Respiratory Flashcards

(62 cards)

1
Q

lwr respir system structures

A
left lung- 2 lobes
right lung- 3 lobes (upper, middle, lower)
pleura
mediastinum
bronchi and bronchioles
alveoli
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2
Q

oxygen transport

A

diffuses across alveolar mem into arterial blood, oxygenated blood carried to tissues (o2 bind w/ hgb= oxyhemoglobin)
*diffuses frm areas of higher partial p to lower partial p

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3
Q

c02 transport

A

end product metabolic combustion
diffuses across alveolar cap mem into venous blood, deoxygenated blood perfuses back to lungs
co2 diffuses easier than o2

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4
Q

respiration def

A

process of gas exchange btw atm air and the blood and blood and cells of the body
removes co2 from airway and oxygenates the blood

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5
Q

oxygen concen in lungs v alveoli

A

lower conc in cap of lungs than alveoli

= o2 diffueses from alveoli into blood (remember o2 goes frm high to low pressure)

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6
Q

ventilation-perfusion inequality or mismatch

A

v/q
air in alveoli/ blood flow in cap
normal = 1-1

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7
Q

shunting

A

low v/q, dec vent to well perfused areas
blood passes alveoli w/out exchange
results- dec o2 sat, dyspnea
causes- pneumonia, atelectasis, respir depression

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8
Q

alveolar dead space

A

high v/q, poor perfusion
cause- inc residual co2, pulmonary emboli
inhaled air not participate in gas exchange= alv damage`

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9
Q

gerontologic considerations

A
dec surface are available for O2
dec elasticity
dead space inc
dec cough reflex, inc mucus
peak lung function-middle age
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10
Q

s/s and cause (shortness of breath, cough, sputum production)

A

SOB- inc airway resistance, dec lung compliance, bronchospasms, anemia (dec 02 carrying capacity)
cough- irritation mucus mem (dry)- ACE inhib (lisinopril)
sputum- yellow-infection
white/pink and frothy- pulm edema

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11
Q

s/s in physical assessment- chronic respir dis (COPD)

A

clubbing fingers, cyanotiic skin color

retraction, use acces musces, tripod position

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12
Q

lung sounds

A

crackles (fluid) pneumonia, heart failure (if gone w/ cough= atelectasis (IS/ mobility important))
wheezing- asthma, COPD (inc airway resistance)
rhonchi- course crackle- bronchitis, pneumonia

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13
Q

diagnostic tests

A

abg
sputum (id malignant cells, rinse mouth and obtain before eat/antib)
chest x-ray
CT
MRI
Radioisotope (lung scan, inject tracer tags specific tissue)
Thoracentesis (remove fluid frm lungs w/ needle)
biopses

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14
Q

purpose of supplemental o2

A

dec work of breathing, reduce stress on the myocardium

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15
Q

risks assoc w/ supp o2

A

oxygen toxicity, dry muc mem, (trt w/ wtr based lubricant), pressure sores frm tubing

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16
Q

COPD supp O2 considertions

A

88-92%

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17
Q

hypoxemia

A

not enough o2 avaliable
cause- abnorm v/q, inc mem thickness, edema, dec surface area
dec in arterial o2 tension in blood

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18
Q

hypoxia

A

dec o2 supply to tissues/ cells

can be lifethreatening

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19
Q

supp o2 admin

A

low v high flow
low- nasal can (variable performance bc breath in RA too
simple mask and non-rebreather mask
high- venturi (COPD) specific inspired control 4-8L

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20
Q

chest tube drainage

A

suction or gravity-powered
collects pleural drainage (position changing can = dumping)
prevents air from re-entering chest w/ inhalation
removes fluid/air from pleural space
wet water seal or dry suction

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21
Q

pneumonia def

A

inflamm lung parenchyma caused by microo (bac, mycobac, fungi and virus)
alveoli fill w/ fluid

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22
Q

pneumonia- community-acquired

A

viral/bac
<48h after admission
incubating before
trt w/ antib

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23
Q

pneumonia- nosocomial (healthcare assoc)

A

non-hospitalzied ppl w/ extensive healthcare contact
pts been in hospital >2 days within last 3 mon, nursing home res, pt on chronic antib, chemo pt, wound care, hemodialysis, pt w/ family member of MDR (mult drug resistant)

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24
Q

hospital aquired pneumon/ ventilator-associated

A

greater than 48 h after admission (not icubating)

