somatosensory 1 Flashcards

1
Q

in the descending pathway cell bodies are located in the

A

primary motor cortex

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2
Q

axon fibers descend to the

A

corticospinal tract

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3
Q

most descending fibers decussate in the

A

medullary pyramids (90%)

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4
Q

fibers synapse on to the LMN in the

A

ventral horn of the spinal cord

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5
Q

in the descending pathway cell bodies in the ventral horn can be found in this laminae

A

9

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6
Q

LMN will always use __ as their Neurotransmitter

A

Ach

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7
Q

Ach binds to the ___ receptors

A

excitatory nicotinic

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8
Q

When ACh binds to the receptor, the channel opens, allowing for ion exchange and __

A

muscle fiber depolarization.

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9
Q

Excess ACh in the synapse is degraded locally by

A

Excess ACh in the synapse is degraded locally by acetylcholinesterase (AChE).

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10
Q

A motor unit is defined

A

A motor unit is defined as a motor neuron and all the fibers it contacts.

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11
Q

small motor units vs large motor units

A

Smaller motor units regulate fine motor activity, while large motor units control more gross movements.

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12
Q

gamma motor neurons and alpha

A

alpha: tells muscle to contract

gamma : intrafusal fibers that control the length/tension of muscle spindles

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13
Q

what happens When alpha motorneurons cause shortening of the extrafusal fibers?

A

sensory input is loss

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14
Q

polysynaptic vs monsynaptic

A

poly: indirect
mono: direct. These afferent proprioceptive sensory neurons directly synapse onto
ipsilateral lower motor neurons.

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15
Q

in reciprocal pathway :Afferent proprioceptive sensory neurons directly synapse onto

A

ipsilateral lower motor neurons to cause quadriceps contraction
and
interneurons that inhibit the lower motor neurons innervating the hamstring muscle.

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16
Q

interneurons are usually

A

inhibitory

17
Q

reciprocal inhibition

A

activate one and relax other muscle ..

18
Q

CROSSED EXTENSOR aka

A

(WITHDRAWAL) REFLEX’

19
Q

explain withdrawl reflex

A

Afferent nociceptive sensory neurons cause contraction of the ipsilateral hamstring and reciprocal inhibition of the ipsilateral quadriceps, for flexion of the ipsilateral leg

Sensory neurons also cause extension of the contralateral leg

20
Q

cerebellum

A

The cerebellum receives and integrates information about balance, body position, motor intention

The cerebellum is also involved in fine motor memories such as typing or playing the piano.

21
Q

vestibulocerebellum

A

The vestibulocerebellum helps maintain balance and controls eye movements.

22
Q

The spinocerebellum

A

The spinocerebellum enhances muscle tone and helps coordinate voluntary movements, especially fast, phasic motor activities

23
Q

The cerebrocerebellum

A

The cerebrocerebellum plays a role in initiating voluntary movement

24
Q

function of basal ganglia

A

Inhibiting muscle tone throughout the body – prevent unintentional movement
Monitoring and controlling slow, sustained contractions

25
Q

Rubrospinal

A
  • Integration of cortical and cerebellar information. Distal musculature preferentially.
26
Q

Reticulospinal

A

Reticulospinal - excite motorneurons to proximal extensors for postural control

27
Q

Tectospinal

A

Tectospinal - Head movement in response to stimuli

28
Q

Vestibulospinal

A

Vestibulospinal – Postural control

29
Q

Corticobulbar axons innervate the

A

Corticobulbar axons innervate the cranial nerve motor nuclei Ipsilateral system

30
Q

in descending pathway Axons exit the spinal cord via the

A

Axons exit the spinal cord via the ventral
root and project in spinal nerves to innervate
target muscles.

31
Q

parkisons is a Neurodegenerative disease of the

A

nigrostriatal dopamine pathway

32
Q

BROWN-SEQUARD SYNDROME
spinal cord hemisection
ipsilateral defects

A

Loss of fine touch, vibration, proprioception

Loss of function-upper motor neurons
Spastic paralysis
Hyper-reflexia

33
Q

BROWN-SEQUARD SYNDROME
spinal cord hemisection
contralateral defects

A

loss of pain and temp

34
Q

NEUROMUSCULAR JUNCTIONMYASTHENIA GRAVIS

A

Autoimmune disorder.

Targets nicotinic receptors at the neuromuscular junction.

Muscle weakness, ptosis

Treated with acetylcholinesterase inhibitors