Sport Physiology A Flashcards

(120 cards)

1
Q

Define Energy?

A

The ability/capacity to perform work
(comes from breakdown of ATP)

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2
Q

What is ATP?

A

Adenosine Triphosphate
- The body’s energy “currency” for all body cells
- Serves as the immediate source of energy that comes from the breakdown of ATP
- Powers all the body’s metabolic activities

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3
Q

What is ADP?

A

Adenosine Diphosphate
- Is created when ATP splits to release energy
- Occurs when one of the three phosphate molecules splits off and energy is released

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4
Q

How many seconds worth of ATP is stored in body cells?

A

1-2secs (max)

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5
Q

Name the Energy Systems for Replenishing ATP

A
  1. Phosphate Creatine System (ATP-PC System)
  2. Anaerobic Glycolysis System
  3. Aerobic Glycolysis System
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6
Q

How is ATP resynthesised?

A

Through fuel/energy substrates and the energy systems

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7
Q

How much ATP is in our muscular systems?

A

50-100g

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8
Q

How much ATP is produced daily?

A

50-180kg - typically upwards of bodyweight

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9
Q

Where is Creatine Synthesised?

A

Liver, kidneys and pancreas

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10
Q

What is PC primarily used for?

A

The ATP-PC system (powerful movements)

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10
Q

How can Creatine be consumed?

A

Through diet
- Fish
- Meat
- Supplements

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11
Q

What are the 3 main fuels for our body?

A

Carbohydrates (carbs/CHOs)
Fats (lipids)
Proteins

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12
Q

What are CHOs?

A

Primary and most versatile source of fuel
- sugars and starches
- can be simple or complex carbs
- should make up 55-60% of diet/60-80% for athletes

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13
Q

What are CHOs transported as and how?

A

Glucose/glycogen and transported through the blood

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14
Q

What is glycogen/glucose?

A

Broken down carbs and/or released by liver to be used for energy (ATP) production

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15
Q

Where are CHOs stored?

A
  • Blood
  • Muscles and liver (glycogen)
  • Excess stored as fat (adipose tissue around body)
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16
Q

What are CHOs stored as?

A

Glucose, glycogen or adipose tissue

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17
Q

Low GI Foods

A

0-55 GI
Slow release of energy providing a constant source
- pasta
- oats
- brown bread

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18
Q

Medium GI Foods

A

55-79 GI
- bananas
- mangoes

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19
Q

High GI Foods

A

79-100 GI
Instant energy from rapid increase in blood sugar levels
- white bread and rice
- energy drinks
- jelly beans

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20
Q

What is the role of the Energy Systems regarding ATP?

A

Serves to replenish stores of ATP through phosphorylation
(ADP + Pi + energy –> ATP)

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21
Q

What are fats?

A
  • Extremely high source of energy
  • Supply the “most” energy but harder to break down
  • Made of free fatty acids (FFA) and triglycerides
  • Should make up 20-30% diet
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22
Q

How are fats transported?

A

Broken down and transported through blood as FFA

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23
Q

How is fat stored?

