Shock/ACLS/Diagnostics Flashcards

(85 cards)

1
Q

ABCDE algorithm

A

Airway
Breathing
Circulation
Disability
Exposure

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2
Q

How do I know the airway is adequate?

A

Patient is alert and oriented.
Patient is talking normally.
There is no evidence of injury to the head or neck.
You have assessed and reassessed for deterioration.

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3
Q

Signs and symptoms of airway compromise

A

High index of suspicion
Change in voice / sore throat
Noisy breathing (snoring and stridor)
Dyspnea and agitation
Tachypnea
Abnormal breathing pattern
Low oxygen saturation (late sign)

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4
Q

what is the most difinitive way to manage airway?

A

oral intubation

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5
Q

How to assess Breathing

A
  1. assess along with airway
  2. determine whether respirations are adequate
  3. determine if both lungs are working equally- auscultation, expansion, palpation, percussion
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6
Q

Percussion of pneumo vs hemo-thorax

A

hypo-resonant= hemothorax
hyper-resonance = pneumothorax

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7
Q

signs and symptoms of tension pneumothorax

A
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8
Q

ABCDE

Assess circulation

A

Ensure adequate tissue oxygenation and delivery and blood volume

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9
Q

What to do for circulation in trauma

A

STOP THE BLEEDING!
EXTERNAL HEMORRHAGE
Usually just need pressure
Coagulating agents (EX: Quikclot) may be helpful
Suture closed quickly (Don’t worry about cosmesis. You can re-do it later.)
Tourniquets on extremities. Try not to use for over 1 hour.

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10
Q

Blood pressure calculation

A

Systemic vascular resistence

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11
Q

What is the first factor to change in BP?

A

The SVR is the first thing to change. It will become higher.

Capillary refill can easily be assessed at the fingers or toes.
Delay of greater than 2 secondsshould be suspicious for blood loss, even if the BP is normal

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12
Q

Heart rate can be an indicator of hemorrhagic shock in about 50% of trauma patients

who are people that can fool you?

A

Children
Elderly—heart blocks, dysrhythmia, medication
Trained athletes
Pacemaker patients
Pregnant patients

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13
Q

when should you take manual BP?

A

All initial trauma BP should be MANUAL not automated.

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14
Q

what do you do if you cannot get pedal or radial pulse?

A

IO access - quickest is tibia

sternal and humeral also acceptable

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15
Q

what do you do if you give 1L of crystalloid (IV fluid) and the patient is still hypotensive

A

assume hemorrhage, GIVE WHOLE BLOOD

If you must givecomponent therapy, give 1:1:1 PRBC:FFP:PLATELETS

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16
Q

Permissive hypotension

A

just know basics for exam

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17
Q

What is a FAST

A

bedside echo

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18
Q

Sx of pericardial tamponade

A

Beck’s Triad is a common board exam question.
ECG Triad
POCUS Triad
Can occur with as little as 50-60cc of blood in the pericardium

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19
Q

Becks Triad

A
  1. hypotension
  2. JVD jugular vein distension
  3. muffled heart sounds
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20
Q

cardiac tamponade & pericarditis

ECG Triad

A
  1. Sinus Tachycardia
  2. low voltage
  3. electrical alternans
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21
Q

Causes other than hemorrhagic shock for hypotension

A
  1. neurogenic shock
  2. cardiogenic shock
  3. septic shock

slide 40

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22
Q

Trauma in Pregnancy

A

O negative mothers get RHOGAM

Fetal distress may be a sign of hemorrhage

Mothers will need a pelvic exam to check for blood. Possible premature labor and placental abruption.

