schizophrenia Flashcards

(44 cards)

1
Q

family therapy

A

-based on idea that family dysfunction can play a role in development of Sz
-due to altering relationships and communication patterns within dysfunctional families
-works by reducing expressed emotion and stress levels which may contribute to patients risk of relapse

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2
Q

how does family therapy work

A

MAIN AIM TO REDUCE LEVELS OF EXPRESSED EMOTIONS/STRESS BY:
-improving families beliefs about and towards Sz
-reducing the stress of caring for a relative with Sz
-decreasing feelings of guilt
-helping family members achieve balance between caring for individual and living their own life

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3
Q

what is positive symptoms on Sz

A

an additional experience beyond those of ordinary existence

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4
Q

what is negative symptoms of Sz

A

a loss of usual abilities and experiences

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5
Q

example of positive symptoms of Sz

A

hallucinations and delusions

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6
Q

what are hallucinations

A

-unusual sensory experiences that have no basis in reality
-can effect any sense eg auditory/visual hallucinations

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7
Q

what are delusions

A

-irrational/false beliefs that have no basis in reality
-e.g delusions of persecution (beliefs you are being harassed by government)
-e.g delusions of control (beliefs that you are being controlled by something external)

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8
Q

what is avolition

A

-severe loss of motivation to carry out everyday tasks
-andreason identified 3 signs of avolition: poor hygiene, lack of persistence in work and lack of energy

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9
Q

what is speech poverty

A

reduction in amount and quality of speech, usually accompanied by a speech delay or lack of fluency

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10
Q

diagnosis

A

-identification of a disorder through symptoms
-e.g hearing voices

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11
Q

classification

A

-criteria that is used to make a diagnosis
-e.g symptom of Sz is hallucinations

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12
Q

symptom overlap

A

-two or more conditions share similar symptoms e.g Sz and depression both involve negative symptoms

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13
Q

co-morbidity

A

-two illnesses occur at the same time e.g Sz and OCD diagnosed at same time as they share similar symptoms
-lead to misdiagnosis

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14
Q

genetic theory ao1

A

-suggests Sz is hereditary and passed on generations through genes
-a person is born with genetic predisposition for Sz
-believed several maladaptive ‘candidate’ genes e.g PCM1 are involved in increasing individual’s vulnerability to Sz
-Gottesman studied 40 twins and found the concordance rate for monozygotic twins was 48% but 17% for dizygotic twins.
-THEREFORE CLOSER THE GENETIC LINK, HIGHER LIKELIHOOD OF DEVELOPING SZ

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15
Q

brain structure or function

A

-enlarged ventricles
-meta analysis by raz and raz found over half of individuals with Sz had enlarged ventricles
-associated with damage to pre frontal cortex which leads to negative symptoms of Sz e.g schizophrenia

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16
Q

dopamine hypothesis

A

-neurotransmitters appear to work differently in the brain of a patient with Sz
-dopamine is widely believed to be involved as individuals with Sz release too much dopamine
-or have large amounts of D2 receptors on post SN

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17
Q

hyperdopaminergia in subcortex

A

-high dopamine activity in central areas of brain e.g broca’s area (responsible for speech production)
-associated with auditory hallucinations

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18
Q

hypodominergia in cortex

A

-low dopamine activity in prefrontal cortex
-associated with negative symptoms of Sz such as avolition

19
Q

biological explanation ao3

A

-biological determinism
-scientific methods
-alternative explanation (family dysfunction)

20
Q

family dysfunction

A

-idea that an individual develops Sz because they’ve been raised in a dysfunctional family environment
-dysfunctional in the way they communicate with each other as they have high levels of tension and arguments
-results in risk factors for development and maintenance of Sz

21
Q

schizophrenogenic mother

A

-idea that Sz is caused by individuals early experience of a schizophrenogenic mother
-a Sz mother is cold, controlling, rejecting and emotionally unresponsive
-builds a family environment based on tension and secrecy
-leads to distrust that later develops into paranoid delusions in Sz

