Spinal cord injury Flashcards

(42 cards)

1
Q

Central nervous system: the spinal cord

A
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2
Q

The Autonomic Nervous System

A
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3
Q

Spinal Injury: Causes

A

SERIOUS INJURY:
Motor vehicle accidents
Falls
Gunshot or
stab wounds
Sports injuries
Diving (66%)

LESS SERIOUS INJURY:
Lifting heavy objects
Minor falls

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4
Q

Spinal Injury: Terminology

A

PLEGIA = paralysis

-Monoplegia
One limb

-Hemiplegia
Both limbs on one side

-Paraplegia
Both upper OR both lower limbs

-Quadriplegia or Tetraplegia
All four limbs

PARESIS = weakness
Ipsilateral = same side
Contralateral = different sides

MUSCLE TONE
-Hypotonia
Less than normal

Flaccidity
Absent

Hypertonia
Excessive

Spasticity
Causes stiff awkward movement

Rigidity
Immovable stiffness

Tetany
Intermittent tonic spasms - paroxysmal

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5
Q

Vertebral Column Injury

A

Fracture
-Fragmentation of the bone
-Pedicle, lamina, processes

Dislocation
-Displacement of vertebral body

Subluxation
-Partial dislocation

Types of injuries:
Flexion
Extension
Compression
Axial rotation

Extent of injury depends on:
Location
Severity

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6
Q

Spinal Cord Injury mechanisms

A

Mechanical disruption of neurons:

Injury-related ischemia & hypoxia
-Contributes to local infarction

Development of micro-hemorrhages or edema:

-Interruption of neuronal function

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7
Q

2-step Pathophysiology

A

PRIMARY –> initial injury
-Small hemorrhages in gray matter, edematous changes in white matter leading to necrosis of neural tissue

-Is IRREVERSIBLE

SECONDARY–> progressive neurologic damage

-Vascular damage 🡪 ischemia, ↑ vascular permeability, edema

-Neuronal injury🡪 loss of reflexes below the level of injury

-Release of vasoactive agents & cellular enzymes leads to delayed swelling, demyelination, & necrosis

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8
Q

Spinal Cord Injury: Types

A

INCOMPLETE TRANSECTION – partial preservation of sensory and motor function:

-Central cord syndrome
-Anterior cord syndrome
-Brown-Sequard syndrome
-Conus medullaris syndrome

COMPLETE TRANSECTION – absence of sensory and motor function:

-Above T1 –> Quadriplegia
-Below T1 –> Paraplegia

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9
Q

Effects of Spinal Cord Injury
by Location

A

the lower the damage on the spinal cord the better

do not need to memorize where the damage is and its effect

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10
Q

Central Cord Syndrome

(do not need to remember each syndrome but different areas of damage can manifest differently)

A

Nature of Injury: Damage to central gray or white matter of cord

Areas less or not affected: Motor function of lower extremities
Bowel, bladder sexual function

Recovery: Often recover to the point of being ambulatory and controlling bowel and bladder, but often are not able to perform detailed or intricate work with their hands

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11
Q

Anterior Cord Syndrome

A

dont need to memorize

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12
Q

Brown-Sequard Syndrome
(hemi- half)

A

dont need to memorize

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13
Q

Conus Medullaris Syndrome

A

dont need to memorize

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14
Q

Spinal Cord Syndromes
-central and conus medullaris

A

do not need to memorize

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15
Q

SCI Syndromes

A
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16
Q

Cauda Equina Syndrome
do not need to memorize

A

Nature of Injury:
Damage to the lumbosacral nerve roots within the canal

Areas MOST affected:
Various patterns of asymmetric flaccid paralysis, sensory impairment, and pain

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17
Q

Neurologic Examination

A

Mental status & speech

Cranial nerves

Central and peripheral sensory function

Motor function

Cranial & peripheral reflexes

Cerebellar function & gait

18
Q

Diagnostics: X-Ray

A

X-ray:

provides detail of the bone structures in the spine

used to rule out:
instability
tumors
fractures

does not capture disc and nerve root structures

cannot be used to diagnose lumbar disc herniation or other causes of nerve pinching.

19
Q

Diagnostics: CT

A

Computed Tomography (CT):
fancy x-ray that can take cross section images of the body

will image large disc herniations but can miss smaller ones

20
Q

Diagnostics:
CT with Myelogram

A

CT with Myelogram:

-radiopaque dyes injected into the sac around the nerve roots, which lights up the nerve roots

-provides substantial information about the nerve roots

-very sensitive test for nerve impingement and can pick up even very subtle lesions

21
Q

Diagnostics: MRI

A

Magnetic Resonance Imaging

single most useful imaging study available for spine surgery

aids in the assessment of certain conditions by providing detail of the disc and nerve roots

provides highly refined detail of the spine’s anatomy

22
Q

Diagnostics: EMG & SSEP

A

Electromyography:
Assesses the electrical activity of a nerve root

Useful to distinguish nerve degeneration (neuropathy) from nerve root compression (radiculopathy)