A

Adipose tissue
FFA
Triglycerides

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24
Where is fat stored?
Around the body (as adipose tissue) Liver Muscles Blood
25
When are fats used as the predominant energy source?
During low intensity/sub-maximal efforts When carbohydrate stores are depleted
26
What are proteins?
Amino acids that are essential for growth, repair and recovery of body tissue Are the emergency fuel source when both carb and fat stores are depleted
27
How can protein be consumed?
Meat, fish, eggs, diary In extreme situations protein is released from breakdown of body tissue
28
How are proteins transported?
As amino acids in the blood
29
How are proteins stored?
Body tissue/muscle Body fluids Adipose tissue
30
What is hitting the wall?
An individuals sudden increase in fatigue and decrease in power
31
How does hitting the wall occur?
When liver and muscle glycogen stores become depleted Fats become the primary source of energy to produce ATP (as oxidisation is slower)
32
How to counteract hitting the wall?
Carb loading Consuming high GI fuel sources during the race Glycogen sparing
33
What is glycogen sparing?
An athletes increased capacity to metabolise on fats to rely on less and hence save glycogen stores Glycogen is not used on early in an event, so there is an increased capacity to use fats through an improved oxidisation ability
34
Energy from ATP
Is limited in storage ATP splitting releases energy Different fuel substrates resynthesise ATP depending on intensity and duration of activity
35
ATP Production: Rest
Produced aerobically as high O2 abundance 2/3 of ATP produced comes from the breakdown of fats Fats breakdown when abundant O2 is available
36
ATP Production: Initially
ATP is produced anaerobically as respiratory and circulatory systems cannot meet demands to supply O2 to working muscles
37
ATP Production: Anaerobic
Produce ATP for powerful and quick movements Limited amounts of ATP--> limited activity time Fatiguing by-products (Lactic Acid)
38
ATP Production: Aerobic
Produce ATP for prolonged periods Cannot produce energy quickly for high intensity efforts only sub-maximal/low intensity Non-fatiguing by-products
39
What is Phosphorlyation?
Chemical addition of a inorganic phosphate back onto ADP to synthesise ATP
40
What is the Phosphagen System?
ATP-PC system (anaerobic) Provides the bulk of ATP for powerful/explosive/short efforts \ Relies on stores of ATP and PC Lasts 10-12secs
41
How does PC resynthesise ATP?
Stored ATP lasts for 2 seconds PC splitting can provide energy to resynthesise ADP to ATP
42
How long does it take for the ATP-PC system to recover?
3-5mins to restore to pre-exercise levels - 50% PC replenishment occurs in first 30s - <10s of effort only 3mins to recover
43
What is the Anaerobic Glycolysis System?
Known as the lactic acid system ATP is produced via incomplete breakdown of glucose Provides for bulk of ATP for high intensity/maximal activity for exercise longer than 10s Relies on muscle stores of glycogen and blood glucose Produces pyruvate acid and H+ ions
44
Sporting Example: ATP-PC
100m sprint
45
Sporting Example: LA
400m sprint
46
LA Sytem: By-products
Fatiguing (lactic acid from incomplete breakdown of pyruvate and hydrogen ions)
47
Aerobic System: By-products
Non-fatiguing (H2O, CO2 and heat)
48
How does Lactic Acid Accumulate?
Through the incomplete breakdown of pyruvic acid In aerobic system O2 in mitochondria facilitates breakdown As no O2 in anaerobic system, pyruvate is unable to breakdown and is converted to LA
49
How is Pyruvic Acid produced?
Via the incomplete breakdown of glucose
50
What are the impacts of LA and H+?
Accumulate in the muscle cells during prolonged high intensity efforts Result in a decrease of blood pH (increased acidity) and cause soreness burning/tiredness Body can tolerate until Lactic Threshold is met (production is higher than removal rate)
51
Lactic Acid Removal/Fate
65% converted to CO2 and H2O 20-25% into muscle/liver glycogen 10% protein 5% glucose
52
How long does the LA last?
2-3mins
53
Duration of Energy Systems
ATP-PC: 0-12s LA System: Aerobic System:
54
What is the Aerobic System?
Uses O2 to drive the production of ATP via the breakdown of carbs, fats, and proteins Process occurs in the mitochondria
55
What are the three main stages of the Aerobic System?
1. Anaerobic glycolysis 2. Krebs Cycle 3. Electron Transport Chain
56
Order of fuel substrates in the Aerobic System
1. Carbohydrates (glycolysis) 2. Fats (lipolysis) 3. Proteins
57
Anaerobic Glycolysis
- Occurs in the muscle cell - Carbs are the prominent fuel source - Glycogen is broken down to form glucose which is broken down to form pyruvic acid - This releases a small amount of ATP
58
