TBI Flashcards

(38 cards)

1
Q

Add pleural effusion

A
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2
Q

Normal ICP for a supine Pt.

A

10-15 mmHg

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3
Q

What is the ceiling ICP in many ICUs

A

20mmHg

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4
Q

What happens if the ICP equals MAP

A

Perfusion ceases and brain dies

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5
Q

What is cerebral perfusion pressure

A

Indicates adequate perfusion to brain, which can be measured directly

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6
Q

Formula for cerebral perfusion pressure (CPP)

A

MAP-ICP

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7
Q

What is a normal CPP value

A

65-80

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8
Q

What GCS indicates mild TBI

A

14-15

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9
Q

What GCS indicates moderate TBI

A

9-13

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10
Q

What GCS indicates severe TBI

A

Equal or less than 8

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11
Q

Why do patients with TBI get blown

A

Optic nerve is paralyzed

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12
Q

What is mydriasis

A

A blown pupil

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13
Q

What is decorticate posture

A

Abnormal flexion

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14
Q

What is decerebrate

A

Abnormal Extension

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15
Q

What are symptoms of impending or active cerebral herniation

A

Decorticate or decerebrate

Pupillary asymmetry

Bilateral or unilateral fixed and dilated pupils

Cushing triad: hypertensio, Brady and irregular RR

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16
Q

What causes brain herniation

A

Increased cerebral pressure

17
Q

What is the danger of brain herniation

A

ICP squeezes brain out of its restraints (skull)

18
Q

What is a midline shift (brain)

A

BBB is compressed (mass effect)

Compresses the ventricle and pushes midline over

19
Q

Examples of primary brain injury

A

Direct impact

Rapid acceleration and deceleration

Penetrating injury

Blast wave

20
Q

What are examples of secondary brain injury

A

Cerebral edema

Inflammation

Electrolytes imbalance

Death of neurons

21
Q

What causes secondary brain injury

A

Subtle changes in the brain and nervous system that follow primary injury

22
Q

Time course of cerebral edema

A

Accumulation is gradual

Peaks in a few days

Contributes to intracranial hypertension

23
Q

What is the most common cause of cerebral edema

A

Vasogenic edema

24
Q

What is the goal for treatment of TBI

A

Treat primary brain injury

Stall or minimize secondary brain injury

25
What is the danger of cerebral edema
At the time of initial injury, a catecholamine surge increases HR and BP potentially damaging brain’s architecture
26
What are the four interventions for treating ICP
Hyperosmolar 4 solution External ventricular drains Evacuation of blood clot Craniectomy
27
What are hyperosmolar solutions
Mannitol (IV) Hypertonic saline 3% (IV)
28
How does hyperosmolar IV work
Pulls fluid back into vessels
29
Danger of intubating a patient with ICP
Intubation can increase it Gentle intubation technique Comatose patients can still generate reflex response (stim of supraglottic larynx)
30
What are respiratory therapist role in avoiding secondary injury of ICP
Prevent 2nd injury Gentle intubation Mgt vent Mgt O2
31
Hazards when hyperventilating patients with ICP
PaCO2 <25mmHg can decrease ICP, but can reduce blood delivery
32
Should hyperventilation be used for ICP
Can be, but should be avoided 25-30mmHg
33
When is hyperventilation with ICP dangerous
24-48 hrs Tolerance increases after
34
Means to relieve ICP
Extraventricular drains Evacuation of blood clot Crainectomy
35
How doe craniectomy work
Open skull Remove clot Give brain room to expand Return
36
Difference between crainectomy vs Crainotomy
Otomy is returned Ectomy is held
37
What does IICP stand for
Increased intracranial pressure
38