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23
Q

ABCDE

Disability Assessment

A

Assess pupils
Level of consciousness
Response to stimuli
AVPU scale
Glasgow Coma Scale (GCS)

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24
Q

AVPU scale

A

Alert
Responsive to vocal stimuli
Responsive to painful stimuli
Unresponsive

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25
if you see tracheal deviation and diminished breath sounds what do you think of?
tension pneumothorax
26
what to do when GCS less than 8
If not already done, intubate Frequently repeat neuro exam and document CT scan ASAP Early neurosurgical consultation
27
# ABCDE Exposure | what to do
Remove all clothes & blankets Thorough physical exam Re-cover with warm blankets Prevent hypothermia
28
If unresponsive to 1000 cc IVF begin | intravenous fluid
blood transfusion
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Contraindications to NG tube placement in trauma
Severe midface trauma
30
# Distributive Shock General
Excess vasodilation and altered distribution of blood flow: decrease CO, decrease SVR, decrease PCWP 4 types
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# Distributive shock 4 types
Septic shock: look for signs infection; treat infection Anaphylactic shock: IgE mediated; look signs allgergic reaction; treat allergy Neurogenic Shock: acute spinal cord injury, anesthesia; fluids/pressors/steroids Endocrine shock: adrenal insufficiency; IV hydrocortisone- does not respond to fluids/pressors
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# Obstructive Shock general
Decrease blood flow due to physical obstruction of heart or great vessels- pressure decreases heart’s ability to pump blood
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# obstructive shock Management
Oxygen, fluid resuscitation, inotropic support, mechanical support ( IABP) Treat underlying cause: Pulmonary emboli: CT PE protocol; heparin, thrombolytics Pericardial tamponade: echocardiogram; pericardiocentesis Tension pneumothorax: xray; needle decompression Dissection: EFAST, CTA, TEE; surgical cx
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# Cardiogenic Shock general
Decrease cardiac output, decrease perfusion, increase systemic vascular resistance (often increase respiratory effort) Etiology: myocardial infarction, myocarditis, valvular disease, congenital heart disease, cardiomyopathy, arrhythmias
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# cardiogenic shock presentation
Decrease cardiac output (with preserved volume; CVP 8-12, PCWP > 15), hypotension, vasoconstriction ( increase SVR)
36
# Cardiogenic Shock Management
Oxygen, fluid resuscitation ( smaller amount bc not a volume problem), inotropic support (rx to increase cardiac output: dobutamine, epinephrine, amrinone), consider Intra-aortic balloon pump
37
# Hypovolemic Shock general
Loss of blood or volume (hemorrhage/volume loss) Etiology: GI bleed, AAA rupture, hemoptysis, trauma, ectopic pregnancy, postpartum hemorrhage, vomiting, bowel obstruction, pancreatitis, burns, diabetic ketoacidosis
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# hypovolemic shock presentaion
Tachycardia, hypotension, decrease urine output, pale cool skin/extremities, decrease capillary refill, poor skin turgor, dry mucous membranes, altered mental status (usually no assoc respiratory symptoms) Vasoconstriction (increased SVR), hypotension, decrease cardiac output and decrease pulmonary capillary pressure
39
# hypovolemic shock management
ABCDE, 2 large bore iv lines or central line, volume resuscitation, control source of hemorrhage, prevent hypothermia, treat coagulopathies Universal blood is O-negative Consider transfusion of whole blood
40
# Circulatory Shock general
In adequate perfusion due to low cardiac output or low systemic vascular resistance 4 types of circulatory shock Hypovolemic ( loss of blood or fluid) Cardiogenic ( reduced cardiac output) Obstructive (obstruction to circulation) Distributive (maldistribution of circulation)
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# Circulatory Shock Presentation of shock
Due to inadequate perfusion/oxygenation will see altered mental status, decrease peripheral pulses, tachycardia, cool/mottled skin, hypotension
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# circulatory shock Management
Airway: assess Breathing: reduce workload assoc with tachypnea ( +/- mechanical ventilation/sedation) Circulation: fluid resuscitation ( NS/LR vs whole blood), monitor urine output, HR, BP Delivery of Oxygen: ABGs, lactate levels Endpoint of Resuscitation: urine output, HR, BP
43
what does epi do
increases CO but decreases SVR systemic vascular resistance
44
