Respiratory Flashcards

(86 cards)

1
Q

Dyspnoea at rest?

A

Cardiovascular disease

(Inadequate perfusion of the lungs)

Cardiac disease

congestive heart failure, acute heart failure

Peripheral circulatory failure

shock and dehydration

Increased blood viscosity

•haemo-

concentration including

polycythaemia

and disseminated

intravascular

coagulopathy

Diseases of blood

A

naemia

A

ltered haemoglobin

e.g.

methaemoglobinaemia

in nitrate poisoning

•Nervous system diseases

Paralysis respiratory muscles

•botulism, tick paralysis

Paralysis respiratory centre

•nicotine sulphate poisoning

Neurogenic dyspnoea

• stimulation of respiratory centre by

irritative

lesion

•General systemic states

–pain

–hyperthermia

–acidosis

•Environmental causes

–Very high temperatures, oxygen lack (high

altitude), exposure to toxic gasses

•Poisons

–Nitrate, hydrocyanic acid, urea,

organophosphate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Examination of the Resp Tract

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Additional tests for respiratory

A

* Percutaneous transtracheal aspiration

  • wide bore needle between tracheal rings
  • insert long plastic catheter: withdraw needle. Leave catheter in place– passed to point approximately 10 cm beyond thoracic inlet
  • infuse 20-40 ml of sterile saline and aspirate back quickly

– culture, cytological examination

* Pleural paracentesis

  • ideally use a teat siphon through a skin stab wound but can use a catheter– insert below fluid line
  • samples for cytology and bacterial culture
  • can be of use in treatment

* Bronchoalveolar lavage (BAL)- may be done using two tubes introduced through the nares– an outer tube which is passed to the level of the trachea and an inner tube which is passed to the level of the bronchus

* Sample obtained can be used for virus isolation and bacterial culture

* Thoracic ultrasonography– pleural and pulmonary surfaces of both lung fields can be examined through the intercostal spaces between the 7th and 11th ribs

  • a 3.5 MHz sector transducer is suitable for this task
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Causes of pulmonary congestion and oedema

A

Causes:

* early pneumonia

* Inhalation smoke, fumes

* anaphylactic reaction

* hypostasis in recumbent animals

* secondary causes- CHF

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Clinical signs of pulmonary congestion and oedema

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Treatment for pulmonary congestion and oedema

A

* Adrenalin for oedema due to anaphylaxis

* Antiobiotics if early pneumonia suspected

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is pneumonia? Causes?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Classification of pneumonia

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Clinical findings of pneumonia

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Diagnosis of pneumonia

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Treatment of Pneumonia

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Aspiration pneumonia– common sequelae to what?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Acute undifferentiated bovine respiratory disease?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Agents associated with BRD

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What is the most common cause of illness and death in AUS feedlot cattle?

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Cause of BRD

A

The major pathogens of BRD are ubiquitous, and

all the major bacterial respiratory pathogens are

commensal

in clinically normal feedlot cattle.

Clinical BRD is the product of the effect of

stressors causing immuno-

suppression, thereby

allowing the colonisation of the lower respiratory

tract by opportunistic pathogens that invariably are

encountered by feedlot cattle.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Risk factors with BRD

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

BRD Clinical findings

A

* Broncho-pneumonia 10-14 days after stress

* In the early stages

  • off feed depressed rapid shallow resp moist cough

* Later stages:

  • febrile (40-41C)
  • deep laboured resp (insp and exp)
  • increased harshness of insp sounds as purulent material gathers in the airways
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

BRD Clinical findings

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q
A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

BRD Treatment

A

* Antiobiotics

  • Usually oxytetracycline, ceftiofur, or trimethroprim/ sulphonamide combinations are used in feedlots

* NSAIDs

* Nursing- shelter and high quality feed

* Early detection of new cases and continual reassessment of the results of treatment are necessary

* Value of mass medication of feed supplies of newly arrived cattle to the feedlot