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25
pneumonia risk factors
Occurs in patients with certain underlying disorders and diseases Heart failure, diabetes, alcoholism, COPD, and AIDS Influenza, COVID-19shallow breathing, smoking, immobilization, NG tube (aspiration (good oral care, head bed elevated >30°)) Cystic fibrosis (lrg amnt mucus, inc risk bacterial infection= pneumonia)
26
pneumonia clinical manifestations- general, strep, viral, other, orthopnea
Varies depending on type, causal organism, and presence of underlying disease general- dyspneic, sputum production, chest painsystemic-cough, fever, chills Streptococcal:-Sudden onset of chills, fever, pleuritic chest pain, tachypnea, and respiratory distress Viral- mycoplasma, or Legionella: relative bradycardia Other-Respiratory tract infection, headache, low-grade fever, pleuritic pain, myalgia, rash, and pharyngitis Orthopnea- crackles, increased tactile fremitus, purulent sputum
27
pneumonia- hx questions
recent respir tract infection
28
pneumonia- what to look for in physical exam
signs respir distress, vitals, IV v Po antibiotics, immed need for help= determine if treated outpatientdec lung sounds, rhonchi, crackles
29
pneumonia- purpose blood culture
Blood culture (assess for risk bacteremia) blood 2 opposite sites (rule out contamination) 2-3 days for results
30
pneumonia diagnostic tests-
blood culture chest x-ray sputum exam physical exam
31
pneumonia- care plan goals (incl complications)
Improved airway patency interv- bronchodil, steroids (dec inflamm), fluid bal, humidif O2 etc… Increased activity Maintenance of proper fluid volume 1000-1200mL Maintenance of adequate nutrition Understanding of the treatment protocol and preventive measures Absence of complications pleural effusion shock, respir failure, bacteremia
32
pneumonia- care plan interventions
``` Oxygen with humidification to loosen secretions Face mask or nasal cannula Coughing techniques Chest physiotherapy postural drainage (vibration- cystic fibrosis) Position changes Incentive spirometry Nutrition Hydration Rest Activity as tolerated Patient teaching Self-care ```
33
pneumonia- drug therapy
Administration of the appropriate antibiotic as determined by the results of a culture and sensitivity Supportive treatment includes fluids, oxygen for hypoxia, antipyretics, antitussives, decongestants, and antihistamines Antibiotics not indicated for viral infections but are used for secondary bacterial infection
34
pneumonia- education
Educate about pneumococcal vaccine (23+ Prevnar 15) reduce incidence pneumon. Reccom. +65 yr even if infected- risk for severe complications much less
35
pnemonia- goals contin/ expected outcomes
Demonstrates improved airway patency (assessed w/ lung sounds, improved pulse ox) Rests and conserves energy and then slowly increasing activities Maintains adequate hydration; adequate dietary intake (assessed w/ I&O/ daily weight) Verbalizes increased knowledge about management strategies Complies with management strategies Exhibits no complications
36
risk factors aspiration
``` Decreased LOC Seizure Stroke Swallowing disorder Flat positioning ```
37
aspiration in relation to pneumonia
complication that can cause pneumonia | *can be silent
38
aspiration def
inhalation foreign material into the lungs
39
s/s aspiration
tachycardia, dyspnea, central cyanosis, hypertension, hypotension, and potential death
40
aspiration- nursing interventions
``` Keep HOB elevated >30 degrees Avoid stimulation of gag reflex with suctioning or other procedures Check for placement before tube feedings Thickened fluids for swallowing problems Oral care ```
41
Pulmonary emboli
Obstruction of the pulmonary artery or one of its branches by a thrombus (or thrombi) that originates somewhere in the venous system or in the right side of the heart
42
surgeries assoc w/ PE
multi-trauma, shortness of breath, long bone surgeries, no mvmnt
43
PE patho
Inflammatory process obstructs area, results in diminished or absent blood flow Bronchioles constrict, further increasing pulmonary vascular resistance, pulmonary arterial pressure, and right ventricular workload
44
PE diagnosed via
diagnosed w/ v/q scan and d-dimer lab (measure fibrin lvl from clot lysis) abg’s (show respir alkalosis early on and acidosis later due to low O2 anaerobic metab)
45
immediate bed side interventions for PE
ex. 84% on 28rr- respir physical assessment, start O2 suppl therapy, sit upright, anticipate EKG (test IV function)