Somatosensory Evoked Potentials:
Assesses the speed of electrical conduction across the spinal cord

If the spinal cord is significantly pinched, the electrical signals will travel slower than usual

Also used to monitor spinal cord function during surgical procedures

23
Q

Spinal Injury: Management

A

Reduce neurologic deficits + prevent additional losses

Immobilization with neck collars and back boards to limit movement, stabilize spinal column, and prevent further damage

Log-roll and secure head with straps or tape

Cervical: Cervical traction

Thoracic & lumbar: Bedrest & logrolling

High dose methylprednisolone w/in 8 hours
-Stabilizes cell membranes, enhances impulse generation, improves blood flow, inhibits free radical formation

CURRENT RESEARCH: neuron regeneration with stem cells

Prevention, early detection, prompt intervention, rehabilitation

24
Q

Alteration in Functional Abilities

A

Alterations in spinal reflexes

Ventilation & communication dysfunctions

Autonomic nervous system dysfunction
-Vasovagal response
-Autonomic dysreflexia
-Postural hypotension

-Alterations in temperature regulation
-Circulatory system dysfunction
-Sensorimotor dysfunction
-Skin
-Pain
-Bladder function
-Bowel elimination
-Sexual Function

25
Alterations in Spinal Reflexes
UMN (upper motor neurons) Lesions: -Affected by any injury at the T12 level or above -Results in spastic paralysis of affected skeletal muscle groups, and muscles that control bowel, bladder, and sexual function LMN (lower motor neurons) Lesions: -Occur with injuries below T12 -Result from damage to the peripheral nerves that exit each segment of the spinal cord -Causes flaccid paralysis of involved skeletal muscle groups and muscles that control bowel, bladder, and sexual function
26
Spinal or Neurogenic Shock
Temporary self limiting: falcid paralysis is not an indicator of lifelong paralysis State of areflexia that occurs after spinal injury Involves loss of all or most of the spinal reflexes below the level of the injury Also involves the motor pathways MANIFESTATIONS: flaccid paralysis, lack of tendon reflexes and autonomic function May last minutes, hours, days, weeks Usually self-limiting
27
Ventilation Dysfunctions
Diaphragm innervated by C3 to C5 via phrenic nerves Intercostals innervated by T1 to T7 Major muscles of expiration innervated by T6 to T12 C1 to C3 Injury -Lack of respiratory effort -Requires assisted ventilation C3 to C5 Injury -Allows partial or full diaphragmatic function but ventilation diminished Below C5 -Ability to take a deep breath and cough less impaired
28
Meeting Communication Needs
Verbal: -Fenestrated tracheostomy tubes Provide airflow for vibration of the vocal cords -Talking tracheostomy tubes -Diaphragmatic pacing -Electrolarynx-type devices -Mechanical ventilation with an air leak Non-verbal: -Communication boards or cards -Computerized scanning programs -Mouth-stick control devices
29
ANS Dysfunction
-Afferent & efferent flow above the level of injury is unaffected 🡪 normal function -Ascending & descending transmission below injury is blocked 🡪 uncontrolled spinal and autonomic reflexes -Autonomic regulation of circulatory function & thermoregulation are most severe problems -The higher the level the injury, the more profound the effect esp. above T6
30
Vasovagal Response
-Vagus nerve – continuous inhibitory effect on HR -Vagal stimulation 🡪 Vasovagal response 🡪 bradycardia or asystole -Deep tracheal suctioning hyperoxygenate -Rapid position change Avoid rapid position changes have anticholinergic drugs immediately available
31
Autonomic Dysreflexia: A Clinical Emergency !
Acute episode of exaggerated sympathetic reflex responses Caused by visceral stimuli that normally cause pain or discomfort in the abdominopelvic region Does not occur until spinal shock has resolved and autonomic reflexes return (within 6 months of injury) Unpredictable in first year; can occur throughout lifetime T6 and above injuries Characterized by: Hypertension Bradycardia Headache Unregulated SNS activity leads to vasospasm, hypertension, skin pallor, piloerection, and baro-reflex mediated vagal bradycardia, but vasodilation, flushed skin, profuse sweating above the level of the injury, nasal congestion
32
Autonomic Dysreflexia: Convulsions - ↓LOC - Death
CAUSES: -Visceral distension – full bladder or rectum -Pain – pressure ulcers, ingrown toenails, dressing changes, diagnostic or operative procedures -Visceral contractions – ejaculation, bladder spasms, uterine contractions INTERVENTION: -Monitor BP Q5’ -Remove/correct cause/stimulus -Position upright -Remove AE hose (allow venous pooling to ↓ BP) -IV peripheral vasodilators Hydralazine (Apresoline) Diazoxide (Hyperstat)
33
Postural Hypotension
