symptoms of tension
– Apprehensive anticipation of future danger

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2
Q

Anxiety

A
  • anxiety: worrying about something that hasn’t come
    e. g. muscle tension

fear: present reaction to threat

normative emotions

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3
Q

Anxiety

A

-

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4
Q

Fear

A

Immediate alarm reaction to present danger

– “fight or flight”

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5
Q

Fear

A

-

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6
Q

Adaptive versus Maladaptive Fear

A

FEAR

Adaptive ===> Maladaptive

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7
Q

Adaptive versus Maladaptive Fear

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realistic concerns => unrealistic concerns
e.g. concern is unfounded - unlikely to happen
proportional => disproportional
the amount of fear experience
excessive distress
subsides upon removal of threat => persists after threat also anticipatory anxiety
worked up agitated state of anticipation

incontrollable and helpless
impairing functioning

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8
Q

Adaptive versus Maladaptive Fear

A

-

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9
Q

Adaptive versus Maladaptive Fear

A

-

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10
Q

Common Symptoms of Anxiety

A

Physical
Cognitive/ Emotional
Behavioural

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11
Q

Common Symptoms of Anxiety

A

physical: goosebumps, nauseated
emotional: irritability
cognitive: difficulty focusing, hyper vigilance, fear losing control, unreality
behavioural: escape vs avoidance, freeze, aggression

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12
Q

Common Symptoms of Anxiety

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13
Q

Common Symptoms of Anxiety

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14
Q

What Makes You Anxious?

A
public speaking
not being good enough
interpersonal social: friends, partner
uncertainty
performance: failure 16%
somatic concerns - health 2%
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15
Q

What Makes You Anxious?

A

54% of college students report feeling overwhelming anxiety

more common type of distress on common

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16
Q

What Makes You Anxious?

A

academic concerns: exams, tests 34%

money: 1%
misc. : 7%

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17
Q

Anxiety Disorders

A

Commonalities
• Basic biological causes
• Basic psychological causes
• Effective treatments

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18
Q

Anxiety Disorders

A

we all experience it and have similar concerns
same commonalities in DSMV disorders
similar causes and treatments

treatments: behavioural treatments

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19
Q

Anxiety Disorders

A

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20
Q

Anxiety Disorders

A

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21
Q

Anxiety Disorders

A
  • Severe
  • Impact quality of life
  • Chronicity and frequency
  • Interfere with functioning
  • Disproportion to real dangers
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22
Q

Anxiety Disorders

A

not everyone has it - beyond normal experiences
severe: affects life + enjoyment
far more frequent and pervasive
becomes an obstacle: e.g. can’t leave house anymore