Krebs Cycle
- Occurs in the mitochondria - O2 and fuel (glucose, protein, fats and pyruvic acid) enter - O2 combines with carbon (CO2 waste product - H+ ions are produced - Some ATP is released
59
Electron Transport Chain (ETC)
- Occurs in the mitochondria - H+ and water combine (H2O as waste product) - Heat is produced - Large amount of ATP (energy is released)
60
What is myoglobin?
An oxygen binding protein found in skeletal muscle cells that attracts O2 from the blood stream to muscle cells
61
What is the function of myoglobin?
To aid the delivery of O2 from the cell membrane to the mitochondria When O2 conc is low, it will release O2
62
How is O2 transported around the body?
Via haemoglobin in blood to the capillary beds on muscle where it is released and diffuses into muscle cells
63
How to increase O2 utilisation?
Aerobic training increases the body's ability to attract O2 from blood to muscle cells
64
Three physiological adaptations that increase O2 utilisation
1. Increased number and size of mitochondria 2. Increased myoglobin stores 2. More capillaries
65
Define Energy Continuum/Interplay
The continual interplay between all energy systems to meet energy demands based upon the type, intensity and duration of activity
66
What are the three factors the determine the dominant energy system?
Fitness Level Intensity Duration
67
Types of Muscle Fibres
Slow Twitch (Type 1) Fast Twitch (Type 2a & 2b)
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Colour of Fibres
Type 1: Red (rich O2/blood supply) Type 2a: Pink (combination) Type 2b: White (limited 02/blood supply)
69
Fatigue resistance of Muscle Fibres
Type 1: high Type 2a: medium Type 2b: low
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Strength of Contraction of Muscle Fibres
Type 1: slow Type 2a: fast Type 2b: fastest
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Characteristics of Muscle Fibres
Type 1: aerobic Type 2a: aerobic and anaerobic Type 2b: anaerobic
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Mitochondria density of Muscle Fibres
Type 1: high Type 2a: moderate Type 2b: low
73
How to improve Muscle Fibres?
Specific aerobic/anaerobic training Own fibres influenced by genetics
74
What fuel substrate produces the most energy?
Fats - 12x more ATP produced than carbs
75
Increased Heart Rate
Heart pumps faster to supply more O2 to working muscles Measured in BPM MHR 220 - age
76
Stroke Volume
Volume of blood ejected from the left ventricle per beat
77
3 Factors that affect Stroke Volume
Frank-Starling Mechanism - increased blood returning to heart results in stronger contraction of left ventricle ∴ increased SV Neural Stimulation - increased nerves increase SV Peripheral Resistance - resistance to passage of blood
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Cardiac Output
Volume of blood pumped by the heart per minute (mL blood/min) Q = SV x HR
79
Blood Pressure
Pressure exerted by the blood against the arterial walls as it forced through the circulatory system by the heart 2 main components: systolic and diastolic Measured by systolic/diastolic e.g 120/80
80
Systolic Blood Pressure
Higher value of the two Pressure recording while contracting
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Diastolic Blood Pressure
Lower value of the two Pressure recording while relaxing
82
Blood Redistribution
Redistribution of blood from organs to muscles during exercise Achieved through vasodilation and vasoconstriction of blood vessels
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Systemic Blood Flow
Blood flow around the body At rest 15-20% to muscles - rest to organs (vasoconstriction) At exercise 80-90% to muscles - rest to organs (vasodilation)
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Vasoconstiction
Capillaries and arterioles restricting blood flow Component of blood redistribution
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Vasodilation
Capillaries and arterioles expanding Component of blood redistribution
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Arteriovenous Oxygen Difference (A-VO2 Diff)
Difference in the conc of O2 in the arterial blood and conc of O2 in the venous blood Measured in ml/100mL of blood
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External Respiration
gas exhange at the alveoli
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Internal Respiration
gas exchange at the muscle fibres
89
Respiratory Rate
Number of breathes per min Increases during exercise
90
Tidal Volume
The amount of air inhaled and exhaled in a breathe Increases during exercise
91
Pulmonary/Minute Ventilation
Volume of air moved in and out of the respiratory tract per min VE = RR x TV
92
Gas Exchange