46
risks for PE
``` Trauma Surgery Pregnancy (hormone- inc risk clotting) Heart failure Hypercoagulability- (birth control inc risk) Immobility, venous stasis (DVT) ```
47
prevention/treatment PE
Exercises to avoid venous stasis Early ambulation Anti-embolism stockings Treatment Measures to improve respiratory and vascular status Anticoagulation and thrombolytic (prevent new clots forming) therapy Surgical interventions embolectomy- remove clotreassurance- use inferior vena cava filter (stops clots frm lwr extremities) temporary, long term anti-coagulant
48
COPD def
COPD is a slowly progressive respiratory disease of airflow obstruction Emphysema, & chronic bronchitis Preventable and treatable but not fully reversible smoking #1 cause Involving the airways, pulmonary parenchyma, or both
49
COPD patho
Airflow limitation is progressive, associated with abnormal inflammatory response to noxious particles or gases Chronic inflammation damages tissue Scar tissue in airways results in narrowing Scar tissue in the parenchyma decreases elastic recoil (compliance) Scar tissue in pulmonary vasculature causes thickened vessel lining and hypertrophy of smooth muscle (pulmonary hypertension)
50
chronic bronchitis def
Cough and sputum production for at least 3 months in each of 2 consecutive years Ciliary function is reduced, bronchial walls thicken, bronchial airways narrow, and mucous may plug airways Alveoli become damaged, fibrosed, and alveolar macrophage function diminishes The patient is more susceptible to respiratory infections Poor lung function @ baseline w/ exacerbation (accompanied w/ components COPD)
51
emphysema def
Abnormal distention of air spaces beyond the terminal bronchioles with destruction of the walls of the alveoli alveoli chronically expanded Decreased alveolar surface area increases in “dead space,” impaired oxygen diffusion Hypoxemia results *Increased pulmonary artery pressure (frm resistance) may cause right-sided heart failure (cor pulmonale) Elevated CO2 lvl ”pink puffers”
52
COPD manif
``` Four primary symptoms Chronic cough Sputum production Dyspnea Wheezing Weight loss due to dyspnea specifically in emphysema “Barrel chest” A/P diameter 1:1 v 1:2 retraction supraclavicular fossa and abdomen ```
53
how to diagnose COPD
``` Pulmonary function tests Spirometry Arterial blood gas Chest x-ray Note ability talk and complete sentences, use of accessory muscles, R sided-heart failure, enlarged neck veins, LOC (hypercapnia), tripod position ```
54
COPD complications
``` Respiratory insufficiency and failure acute nonchronic respir failure= O2 @ home Pneumonia Chronic atelectasis Pneumothorax Cor pulmonale ```
55
COPD nursing interventions
Promote smoking cessation Reducing risk factors hygiene, avoid lrg crowds Managing exacerbations SABA, ICS, LABA etc Providing supplemental oxygen therapy Pneumococcal vaccine Influenza vaccine Pulmonary rehabilitation
56
COPD drug therapy
Bronchodilators, MDIs Beta-adrenergic agonists Muscarinic antagonists (anticholinergics) Combination agents Shake, use spacer, inhale, wait 5 sec then use again Prioritize bronchodil before corticosteroid Corticosteroids- rinse after use, dec risk thrush Antibiotics Mucolytics Antitussives
57
COPD patient education
COPD- diaphragmatic breathing (blow out w/ pursed lip breathing), 88-92% O2 metered dose inhaler- check physical/ cog ability to use
58
COPD goals
Achieving airway clearance Improving breathing pattern Improving activity tolerance
59
asthma def
Chronic inflammatory disease of the airways that causes hyperresponsiveness, mucosal edema, and mucus production- reversable Inflammation leads to cough, chest tightness, wheezing, and dyspnea (Fig. 24-6) Asthma is largely reversible; spontaneously or with treatment Allergy is the strongest predisposing factor
60
asthma clinical manif
``` Cough, dyspnea, wheezing Exacerbations Cough, productive or not Generalized wheezing Chest tightness and dyspnea Diaphoresis Tachycardia Hypoxemia and central cyanosis ```
61
asthma- drug therapy
``` Quick-relief medications Beta2-adrenergic agonists Anticholinergics Long-acting medications Corticosteroids Long-acting beta2-adrenergic agonists Leukotriene modifiers ```
62
asthma- patient ed
``` How to identify and avoid triggers Proper inhalation techniques How to perform peak flow monitoring How to implement an action plan When and how to seek assistance ```