Session 8: Conditions Affecting the Elbow Flashcards

1
Q

Common conditions affect the elbow:

A

Supracondylar fracture of the distal humerus Dislocated elbow Pulled elbow (nursemaid’s elbow) Radial head and neck fractures OA of elbow Rheumatoid Arthritis Lateral elbow tendinopathy (tennis elbow) Medial elbow tendinopathy (golfer’s elbow) Olecranon bursitis (student’s elbow) Rheumatoid nodules Gouty tophi Cubital tunnel syndrome

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2
Q

What is this?

A

A supracondylar fracture of the distal humerus.

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3
Q

Mechanism of injury of a supracondylar fracture

Usual age of injury

Usual gender

Symptoms

A

Usually from falling from a moderate heigh onto an outstretched hand with elbow hyperextended (child fallong off monkey bars e.g.)

Younger than 10 years of age (peak age is 5-7 years)

Males

Pain, deformity and loss of function.

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4
Q

What does a supracondylar fracture look like?

A

Flexed and distal fragment is displaced and protrudes posteriorly.

Proximal fragment might protrude anteriorly.

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5
Q

Three main complications of supracondylar fractures.

A

Malunion resulting in cubits varus. Also called gunstock deformity.

Damage to median nerve (most common), radial nerve or ulnar nerve.

Ischaemic contracture

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6
Q

Further explain ischaemic contracture.

A

Since the brachial artery passes close to the fracture site it can become damaged or occluded. If there are reflex spams of the collateral circulation around the elbow there will be ischaemia of the muscles in the anterior compartment of the forearm.

Results in oedema and a rise in compartment pressure (compartment syndrome). If left untreated muscle bellies will undergo infarction and can die. During repair phase the dead muscle tissue becomes replaced by scar tissue by fibrosis. Fibrotic tissue contracts reuslting in a flexion cotnracture known as Volkmann’s ischaemic contracture.

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7
Q

What does an ischaemic contracture leading to a Volkmann’s ischaemic contracture look like?

A

The wrist is typicall flexed and fingers are extended at metacarpophalangeal joints and flexed at interphalangeal joints. Forearm is pronated and elbow is flexed.

This is due to the fibrotic tissue contracting.

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8
Q

To minimise risks of these complications. What is done?

A

Prompt and thorough neurovascular examination. Any compromise to supply of forearm or hand will lead to emergency reduction and fixation of the fracture.

You examine by checking radial pulse, OK sign, paraesthesia and capillary refill.

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9
Q

What is this?

A

Dislocated elbow. Specifically posterior dislocation.

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10
Q

What type of elbow dislocation is most common?

A

Posterior dislocation (90%)

Anterior disclocation (10%)

It is called posterior because the distal fragment has displaced. It is named after the displacement of the distal fragment.

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11
Q

Mechanism of injury of posterior dislocation of elbow.

Age

Internal mechanism

A

FOOSH = fall on outstretched hand with the elbow partially flexed

Most common joint displacement in children (second most common in adults after shoulder)

Distal end of the humerus is driven trough the joint capsule anteriorly. Ulnar collateral ligament is usually torn and maybe also ulnar nerve damage.

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12
Q

Why is it more common to dislocated elbow if it is partially flexed?

A

Because when it is flexed most of the stability relies on the ligaments.

If it is extended fully most of the stability relies on the bones.

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13
Q

Mechanism of injury of anterior dislocation of elbow.

Associated fractures

A

Direct blow to the posterior aspect of a flexed elbow.

Fracture of olecranon due to high degree of force needed to dislocate the joint anteriorly.

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14
Q

What does a posterior dislocated elbow look like?

A
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15
Q

What is nursemaid’s elbow or pulled elbow?

A

It is also called subluxation of the radial head which is a partial disruption of a joint. Usually subluxation from anular ligament.

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16
Q

Mechanism of injury of subluxation of radial head.

Age

Symptoms

A

Longitudinal traction applied to arm with forearm pronated. For example grabing an uncooperative child or swinging child by their arms during play.

2-5 years of age.

Reduced movement of elbow and pain of lateral aspect of proximal forearm. Parents usually say that their child doesn’t use their arms.

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17
Q

Why doesn’t subluxation of the radial head usually occur <2 years of age?

Why is it less common as you get older?

A

Because most children don’t walk yet and there is no need to tug them to be cooperative therefore.

Neglection signs.

Because the annular ligament naturally strenghtens.

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18
Q

Why is subluxation of radial head more common during pronation of forearm?

A

Because the annular ligament is taut in supination and more relaxed in pronation.

