Supraventricular Tachycardia Flashcards

1
Q

What is supraventricular tachycardia caused by?

A

electrical signal re-entering the atria from the ventricles.
Once the signal is back in the atria, it again travels through the atrioventricular node to the ventricles, causing another ventricular contraction.
This causes a self-perpetuating electrical loop without an endpoint, resulting in narrow complex tachycardia.

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2
Q

What length of QRS is seen in SVT?

A

Narrow complex tachycardia
<0.12s
<3 small squares

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3
Q

What does SVT look like?

A

QRS complex immediately followed by a T wave and so on
P waves are buried in the T waves

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4
Q

What does atrial fibrillation look like?

A

Absent p waves
Irregularly irregular

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5
Q

What does atrial flutter look like?

A

Sawtooth
Atrial rate around 300bpm

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6
Q

When can SVT cause a broad complex tachycardia?

A

When patient also has a bundle branch block

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7
Q

What are the 3 main types of SVT?

A

AVNRT
AVRT
Atrial tachycardia

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8
Q

What is AVNRT?

A

• Atrioventricular nodal re-entrant tachycardia (AVNRT) = is where the re-entry point is back through the atrioventricular node.

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9
Q

What is AVRT?

A

• Atrioventricular re-entrant tachycardia (AVRT) = is where the re-entry point is an accessory pathway.
○ An additional electrical pathway, somewhere between the atria and the ventricles, lets electricity back through from the ventricles to the atria.

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10
Q

What is atrial tachycardia SVT?

A

• Atrial tachycardia = is where the electrical signal originates in the atria somewhere other than the sinoatrial node.
○ This is not caused by a signal re-entering from the ventricles but from abnormally generated electrical activity in the atria.
○ Ectopic atrial activity
○ Atrial rate >100bpm

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11
Q

What is wolf Parkinson white syndrome?

A

• Wolff-Parkinson-White syndrome (WPW) is caused by an extra electrical pathway connecting the atria and ventricles.

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12
Q

What is the extra pathway in wolf Parkinson white syndrome called?

A

Bundle of Kent

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13
Q

What ecg changes are seen in wolf Parkinson white syndrome?

A

• Short PR interval, less than 0.12 seconds
• Wide QRS complex, greater than 0.12 seconds
• Delta wave = slurred upstroke of the QRS complex
○ It is caused by the electricity prematurely entering the ventricles through the accessory pathway.

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14
Q

What is the management of Wolff Parkinson white syndrome?

A

Radio frequency ablation of the accessory pathway

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15
Q

Why are most anti-arrhythmic drugs e.g. beta blockers, CCB, digoxin contra-indicated in Wolff Parkinson white syndrome?

A

increase this risk by reducing conduction through the AV node and promoting conduction through the accessory pathway.

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16
Q

What is the stepwise management of SVT? (4)

A

a. Step 1: Vagal manoeuvres
b. Step 2: Adenosine
c. Step 3: Verapamil or a beta blocker
d. Step 4: Synchronised DC cardioversion

17
Q

What is the usual management for patients with Wolff Parkinson white syndrome who develop atrial fibrillation or flutter?

A

Procainamide (if stable)
Electrical cardioversion (if unstable)

18
Q

What is the action of vagal manoeuvres?

A

• Vagal manoeuvres stimulate the vagus nerve, increasing the activity in the parasympathetic nervous system.
• This can slow the conduction of electrical activity in the heart, terminating an episode of supraventricular tachycardia.

19
Q

What are 3 types of vagal manoeuvres?

A

Valsalva manoeuvres
Carotid sinus massage
Diving reflex

20
Q

What is done in a valsalva manoeuvre?

A

• Valsalva manoeuvres involve increasing the intrathoracic pressure.
○ This can be achieved by having the patient blow hard against resistance, for example, blowing into a 10ml syringe for 10-15 seconds.

21
Q

What is the action of adenosine?

A

• Adenosine works by slowing cardiac conduction, primarily through the AV node.
It interrupts the AV node or accessory pathway during SVT and “resets” it to sinus rhythm

22
Q

How is adenosine given?

A

○ It needs to be given as a rapid bolus to ensure it reaches the heart with enough impact to interrupt the pathway for a short period.

23
Q

What do patients need to be warned of when being given adenosine?

A

○ It will often cause a brief period of asystole or bradycardia that can be scary for the patient and doctor. However, it metabolises quickly, and sinus rhythm will return.

24
Q

When is adenosine avoided? (6)

A

• Asthma
• COPD
• Heart failure
• Heart block
• Severe hypotension
Potential atrial arrhythmia with underlying pre-excitation

25
Q

How does synchronised dc cardioversion work?

A

• A defibrillator machine monitors the electrical signal, particularly identifying the R waves.
• An electric shock is synchronised with a ventricular contraction, at the R wave on the ECG.
• If successful, the shock will be followed by sinus rhythm.

26
Q

Why is synchronised cardioversion used in patients with a pulse rather than unsynchronised?

A

Avoid shocking during a t wave
○ Delivering a shock during a T wave can result in ventricular fibrillation and, subsequently, cardiac arrest.

27
Q

How is paroxysmal SVT managed? (2)

A

• Radiofrequency ablation
• Long-term medication (e.g., beta blockers, calcium channel blockers or amiodarone)

28
Q

How does radio frequency ablation work?

A

radiofrequency ablation (heat) is applied to burn the abnormal electrical pathway.
• This leaves scar tissue that does not conduct electrical activity.
• Destroying the abnormal electrical pathway aims to remove the source of the arrhythmia.

29
Q

What arrhythmias can be permanently resolved with radio frequency ablation? (4)

A

○ Atrial fibrillation
○ Atrial flutter
○ Supraventricular tachycardias
○ Wolff-Parkinson-White syndrome