RRAPID - C Flashcards

(31 cards)

1
Q

Causes of circulatory problems

A

Primary cardiac = MI, ischaemia, HF, tamponade

Secondary = asphyxia, hypoxia, hypothermia, sepsis, shock

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2
Q

How do circulatory problems kill?

A

Cardiac arrest

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3
Q

Definition of shock

A

State of tissue hypoperfusion

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4
Q

What is stroke volume?

A

Volume of blood ejected by ventricle per heartbeat

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5
Q

What is preload?

A

Volume of blood returning to ventricle during diastole

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6
Q

What is Starling’s law?

A

Force of myocardial contraction is proportional to initial myocardial fibre length

Means larger the preload = larger the end diastolic volume = bigger the stretch of myocardial fibres = bigger the force of contraction = bigger the stroke volume

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7
Q

What is myocardial contractility?

A

Force with which myocardium contracts

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8
Q

What increases contractility?

A

Increasing preload or sympathetic nervous system activity

Adrenaline or inotropic drugs

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9
Q

What decreases contractility?

A

Hypovolaemia, cardiac disorder, hypoxia, hypercapnoea, acidosis, electrolyte disturbances

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10
Q

What is afterload + what is the consequence of increased afterload?

A

Tension in L ventricle during systole. 1 component is systemic vascular resistance

Increased afterload = increased myocardial work = decreased stroke volume

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11
Q

What is systemic vascular resistance?

A

Resistance of arteries to flow of blood
Directly proportional to BP
Increases with age leading to HTN

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12
Q

What is cardiac output?

A

SV X HR

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13
Q

What is MAP?

A

Mean arterial pressure = CO X SVR

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14
Q

5 categories of shock

A
Hypovolaemic 
Vasodilatory (septic, anaphylactic)
Cardiogenic 
Obstructive (PE, tamponade, tension pneumothorax)
Neurogenic
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15
Q

What type of shock affects preload?

A

Hypovolaemia

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16
Q

What type of shock affects cardiac contractility?

A

Arrhythmias
Congestive HF
Obstructive causes

17
Q

What type of shock affects afterload?

A

Sepsis
Anaphylaxis
Neurogenic

18
Q

Early signs of septic shock

A

Hypotension
Bounding pulse
Low urine output

19
Q

Presentation of cardiogenic shock

A

Hypotension
Raised JVP
Raised SVR
Signs of pulmonary oedema

20
Q

Signs of neurogenic shock

A

Due to spinal transection
Raised JVP
Hypotension + bradycardia

21
Q

S+S shock (+ which are early + late)?

A

Tachycardia - EARLY
SOB - EARLY

Cold peripheries 
Prolonged CRT
Reduced JVP 
Poor urine output 
Agitation 

Hypotension - LATE
Lactic acidosis - LATE

22
Q

presentation + signs of hypovolaemic shock

A
Thirsty 
Cold + clammy 
Decreased CRT 
Raised HR, low BP 
Reduced JVP
23
Q

How to assess C

A
Feel peripheries 
CRT
PR 
BP
O2 sats 
RR 
Urine output 
ABG
24
Q

How to assess fluid status?

A
CRT 
PR 
BP 
JVP 
Skin turgor (over clavicle) 
Heart sounds 
Lungs (pulmonary oedema)
Peripheral oedema 
Urine output
25
What 5 questions to consider for cause of circulatory disease?
1) Are they shocked? 2) Signs of hypovolaemia? 3) Cause of hypovolaemia? 4) evidence of sepsis? 5) evidence of cardiogenic cause?
26
What to give in a fluid bolus?
500ml (250ml for HF) of Hartmanns or saline if K >5.5, rhabdomyolysis, AKI
27
What is the definition of SIRS?
Temp <36 or >38 HR >90 RR >20 or CO2 <4.3 WCC <4 or >12
28
What is the definition of sepsis + severe sepsis?
Sepsis = SIRS secondary to infection | Severe sepsis = sepsis + organ dysfunction, hypotension or hypoperfusion
29
What is the definition of septic shock?
Sepsis + hypotension despite fluids
30
What is the definition of narrow + broad complex tachycardias
QRS <0.12 | QRS >0.12
31
What are the signs that indicate patients with arrhythmia are unstable?
Shock Syncope MI HF