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19
Q

What is this?

A

Radial head and neck fracture.

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20
Q

Mechanism of injury of radial head and neck fractures.

Age

Symptoms

A

FOOSH = fall on an outstretched hand when the radial head impacts on the capitellum of the humerus

Most common in adults (most common elbow fracture as well in adults)

Pain in lateral aspect of their proximal forearm and loss of range of movement. Swelling associated with fractures is usually modest in comparison with supracondylar fractures.

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21
Q

What are signs of radial head and neck fractures on an x-ray?

A

Can be difficult to see.

Fat pad signs also called sail sign which indicates effusion. Displaced fat looks black.

Haemarthrosis which is blood in the joint.

Sail sign is caused by displacement of the anterior fat pad.

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22
Q

Why is OA relatively uncommon in elbow?

A

Well-matched joint and strong stabilising ligaments. Less weak and tear and stable.

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23
Q

In what demographics is OA of elbow most common?

Symptoms

A

More in men than women (4:1)

Most common in manual workes and athletes who engage in sports involving throwing (baseball etc.)

Grating sensation (crepitus)

Swelling occurs late

Osteophytes can impinge on ulnar nerve causing paraesthesia and muscle weakness.

Stiffness and loss of extension.

24
Q

Mechanism of pathology of rheumatoid arthritis.

A

Autoantibodies (rheumatoid factor) attack synovial membrane. Inflamed synovial cells proliferate to form a pannus which penetrates through the cartilage and adjacent bone leading to joint erosion and deformity.

25
Q

Which joints does RA most commonly affect?

Complications.

Age

A

Metacarpophalangeal joints (MCPJ) and proximal interphalangeal joints (PIPJ) of the hands, the feet and cervical spine.

Leads to damage to other organs, including eyes, skin, lungs, heart and blood vessels and kidneys.

Commonly have anaemia of chronic disease.

Peak age of onset is 40-50 yrs of age.

26
Q

X-ray features of rheumatoid arthritis.

A

Joint space narrowing

Periarticular osteopenia

Juxta-articular bony erosions

Subluxation and gross deformity

27
Q

How is RA managed?

A

Usually medically rather than surgically by disease-modifying medication.

Surgery can be required in severe cases to relieve pain and improve mobility.

In this image you can see erosion of humeroulnar joint with most of the trocheal of the humerus having been destroyed. Sigmoid fossa (trochlear fossa) is enlarged. This patient may be a candidate for total elbow replacement.

28
Q

What is lateral elbow tendinopathy?

A

Tendinopathy (overuse) of the common extensor tendon at the lateral epicondyle.

29
Q

Causes of lateral elbow tendinopathy (Tennis elbow).

A

The extensor carpi radialis brevic (ECRB) helps stabilise the wrist when the elbow is straight like in a tennis groundstroke.

When ECRB is weakened from overuse microscopic tears form in the tendon where it attaches to lateral epicondyle.

Tennis players, painters, plumbers and carpenters are particularly prone to tennis elbow.

30
Q

Signs and symptoms of tennis elbow (lateral elbow tendinopathy)

A

Pain over the lateral epicondyle during extension of the wrist, especially if this is against resistance.

31
Q

Treatment of lateral elbow tendinopathy.

A

Modify their activities to give the tendon an opportunity to heal. 90% of patients recover within 1 year.

Sometimes physiotherapy and bracing are required.

Small number of patients need injections or surgery.

32
Q

What is medial elbow tendinopathy?

A

Also called Golfer’s elbow.

Affects the common flexor origin at the medial epicondyle.

10 times less common than lateral elbow tendinopathy.

33
Q

Causes of medial elbow tendinopathy.

A

Associated with golfing and with throwing sports that place valgus stress on the elbow. Also been reported in bowlers, archers and weightlifters.

34
Q

What is the most common site of pathology in medial elbow tendinopathy?

A

Interface between the pronator teres and the flexor carpi radialis (FCR) origins.

35
Q

Clinical presentation of golfer’s elbow.

A

Aching pain over the medial elbow often associated with the acceleration phase of throwing.

Pain is produced on resisted flexion or pronation of the wrist.

Ulnar nerve symptoms can occur and are present in about 20% of cases due to the proximity of the ulnar nerve running in the cubital tunnel close to the medial epicondyle.

36
Q

Treatment of golfer’s elbow.

A

Similar to lateral elbow tendinopathy (tennis elbow).

Modify their activities and promote rest.

Physiotherapy and bracing can be required and also surgery if persistent.

37
Q

Three common swelling of the elbow.