Replenishment of O2 and removal of CO2 Occurs at lungs and muscle tissue (external and internal respiration)
93
O2 Uptake/VO2
Amount of O2 transported to, taken up, and used by the body for energy production Increases during exercise due to muscles need for O2
94
Oxygen Deficit
Shortfall (discrepancy) between oxygen supply and demands for exercise, where ATP must be produced anaerobically
95
Aerobic Steady State (ASS)
The state of which oxygen supply meets oxygen demands and virtually all ATP is produced aerobically Trained athletes can reach ASS quicker due to increased myoglobin and haemoglobin stores Usually 60-85% MHR
96
EPOC
Excess Post-Exercise Oxygen Consumption
97
What is EPOC?
After cessation of exercise, oxygen uptake/consumption remains elevated about normal levels
98
VO2
Amount of O2 per minute transported by, taken up and used by the body to produce energy (ATP)
99
VO2 Max
Max amount of O2 per minute transported by, taken up and used by the body to produce energy (ATP) or The highest rate of oxygen consumption attainable during maximal or exhaustive exercise
100
Types of VO2
Absolute Relative (bodyweight)
101
Absolute VO2
Expressed in L/min Does not factor body weight ∴ less comparable Divide by bodyweight to find relative value
102
Relative VO2
Expressed in mL/kg/min Factors body weight ∴ quantifiable and comparable Times by bodyweight to find absolute value
103
Factors that influence an individual's VO2 (5)
1. Aerobic Fitness 2. Body Size 3. Gender 4. Heredity 5. Age
104
Lactate Threshold Point (LIP)
The point beyond the intensity of an effort cannot be maintain by an athlete Beyond this point LA accumulation is greater than its removal ∴ causing fatigue
105
Why is LIP important?
Athletes can exercise at a higher intensity for longer before fatigue sets in Is a better indicator of aerobic performance than VO2 Max
106
Buffering
Utilising lactate to assist in neutralising H+ and remove lactate to improve performance after LIP is reached (CHECK) e.g bi-carb soda
107
What are Acute Responses?
The immediate changes the body experiences in response to exercise that only last for entirety of exercise
108
LIP VALUES (Trained and Untrained athletes)
Untrained: 60% MHR or 70-80% VO2 Max Trained: 90% MHR or 70-80% VO2 Max (CHECK THIS)
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Acute Cardiovascular Responses (7)
Increased HR INcreased SV Increased BP Increased A-VO2 Dif Increased Cardiac Output (Q) Increased blood flow Blood Resdistribution
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Acute Respiratory Responses (4)
Increased RR Increasde TV Increased minute ventilation Increased O2 uptake (VO2)
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Acute Muscular Responses
Increased O2 supply and usage Increased muscular temp Increased blood flow to muscles Depletion of muscle energy stores (ATP, PC, glycogen, triglycerides)
112
What are Chronic Adaptations?
The long-term physiological adaptations the body makes to training over an extended period (6-8 weeks)
113
Factors that affect the nature of Chronic Adaptations (4)
1. Genetics and Fitness Capacity 2. Frequency, Duration and Intensity 3. Anaerobic/Aerobic Training 4. Type and Method of Training
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Cardiac Hypertrophy
Is the enlargement of the heart based on aerobic or anaerobic training Endurance/aerobic athletes will have a bigger left ventricle Power/anaerobic athletes will have an bigger heart wall enlargement of the heart chambers (especially the left ventricle) and thickening of the myocardium (heart muscle) as a result of training.
115
Chronic Circulorespiratory Adaptations (REST) (7)
Decreased resting HR Cardiac Hypertrophy Increased SV Decreased BP Increased blood volume and haemoglobin Unchanged/decreased cardiac output Increased capillarisation of heart and skeletal muscle
116
Chronic Circulorespiratory Adaptations (SUB-MAX) (7)
Decreased HR Cardiac Hypertrophy Increased SV Decreased BP Increased A-VO2 Dif Improved HR recovery rates Decreased minute ventilation
117
Chronic Circulorespiratory Adaptations (MAX) (5)
Cardiac Hypertrophy Increased SV Increased cardiac output Improved HR recovery rates Increased minute ventilation
118
Chronic Muscular Adaptations (Endurance) (8)
Increased O2 utilisation Increased myoglobin conc Increased size/number of mitochondria Increased size of slow twitch fibres Increased oxidisation of CHOs and fats Glycogen sparing - increased oxidisation of fats Increased ATP-PC stores Increased sotres of muscle glycogen/triglycerides
119
Chronic Muscular Adaptations (Non-Endurance)
Increased glycogen stores Increased ATP-PC stores Increased flexibility Increased size of fast twitch fibres Increased number of muscle capillaries