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23
Q

Anxiety Disorders

A

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24
Q

Anxiety Disorders

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Anxiety Disorders
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Panic Attacks (PAs)
* Not a disorder * Intense fear/discomfort * Sudden onset and peaks rapidly (
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Panic Attacks (PAs)
-symptoms: sweating, heart rate, muscle tension cued: suddenly in response to specific stimulus uncued: out of the blue suddenly develop all of these symptoms at once
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Panic Attacks (PAs)
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Panic Attacks (PAs)
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Panic Attacks (PAs)
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Panic Disorder (PD)
• Recurrent, unexpected panic attacks (PAs) • Followed by 1+ month of (at least one): – Persistent concern about having another PA – Persistent concern about implications of PAs – Significant change in behaviour
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Panic Disorder (PD)
important for diagnosing panic disorder: only when there’s at least 2 uncued panic attacks at least 1 month or longer after - constant fear of having another attack and/or the implications of having a heart attack change in behaviour: e.g. stop riding the subway because it happened there last time - can’t go to work uncued becomes cued - come to fear that situation or place - would still be diagnosed as PD
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Panic Disorder (PD)
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Panic Disorder (PD)
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Agoraphobia
* Anxiety about places / situations where escape might be difficult or help is unavailable * Situations avoided or endured with distress
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Agoraphobia
-condition in itself fear of wide open space 50% of PD also have agoraphobia might be able to do it if they had someone with them
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Agoraphobia
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Examples of Agoraphobia
e.g. fear of crowds, lines, on trains, on a bridge, being in a house alone or leaving house alone
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Examples of Agoraphobia
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Separation Anxiety Disorder
Excessive anxiety concerning separation from the home or primary caregiver(s) previously in developmental disorder => now anxiety disorder physical symptoms: being sick - can’t go to school complain so they can stay - can be real frequent nightmares of separation
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Separation Anxiety Disorder
4+ weeks (6 mos in adults): avoid over-pathologizing responses to transitions
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Separation Anxiety Disorder
not a lot of evidence of continuity - maybe it doesn’t continue or a measurement artifact e.g. earlier in life, parent interview, later in life, self-report generally when child first goes to school can come up later in life - can’t recall disorder
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Separation Anxiety Disorder
e.g. going to college e.g. over concern of offspring or spouse we don’t know as much about condition in adulthood
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Selective Mutism (DSM-5)
* Consistent failure to speak in social situations * Interfereswithachievement * Duration>1month * Not due to lack of knowledge of language * Not better accounted for by other disorders
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Selective Mutism (DSM-5)
emerges in childhood - likely to manifest in school age don’t know much about longitudinal course of disorder rule out other possibilities such as developmental disorder, difficulty in interaction due to anxiety or language problems
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Selective Mutism (DSM-5)
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Selective Mutism (DSM-5)
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Selective Mutism (DSM-5)
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Specific Phobia
* Marked and persistent fear * Exposure almost invariably provokes fear/anxiety* • Situation avoided / endured with distress * Fear/anxiety ≠ actual danger posed
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Specific Phobia
cannot under any circumstance endure situation or stimulus intense amount of distress in childhood: fear may be expressed through screaming, not speaking, tantrums similar symptoms, but tied to specific stimulus or situation
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Specific Phobia
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Specific Phobia
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Specific Phobia – Subtypes
``` Animal Natural / Environment Blood-injection- injury Situational Other ```
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Specific Phobia – Subtypes
e. g. could have been bitten by dog e. g. heights e. g. seeing blood, getting shots e. g. claustrophobia in elevators e. g. clowns, #13
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Specific Phobia – Subtypes
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Specific Phobia – Subtypes
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Specific Phobia – Subtypes
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Social Anxiety Disorder (Social Phobia)
Fear of being focus of attention/scrutiny • Fear of being humiliated – Capacity for age-appropriate relationships* – Must occur in peer settings* • Exposure almost invariably provokes anxiety • Fear is persistent (6+ months)
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Social Anxiety Disorder (Social Phobia)
specific social situations that evoke fear common 20% of US college students report fear of public speaking wording change in DSM-V fearful of humiliation early on - other disorders may cause symptoms like Autism anxiety must be occurring not just with adults, but with peers primary concern to be judged as stupid or weak significant impairment: e.g. run out of room screaming during a presentation
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Social Anxiety Disorder (Social Phobia)
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Social Anxiety Disorder (Social Phobia)
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Social Anxiety Disorder (Social Phobia)
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Generalized Anxiety Disorder (GAD)
``` Chronic/exaggerated worry / tension (6+ mos) • Unable to control worry • Physical symptoms (3+)* – Restlessness, keyed up, on edge – Easily fatigued – Difficulty concentrating – Irritability – Muscle tension – Sleep disturbance ```
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Generalized Anxiety Disorder (GAD)
worries about a lot of things feel like you can’t control the worry in adults at least 3, in kids (
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Generalized Anxiety Disorder (GAD)
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Generalized Anxiety Disorder (GAD)
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Generalized Anxiety Disorder (GAD)
