TBI's and SCI's Flashcards

(55 cards)

1
Q

what is a primary injury?

A

effect on the brain at the time of injury

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2
Q

examples of causes of a primary injury?

A

trauma- contusion, damage to blood vessels, axonal shearing, contusion, epidural/subdural hematoma, SAH, DAI

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3
Q

what is axonal shearing?

A

when the axons of neurons are stretched and torn

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4
Q

what is a secondary injury?

A

complications that result in additional changes and dysfunction of brain tissue
after injury resulting in further damage or even death

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5
Q

examples of secondary injuries?

A

hypoxemia, hypotension, hypo/hyperglycemia, increased metabolic demands, loss of auto regulatory mechanisms, increased ICP, hypo or hypercapnia

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6
Q

what is a DAI?

A

diffuse axonal injury, is a form of TBI

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7
Q

when do DAIS occur?

A

when the brain rapidly shifts inside of the skull as an injury is occurring; the axons in the brain are sheared as the brain accelerates and decelerates inside of the skull

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8
Q

what part of the brain do DAI’s occur in?

A

many parts of the brain

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9
Q

what happens to most patients with a DAI?

A

comatose state

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10
Q

how is clinical presentation assessed?

A

using the GCS

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11
Q

what GCS score indicates a mild TBI?

A

13-15

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12
Q

what GCS score indicates a moderate TBI?

A

9-12

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13
Q

what GCS score indicates a severe TBI?

A

<8

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14
Q

what might a mild TBI cause?

A

functional deficits weeks or months following injury

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15
Q

What might we be able to see on a CT?

A

hematomas, bleeding, fractures, cerebral edema

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16
Q

what might we be able to see on an MRI?

A

DAI, brain stem injury, traumatic aneurysms

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17
Q

what does management of TBIs focus on?

A

optimizing functional recovery and prevention of secondary injury

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18
Q

things assessed and monitored to prevent secondary injury?(8)

A
airway
oxygenation
ventilation 
fluid management
ICP
cerebral perfusion
preventing increased cerebral oxygen demand
preventing complications
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19
Q

ways to support cerebral perfusion?

A
pressors:
dopamine
levo
neosynephrine
epinephrine
EEG
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20
Q

what are some examples of secondary complications?

A

pneumonia
PE
DVT
skin integrity

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21
Q

Ways to prevent secondary complications?

A

blood thinners
lung assessments
oral care
q2h turns

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22
Q

what are some examples of skull fractures?(3)

A

linear
depressed
basilar

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23
Q

what is a linear fracture?

A

generally not displaced, a hairline fracture

24
Q

what is the treatment for a linear fracture?

25
what is a depressed fracture?
inward depression of bone fragments
26
what is a treatment for a depressed fracture?
surgery
27
what is the main concern with depressed fractures?
brain damage
28
what is a basilar fracture?
involves the base of the skull | CSF leaks from nose/ears indicates dural tear
29
what is a major indicator of a basilar fracture?
battles sign- bruising behind the ears
30
what is the best way to test for a CSF leak?
pH paper
31
what are the most common causes of SCI's?
- MVA - falls - acts of violence - sports-related
32
what is the most common cause of a SCI?
MVA with 48% resulting from them
33
what is the most common population to get a SCI from a fall?
the elderly
34
what does an SCI result from?
bone, disk material, or foreign object entering the spinal canal and disrupting the spinal cord or blood supply
35
What are the mechanisms of injury for an SCI?
- whiplash (hyperflexion/extension) - axial loading, compression (diving and hitting head) - rotation - penetrating trauma (knife, GSW)
36
types of SCI damage?
- concussion (shaking of brain, spinal cord) - contusion (bruising) - laceration(cut) - transection (severing) - hemorrhage (bleeding out) - blood vessel damage
37
what will you see with secondary injury?
HA, changes in LOC, respiratory fx
38
what is neurogenic shock shown by?
``` MAP, CVP, CPP loss autonomic nervous system influence (T1-L2) decreased HR vasodilation edema ```
39
what is an incomplete SCI?
varying degree of sensory and motor function disruption because some tracts intact
40
what is a complete SCI?
total loss of sensory and motor function below level of injury caused by complete interruption of pathways
41
what is the presentation with neurogenic shock?
hypotension, warm-flushed skin, diaphragmatic breathing
42
treatment for neurogenic shock?
- dopamine - vasopressin - ephedrine - norepinephrine - phenylephrine - atropine
43
what is the first line treatment for neurogenic shock?
phenylephrine
44
what is an example of a muscle relaxant?
cyclobenzaprine
45
what is an example of an anticonvulsant?
levatiracetam
46
what is an example of an antineuropathic?
gabapentin
47
what is autonomic dysreflexia?
a life threatening condition in pts with injury at or above T6
48
what can autonomic dysreflexia precipitate?
seizure or stroke
49
what is autonomic dysreflexia caused by?
unopposed sympathetic response below level of injury
50
when can autonomic dysreflexia occur?
anytime after spinal shock resolves, usually in first year after injury
51
what triggers autonomic dysreflexia?
- over distended bladder - full rectum - infection - pressure sore - pain
52
what is the most common cause of autonomic dysreflexia?
over distended bladder
53
what does the stimulus from causes of autonomic dysreflexia cause?
marked elevation in blood pressure
54
symptoms of autonomic dysreflexia?
- severe HA - nasal congestion - SOB - nausea - blurred vision - facial flushing - diaphoresis - piloerection - anxiety
55
what is the difference between neurogenic shock and autonomic dysreflexia?
shock has hypotension and bradycardia | autonomic dysreflexia is a hypertensive crises