S8. headache from quizlet Flashcards

1
Q

What are the two types of headache?

A
  1. Primary headache due to a headache disorder and is usually non life/site threatening
  2. Secondary due to another condition which is less common but can potentially be life/site threatening
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2
Q

What are some primary headaches?

A

Tension type headaches
migraine
cluster headache

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3
Q

What are some secondary headaches?

A

-life threatening eg tumour, haemorrhage, meningitis

  • Site threatening eg giant cell ateritis and closed angle glaucoma
  • non life threatening such as medication overuse, sinusitis, trigeminal neuralgia
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4
Q

How do we take the history of a headache? -

A

History of presenting complaint eg SQITARS

  • Past medical history
  • Drug history
  • Family history
  • Social history eg stress, lack of sleep, alcohol and caffeine
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5
Q

How do we remember the red flags for headache and what do they stand for?

A

SNOOP
S-systemic signs and disorders
N-Neurological symptoms
O- onset new or changes symptoms in patients over 50
O- onset in thunderclap
P-papilledema, positional provocation, precipitated by exercise

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6
Q

What clinical examination do we carry out for headache?

A
  • Vital signs
  • Neurological exam including cranial and peripheral nerves, GCS
  • Other relevant systems from the history
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7
Q

What are the most common types of headache?

A

Tension type headache, Migraine, Medication overuse, Cluster headache

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8
Q

What is the pathophysiology of a tension type headache?

A

is more common in females and more common in younger patients and is thought to be due to tension in the muscles of the head and neck

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9
Q

How does a tension type headache present? -

A

tight/band like

  • Generalised pain commonly in frontal and occipital - radiate into the neck a
  • mild/moderate intensity
  • responding to simple analgesics
  • pain worst at the end of the day
  • recurrant lasting 30- 60 minutes
  • aggrivated by stress, poor posture and lack of sleep
  • few associated symptoms with normal neurological exam
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10
Q

What is the pathophysiology of migraines?

A

more common in females presenting in early to mid life with most having a first attack by 30, unclear how it comes about but may be due to:
-neurogenic inflammation of CNV sensory neurones innervating the large vessels and meninges altering how the brain processes pain, sensitising us to normally ignored stimuli

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11
Q

What is the presentation of migraines?

A
  • Unilateral temporal or frontal pain
  • throbbing and pulsating,
  • moderate to severe
  • disabling
  • respond to simple analgesia but may need triptans
  • prolonged lasting between 4-72 hours
  • Triggers- food, mesntrual cycle, stress
  • Aura, photo and phono phobia but a normal clinical examination between episodes
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12
Q

What is the pathophysiology of a medication overuse headache?

A

more common in middle aged females and occurs in patients with a pre-existing headache disorder who are using regular anaglesics for more than 10days/month leading to upregulation of pain receptors so the headache does not respond- cure by discontinuing medicine

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13
Q

How does a medication overuse headache present?

A
  • Present on at least 15 days/month with variable character

- co-exists with depression and sleep disturbance

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14
Q

What is the pathophysiology of cluster headaches?

A

more common in males with a history of smoking occuring between 30-40 years and may be due to hypothalamic activation with trigeminal autonomic involvement

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15
Q

How do cluster headaches present?

A
  • unilateral sharp, stabbing intense disabling pain around the eye with the patient being agitated and usually lasting 15 mins-3 hours with periods of remission
  • triggers- alcohol, cigarretes, smells, temp
  • analgesia ineffective and oxygen and triptans are used
  • ipsilateral autonomic symptoms eg red watery eye, ptosis (may be seen on exam during an attack)
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16
Q

How does a headache due to a space occupying lesion present?

A
  • gradual headache which is progressive in severity and is worse in the mornings and worsend with leaning forwards, cough, valasava manouver
  • analgesia may be effective in early stages
  • nausea and vomiting, focal neurological and visual symptoms, papilloedema
17
Q

What is the pathophysiology of trigeminal neuralgia?

A

more common in females presenting between 50-60 years due to compression of CNV by a loop of a blood vessels (or possibly due to tumour or AV malformations)

18
Q

What is the presentation of trigeminal neuralgia?

A
  • unilateral pain felt in at least 1 division of the CNV as a sharp, electric shock which is severe and lasts seconds to minutes with a sudden onset
  • precipitated by light touch, eating, cold, combing hair and may have preceding symptoms such as tingling or numbness
  • simple analgesia not effective
19
Q

What is the pathophysiology of temporal arteritis?

A

vasculitis of the large and medium sized arteries of the head, most commonly the superficial temporal artery, presenting most commonly in females over 50
CONSIDER IN ANYONE OVER 50 WITH SUDDEN ONSET, VISUAL DISTURBANCE AND JAW CLAUDICATION

20
Q

What are the risks of temporal arteritis?

A

-risk of irreversible vision loss due to involvement of blood vessels supplying CN II