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Epidemiology
Disorder l Age of Onset l Prevalence l Gender Separation Anxiety l ~ preschool l 0.9%–4.0% l F>M Selective Mutism l M Social Anxiety l 8 – 15 years l 7.0–12.0% l F>M Agoraphobia l 17 years l 1.7% l F>M Panic Disorder l 20 – 24 years l 2.0–3.0% l F>M Generalized Anxiety l M
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Epidemiology
-some disorders such as social anxiety are more common, while mutism is rare because we are ruling it out anxiety disorders more common in female manifestation may be different by gender boys may show indirect symptom - e.g. boys say they have nightmares instead of I don’t wanna go to school gender differences emerge depending on specific object of phobia emerge most frequently in adolescents, but in youth may show remission later on social phobia: males more often diagnosed with because it is inconsistent with expectations of masculinity
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Epidemiology
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Epidemiology
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Epidemiology
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Comorbidity
* Anxiety and anxiety | * Anxiety and depression (i.e., internalizing) • Anxiety and substance use disorders
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Comorbidity
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Comorbidity
internalizing disorders inwardly directed emotional problems externalizing: violence, acting out, substance abuse in children it’s less clear cut the difference between the two than in adults social anxiety: e.g. have a drink to calm nerves issue of self-medicate with alcohol in most cases, anxiety comes first
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Comorbidity
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Biological Approach: Structural Theories
Dual pathway model of fear | Stimulus => Thalamus => Amygdala
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Biological Approach: Structural Theories
Stimulus => Thalamus => Cerebral Cortex
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Biological Approach: Structural Theories
-understand how brain processes fear get info from environment that triggers fear response thalamus: gateway amydala: registers danger, stores emotional memory triggers fast reaction cerebral cortex: to brain for more thoughtful processing of info
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Biological Approach: Structural Theories
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Biological Approach: Structural Theories
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Biological Approach: Structural Theories
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Biological Approach: Psychophysiological Theories
* Poorly regulated fight-or-flight responses | * Overreactive autonomic nervous system
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Biological Approach: Psychophysiological Theories
-might be sensitive to bodily reactions when exposed to fear stimulus same experience of hyperventilating can cause more intense reactions in other ppl might be sensitively attuned to bodily reactions when exposed to fear stimulus
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Biological Approach: Genetic Theories
``` twin studies: genetic transmission more frequent in families with probands of anxiety disorders range of heritability: PD - more narrow confidence intervals SAD - larger range across studies- ```
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Biological Approach: Genetic Theories
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Biological Approach: Genetic Theories
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Biological Approach: Genetic Theories
Molecular Genetics Studies • Serotonergicsystem (5-HT;SLC6A4) • Dopaminergicsystem (DRD4;DRD2) • Modulation of monoamine metabolism (MAOA; COMT)
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Biological Approach: Genetic Theories
-serotonergic: involves negative emotions dopaminergic: excessive or lack of receptors through generations degradation of serotonin proposed to be related proposed for candidate gene studies due to expense - not enough replication
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Biological Approach: Genetic Theories
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Biological Approach: Genetic Theories
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Biological Approach: Neurotransmitter Theories
Poor regulation of NTs – NE (stimulating) – 5-HT (mood regulation) – GABA (inhibitory) • GAD: Deficiency of GABA or GABA receptors
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Biological Approach: Neurotransmitter Theories
Norepinephrine - physiological hyperactivity in anxiety disorders serotonin - insufficient may be implicate in anxiety disorders GABA: calming effect - regulating emotion insufficient not being able to habituate to novel stimuli related to GAD - worrying about everything because we’re not getting used to them
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Biological Approach: Neurotransmitter Theories
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Biological Approach: Neurotransmitter Theories
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Personality/Temperament Traits & Anxiety
• Neuroticism/Negative Affectivity (Kotov et al., 2010) • Behavioral inhibition (Kagan) – Risk for social phobia
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Personality/Temperament Traits & Anxiety
-more phenotypic, but influenced by heritable genes high in neuroticism: more prone to anxiety disorder tendency to display inhibition high: more likely to be extremely inhibited in new situations e.g. vulnerable to social phobia
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Personality/Temperament Traits & Anxiety
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Psychological Approach: Cognitive Theories
* High anxiety sensitivity * Hyper-vigilant to potential threat * Cognitive self-evaluation model (social phobia)
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Psychological Approach: Cognitive Theories
attribution to physical symptoms catastrophic interpretation of events misinterpretation of events people with extremely high standards in social performance situations assume everyone will judge them harshly sensitive to misinterpreting social judgement cues
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Psychological Approach: Cognitive Theories
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Triple Vulnerability Model
Biological Vulnerability Heritable contribution to negative affect Specific Psychological Vulnerability Catastrophic interpretations (e.g., of physical sensations) Generalized Psychological Vulnerability External locus of control
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Triple Vulnerability Model
possession of all three most at risk to developing a disorder specific psychological vulnerability: maladaptive interpretations
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Triple Vulnerability Model
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Triple Vulnerability Model
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Psychological Approach: Behavioural Theories