# glossary of terms GDMT OMT EF HFrEF HFpEF HEmrEF RSVP
GDMT: Guideline-directed medical therapy OMT: optimal medical therapy EF: Ejection fraction HFrEF: Heart failure with reduced left ventricular ejection fraction (EF ≤0.40) HFpEF: Heart failure with preserved left ventricular ejection fraction (EF ≥0.50) HFmrEF: Heart failure with midrange ejection fraction (EF <0.50 but >0.40) RVSP: Right ventricular systolic pressure
45
what is normal EF
55-65%
46
Tests of perfusion
Treadmill ( bruce protocol) Treadmill with MPS Stress echo PET arteriogram | determine if the heart is getting enough O2
47
Plain stress test or exercise stress test
Information about perfusion, chronotropic competence, METs, arrhythmia, exercise tolerance look at ST on ECG - checking for blockage bruce protocol- start walking w specific increase in incline and speed on treadmill Lexiscan- medication you can give a pt that cannot ambulate to get HR up
48
chronotropic competence/incompetence
Incompetence is the inability to get HR up to expected max (age adjusted)
49
# stress test Instructions to patient:
hold BB/ CCB (diltiazem) 48 hours if treadmill, no caffeine 24 hours
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51
Patient presents to stress lab for stress test. bp 220/100 hr-125 rr-16.
cancel stress test
52
Patient presents to stress lab for plain treadmill r/o ischemia. Resting ECG reveals LBBB
contraindicated bc left bundle branch block could mask ST changes they would need a stress test with imaging
53
Patient presents for plain treadmill to evaluate for chronotropic incompetence. They did NOT hold their BB
stop test, reshcedule or switch to lexiscan
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Sress Echo
Information about heart valves and est RVSP (R ventricular systolic pressure) Can look for ASD/VSDs ( bubble study) test for perfusion limited by larger body habitus
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tests for anatomy | 6
Cardiac CTA Heart Catheterization Cardiac MRI 2-d echocardiogram (echo) Transesophageal echocardiogram (TEE) Cardiac MRA
56
Cardiac CTA | A=angiogram
A coronary computed tomography angiogram (CTA) uses advanced CT technology, along with intravenous (IV) contrast material (dye), to obtain high-resolution, 3D pictures of the moving heart and great vessels at time of test HR must be <60 - might give BB to pt must be able to hold breath for 20 sec intervals
57
cardiac CTA
57
Alternative to stress echo
MPS- but has radioactive dye?
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Heart Cath
Dye exposure LEFT heart cath they go into artery usually femoral or radial, then put puffs of due to look at all the arteries Right cath looks at hemodynamics of the heart, checks pressures of ventricles and atria, does not go into arteries
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Flow wire (FFR) heart cath
put wire in that checks pressure where there are lesions in vessels to it it is hemodynamically significant
60
Heart cath intravascular ultrasound (IVUS)
get US of actual lesion inside vessel/heart
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Left heart cath PROs
PRO: 99.9% accurate “the dye don’t lie” Can evaluate anatomy and perfusion, details of lesion Quick turn around/ immediate interpretation Final stop- can determine +/- “fix” while there
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What do you do for following findings in patient pending heart cath? A- GFR 45, creat 1.9 on metformin and ace? B-severe hx peripheral artery disease with decreased femoral pulse C- allergy to contrast dye D- patient on warfarin/apixaban E- patient on asa and Plavix daily
A- hold metformin/ace 48 hours, pre hydrate NS, watch creatinine for spike first 48 hours, NO LV gram- no dye exposure! ( can get echo to evaluate LV) B- notify surgeon, evaluate other access sites esp radial arteries vs L groin C- what is allergy? Pre medicate D- when was last dose? Why are they on it/ can it be held? E-do nothing, they can continue meds
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after stent placement pts are prescribed for a year
dual antiplatelet
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When will I order a LHC? Left heart cath
-STEMI/ACS + ischemia on stress test + >80% lesion on CTA Pre-valve work up If you have high suspicion for blockage- this is the only was to “fix” it
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Right heart cath
Measures pressures in the chambers of the heart: RA: right atrial pressure RV: right ventricular pressure PA: pulmonary artery pressure PCW: pulmonary capillary wedge LA: left atrial pressure LV: left ventricular pressure
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Echocardiogram
for anatomy of heart and perfusion Trans thoracic ( TTE) vs trans esophageal (TEE) Ultrasound to evaluate the heart tissues ( muscle and valves) and their movement MOST ACCURATE TEST for ejection fraction (EF)