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Vaccination against BRD

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Pneumonic pasteurellosis
• Combination of: – Viral infections of the respiratory tract – Effects of transportation, temporary starvation, weaning, fluctuations in temperature, mixing, excess handling an increase in the total numbers and virulence of Pasteurella in the nasopharynx • Most commonly isolated – Mannheimia hemolytica – Pasteurella multocida
26
27
Pasteurellosis
28
Managing a Manheimia outbreak
29
Monitoring for Manheimia hemolytica
30
Histophilus somni disease complex?
31
Histophilosis respiratory form
32
Histophilosis Septicaemic Form
33
Histophilosis nervous form
34
Diagnosis of Histophilosis
35
Atypical Interstitial Pneumonia
36
Interstitial pneumonias include
37
ABPEE
38
ABPEE clinical findings and necropsy?
39
ABPEE Treatment?
40
Other interstitial pneumonias
41
Contagious Bovine Pleuropneumonia
42
Epi of CBPP
43
CBPP signs and symptoms
44
PM Findings in CBPP
45
Control of CBPP
46
Enzootic pneumonia of calves
47
Enzootic pneumonia of calves signs and symptoms
Enzootic pneumonia of calves • Areas of collapse with little bronchiolar reaction • In the later stages a dark red consolidation with little or no fluid in the lung is detected. • Lesions are bilateral and histologically there is an interstitial pneumonia.
48
Lungworm in cattle
• Dairy calves are most vulnerable to lungworm disease, as they are often placed on paddocks grazed each year by successive groups of calves • With high stocking rates, pasture challenge can then reach pathogenic levels within 2 to 4 months. • Usually, only relatively small numbers of larvae survive to induce low -grade, asymptomatic infections in a new group of susceptible calves, which then start to re- contaminate the pasture and recycle the infection
49
Acute verminous pneumonia
50
Sub-acute verminous pneumonia
51
Lungworm diagnosis
52
Infectious bovine rhinotracheitis
• May also cause an encephalitis in new born calves • Infectious pustular vulvo - vaginitis (IPV), balano- posthitis in adults.
53
Infectious bovine rhinotracheitis signs and symptoms
• Sudden onset • Anorexia, • Fever up to 42 degrees C, • Severe hyperaemia of the nasal mucosa - greyish plaques of necrosis nasal septum • Serous discharge from the eyes and nose • Respiratory rate increased and shallow • Lung sounds are normal on auscultation. • May be short explosive cough • Conjunctivitis is seen in some outbreaks.
54
55
Infectious bovine rhinotracheitis calves
• New born calves systemic form is severe and highly fatal. • Signs include sudden anorexia, fever, rhinitis, erosion of the soft palate, acute pharyngitis. Broncho- pneumonia is common.
56
Infectious pustular vaginitis (IPV)
• Vaginal examination reveals plaques or pustules of white necrotic material on the vulva and vaginal mucosa, and a pool of odorless mucopurulent material on the vaginal floor. • In bulls, IPV causes a balanoposthitis.
57
IBR diagnosis
Overseas, abortion is a common sequel in pregnant cows
58
IBR Control
59
Oral and laryngeal necrobacillosis
60
In a calf
61
Oral necrobacillosis- necrotic stomatitis
62
• Quite irritant - affected animals will often rub their noses on sticks, thistles • On palpating the nasal mucosa of chronically infected granulomatous nodules of 1 to 4 mm in diameter and height can be detected in both nostrils • The severity of the condition in affected cattle will often wax and wane
63
TB in cattle? Epi?
Caused by Mycobacterium bovis and is characterised by the progressive development of tubercles in any of the organs in most species. More about Epi: \* Inf by ingestion is more likely at pasture when faeces contaminate the feed and communal drinking water and feed troughts \* Drinking of infected milk by young animals is a commoin method of spread
64
TB spread in the body
65
TB clinical findings
\* TB mastitis can be of major importance because of its danged to PH \* palpation of the supramammary LNs is essential in all cases of suspected tuberculous mastitis
66
TB clinical pathology
Basis of tuberculosis eradication campaigns is the tuberculin test and a knowledge of the various tests used, their deficiencies and advantages, is essential