Acquired through classical conditioning – Can occur via observational learning • Maintained through operant conditioning
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Psychological Approach: Behavioural Theories
e. g. got stung by bee, conditioned to fear them e. g. or seeing someone get stung by a bee e. g. running away from bee, anxiety subsides so operantly conditioned to run away
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Psychological Approach: Behavioural Theories
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Psychological Approach: Behavioural Theories
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Biological Treatments
Tricyclic antidepressants  Increase levels of NTs (e.g., norepinephrine)  Serotonin reuptake (SSRIs)  Increase levels of serotonin  Benzodiazepines  Suppress CNS and influence NT functioning ▪ NE, 5-HT, and GABA systems
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Biological Treatments
shown to work on certain anxieties e.g. but don’t give tricyclic to PD, but maybe to GAD improve regulation of negative disorders increase GABA - inhibiting physiological responses addictive, so not given as first treatment
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Biological Treatments
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Biological Treatments
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Behavioural Treatments
* Modelling * Flooding (aka Implosive Therapy) * Systematic desensitization
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Behavioural Treatments
most effective modelling: therapist models appropriate reaction flooding: exposing it all at once e.g. sending them to a bee farm not preferred approach bodies can only sustain reaction for so long, then they realize that it’s not so bad more gradual exposure
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Behavioural Treatments
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Behavioural Treatments
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Systematic Desensitization
1. Develop a fear hierarchy Behavioural Strategies 2. Teach relaxation and breathing exercises 3. Practice gradual exposure to feared situations 4. Practice relaxation while experiencing panic symptoms Cognitive Strategies • Identify maladaptive cognitions • Challenge catastrophizing thoughts
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Systematic Desensitization
1. least feared to most feared 2. teach relaxation, only when they can perform it can they move on to gradual exposure 3. start small so they can extinguish fear to smallest fear and show they can manage it e.g. fear of spider bite, gradual exposure provides evidence that it’s not gonna kill them
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Systematic Desensitization
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Systematic Desensitization
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Systematic Desensitization
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Systematic Desensitization
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What works for Anxiety?
• GAD: – Treatment > No treatment (Cuijpers et al., 2014) – Improve in short-term, but not at follow-up (Westen & Morrison, 2001) – Best results for CBT (Dugas et al., 2003) • Panic: – Improve and remain improved at follow-up (Westen & Morrison, 2001) – CBT = pharmacotherapy (Mitte, 2005)
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What works for Anxiety?
attacking maladaptive thoughts and replacing them gains are sustained as effective as medication cessation of medication, the symptoms return
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What works for Anxiety?
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What works for Anxiety?
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What works for Anxiety?
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Obsessive-Compulsive Disorder
Either obsessions OR compulsions | • Time-consuming (>1hr/day) • Cause distress/impairment
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Obsessive-Compulsive Disorder
-has to interfere with functioning or distress
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Obsessive-Compulsive Disorder
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Obsessive-Compulsive Disorder
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Obsessions (DSM-5)
• Recurrent and persistent thoughts, urges, or images • Intrusive and unwanted, cause anxiety/distress • Thoughts,impulses,orimagesthat: – Are not simply excessive worries about real life problems – The person recognizes are a product of own mind – The person attempts to ignore or suppress or to neutralize with some other thought or action
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Obsessions (DSM-5)
thoughts of germs, killing someone, need for symmetry reality testing excessive - required that they realized it was out of proportion with kids you don’t expect this insight
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Obsessions (DSM-5)
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Obsessions (DSM-5)
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Compulsions (DSM-5)
• Repetitive behaviours or mental acts that the person feels driven to perform in response to an obsession or rigid rules • Aimed at preventing/reducing distress or preventing some dreaded event or situation – Unrealistic – Excessive
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Compulsions (DSM-5)
-fit well with obsessions e.g. entire elaborate ritual of handwashing if not done right causes substantial anxiety checking counting up cleaning or washing doing things in order ordering and organizing neutralize obsessive thought
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Compulsions (DSM-5)
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Compulsions (DSM-5)
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Compulsions (DSM-5)
Stimulus (Internal / External) => Obsession Stimulus (Internal / External) => Distress & Anxiety Obsession Distress & Anxiety
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Compulsions (DSM-5)
Distress & Anxiety => Ritualized Behaviour (Compulsion) | Temporary Relief Ritualized Behaviour (Compulsion)
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Compulsions (DSM-5)
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OCD Example
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OCD Example
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Biological Theories of OCD
• Brain dysfunction in areas that control primitive behaviour • Linkedto5-HT • Evidence for a genetic predisposition (Nestadt et al., 2010) – 7-15% in first-degree relatives of probands – Limited knowledge of potential candidate genes
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Biological Theories of OCD
-e.g. aggression and sex serotonin function - linked to negative emotions common within families with OCD don’t know which genes
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Biological Theories of OCD
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Biological Theories of OCD
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Theories of OCD
Cognitive-Behavioural • PeoplewithOCDcannot“turnoff”obsessivethoughts Psychodynamic (Chlebowski & Gregory, 2009) • Obsessionsandcompulsionsrepresentsymbolicconflicts