A

Olecranon bursitis

Rheumatoid nodules

Gouty tophi

38
Q

What is olecranon bursitis?

A

Inflammation of the olecranon bursa situated between the skin and the olecranon process of the ulna.

39
Q

Causes of olecranon bursitis.

A

It is sometimes called student’s elbow where repeated minor trauma leads to something bigger like leaning with their elbows on the desk for many hours whilst studying.

It can also be due to infection of the bursa like in septic bursitis following a minor penetrating injury to the elbow.

40
Q

What is the major difference between olecranon bursitis due to repeated minor trauma and infectious olecranon bursitis like due to septic bursitis?

A

Minor trauma: Serous fluid and swelling is soft, cystic and transilluminate

Infection: Pus fluid

41
Q

Clinical presentation of olecranon bursitis.

A

Usually present with cosmetic concern.

Unsightly swelling. Soft, cystic and transilluminate swelling.

42
Q

Treatment of olecranon bursitis.

A

Usually conservative with compression bandagin +- aspiration.

Hydrocortisone injection can be needed in chronic cases.

In case of infection treat the infection (antibiotics) + aspiration and compression. Occasional surgical drainage and washout will be needed to resolve the infection.

43
Q

What is this?

A

Olecranon bursitis

44
Q

What is this?

A

Rheumatoid nodules

45
Q

What are rheumatoid nodules?

A

Commonest extra-articular manifestation of rheumatoid arthritis and affect 20% of patients with RA.

46
Q

What risk factor is very major in developing rheumatoid nodules?

A

Smoking as they tend to have more aggressive joint disease. They are also more prone to other extra-articular manifestations of rheumatoid arthritis including vasculitis and lung disease.

47
Q

Where do you most often find rheumatoid nodules?

A

Usually occur over exposed regions that are usually subject to repeated trauma.

As well as affecting the elbow** region they can be seen in the **fingers** and **forearms** and occasionally over the **back of the heel.

They are usually non-tender although the overlying skin can occasionally ulcerate and become infected.

48
Q

What is this?

A

Gouty tophi

49
Q

What is gout?

A

An inflammatory condition resulting from defective purine metabolism leading** to an **increased production of uric acid.

When the uric acid concentration increases in the blood**, **supersaturation** and **precipitation** occur which will form **crystals of monosodium urate** in the **synovial joints, tendons and surrounding tissue.

The urate crystals lead to acute inflammation.

50
Q

Treatment of gout.

A

Treated medically with anti-inflammatory drugs** during the **acute phase.

When the acute phase is over and the gout has resolved, xanthine oxidase inhibitors such as allopurinol which is a purine analogue can be prescribed to reduce the production of uric acid and reduce the risk of further attacks.

51
Q

What is gouty tophi?

A

Tophi are nodular masses of monosodium urate crystals deposited in soft tissues due to gout.

They are late complication of hyperurciaemia and develop in over 50% of patients.

52
Q

Clinical presentation of gouty tophi.

A

Usually painless nodules but complications can include pain, soft tissue damage and deformity, joint destruction and nerve compression.

53
Q

Where can you most commonly find gouty tophi?

What do they look like?

A

Most common sites are the fingers and ears. Can also be found in the olecranon bursa and the subcutaneous tissues of the elbow.

They contain white pasty material and they enlarge. They work their way towards the skin surface to drain either forming a sinus tract or a continuously draining ulcer.

54
Q

What is cubital tunnel syndrome?

A

The ulnar nerve passes behind the medial epicondyle of the humerus to enter the forearm. Above the ulnar nerve as it passes through the cubital tunnel the flexor carpi ulnaris tendinous arch can be found.

The flexor carpi ulnaris muscle has two heads, one from common flexor origin on medial epicondyle and one from the medial margin of the olecranon. The two heads unite to form a tendinous arch.

The ulnar nerve passes beneath the tendinous arch to enter the cubital tunnel. This area forms a common site for ulnar nerve compression known as cubital tunnel syndrome.

55
Q

What is ‘catching your funny bone’?

A

Minor tauma to the ulnar nerve in the cubital tunnel.

Sharp transient pain radiating from the elbow to the cutaneous ulnar nerve territory.

56
Q

Signs and symptoms of cubital tunnel syndrome.

A

Paraesthesia in the cutaneous territory of the ulnar nerve 1 1/2 most medial digits as well as the medial palmar and dorsal surface of the hand.

May also result in weakness in the muscles supplied by the ulnar nerve.

57
Q

Treatment of cubital syndrome.

A

To decompress the nerve by surgically release it and transpose it anteriorly to the medial epicondyle.