Shoulder Complex III (Rx) - exam 3 Flashcards

(34 cards)

1
Q

What are some Distinctive Tendon Rx’s- Tendinitis and Tendinosis: (4)

A

Pt. education - load management
POLICED
NSAIDs
Bracing

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2
Q

Effects of NSAIDs regarding Tendinitis and Tendinosis?

A

-Short-term P! relief in acute presentation
-Delays healing in injury at insertion (needs inflammation to heal)

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3
Q

iontophoresis, ultrasound, phonophoresis and low-level laser treatment lack sufficient evidence at this time:

A

modalities

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4
Q

Soreness rule:

A

no more than mild P! during or up to 24 hours after exercise and quality of movement not affected

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5
Q

What is the PRIMARY purpose of Tendinosis MET: (2)

A

Tendon proliferation
Possible spinal stabilization with regional interdependence

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6
Q

Tendinosis Rx paramenters:
implement time?
load type?
action type?

A

-implement after any acuity settles
-heavy loads
actions
–slower eccentrics
–3 sec. mm. actions (conc, isometric, and ecc.)

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7
Q

Tendinosis Rx parameters:
sets x reps?
exercises?

A

2-3 set of 10-15 reps to fatigue
2-3 exercises involved tendon

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8
Q

Activity response of Tendinosis Rx Parameters:

A

-mild to moderate increase in P! - possibly up to 5/10
-timeframe P! should ease back to baseline levels before preparing for exercise 24-48hrs

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9
Q

Tendinosis Rx MET parameters:
_______ wk. program
_______ with _______ to fatigue

A

8-12 wks
precautions w/heavy loads

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10
Q

Excessive fat absorbs inflammatory cells away from the tendon is known as___________

A

obesity

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11
Q

Excessive glucose impairs collagen production and remolding is known as________

A

diabetes

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12
Q

Both diabetes and obesity improve the healing phase of tendinosis. T or F

A

False- impairs

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13
Q

Low-grade inflammation is associated with:
Persistent inflammation limits:

A

systemic diseases and/or poor diet
proliferation and remodeling

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14
Q

MD Rx:
Cortisone injection:
Glycerin trinitrate patches:
Sclerosing injections:

A

-shorts term benefits
-effective by increasing circulation
-stiffen tendon for P! relief

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15
Q

MD Rx tendinosis- worst-case scenario/last option
Future options:

A

surgical debridement
growth factors and stem cells

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16
Q

PT Rx for impingement syndrome: (3)

A

POLICED
Modalities
Scapular Taping

17
Q

MOST modalities are beneficial. T or F

A

False; MOST are not beneficial

18
Q

Scapular taping has long-term P! relief. T or F

A

False; improved short-term P!
-may provide an early “window” for MET and limit ADL provocation

19
Q

PT Rx: JM for impingement syndrome
________ recommendation
________jt.

20
Q

JM supports regional interdependence as shown in the cervicothoracic. JM should be added to ________ for more effectiveness.

21
Q

What is the PRIMARY treatment option for impingement syndrome?

22
Q

What should MET dosage be for impingement syndrome?
Should you do MET every day?

A

-High-dose MET superior to conventional low-dose exercise
-It depends; every other day, later progress to every day, never 2x/day

23
Q

PT Rx for tendinosis:
> ______mths of symptoms
________ and _______ exercises
______ w/ supporting PT visits
MET parameters for tendinosis______
________JM

A

-6 months
-Cuff (SIT) and Scapular exercises (MT/LT/ Rhom/SA)
-HEP
-1-2x/day
-Post GH

24
Q

At 3 mths. of a successful PT Rx for tendinosis:

A

70% with improved P!/function vs 25% traditional exercises
reduced need for Sx

25
Subacromial decompression reccomendation:
should NOT be performed if atraumatic and present > 3 mths. aka tendonosis
26
Is subacromial decompression more effective than exercise alone?
equally or no more effective and more expensive
27
The theory is that differing body regions are biomechanically and neurophysiologically interdependent and impairment in one region can contribute to impairment in another, particularly persistent. example:
regional interdependence -lower hip and LB strength with persistent neck P! -spine P! can cause extremity P!
28
What cervical segment is most common for impingement?
C5-6 dysfunction MOST common segment for impingement
29
Dysfunction w/overhead reaching: What shares the innervation and excessively recruited from C6 with C5,6 jt.
Internal Rot. (subscapularis, lats, and pec major)
30
What happens when you have excessive recruitment of IRs?
humeral head pulled anterior of coracoid process creates excess tension and compression underneath LHB leads to possible tendinopathy
31
Dysfunction w/overhead reaching: What happens when you have inhibition and protective hypertonicity of ERs?
-Greater tubercle won't effectively move fully out from under the acromion. -Impingement of supraspinatus and LHB that may lead to tendinopathy --> more impingement
32
What can you expect to see with C2, 3 dysfunctions regarding impingement syndrome?
excessively recruited scapular elevators --- creates excessive tension and compression on supraspinatus
33
Dysfunction w/overhead reaching: If impingement is occurring more often at higher levels what is happening?
-inhibition and protective hypertonicity of depressors -scapula won't depress effectively -impingement > 150 b/c scapula won't go back and down -supraspinatus and LHB tendons will impinge ----lead to tendinopathy
34
GH and AC jt. may also ________ with hypermobility/instability to reach higher.
compensate