-Occurs in persons with injuries at T4 to T6 and above -Related to interruption of descending control of sympathetic outflow to blood vessels in the extremities & abdomen -Results in pooling of blood leads to decreased CO SIGNS: -Dizziness, pallor, excessive sweating above level of lesion, blurred vision, fainting PREVENTION: -Slow changes in position -Measures to promote venous return
34
Alterations in Temperature Regulation
SNS functions in regulation of body temp Central mechanisms for temperature regulation located in the hypothalamus Cold --> vasoconstriction + shiver--> conservation & production of heat Heat --> vasodilation + sweating --> dissipative & evaporative heat loss Sympathetic effector responses below the level of injury are disrupted : -Lack of ability to conserve/dissipate heat + sweat -Higher levels of injury (on the spine) produce greater disturbances -Poikilothermy – patients assume external temperature because of impaired temp regulation -EDUCATION: clothing & awareness of environment
35
Circulatory System Dysfunction
Edema & DVT common problems ↓ PVR , areflexia or hypotonia, immobility --> ↑ venous pressure & pooling of blood in abdomen, lower limbs, and upper extremities Orthostatic or dependent edema --> elevation & compression (AE hose) DVT --> low dose heparin, ROM, compression devices, assessment
36
Sensorimotor Dysfunction
After spinal shock, isolated reflex activity + muscle tone not under control of higher centers returns Results in hypertonia or involuntary spasticity of skeletal muscles below the level of injury May be tonic (sustained tone) or clonic (intermittent) Occurs in injuries above T12; below T12 reflex response damaged at the cord or spinal nerve level Spasticity has some benefits but also places individual at risk for injury STIMULI: muscle stretching, bladder infection, bowel distension or impaction, pressure areas, infections INTERVENTION: PROM, avoid stimuli, antispasmodics
37
Skin
Innervation by cranial & spinal nerves in dermatomes Afferent/sensory information 🡪 Brain🡪 Efferent/motor control & reflex activity at each dermatome SNS: control of vasomotor and sweat glands provides adequate circulation, excretion of body fluids, & temperature regulation Spinal cord injury 🡪 major risk for altered skin integrity FACTORS: pressure, shearing forces, trauma & irritation INTERVENTION: relieve pressure, encourage circulation, inspect for breakdown MOST PREVENTABLE COMPLICATION !!!
38
Pain
Diverse & unpredictable pain syndromes Mechanical or fracture pain -Dull, aching pain that occurs at level of injury from soft tissue damage Radicular or spinal nerve root pain -Aching or shooting pain that radiates along distribution Visceral -Poorly localized, burning abdominal/pelvic discomfort r/t bladder distension or UTI Central -Diffuse burning sensation below level of injury -Aggravated by touch, movement, & visceral distension INTERVENTION: TENS, TCAs, Anticonvulsants, NSAIDs, PT
39
Bladder Function
Sensory signals from bladder stretch receptors (S2 to S4) 🡪 Reflex voiding center 🡪 Motor neurons (S2 to S4) SNS: detrusor relaxation (bladder filling) PNS: detrusor contraction (voiding) UMN Injury – spastic bladder dysfunction -Lack awareness of bladder filling and voluntary control of voiding 🡪 incontinence LMN Injury – flaccid bladder dysfunction -Lack awareness of bladder filling and lack of bladder tone 🡪 unable to void 🡪 retention & overflow INTERVENTION: continuous or intermittent drainage, external collection, manual techniques
40
Bowel Elimination
SNS: T6 to L3 --> decreased intestinal motility + increased internal sphincter tone PNS: S2 to S4 --> increased intestinal motility + decreased internal sphincter tone Spinal cord injuries at S2 to S4 --> flaccid functioning of the defecation reflex + loss of voluntary control of external anal sphincter Spinal cord injuries above S2 to S4 --> spastic functioning of the defecation reflex + loss of anal sphincter tone --> intrinsic contractile responses intact but no defecation reflex INTERVENTION: high-fluids, high-fiber diet, mobility, consistent pattern, privacy, positioning, laxatives, digital stimulation (questionable)
41
Sexual Function
Physical act of sex vs. sexual/caring relationship T11 to L2 -->mental stimuli or psychogenic sexual response S2 to S4 --> sexual touch or reflexogenic sexual response Spinal cord injury at any level 🡪 disrupts neural pathways (S2 to S4) between genital and higher centers -UMN Lesion (T10 or higher): reflex sexual response to touch but not to mental stimuli (spinal lesion is blocking pathway) -LMN Lesion (T12 or below): sexual reflex center may be damaged 🡪 no response to touch; below T12 sexual arousal by mental stimuli; L2 to L1 may have sexual response to mental or touch stimuli INTERVENTION: erectile aids, lubricants, Fertility may not be lost ! Pregnancy, labor & birth control requires caution
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Herniated Disks