22
Q

double bind communication

A

-bateson et al argues Sz is due to faulty communication patterns that exist within families
-occurs when parent communicates a verbal message which is not matched with physical behaviour
-child receives mixed messages
-conflicting, confusing forms of communication can lead to development of Sz
-child feels they can’t do the right thing, increasing anxiety, loses motivation leads to negative symptoms of Sz e.g avolition

23
Q

high expressed emotion

A

-high levels of expressed emotion towards Sz patient such as verbal criticism, hostility e.g anger and rejection and emotional over involvement
-cause stress in patient and can trigger onset Sz
-the stress caused is a primary explanation for relapse in patients with Sz as they’re placed back in a stressful environment causing relapse of positive/negative symptoms

24
Q

family dysfunction ao3

A

-RTS schizophrenogenic mother conducted by Mednick et al
-RTS double bind communication by Berger (DISCUSSION: retrospective data)
-practical applications: family therapy

25
meta representation
-cognitive ability to reflect on thoughts and behaviour -allows us to understand our actions and actions of others
26
dysfunction in meta representation
-disrupt our ability to recognise our own actions and thoughts as being carried out by ourselves and others -could explain auditory hallucinations as individuals can’t understand voices in head -lead to distress
27
central control
cognitive ability to supress automatic responses whilst performing a deliberate action instead
28
dysfunction in central control
-could explain speech poverty and thought disorder as individual is not able to suppress automatic thoughts and speech triggered by other thoughts/words spoken -can experience disrupted spoken sentences: derailment
29
cognitive explanation ao3
-RTS conducted by stirling et al -practical applications: CBT -alternative exp: biological explanation
30
drug therapy
-most common treatment for Sz -dosage is dependent on severity on psychosis -two types: typical and atypical
31
typical antipsychotics
CHLORPROMAZINE -dopamine antagonists -reduces dopamine activity in brain -bind to D2 receptors on post SN, reducing dopamine action -reduces positive symptoms e.g hallucinations -can be used as a sedative
32
atypical antipsychotics
CLOZAPINE -seretonin agonists -dopamine antagonists -binds to D2 receptors to reduce positive symptoms -acts as agonists upon seretonin receptor sites -reduces negative symptoms such as lack of emotions
33
drug therapy ao3
-require little motivation (unlike CBT) -negative side effects -RTS by thornley et al
34
aim of CBT
-aim is to help patients identify irrational/delusional thoughts and change them into more rational ones through empirical disputing
35
CBT process
-once irrational thoughts have been identified e.g… the psychiatrist would challenge the patients thoughts in order to encourage more rational ones -use of empirical disputing, asking for evidence -helps patients understand their delusions aren’t real -offer more rational explanations can reduce patients anxiety and help patients realise their delusions aren’t based in reality -positive self talk to challenge auditory hallucinations -teach self distraction strategies
36
CBT ao3
-RTS by jauhar et al -requires motivation and commitment -avoids chemical dependence (unlike drug therapy)
37
token economies
-behaviourist approach to manage behaviour of patients with Sz -mainly used with patients who have developed maladaptive behaviour e.g bad hygiene
38
token economies aim
-change a patients behaviour so they are easier to manage -patient will have a better quality of life -thus enabling them to live outside of a hospital setting
39
how do token economies work
-uses skinners operant conditioning principles -patients receive reinforcements in the form of token after producing a desired behaviour e.g self care -tokens can later be exchanged for goods or privileges
40
token economies ao3
-RTS by dickerson et al -ethical issues -does not actually treat Sz
41
interactionist approach
suggests Sz is developed through a combination of biological, psychological and social factors
42
explaining Sz: diathesis stress model
-meehls original diathesis stress model suggested that diathesis was entirely genetic -down to a single ‘schizo-gene’ -if you have the gene then stress through childhood e.g Sz mother could lead to Sz -now believed many genes increase genetic vulnerability to Sz -believed other factors can be a diathesis such as psychological trauma -cannabis use causes increased risk of Sz by up to 7 times due to its interference with dopamine system
43
treating Sz: diathesis stress model
-considers bother biological and psychological factors in development of Sz -therefore compatible with both biological and psychological treatments -combination of Sz and antipsychotics
44
interactionist approach ao3
-RTS explaining Sz comes from tienari et al -individual differences -RTS treating Sz comes from tarrier et al