Reverse Test 1 Flashcards by user delete

Failure of processes to merge. Prevelance: Native American, Asian, White, Black. Lateral: Maxillary and Mandibular. Oblique: Lateral nasal process and Maxillary. Cleft Lip: Maxillary and medial nasal. Median Cleft: Two medial nasal processes. CP= failure of palatal shelves to fuse (bifid uvula is minimum manifestation) if syndromic its CP only, if nonsyndomic (more common) its CP and CL. Submucosal cleft= palate shelves get close enough that the mucosa cover it but bones aren’t fused.

Cleft Lip and Palate (cause, prevalence):

How well did you know this?

Not at all

Perfectly

CP, Mandibular Micrognathia, glossoptosis

Peirre Robin Sequence

How well did you know this?

Not at all

Perfectly

Corners of the mouth, on vermillion border

Commissural lip pits

How well did you know this?

Not at all

Perfectly

Symmetrical just off center pits on the lower lip. Van der woude

Paramedian lip pits

How well did you know this?

Not at all

Perfectly

Double lip, blepharochalasis, nontoxic thyroid enlargement

Ascher syndrome

How well did you know this?

Not at all

Perfectly

ectopic sebaceous glands. Raised yellow papules

Fordyce granules

How well did you know this?

Not at all

Perfectly

White appearance on buccal mucosa, disappears when stretched and comes back when released

Leukoedema

How well did you know this?

Not at all

Perfectly

big tongue, beckwidth-weiddermans syndrome. Caused by 1) muscular hypertrophy 2) vascular malformations 3) other (lymphangioma, downs syndrome ect)

Macroglossia

How well did you know this?

Not at all

Perfectly

big thyroid in back of throat. Between foramen cecum and epiglottis. Has difficulty swallowing (dysphagia) talking (dysphonia) breathing (dyspnea). Dx by iodine/technetium-99/ct/mri

Lingual Thyroid

How well did you know this?

Not at all

Perfectly

PMN swelling on tongue. White/yellow serpentine lines that move around. Erythema migrans when not on tongue

Geographic tongue:

How well did you know this?

Not at all

Perfectly

AKA scrotal tongue. Deep fissured tongue. Pts complain of bad breath, burning, and bad taste

Fissured tongue

How well did you know this?

Not at all

Perfectly

Overgrowth of coronoid. Deviates to ipsilateral side. Typically only during open

Coronoid hyperplasia

How well did you know this?

Not at all

Perfectly

Overgrowth of the condylar process. Deviates to contralateral side. Even at rest

Condylar hyperplasia

How well did you know this?

Not at all

Perfectly

Saliva gland gets in mandible formation, radiopacity below the mandibular canal “lingual mandibular salivary gland depression” submandibular gland (mostly serous with some mucins)

Stafne defect

How well did you know this?

Not at all

Perfectly

pathologic cavity lined by epithelium. Typically fluid filled and continue to grow bc of hydrostatic pressure

Cyst

How well did you know this?

Not at all

Perfectly

radiolucency in the anterior maxilla. Commonly is radicular cyst or periapical granuloma. Usually in the lateral incisor area.

Globulomaxillary radiolucencies

How well did you know this?

Not at all

Perfectly

incisive canal cyst” cyst below the incisive papilla. Duct should be less than 6 mm wide.

Nasopalatine duct cyst

How well did you know this?

Not at all

Perfectly

has dermis structures in the lining (hair, and sebaceous glands)

Dermoid cyst

How well did you know this?

Not at all

Perfectly

FOM (most common), ventral tongue, soft palate. waldeyers ring (palatine tonsils, lingual tonsils, pharyngeal adenoids) White/yellow asymptomatic mass less than 1 cm. has lymph tissue in it.

Lymphoepithelial cyst

How well did you know this?

Not at all

Perfectly

atrophy on one side of the face. Possible hx of trauma. Lyme disease can be a big cause. Look at tongue

Progressive hemifacial atrophy

How well did you know this?

Not at all

Perfectly

downward slant of lateral palpebral fissures. Mouth is trapezoid shape. 75% have cleft soft palates or bifid uvula. Syndactyly (malformation of limbs)

Apert syndrome

How well did you know this?

Not at all

Perfectly

defects of 1 and 2 branchial arches. Coloboma (notch on outer portion of eyelid) hypoplastyic zygomatic arch. micrognathia

Mandibulofacial dysostosis/treacher-collins syndrome

How well did you know this?

Not at all

Perfectly

periapical inflammation of the primary tooth

Turner’s hyperplasia/turner’s tooth

How well did you know this?

Not at all

Perfectly

hutchisons incisors, mulberry molar, interstitial keratitis (blindness), 8 nerve deafness

Congenital syphilis

How well did you know this?

Not at all

Perfectly

Attrition: tooth on tooth, Abrasion: tooth on not tooth. Erosion: chemical (perimolysis is erosion caused by gastric secretions)Abfraction: loading causes cervical notches

Post developmental loss of tooth structure (attrition, abrasion, erosion, abfraction):

one or more missing teeth. anodontia: no teeth oligodontia: six or more missing teeth. If missing a primary tooth you will most likely miss permanent tooth

Hypodontia

more than normal teeth. Mesiodens= extra tooth between centrals.