67
Single intradermal tuberculin
68
TB pathology
69
TB Diagnosis
70
SBE
• caused by a chlamydia • characterised by inflammation of vascular endothelium and mesenchymal tissue.
71
SBE Signs and symptoms
• caused by a chlamydia • characterised by inflammation of vascular endothelium and mesenchymal tissue. • Later, difficulty in walking stiffness with knuckling at the fetlocks is evident at first, followed by staggering, circling and falling • Opisthotonus may be present but there is no excitement or head pressing .
72
SBE Pathology
Sporadic Bovine Encephalomyelitis Pathology: • Chlamydia can be isolated from the blood in the early clinical • Serological tests, including a complement fixation test, are available . Pathology: • On post mortem a fibrinous peritonitis, pleurisy and pericarditis accompanied by congestion and petechiation are characteristic.
73
Brisket Disease in Cattle
Epidemiology • the low density of the atmosphere at high altitudes results in an environmental anoxia • most commonly in yearlings. • Brisket disease is reported commonly only in animals maintained for some months at altitudes above 1800 metres. • Continued hypoxia can cause sufficient myocardial weakness to interfere with cardiac compensation and thus lead to congestive heart failure.
74
Poisonings which may be associated with dyspnoea
• A number of poisons cause dyspnoea as the prominent sign. • Farm chemicals including methaldehyde and dinitrophenols, organophosphates and carbamates, urea • Poisonous plants including fast death factors of algae, the weeds Albizia , Helenium , Eupatorium, Ipomea, Taxus spp., Laburnum and Erythrophloeum spp. can all cause dyspnoea.
75
Nitrite/ Nitrate poisoning
Nitrate/nitrite poisoning – common plant sources • Cereal crops include: immature green oats, millet, barley, wheat, rye, Sudan grass, corn, sorghum fodder, mangles, turnips, sugar beet tops and rape, certain specific plants and water from deep wells. • Specific plants include variegated thistle, mintweed and a series of other plants Nitrate/nitrite poisoning. - epidemiology • Cereal and root crops are likely to contain high concentrations of nitrate when heavily fertilised with nitrogenous fertilisers • When growth is rapid during hot humid weather. • In cattle the maximum methaemoglobin level usually occurs about 5 hours after ingestion of nitrate.
76
Findings in nitrate/nitrite poisonings
77
Nitrite/nitrate poisoning- pathology
• Post mortem - blood is coffee brown in colour and clots poorly.
78
Nitrate/ Nitrite Treatment
79
Hydrocyanic Acid Poisoning
Sources • Many plants contain cyanogenetic glucosides • Common pasture plants including sorghum, Sudan grass and Johnson grass.
80
Hydrocyanic Acid poisoning epi
Epidemiology • High soil concentrations of nitrogen predispose to higher levels of hydrocyanic acid in plants. • Wilted, frost bitten and young plants are likely to more toxic
81
Hydrocyanic Acid poisoning • Animals may show signs within 10- 15 minutes of eating the toxic material and die within a few minutes. • Signs observed include dyspnoea, restlessness, moaning, recumbency and terminal clonic convulsions with opisthotonus. • Mucosae are bright red in colour.
82
Hydrocyanic Acid Pathology
83
Hydrocyanic acid poisoning treatment
Treatment • Hydrocyanic acid is taken up by thiosulphate to form thiocyanate which is not toxic and is readily excreted. • Sodium thiosulphate should also be given orally to fix the free hydrocyanic acid in the rumen
84
Poisoning with OPs signs and symptoms
Organophosphate compounds and carbamates inactivate cholinesterase and produce a syndrome of salivation, diarrhoea and muscle stiffness indicative of stimulation of the parasympathetic nervous system.
85
Poisoning with OPs Treatment
86
Urea Poisoning
Urea poisoning • Urea is used as a feed additive for ruminants as a source of non protein nitrogen and as a fertiliser • Urea should not constitute more than 3% of the concentrate ration of ruminants. • Toxic affects are due to the sudden production of large quantities of ammonia and signs occur within 20 to 30 minutes of feeding.