TBI Flashcards

1
Q

Causes of acquire brain injuries-7

A
  1. strokes
  2. Tumors
  3. anoxia
  4. hypoxia
  5. toxins
  6. Degenerative disease
  7. near drowing
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2
Q

Definition of TBI

A

NOT DEGENERATIVE OR CONGENITAL

  • caused by external physical force
  • alteration in brain function caused by an external force
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3
Q

Outcomes of TBI depend on 5

A
immediate damage
cumulative effects
pre-morbid
substance abuse
interpersonal relationships and work history
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4
Q

Open Vs Close brain injuries

A

OPEN:

  • penetrating types of wounds
  • gunshot, knife
  • skull is either fractured or displaced
  • brain injury follows path of object entry and exit
  • risk of infection increased from open wound and hair, fragments

Closed:

  • impact to the head but the skull does not fracture or displace
  • brain tissue damaged and dura remains intact
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5
Q

Contusion

A
  • bruising on the surface of the brain

- small blood vessels on the surface hemorrhage

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6
Q

Coup vs CounterCoup

A

Coup:
- same side of brain where impact is

Counter:

  • opp side of trauma
  • acceleration can cause further vessel occlusion and edema formation
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7
Q

Epidural Hematomas

A
  • between dura mater and skull
  • seen with MVA blow to head and side of skull fracture
  • Arterial Hematoma

Signs:

  • unconsciousness and then alert&lucid
  • immediate surgical intervention needed
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8
Q

Subdural Hematoma

A
  • venous hematoma
  • rupture of cortical bridging of veins
  • between dura and arachnoid
  • common in older adults who fall
  • slow to build up and detect

signs:
- resemble CVA and flucuate

Small clots can be reabsorbed vs. large need to be removed

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9
Q

Subarachnoid hematoma

A

space below arachnoid and above the pia mater

  • associated with cerebral aneurysm
  • -but can be caused by skull fx
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10
Q

Diffuse Anoxal injury

A
  • common

- usually from shaking brain back and forth

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11
Q

Symptoms of Diffuse Anoxal Injury

A
disorientation or confusion
headache
nausea or vomit
drowsiness or fatigue 
trouble sleeping
sleeping longer than normal
LOB or dizziness
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12
Q

Concussion

A

most common
can be caused by open or closed injury
momentary loss of consciousness and reflexes
repeated concussion leads to CTE

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13
Q

Symptoms of concussion

A
dizziness
disorientation
blurred vision
difficulty concentrating
altered sleep patterns
nausea
headache 
LOB
amnesia
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14
Q

3 types of secondary problems

A

*cerebral damage occurs in result to initial injury
1. Increased ICP
2 anoxic injuries
3. postraumatic epilepsy

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15
Q

Increased ICP

A
  • skull is rigid and does not expand to accommodate edema
  • leads to compression of brain tissue, decreased perfusion of blood and possible herniation

Treatment: monitor, drugs, shunt

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16
Q

S&S of increased ICP

A
decreased responsiveness
impaired consciousness
severe headache
vomiting 
irritability
papilledema
changes in vital signs--increased BP and decreased HR
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17
Q

normal ICP range

A

5-10mmHg—> above 20 is abnormal

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18
Q

anoxic injuries

A

brain tissue demands 20% of bodys O2 intake to maintain proper o2 sats and metabolic functions

  • cardiac arrest most frequent cause
  • causes diffuse damage within brain tissue
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19
Q

What areas of the brain are vulnerable from anoxic injuries

A

hippocampus
cerebellum
basal ganglia

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20
Q

Post traumatic Epilepsy

A
  • at increased risk after TBI
  • open injury: subdural hematoma->older adults
  • vestibular stimulation is contraindicated
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21
Q

what triggers a seizure

A
stress
poor nutrition
electrolyte imbalance 
missed meds
flickering lights
infection
lack of sleep
fever 
anger 
worry 
fear
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22
Q

what do you do if a pt. has a seizure

A

bring them to lowest safe level
do not put anything in mouth
protect them from hitting head but do not restrain
flip on side if they are starting to vomit

