Task 5 Flashcards

(52 cards)

1
Q

What is meant by James’ peripheral feedback theory ?

A
  • bodily reaction influence emotional feeling

- we are afraid because we run

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2
Q

What is meant by Cannon and Bard’s central theory ? Also, what is the experiment which leaded him to his thesis ?

A
  • First experience the emotion and then bodily reaction
  • figured out part of the behaviourl pathway
  • hypothalamus is key region in the control of fight or flight responses and other emotions
  • cat experiment
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3
Q

According to cannon and Bard’s central theory what is the function of the hypothalamus ?

A
  • produce emotional responses such as rage or fight and flight (shame rage)
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4
Q

According to cannon and Bard’s central theory what is the function of the cerbral cortex ?

A
  • it acts as an evaluator of action

- it figures out the “purpose”

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5
Q

What was papez theory ?

A
  • he saw a neural cicuit as a cause for emotional behaviour

- added mamillary bodys and thalamus as important structures to brocas idea !

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6
Q

How dord papez circuit looks like ?

A
  1. hippomcampus ventral -> fornix -> Mamillary body -> Anterior nucleus thalamus -> cingulate gyrus exchange -> back to hippocampus ventral
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7
Q

What did Mc lean discover ?

A
  • emotion is the product of “visceral brain” = limbic system

- and mainly added to the papez circuit the amygdala prefrontal cortex and hypothalamus

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8
Q

What is the differnce between papez and Mc lean ?

A
  • papez saw cingulate cortex as point of convergance

- McLean saw hippocampus as point of convergence

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9
Q

What were some other significant findings regarding Mc Lean ?

A
  • Saw emotional responses as essential for survival
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10
Q

Why was Mc Leans theory in comparison to papez theory wrong ?

A
  • because people with hippocampical damage were still able to experience emotions
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11
Q

What is meant by the Klüver-Bucy syndrome ?

A
  • abnormal behaviour seen in monkeys after the removal of much of the limbic system
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12
Q

What happens when much of the limbic system is removed ?

A
  • Hyperactivity and hypersexuality
  • No motor or vocal reaction regarding anger or fear was present
  • Visual agnosia: unable to recognize (but not blind)
  • memory problems
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13
Q

What were the explanations for the Klüver-Bucy syndrome ?

A
  1. First: interruption of the pathways explained by Papez

2. Later: demonstrated that removal of amygdala alone was enough

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14
Q

Explain the downer experiment:

A
  • removed amygdala on only one side

- cutting the optic chiasm and collosum -> not anymore contralateral function

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15
Q

What were the fndings of the Downer experiment ?

A
  • Aggressive behaviour is due to amygdala
  • Due to the fact that the syndrom was only present while seing with the leasiond eye
  • The syndrome only ocurs if just visual input are present
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16
Q

Explain the Le doux experiment:

A
  • conditioned fear experiments with rats (tone+shock) -> measured by blood pressure and freezing
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17
Q

What were the findings of the Le doux experiment ?

A
  • Cortex (conscious experience ) and amygdala (unconsciously) faster
  • Both have the same onset
  • figured out the direct and indirect pathway
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18
Q

What was Le doux final hypothesis ?

A
  • Amygdala establishes association between neutral stimuli and stimuli with reinforcement value
  • Got support by the fact that LTP occurs in amygdala
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19
Q

Where is the amygdala more involved in regarding conscious feelings or unconscious feelings ?

A
  • mainly involved in unconscious emotional states/feelings
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20
Q

Explain pavlov fear conditioning:

A
  • it is a learned condition between US (eg, shock) and the conditioned stimuli (CS) that predict the US
  • > being exposed by only CS leads to same bodily symptom as being exposed to CS and US
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21
Q

What happens when the amygdala is lessioned and what happens when hippocampus is leasioned ?

A
  • Amygdala lessioned = conditioning can not occur
  • Hippocampus lesioned = conditioning can still occur
  • > Function of hippocmapus = explicit learning (conscious learning)
  • > Function of amygdala = implicit learning (unconscious learning)
  • > interaction between both
22
Q

Which major parts inside the amygdala play an important role in the amygdala ?

A
  • dorsal part of Lateral nucleus = initiate learning when the CS and US are paired
  • ventral part of olateral nucleus = mediate the long-term memory of the CS–US association -> LTP
23
Q

What controls unconditioned fear ?

A
  • are mediated by the olfactory system to the medial amygdala
  • fear which is not conditioned so not based on experiment
24
Q

What is special for humans regarding fear conditioing ?