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Theories of OCD
brains are wired differently manifestations of underlying conflict
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Theories of OCD
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Theories of OCD
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Psychodynamic Case Formulation
• 22-year-old single female • Feels guilty about past mistakes • Obsessions of contamination • Cleansingcompulsions Formulation:
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Psychodynamic Case Formulation
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Psychodynamic Case Formulation
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Psychodynamic Case Formulation
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Treatments for OCD
BiologicalTreatments – 5-HT-enhancing drugs (e.g., Paxil, Prozac, Zoloft) – Psychosurgery • Anterior Cingulotomy • Anterior Capsulotomy
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Treatments for OCD
-similar to ones used for depression 50% success rates only in very severe cases where remission does not occur for other forms of treatment
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Treatments for OCD
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Treatments for OCD
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Treatments for OCD
• BehaviouralTreatments – Exposure and Response Prevention • CognitiveTherapy – Encourage change in thinking (e.g., acceptance)
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Treatments for OCD
trigger obsessive thoughts and prevent them from engaging in ritual see that response will subside even when still exposed to stimulus accepting that my brain is wired differently
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Treatments for OCD
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Treatments for OCD
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COGNITIVE THERAPY: EXPOSURE & RESPONSE PREVENTION
MEDICATION Stimulus (Internal / External) => Obsession Stimulus (Internal / External) => Distress & Anxiety Obsession Distress & Anxiety
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COGNITIVE THERAPY: EXPOSURE & RESPONSE PREVENTION
Distress & Anxiety => Ritualized Behaviour (Compulsion) | Temporary Relief Ritualized Behaviour (Compulsion)
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COGNITIVE THERAPY: EXPOSURE & RESPONSE PREVENTION
- cognitive therapy: labelling thoughts as not facts | exposure: reduce anxiety response without performance of ritual
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Posttraumatic Stress Disorder
* Experienced traumatic event * Actual/threatened death or injury * Fear/anxiety persisting after trauma (> 4 weeks) – If
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Posttraumatic Stress Disorder
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Posttraumatic Stress Disorder
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Posttraumatic Stress Disorder
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Posttraumatic Stress Disorder (DSM-5)
1. Exposuretotraumaticevent 2. Intrusivesymptoms 3. Avoidance of stimuli associated with trauma 4. Negative alterations in cognitions and mood 5. Marked alterations in arousal/reactivity
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Posttraumatic Stress Disorder (DSM-5)
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Posttraumatic Stress Disorder (DSM-5)
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Posttraumatic Stress Disorder (DSM-5)
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PTSD in the News
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PTSD in the News
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PTSD in the News
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Other Trauma/Stressor-Related Disorders
Adjustment Disorder: • Maladaptivereactionsandimpairedfunctioning/distress following exposure to a stressor – Within 3 months of stressor(s) Acute Stress Disorder: • A traumatic stress reaction occurring in the days and weeks following exposure to a traumatic event. –
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Other Trauma/Stressor-Related Disorders
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Other Trauma/Stressor-Related Disorders
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Other Trauma/Stressor-Related Disorders
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Treatments for PTSD
CBTapproach: – Systematic desensitization (extinguish fear reactions) – Cognitive techniques (challenge irrational thoughts) • Stressmanagement: – Assist in problem solving (reduce stress) – Use “thought stopping” strategies (reduce intrusive thoughts) • Biologicaltherapies: – Anti-anxiety and antidepressant drugs
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Treatments for PTSD
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Treatments for PTSD
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Treatments for PTSD
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Treatments for PTSD
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PTSD Vulnerability
``` Sociocultural factors • Severity, duration, & proximity • Social Support Psychological factors • Personal Assumptions • Distress • Coping Styles ```
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PTSD Vulnerability
Biological factors • Physiologicalhyperactivity • Genetics • Epigenetics • HPA-axishypoactivity
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PTSD Vulnerability
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PTSD Vulnerability
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PTSD Vulnerability
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Treatments for PTSD
Eye Movement Desensitization and Reprocessing (EMDR) – Side-to-side eye movements (saccades) while the client attends to traumatic stimuli, thoughts about the trauma, and the physical sensations of anxiety aroused by the trauma. – Highly controversial
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Treatments for PTSD
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Treatments for PTSD
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Treatments for PTSD
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Summary of Major Anxiety Disorders
Disorder l Description | Phobia l Fear and avoidance of objects or situations that do not present any real danger
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Summary of Major Anxiety Disorders
Panic Disorder l Recurrent panic attacks involving a sudden onset of physiological symptoms. Generalized Anxiety Disorder l Persistent, uncontrollable worry, often about minor things.
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Summary of Major Anxiety Disorders
Separation anxiety l The anxious arousal and worry about losing contact with and proximity to other people, typically significant others. Agoraphobia l A fear of being in public places.
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Summary of Major Anxiety Disorders
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Summary of Major Anxiety Disorders
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Case Studies
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Case Studies
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Case Studies
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