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Cardiac MRI magnetic resonance imaging
Imaging for anatomy of heart information re cardiovascular tissue/muscle (congenital defects, valves, vasculature), muscle function (inflammatory) and tumors May be used for pre-procedural/surgical “mapping”
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55 yo male referred after work up for syncope revealed significant left ventricular hypertrophy on echo with severe diastolic dysfunction- no valvular disease, ef 65% What test do you order to find out more about his muscle?
Cardiac mri worried about amloidosis
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Cardiac MRA
seen in stroke protocol, no radiation, with or w/o dye information about vessels not muscle or tissue
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Test for electrophysiology | 5
ECG Telemetry Ambulatory Electronic monitoring (AEM) Treadmill EP study
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telemetry
Continuous tracing usually 2-5 leads Primarily for arrythmia detection but can see ST segment
73
Ambulatory Event Monitoring
Worn by patient for period of time Can be continuous or triggered Duration 24 hours-4 weeks Important to order correctly based on symptoms Keep in mind skin sensitivity
74
29 yo presents for episodes of palpitations 2-3 hours in duration 1-2 x a week. Most conclusive initial test: 1- ECG 2- AEM 48 hours 3-AEM 30 days 4- refer for EP test
3-AEM 30 days
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Treadmill
perfusion and electrical Ecg tracing during test will show heart rate variability, exercise/rate induced changes Important concept: patient terminated vs technician terminated
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EP study
advanced invasive testing of electrical system of the heart performed by EP Can perform mapping, induce arrythmias Requires venous access
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Arterial Brachial Index (ABI)
Looking for peripheral arterial disease if range is .7 or less refer to cardio if mild you treat with asa and statan
78
58 yo female presents with complaints of R buttock burning when she walks. PMHX: DM, HTN, elevated BMI. Improves with rest, worsens w activity. No trauma/injury. Physical exam unremarkable 1- ultrasound lower extremities 2- CTA 3- arteriogram 4- ABIs
4- ABIs bc non invasive
79
62 yo Male pmhx: htn, tobacco, fam hx AAA presents for yearly follow up. you note enlarged abdominal aorta on physical exam. 1) echo 2) CTA 3) abdominal ultrasound 4) mra
3) abdominal ultrasound
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84 yo presents visual disturbances. Reports intermittent “blind spots” resolve spontaneously. Just had eye exam and told normal. Pmhx: HTN ( diet), arthritis. No Rx. VS WNL. On physical exam R carotid bruit 1- MRA 2-CTA 3-carotid doppler 4-coronary calcium score
3-carotid doppler
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Patient PMHx CAD, HTN, HLP presents for follow up stress test results. Asymptomatic with no complaints MPS report reads: 10 minutes of bruce protocol , 12 METS , no CP, no ecg changes. Nl vs. No reversible ischemia with global hypokinesis EF 40%. What do you do? 1-tell them test results look good. Continue tlc/rx 2-send for heart cath 3-order echocardiogram 4-order MUGA
3-order echocardiogram Asx but echo to check EF
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# what do you need to know You need to be comfortable with when to/not to/how to order: plain stress test, stress test w imaging ( MPS/stress echo), echocardiogram, heart cath * if echocardiogram shows hypertrophy ( > 1.5mm) and you need to rule out infiltrative disease order MRI * to concretely r/o blockage need heart cath *echo is starting place to work up valves/murmurs
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62yo pt pmhx: HTN, DM presents yearly ov. No complaints. Rx: lisinopril 10 mg po BID, metformin 500 mg po bid. Vs: 148/90, 88, 16. on physical exam you hear a II/VI crescendo decrescendo murmur. 1-ECG 2- stress test 3-echo 4-stress echo
3-echo to check valves
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56 yo male CAD hx PCI last year, HTN, HLP, DM, + tobacco presents with exertional chest wall discomfort , assoc DOE, DAT. No symptoms at rest. Rx: asa 81mg daily, atorvastatin 40 mg hs, metoprolol 50 mg po bid, metformin 500 mg po bid. Vs: 128/72, 68, 16. physical exam: WNL 1- ecg 2-stress echo 3-stress MPS 4-heart cath 5-troponin *any instructions to the patient?
2-stress echo- if blockage found then heart cath not MPS because probably took metropolol that day, could do lexiscan | stable angina