Respiratory Surgery Flashcards

(44 cards)

1
Q

Laryngeal collapse: Cx

A

Stertor (expiratory); Stridor (Inspiratory); Exercise intolerance; Coughing; Regurg and vomiting

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2
Q

Tracheal obstruction: Cx

A

Cough; Dyspnea; Cyanosis; Collapse

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3
Q

Tracheal obstruction: Tx

A

Resection and anastomosis; Tracheostomy

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4
Q

What is the main surgical treatment for lung lobe consolidation, bronchiectasis; lung lobe torsion or pulmonary neoplasia?

A

Partial or complete lung lobectomies

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5
Q

What are the four main components of BOAS?

A

Stenotic nares; Elongated, thickened soft palate; Everted laryngeal saccules; Hypoplastic trachea; (also can have abherent turbinates; lorg tongue; tracheal collapse)

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6
Q

List at least 3 clinical signs of BOAS

A

Stertor; Obstructive sleep apnea; Heat intolerance; Cyanosis and collapse; GI Signs (difficult swallowing, regurg, reflux, hiatal hernia)

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7
Q

How do we diagnose BOAS?

A

Typical way is to evaluate the dog’s breathing at rest and exercise (walk), and do a sedated oral and laryngeal exam, based on these parameters we can assign a severity score; CT?radiology can be nice for issues like tracheal/bronchial hypoplasia, collapse, hiatal hernias, aspiration pneumonia

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8
Q

Medical management of BOAS

A

Weight management; Avoid overheating

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9
Q

List 5 surgical procedures we can do to manage BOAS

A

Rhinoplasty; Endoscope-guided turbinectomy; Shorten soft palate (e.g. Staphylectomy); Partial tonsillectomy; Laryngeal sacculectomy

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10
Q

Prognosis of BOAS

A

The more severe, the worse the prognosis

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11
Q

What does post-operative management look like for dogs with BOAS?

A

Susceptible to post-operative airway inflammation or pneumonia so address w/ supplemental oxygen, anti-inflammatories and possible a tracheostomy

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12
Q

Pathomechanism of Laryngeal Paralysis (LarPar)

A

Idiopathic disorder typically in older large breed dogs (e.g. Labs and Goldens) where there is damage to the recurrent laryngeal nerve/caudal laryngeal nerve leading to failure of the cricoarytenoideus dorsalis muscle to abduct the arytenoids. Typically bilateral

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13
Q

GOLPP

A

Geriatric Onset Laryngeal Paralysis and Polyneuropathy

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14
Q

Three components of GOLPP

A

Laryngeal paralysis; Radial nerve dysfunction; Tibial nerve dysfunction

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15
Q

Clinical signs of Laryngeal Paralysis

A

Change in phonation; Gagging; Exercise intolerance; Laryngeal stridor (during inspiration the arytenoids and vocal folds get pulled into the larynx closing the airway); Cyanosis; Dyspnea

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16
Q

How is laryngeal paralysis diagnosed?

A

Radiographs to rule out other causes and look for supportive pathology such as aspiration pneumonia, non-cardiogenic pulmonary edema and megaesophagus); Laryngoscopy under light anaesthesia

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17
Q

How would you address an emergency case of LarPar?

A

Cooling, oxygen therapy and anxiolytics +/- emergency intubation or tracheostomy

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18
Q

For mild cases of LarPar, medical management solely tends to be appropriate. Describe medical management of this condition

A

Weight management, stress reduction, exercise restriction and avoiding heat. Warn owners that it will progress and can cause an emergency

19
Q

For moderate to severe cases of LarPar, how can we surgically manage the condition? What is a drawback?

A

Unilateral cricoarytenoid lateralization (“tie-back”); risk of aspiration (no swimmy :( )

20
Q

Prognosis of LarPar

A

Fair to poor (worsens as disease progresses)

21
Q

For tracheal collapse, which type of stenting is preferred, intraluminal or extraluminal?

A

Intraluminal (easier, and less severe complications especially in dogs where sx approach could be difficult)

22
Q

Does stenting a tracheal collapse stop the dog from coughing?

23
Q

Provide at least three causes of a spontaneous pneumothorax

A

Pulmonary emphysema; Pulmonary neoplasia; Chronic pneumonia; Migrating plant material; Asthma; Lungworm; Heartworm

24
Q

Clinical signs of spontaneous pneumothorax

A

In moderate to severe cases, we get restrictive breathing, hypoventilation, diminished lung sounds and respiratory distress

25
Diagnosis of pneumothorax
US and PE to confirm presence of pneumothorax
26
Four treatments for spontaneous pneumothorax
Thoracocentesis; Thoracotomy + continuous suction; Autologous blood patch (basically using animal's own blood, injecting it into the pleural space, and incuding the formation of a fibrin clot); Surgery
27
Pathomechanism of pyothorax
Accumulation of septic purulent fluid in the pleural space
28
Clinical signs of pyothorax
(May not be evident): tachypnea; dyspnea; cough; lethargy; weight loss; anorexia; restrictive breathing pattern
29
Treatment of pyothorax
Typically, thoracocentesis or chest tube to drain +/- lavage + antibiotics (Fluoroquinolone and penicillin or clindamycin)
30
Diagnosis of a pyothorax
Thoracocentesis; Cytology and culture
31
When would surgical management of a pyothorax be indicated? What does this involve?
If you have failure of medical management (3-7d) or complications with the thoracostomy tube; remove inciting cause of pyothorax
32
Prognosis of pyothorax
Guarded to good; Dogs tend to do better than cats with surgery
33
Pathomechanism of a diaphragmatic hernia
Trauma leads to deatchment/rupture of part of the diaphragm from the body wall, allowing abdominal organs (liver most often) to enter the thoracic cavity
34
Clinical signs of a Diaphragmatic hernia
Respiratory distress; Exercise intoelrance; Muffled heart and lung sounds: +/- borborygmi
35
Diagnosis of a diaphragmatic hernia
radiographs; Ultrasound
36
How are patients with diaphragmatic hernias treated?
Taken into surgery ASAP once stabilized; hernia is reduced via an abdominal approach while patient is on ventilatory support
37
Briefly describe diaphragmatic hernia reduction surgery
Gently reduce the abdominal contents back into the abdomen, breaking adhesions where possible; Close opening dorsal to ventral using a continuous pattern +/- surgical mesh if chronic; Place thoracostomy tubes
38
Prognosis of diaphragmatic hernia patients that receive surgeru
80-90% survival to discharge
39
True or false. Death w/in 24 h of DH reduction is typically due to issues related to the GI tract.
False. Death w/in 24 h of DH reduction tends to be due to either pulmonary or cardiac dysfunction. Following 24hj, death typically due to GI damage or an unrelated issue
40
True or false. Diaphragmatic hernias and peritoneopericardial diaphragmatic hernia are acquired.
False. PPDH are congenital while DH are acquired
41
PPDH
Opening in the ventral diaphragm which allows the pericardial sac and peritoneal cavity to interact
42
Clinical signs of PPDH
Muffled heart sounds, Boroborygmi in the thoracic cavity; Dyspnea; Cardiac dysnfunction
43
Diagnosis of PPDH
radiographs; Ultrasound
44
Surgery used to correct PPDH
Midline celliotomy (may have to enter pleural space and pericardium)