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23
Q

medications for seizures

A

Phenytoin
phenobarbital
carbamazepine

**also used to control behavior

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24
Q

Glasgow Coma Scale

A
  • used in ER to assess the individuals level of arousal and function of cerebral cortex
  • TYPICALLY: 3-4 scores do not make it
  • most powerful predictor of prognosis and outcome of TBI
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25
Mild TBI
- GCS of 13 or higher - LOC less than 20 minutes - normal CT - awake when arriving to hospital - may present dazed, confused - c/o headache or fatigue
26
Moderate TBI
GCS 9-12 - confused and unable to answer questions - most have permanent physical, cognitive and behavioral deficits
27
Severe TBI
GCS of 3-8 individual is in coma permanent functional and cognitive impairments
28
7 manifestations of TBI
``` decreased level of consciousness cognitive deficits motor deficits sensory deficits communication deficits behavioral deficits associated problems ```
29
Definition of Coma
decreased level of awareness state of unconsciousness presents with: eyes remain closed unable to initiate voluntary activity sleep/wake cycle not present
30
vegetative state
person who demonstrates a return of brain stem functions - respiration - digestion - BP control no sleep/wake cycle may experience arousal and spontaneous eye opening pain responses may be evident unaware of external and internal needs
31
persistent vegetative
been in this state for a year or longer with no improvements in neurological status
32
arousal
regulated by RAS
33
awareness
consciousness of int/ext env. stimuli
34
consciousness
state of being aware
35
stupor
condition of general unresponsiveness
36
obtundity
people who sleep a great deal of the time when aroused demonstrate disinterest in env. and slow to respond to sensory stim.
37
delirium
disorientation, fear, misperception of sens. stim
38
clouding of consciousness
state in which a person is confused, distracted and has poor memory
39
Cognitive deficits
dysfunction can include: disorientation, poor attention span, loss of memory, poor orginizational and reasoning skills and inability to control emotional response affect ability to learn new skills
40
motor deficits
*when unconscious pt unable to initiate active movement | abnormal posture are frequently seen from brain stem injury
41
Decerebrate Rigidity
``` LE: Hip add Hip IR Knee ext PF supintation ``` UE: Shoulder ext and IR elbow ext pronation wrist/fingers flex
42
what causes decerebrate rigidity
severing of the neuroaxis in midbrain region Pons medulla and SC remain functional
43
decorticate rigidity
``` UE: shoulder flex, add, IR Elbow flex pronation wrist flex ``` LE: extension
44
what causes decorticate rigidity
dysfunction above the level of the red nucleus | between basal nuclei and thalamus
45
sensory deficits
loss of sense of smell impairment or absence of tactile or kinesthetic sensations visual deficits perceptual and proprioceptive deficits
46
communication deficits
initially lost or severely impaired decreased awareness of env. can limit opportunities for interaction may not be able to initiate communication because of abnormal tone or posturing
47
behavioral deficits
- most enduring and socially disabling deficits | - can exhibit neuroses, pychoses, sexual disinhibition, apathy, irritability, agression, low frustration tolgerance
48
associated problems
- 70% of TBI pts. will have other injuries | - may make care and rehab
49
Early intervention goals of PT for TBI
- increase pt. level of arousal - preventing development of secondary impairments - improving pt. function - provide pt. and family with education **avg. length of stay in hospital is 2 weeks
50
position of pts.
- supine-facilitate extensor tone and tonic labyrinthine - sidelying or semi-prone: reduce influence of tonic labyrinthine refelx - UE: abd and ER to inhibit abnormal tone - Position out of decebrate or decorticate postures * contractures occur quickly
51
reflex inhibiting postures
static then progressing to active movements | superimposed on static positions
52
heterotopic ossification
abnormal bone formation in soft tissues and muscles presents with loss of ROM, pain, swelling and erythema no effective treatment DRUGS: NSAIDS
53
ways to increase patients awareness
important even with coma pts. assume pt. can hear you and undestand explain what youre doing all the time orient pt. x4 and converse with pt. & family
54
RLA 1-3 characteristics
decreased level of responsiveness | little interaction with env.
55
RLA 1-3 goals and outcomes
- physical function and alertness increased - risk of secondary impairments reduced - motor control improved - effects of tone managed - postural control improves - tolerance of activities and positions increased - joint integrity and mobility improved or remain functional - education of family and caregivers
56
RLA 1-3 intervention
-prevent indirect impairments -proper positioning both in bed and sitting -turn every 2 hrs. recline w/c or tilt in space -postural drainage, percussion and vibration -ROM and orthotics to prevent contractures
57
RLA 1-3 improving arousal
``` short sessions-15 to 30 mins avoid over stim normal toone and meaningful topics of auditory photographs-visual 10-15 sec of olfactory swabs low freq vibration or stroke/rub with wash cloth rolling and neck rolling ```
