sakit sa ulo Flashcards

1
Q

define headache

A

pain or discomfort over upper head - orbits to suboccipital

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2
Q

general mechanisms of headache

A

traction, distention or dilation of intracranial vessels

inflammation near pain sensitive structures

direct pressure on cranial/cervical nerves

sustained contraction of scalp or neck muscles

stim from disease of EEN and sinuses

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3
Q

give pain sensitive structures

A

cranial venous sinus

arteries at base of brain and dura

dura near base of brain and large arteries

all extracranial structures

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4
Q

give pain insensitive structures

A

brain parenchyma

ependyma

choroid pleus

pia, arachnoid and dura over convexity

skull

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5
Q

what is brain parenchyma

A

brain functional tissues in the brain made up of neurons and glial cells

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6
Q

what is ependyma

A

membrane of glial cells lining the ventricles and central canal of the spinal cord

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7
Q

discuss primary HA

A

no underlying cause

migraine, tension and cluster

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8
Q

diff migraine HA vs tension-type HA

A

migraine: pain, nausea and visual changes on one side and loc varies

tension-type - like a band squeezing head (B); may radiate to neck and shoulder

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9
Q

discuss cluster HA

A

pain is in and around one eye

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10
Q

discuss secondary HA

A

with an underlying cause:
- CNS infection
- neoplasm
- CV diseases
- sinusitis

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11
Q

discuss sinus HA

A

pain is behind brow bone and/or cheekbones

frontal and maxillary sinuses

triggered by sinusitis - inflammation of sinuses

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12
Q

what is the most common type of primary HA

A

tension but migraine is often discussed

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13
Q

importance of HA to neuro diagnosis

A

HA alarms - may be considered life threatening

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14
Q

HA red flags

A

sudden or new onset

wakes pt from sleep

assoc c/:
- vomiting
- visual sx
- weakness/numbness
- meningeal irritation

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15
Q

HA diagnostic alarms

A

onset after 50 yo

sudden on set

inc freq and severity

new onset c risk for HIV/cancer

assoc c systematic illness

altered consciousness or focal neuro deficits

papilledema

significant trauma

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16
Q

danger signs of HA

A

severe and progressive

alteration of consciousness; sudden explosive

fever and neck rigidity

double vision or CN involvement

papilledema

other focal neuro deficits

17
Q

exp vascular theory of migraine

A

vasoconstriction - reduced BF - cerebral hypoxia - abnormal brain activity - aura - HA

18
Q

exp neuronal/nuerogenic inflammatory theory

A

CN V initiate and promote tissue inflaam - TGMV conveys pain = vasodialtion and local edema = pain

19
Q

discuss neurovascular theory

A

trigger factors can stim diff structures - activates certain parts = vasocontriction - lack of O2, abnormal fucntion and aura = HA

also activates CN V = inc BV permeaability to proteins - build up of fluid - inflamm = turnover of 5-HT

depletion of 5-HT = HA

20
Q

discuss the migraine pathophysiology

A

HA is both vascular and neural problem

lack of BF and disturbance of serotonergic system

21
Q

emotion and stress triggers

A

cerebral cortex

22
Q

thalamus is triggered by

A

bright lights, noises and smells

23
Q

hypothalamus is triggered by

A

internal bodily environment and mechanisms

24
Q

vasodilator triggers

A

carotid vessels

25
Q

migraine vs tension HA in terms of duration

A

migraine: 5 attacks lasting 4-72 hrs

tension: 10 attacks lasting 30 mins-7days

26
Q

migraine vs tension HA in terms of character

A

migraine: unilat, throbbing, mod-severe; inhibits/prohibits work and worsened by activity

tension: bilat, not throbbing, mild-mod; no effect or may inhibit work and not worsened by activity

27
Q

migraine vs tension HA in terms of assoc sx

A

migraine: nausea, photophobia and phonophobia

tension: (-) nausea; but can have photophobia or phonophobia or neither