Hyperdontia

Count teeth and normal number but one is big and enarged

Gemination

Two teeth have fused together

Fusion

tooth fusion on the cementum. Extractions are hard, need to do both

Concrescence

curved root

Dilaceration

): tooth inside a tooth. Deep invagination of a tooth that is lined by enamel. Most common is maxillary lateral incisors. radiograph don’t be confused with pulp champber

Dens-ino-dente (dens ivaginatus

most common is mandibular premolar: tooth on a tooth

Dens evaginatus

lowered pulp chambers. Look like mermaid fins 1) klinefelters 2) tricho-dento-osseus 3) amelogenesis imperfecta

Taurodontism

radiographically- sharp pointy teeth with thin enamel

Amelogenesis imperfecta

inadequate deposition of enamel. Pocketed enamel

hypoplastic

laid down properly but doesn’t mature: snow capped

hypomaturation

doesn’t mineralize properly. Brown and yellow. Flakes off easily

hypocalcified

mutation of DSPP. Blue tinted teeth. No pulp in radiograph, bulbous crowns and thin roots

Dentinogenesis imperfecta

rootless teeth

Dentin dysplasia (type I

hotdog on a stick pulp and blue tint

dentin dysplasia type II

non hereditary. Ghost teeth. Shell of teeth

Regional odontodysplasia

sinus tract (intraoral and cutaneous). 2) osteomyelitis (infection of bone. Causes sequestra) 3) cellulitis (infection of soft tissue) 4) condensing osteitis (localized area of bone sclerosis. RO area by roots)

Sequelae of periapical pathology

Radiolucent area by root tip. On histo filled with granulation tissue ( lymphocytes, plasma cells-purple and blue with pink)

Periapical granuloma

RL filled with pus (PMN all pink)

Periapical abcess

has epithelium around it (“spider web” looking thing)

Periapical cyst

expanding lytic destruction, suppuration, sequestra formation, invulcrum (necrotic bone surrounded by healthy bone)

Osteomyelitis

cellulitis in the neck (mandibular tooth [molar])

Ludwig’s angina

cellulitis in the upper face (maxillary tooth [molar])

Cavernous sinus thrombosis

bright red, hypersensitivity reaction (big red gum causes this- cinnamaaldehyde)

Plasma cell gingivitis

fiery red, bleeds easily, epithelial cells spread apart

Localized juvenile spongiotic gingival hyperplasia:

red, raw nasty looking, can pull gums apart with minor force. Ddx: lichen planus, mucous membrane pemphigoid, pemphigoid vulgaris, systemic lupus erythemous, hypersensitivity

Desquamative gingivitis

cyclosporine (25%), phenytoin (highest percentage 50%), nifedipine (25%)

Drug-related gingival hyperplasia

tissue covers a tooth- operculum

Pericornitis

cathepsin c gene, accelerated periodontitis, A.A., palmar plantar keratosis

Papillon-Lefevre syndrome

honey colored crusts. Strep pyogenes or staph aureus

Impetigo

disseminated group A beta hemolytic strep. 2 days of white strawberry tongue then red strawberry tongue desquamation in 3-8 weeks. Pastias lines: rash in areas where pressure and skin folds are

Scarlet Fever

cornebacterium , humans are sole resevoirs

diptheria

treponema pallidum. Primary: painless chancre. Secondary: painless lymphadenopathy, rash tertiary: latent

Syphilis

neisseria

Gonorrhea

mycobacterium, acid fast, aerosol dispersed. Lupus vulgaris (TB of skin) scrofula (tb from contaminated cows milk)

Tuberculosis

isreali: sulfer granules. Mandible. Can occur in tonsillar crypts, plaque, and carious dentin.

Actinomycosis

bartonella henselae. Self resolves in four months

Cat-scratch disease

thrush

Psuedomembranosis candidia

bald pink symmetrical circle on posterior dorsal tongue

Median rhomboid glossitis

median rhomboid glossitis and infection elsewhere (angle of mandible)

Chronic multifocal candidiasis

red rash on corners of mout. Candidiasis and staph aureus

Angular chelitis

infection under a denture- maxillary

Denture stomatitis

antibiotic sore mouth

Acute atrophic candiasis

candida leukoplakia- white that won’t come off. Anterior buccal mucosa

Chronic hyperplastic

APECED. Endocrine

Mucocutaneous candida

most common in US. capsulatum, ohio and Mississippi river valley. Bird and bat excrement. Mobile and spongey

Histoplasmosis

dermatitidis. Us and Canada. Breathe ins pores after rain. Can introduce a psuedoepitheliomatous hyperplasia (benign reaction in epithelium that looks like cancer)

Blastomycosis

brasiliensis: armadillo. Mickey mouse or mariners wheel organisms

Paracoccidioidomycosis

immitis. San joaquin river valley fever- SW and mexico. Bag of marbles on histo slide. Flu like symptoms. Arthrospores from mold

Coccidioidomycosis

neoformans: pigeon excrement. Mucopolysacharide capsule that protects it

Cryptococcosis

mucormycosis: enhanced by iron. Rhinocerebral form is what dentists worry about. Diabetes at risk nasal infection, facial pain, visual disturbances. Black necrotic tissue in middle of face

Zygomycosis

second in frequeny to candidiasis. Antrolith if calcified. A. fumigatus and a. flavus. Surgically debride

Aspergillosis

Reverse Test 1 Flashcards

(78 cards)