A
  • simply telling the subject about CS and US association is enough
  • via observing someone undergoing a fear condition is also enough
25
Besides anger and fear conditioning, what are other functions of the amygdala ?
- positive emotions and processing of reward
26
What is so sepcial about the Visceral motor system ?
- shows the observable emotional body reaction of aroused | - sweating our blushing
27
What is the definition of emotions ?
- automatic, largely unconscious behavioural and cognitive responses triggered when the brain detects a positively or negatively charged significant stimulus.
28
What is a feeling ?
- conscious experience of somatic and cognitive changes
29
What are the 3 systems which control emotionally competent via the amygdala?
- The endocrine system - Autonomic system - the behaviour system
30
What is the function of the The endocrine system ? And what is the pathway of the Endocrine system:
- hormone pathway (cortisol) - slow respond - amygdala -> hypothalamus (ParaVentralNucleus) release CRF -> anterior pituitary -> release (ACTH) adrenal cortex ->cortisol
31
What is the function of the The autonomic system ?
- neurotransmitter pathway (adrenaline) - fast pathway - central amygdala (output)-> lateral hypothalamus -> adrenals cortex (medulla) -> spinal cord -> adrenaline
32
What is the function of the behaviour motor system ?
- system mediates overt/visible behaviours such as freezing | - Medial hypothalamus -> PAG -> beahvioural fear repsond
33
According to Bard’s central theory, what would happen if the hypothalamus is damaged ?
- no raging behaviour
34
According to Bard’s central theory, what would happen if the hypothalamus is intact but the cerebral cortex is damaged?
- u just rage with no purpose
35
Who was the first guy who indicated the idea of a limbic system ?
Paul broca - a ring of cortical areas (cingulate gyrus and hippocampus) in connection with olffactoric system
36
Explain the indirect pathway regarding fear conditioning:
-> US Pathway -> somatenory thalamus -> somatensory cortex -> amygdala -> (basolateral nucleus -> central nucleus) -> towards one of the 3 pathways mentioned above (indirect pathway) - > CS Pathway-> auditory thalamus-> auditory cortex -> amygdala -> (basolateral nucleus -> central nucleus) -> towards one of the 3 pathways mentioned above (indirect pathway) - > not necessary for conditioning
37
Explain the direct pathway regarding fear conditioning:
- Same as indirect pathway besided the cortex part is missing - > necessary for conditioning
38
In what two major parts is the amygdala splited up in ?
1. Basolateral (input) -> Papez circuit + PFC | 2. central amygdala (output) -> 3 pathways
39
Name the two types of the automatic nervous system ?
- Sympathetic system prepare for action-> works | - parasympathetic -> prepare for rest
40
Where do all of the 3 major pathways gain there knowlegde from ?
- Basolateral (input) -> Papez circuit + PFC - hippocampus (ventral) -> fornix -> Mamillary body -> Anterior nucleus thalamus -> cingulate gyrus (exchange also with (PFC))-> back to hippocampus ventral (also in exchange with PFC)
41
Name the 2 descending pathways and some unique features ?
- Volitional Movement and Actual Emotional Expression
42
Explain the volitional movement:
- via pyaramedial trac - projection from corex and brainstm - lateral motor cortex = distal movements (fine cntrolling) - Medial motor cortex = proximal movements (posture) - > via reticular formation towards cranial nerve nuclei and ventral horn -> movement !
43
Explain the emotional expression:
- via extrapyramidal projection - projection from forebrain and hypothalamus - medial motor cortex = ryhtmetical reflexes - lateral motor cortex = sepcific emotional behvaiours - > via reicular formation -> autonomic neurons -> moevemnet
44
What does pyramidal smile mean ?
- Forced smile | - follows the volontational pathway
45
What does the duchenne smile mean ?
- Emotional smile | - follows the emotional pathway
46
What is meant by emotional facial paresis and what are the causes of it ?
- Actual expression of emotion is not present | - damage in forebrain or hypothalamus ( emotional pathway)
47
What is meant by Voluntary facial paresis and what are the causes of it ?
- can not smile forecfully because of damage within brainstem and motor cortex (volitational pathway)
48
How can we define anxiety disorder ?
- based on gentic and enviormental influence Stres - CRH is overexpressed = increase anxiety - hyperactivity of the amygdala and diminished activity of the hippocampus
49
How does an increased lvl of cortisol occur regading anxiety disoreder ?
1. Stress releases CRH hormones in hypothalamus -> which leads to release of by Acth in pituitary gland -> adrenal cortex -> cortisol - to much cortisol damages hippocampus - This is the reason why hippocampus based on a negative feedback loop can influence the state of HPA
50
How does the monoamine hypothesis explain depression ?
- mood is connected to levels of released monoamine neurotransmitters in brain (norepinephrine and/or serotonin ) - To less monoamines - only biological based
51
How does the Diathesis-stress hypothesis explain depression ?
- mood disorders caused by genes + bad childhood experience | - caused by a HPA hyperactive -> which is caused by a dsruption in the feedback loop controlled by hippocampus
52
What were the limitations of the monoamine hypothesis?
- transmission effects of drugs occurs right away but it takes a while for symptoms to get better - cocaine alos increases monoamine but does not treat depression