Stress Flashcards
(160 cards)
What is stress? When do you experience it?
A state of physiological or psychological strain, caused by a stressor, that disturbs the functioning of the body - it’s the body’s reaction to an event.
-You experience stress when there is a mismatch between the demands made upon an individual and their ability to meet these demands.
What causes stress?
Stressors - anything that induces stress.
-E.g an event.
How can stress be a good thing? What can excessive stress lead to? Why are psychologists interested in stress?
A little bit of stress can be a good thing - it can boost performance, give you motivation and increase productivity.
-However, different people have different thresholds and excessive stress can be unhealthy for mind and body, leading to long term illness. E.g many cases of depression and anxiety are due to stress.
-Ultimately, if psychologists can better understand stress and how to treat it, there will be a less stressed workforce and society - positive implications on the economy.
What 2 pathways of the ANS are involved in the physiological stress response?
1) SAM - Sympathomedullary Pathway
2) HPA / PAS - Hypothalamic-Pituitary-Adrenal System / Pituitary Adrenal System
What is the function of each pathway? How do the 2 pathways differ?
-SAM pathway - activates the sympathetic branch of the ANS - controls the body’s initial response to an acute stressor (short-term) - triggers fight or flight response.
-HPA / PAS - activates the parasympathetic branch of the ANS - controls the body’s response to a chronic stressor (long-term) - helps the body to cope with the demands of a persistent stressor and to eventually bring about a normal state (homeostasis) - rest and digest system.
-The SAM and HPA / PAS pathways actually start at the same time - but the PAS takes longer to activate as it involves hormones which travel via the blood, as opposed to the electrical signals involved in the SAM system.
How does the SAM pathway work? How fast is it?
SAM - acute stressor perceived - fight or flight response triggered:
1) The amygdala sends a distress signal to the hypothalamus.
2) The hypothalamus sends an electrical signal to the pituitary gland.
3) The pituitary gland transmits the electrical signal to the adrenal medulla, via the sympathetic ganglia.
4) The adrenal medulla is stimulated (sympathetic arousal) and releases adrenaline (and also noradrenaline) into the bloodstream.
5) Adrenaline travels to the vital organs via the bloodstream - a sympathetic state is induced.
-It’s faster acting (instantaneous) than HPA / PAS (because electrical signals are involved), but doesn’t stay around for long.
What physical state does adrenaline cause? What are the physical effects?
Sympathetic state:
-Increased heart rate/breathing rate - greater flow of oxygenated blood.
-Dilated pupils - get as much light as possible.
-Glycogen rapidly converted into glucose.
-Sweat production increases.
-Inhibited digestion - diverts blood away.
-Inhibited saliva production - dry mouth.
-Contracts the rectum.
When does the sympathetic response to a stressor end?
Either when the parasympathetic system takes over (no longer a threat), or if the stress becomes chronic.
How does the HPA system work? How fast is it? How does the HPA system use a negative feedback loop to self-regulate?
HPA - activated by a long-term stressor:
1) Hypothalamus releases corticotropin-releasing factor (CRF).
2) CRF triggers/stimulates the anterior lobe of the pituitary gland to release adrenocorticotropic hormone (ACTH) into the bloodstream.
3) ACTH levels are detected by the adrenal cortex which secretes corticosteroids such as the the hormone cortisol in response.
4) A parasympathetic state is induced - any fight or flight response is reversed - rest and digest response activated as priority is for energy conservation.
-Think about HPA or PAS as the resting state - but its initial effects are actually to help prepare the body for the fight or flight system.
-HPA takes longer to activate (20 minutes) compared to SAM because it is slower as endocrine glands and hormones involved - the HPA and SAM response actually start at the same time. The effects of the HPA response are longer-lasting however, persisting for several hours or longer.
-Negative feedback loop - levels of cortisol in the bloodstream are then monitored back to the hypothalamus and pituitary gland, which can then inhibit the production of CRF (also known as CRH) and ACTH if cortisol levels are too high.
What physical state does the HPA system eventually cause when there is no longer a threat? What happens to control the body’s longer response to stress?
Parasympathetic state:
-Decreased heart rate/breathing rate - normal flow of blood.
-Constricted pupils.
-Sweat production decreases.
-Stimulates digestion - blood needed for digestion.
-Stimulates saliva production.
-Relaxes the rectum.
-However, if the stressor is chronic or long-term, primarily the HPA system needs to help the body cope with the demands of the stressor - it does this through the role of cortisol.
What is cortisol? What are some of its general functions?
Cortisol is a hormone produced by the adrenal cortex.
-It is often called the stress hormone, due to its central role in helping the body cope with stressors by controlling the body’s use of energy - it also has a number of other general functions.
Functions:
-Regulates the body’s use and metabolism of fats, proteins and carbohydrates.
-Regulates blood glucose - a glucocorticoid.
-Maintains blood pressure and cardiovascular functions.
-Helps reduce inflammation - it does this by slowing down the immune system (suppressing it) - cortisol suppresses leucocyte activity and reduces the production of T cells.
How are cortisol levels monitored? What can happen if levels are too high or too low?
Cortisol is always in the blood - its levels are monitored by the hypothalamus and pituitary gland - but cortisol levels can be too high or too low.
-Too low = Addison’s disease - adrenal cortex cannot produce any or enough cortisol so the body cannot mobilise energy to deal with the stressor.
-Too high = Cushing’s syndrome - body makes too much cortisol over a long period of time - i.e in response to long-term stressors (chronic stressors).
What is the role of cortisol in the stress response? How is this advantageous in the short-term?
-Cortisol provides a quick burst of energy and permits a steady supply of blood sugar - as a glucocorticoid it increases the availability of blood glucose to the brain - this increases blood sugar levels and restores energy supplies to power the stress response.