58
RLA 1-3 monitoring
``` HR BP RR Diaphoresis Facial grimicing changes in posture head turning vocalization ```
59
1-3 RLA managing effects of tone and spasticity
positioning rom orthotics inhibitory tech.
60
RLA 4
confused | agitated
61
goals for RLA 4
``` pt. edurance improved joint mobility and integrity maintained risk of 2ndary impairments decreased increase tolerance of activities pt family educated on dx, prognosis, pt outcomes ```
62
RLA 4 INTERVENTION
``` utilize same therapist, time and place of tx expect no carry over model calm behavior redirect provide options expect egocentricity ```
63
RLA 4 PT. FAMILY EDUCATION
difficult to provide education directly at this level pt. does not have control of behavior entering this stage is good because it indicates improvement aggressive behaviors are usually short-lived keep consistency
64
RLA 5&6
confused but no longer agitated simple commands improved improved carry over
65
RLA 5&6 GOALS AND OUTCOMES
performance of fun. mobility and ADL skills increased gait, mobility and balance improves motor control, postural control increase risk of 2ndary impairments reduced strength and endurance increased safety with fun mobility & ADL skills improved pt. fam education tolerance of activities increased
66
RLA 5&6 INTERVENTION
ROM exercise & task physical conditioning focal lesions: balance and ataxia
67
RLA 5&6 2 TREATMENTS STRATEGIES
1. Compensatory - improve functional skills by compensating for the lost ability 2. Restorative - locomotive training via BW support and treadmill - constraint induced movement therapy for UE function
68
RLA 5&6 INTERVENTION CONSIDERATIONS
``` maintain structure emphasize safety simple instructions short term goals: -gradual increased in the number of reps&exercise time practice should be distributed -sufficient rest periods to minimize physical and mental fatigue extrinsic feedback ```
69
RLA 5&6 FAMILY EDUCATION
emphasize safety awareness ed family should learn to assit pt. with fun mobility fam should learn to assist pt. with strength &ROM fam should be aware of methods to enhance decision making skills and safety
70
RLA 7&8 CHARACTERISTICS
cognitive and emotional deficits are usually greater than the physical deficits
71
RLA 7&8 GOALS AND OUTCOMES
pt.& fam education safety of pt. & family is improved ability to perform physical tasks related to ADL skills, community, work integration and leisure is improved motor control, balance and postural control increased Strength & endurance increased level of supervision and assistance for task performance is decreased
72
RLA 7&8 INTERVENTIONS
ROM exercise and functional tasks physical conditioning ADL and IADL training - pt. integrating cognitive, physical and emotional skills necessary to function in community
73
RLA 7&8 PATIENT AND FAMILY EDUCATION
pt. should be educated how to best compensate for residual impairments and disabilities pt. and family should contact local support group
74
RLA level 1 characteristics
NO RESPONSE; TOTAL ASSIST | -complete absence of change in behavior when stimuli presented
75
RLA Level 2 Characteristics
GENERALIZED RESPONSE; TOTAL ASSIST - reflex response to painful stimuli - response to repeated auditory stimuli - response to ext. stimuli with gross body movement or vocalization
76
RLA 3 Characteristics
LOCALIZED RESPONSE;TOTAL ASSIST - withdrawal or vocalization to pain stim - head turns with auditory stim - follows objects with eyes - responds to discomfort
77
RLA 4 Characteristics
CONFUSED/AGITATED; MAX ASSIST - alert and heightened state of activity - purposeful attempts to remove tubes and restraints - non purposeful movements of divided attention - may cry or scream to stimulus - unable to cooperate with treatment - mood swings for no apparent reason - incoherent verbalization
78
RLA 5 Characteristics
CONFUSED, INAPPROPRIATE NON AGITATED;MAX ASSIST - Alert not agitated - not oriented to person, place or time - may be able to perform learned tasks with cues - simple commands can be followed but random - able to converse on social, level for brief periods of time with cues
79
RLA Level 6 Characteristics
CONFUSED, APPROPRIATE; MOD ASSIST - inconsistently oriented - able to attend to familiar tasks for 30 minutes - able to use assistive memory aide with max assist - mod assist to problem solve - unaware of impairments - verbal expressions are appropriate
80
RLA Level 7 characteristics
AUTOMATIC, APPROPRIATE; MIN ASSIST - consistently oriented - min supervision with new learning and with safety in routine at home and community - overestimates abilities - unable to recognize inappropriate social-interaction behavior - unaware of others needs and feelings
81
RLA Level 8 characteristics
PURPOSEFUL, APPROPRIATE; STAND BY ASSIST - consistently oriented - independently attends and completes task for 1 hour - uses assitive memory devices to recall schedule - thinks about consequences - aware and acknowledges impairments - over or under estimates abilities - able to recognize inappropriate social interactions - self centered and uncharacteristically independent/dependent
82
Locked in Syndrome
rare neurological disorder that results after TBI - complete paralysis of all voluntary muscles except those that control eye movement - pt. remains conscious and possesses cognitive function but is unable to move