-Constricts the blood vessels to increase blood pressure so that oxygenated blood can be delivered to the muscles.
-In the short-term this is advantageous for fight or flight and is not a problem.
What happens if there are high cortisol levels over a long period of time? How can it be damaging?
If the body is consistently responding to stress (i.e when chronic stress is experienced), high cortisol levels can be damaging:
-Elevated cortisol over the long-term consistently leads to increased blood sugar levels (which can lead to impaired cognitive ability), disruption of sleep, reduced immune functioning (cortisol suppresses leucocyte activity and reduces the production of T cells) and type 2 diabetes (which in turn can lead to atherosclerosis and CHD).
-Arterial constriction and high blood pressure can lead to blood vessel damage and the build up of plaque - this can lead to cardiovascular disorders as not enough blood can get through.
-Also, by suppressing the immune system to reduce inflammation, over time cortisol can reduce immune functioning.
What 2 studies show how increased levels of cortisol can cause problems?
1) Newcomer et Al (1999) - gave participants cortisol to replicate blood sugar levels similar to those experiencing major stress events - found that their ability to recall prose passages was poorer compared to those only given cortisol to produce a stress response similar to minor events.
2) Vgontzas et Al (2013) - found that chronic insomniacs had increased activity of their PAS - proves that high cortisol levels can lead to sleep disruption.
What are the strengths and limitations of research into the physiological stress response?
Strengths:
-Treatment of Addison’s disease - adrenal glands cannot produce cortisol so the body cannot mobilise energy to deal with the stressor - individuals can self-administer hydrocortisone daily and must be aware of stressful situation when they might need an extra injection.
Limitations:
-Physiological explanations ignore psychological factors - e.g cognitive appraisal - working out whether a stressor is a threat.
-Speisman et Al (1964) - changes to heart rate whilst watching a gruesome medical procedure depended on the students interpretation of what was happening - i.e heart rate increased if they believed it to be traumatic, and heart rate decreased if they were told that the procedure was part of a voluntary and joyful rite of passage - these findings suggest that these changes cannot purely be explained a physiological theory, but also by psychological factors like cognitive appraisal.
What model did Hans Selye develop in 1936?
The General Adaptation Syndrome (1936), or GAS model.
*Likely to come up
How did Selye view stress? What did he believe was the purpose of this adaptation?
Selye viewed stress as the body’s attempt to adapt to a stressor - a non-specific response to any stressor.
-In the short-term (acute stressor), the adaptation protects the body.
-But in the long-term, serious damage can occur if the stressor persists.
What experiment with animals did Selye base his model on?
Rat experiments:
-Selye exposed rats to surgical injury, excessive muscular exercise or injected them with sublethal doses of various drugs.
-He found that no matter what he did to the rats, they all shared the same collective response (a syndrome), even when the rats were given harmless injections: stomach ulcers, enlarged adrenal glands and a shrunken thymus gland.
After working with hospital patients and seeing a similar pattern of results, Selye concluded that regardless of the stressor, the same general, non-specific response is the outcome - the GAS model was born.
What are the 3 stages of the General Adaptation Syndrome (GAS)?
1) Alarm reaction - threat/stressor recognised.
2) Resistance - body attempts to adapt to the stressor by resisting its impact.
-If the stress continues:
3) Exhaustion - the body cannot maintain normal functioning.
What happens at the alarm reaction stage? What happens in terms of the body’s resources?
-When a stressor is perceived, the immediate reaction is shock.
-At this time, the body’s resources (energy) are briefly decreased (i.e not used / resistance is lowered) before quickly recovering.
-Fight or flight system (SAM) deployed - adrenaline and noradrenaline produced. Heart rate, blood pressure, breathing rate and sweating increases. Sugar is released into the blood.
-PAS also deployed to help cope with the demands of the fight or flight system in response to the stressor.
What happens at the resistance stage? What happens in terms of the body’s resources?
-After the initial shock, the body begins to repair itself as the parasympathetic nervous system takes over - focus now is to conserve energy for the long term (in case the stressor persists - chronic).
-Resistance peaks at this stage as the body attempts to adapt.
-The body’s resources (sugars, neurotransmitters, proteins and hormones) are fully mobilised.
-The individual appears to be coping as the alarm symptoms disappear (heart rate reduces), but the body’s resources are being consumed at a potentially harmful rate.
-Resources begin to deplete - e.g cortisol (which is primary concerned with the body’s use of energy) produced in huge quantities to help cope, but will soon be depleted - the body cannot continue to resist the stress indefinitely because it cannot generate new resources.
What happens at the exhaustion stage? What happens in terms of the body’s resources?
-This stage is the result of chronic stress. If the stressor has continued to persist, exhaustion occurs.
-Struggling with stress for long periods leads to the depletion of the body’s resources - they are completely drained.
-Resistance plummets and gives way to exhaustion.
-Damaged and enlarged adrenal glands cause the parasympathetic branch to fail and the alarm symptoms from the first stage begin to reappear (increased cortisol can also cause problems).
-The physical effects of this stage also weaken/compromise the immune system as the production of proteins necessary for its functioning have been reduced. This puts the body at risk of stress-related illnesses, or ‘diseases of adaptation’ as Selye called them - e.g raised blood pressure, coronary heart disease, ulcers and depression.
What mistake did Selye make about the exhaustion stage?
Selye said we are depleted of resources - this is not the case.
-Whilst it may appear that way, stress only lowers immunity and does not directly cause disease.
-E.g ulcers are caused by bacteria - the bacteria can grow if the immune system is weakened - therefore stress is only linked indirectly.