TEST 1 BURNS Flashcards

1
Q
  1. Burns diet (pg.917)
A

 increase calories & protein
 increase fluid intake
 increase vitamin A (fatty fish, egg yolks, dark yellow/orange fruits
& veggies: yam, carrots, apricots, squash, cantoloupe)
 increase vitamin C (oranges, orange juice, strawberries, green leafy veggies, tomatoes, lemons, peppers)
 increase vitamin zinc (meat, fish, seafood, oysters, lentils, Greek yogurt, broccoli, kidney beans, blueberries)

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2
Q
  1. Burns nursing actions
A

 Use a cradle sheet to maintain body warmth
 Assess for patent airway or evidence of inhalation injury, monitor respiratory
 Avoid using sponges for baths
 Change gloves between different areas of burns
 Evaluate pain & administer IV analgesics

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3
Q
  1. Burns priority care
A

 Give IV fluid (crystalloid & colloid)
 Give IV analgesics
 Give O2 as ordered
 Restrict oral meds & fluids
 Elevate extremities above heart level to prevent shock
 Use powder free sterile gloves
 Client may have slight hoarseness, if it gets worse this indicates respiratory distress or if nose hairs have black soot beware it can also sign of respiratory distress due to inhalation injury

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4
Q
  1. Skin grafting client education
A

 Use of sunscreen w/high sun protection factor when outdoors
 Use pressure garment after healing
 Use mild soap, wash & rinse thoroughly, patted dry.
 Avoid things w/irritants(lanolin, alcohol, perfumes)
 Wear warm clothing w/skin graft
 Film dressing to cover at first

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5
Q
  1. Skin grafting nursing actions (pg.915)
A

 Administer pain analgesics
 Monitor for infections
 1st 2-4weeks restrict movement

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6
Q
  1. Hypovolemic shock evaluation outcomes
A

-urinary output w/in normal limits
-normal V/S

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7
Q
  1. Hypovolemic shock medical management (ATI pg.234)
A

 Fluid replacement w/colloids & crystalloids- LR & NS (mainly as first line) Ratio of 3:1 fluids

 Vasoconstrictors (dopamine, norepinephrine, phenylephrine)

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8
Q
  1. Hypovolemic shock nursing actions (pp slide 3)
A

 Prepare for administration of blood & fluids (NS & or LR w/3:1 ratio)

 O2 administration (on forehead for o2 stat)

 Monitor weight, VS, including temperature (risk of hypothermia w/large volumes of fluid)

 Position client supine or Trendelenburg w/feet slightly elevated. Never high fowlers. (adjust for respiratory s/s)

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9
Q
  1. Septic shock early manifestations (initial phase) pp slide 6
A

 Elevated WBC
 Fever
 Warm, flushed skin —>Progresses to cold pale skin
 Rapid, bounding pulse

Later manifestations:
 Hypotensive
 Delayed capillary refill
 Disorientation

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10
Q
  1. Impending Shock: nursing actions (pp slide 13)
A

Decreased cardiac output impending shock
 Restrict activity to total rest
 Establish IV sites
 Assist w/fluids/bloods

IMPAIRED TISSUE PERFUSION
 Assess w/Neurovascular checks
 Control frank bleeding
 Elevate legs

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11
Q
  1. Hemorrhagic shock: medical management
A

 Fluid replacement w/colloids & crystalloids
 Vasoconstrictors (dopamine, norepinephrine, phenylephrine)
 Transfuse O- blood to client who is bleeding profusely

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12
Q
  1. Neurogenic shock manifestations (pp slide 7):
A

 Dry, warm skin
 Hypotension
 Bradycardia

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13
Q
  1. Stages of shock: manifestations (pp slide 9)
A

Initial:
 Too little O2 in blood to feed organs-anaerobic metabolism (mostly absent s/s)

Compensatory:
 Increased HR
 RR increases
 BP can be stable

Progressive:
 Cold & clammy skin
 Early sign: body cant compensate

Irreversible:
 multi
 Multi-organ failure & death

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14
Q
  1. Hypovolemic & Cardiogenic Shock causes?
A

 Blood or plasma loss, surgery, trauma, birth
 Fluid loss, burns, large draining wounds, suctioning, diabetes insipidus

Cardiogenic shock causes:
MI,HF

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15
Q
  1. Septic shock causes
A

 Bacterial infections (E-coli, Staphylococcus aureus, psuedomonas)
 Endotoxins: harmful chemicals

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16
Q
  1. Neurogenic Shock causes:
A

 General anesthetic
 Opioid overdose
 Tranquilizers
 Spinal cord injury T6 or higher

17
Q
  1. Anaphylactic shock causes
A

 Bee venom
 Latex
 Fish
 Nuts
 PCN (penicillin)

18
Q
  1. Obstructive shock causes:
A

 Cardiac tamponade
 Ascites or enlarged liver
 Tension Pneumothorax

19
Q
  1. Primary shock assessment (pp slide 10)
A

 Compromised blood volume or circulation
 VS
 Peripheral pulses
 Changes in mentation
 Skin
 Urine output
 Bowel sounds

20
Q
  1. Continued shock assessment (pp slide 12)
A

 Respirations: shallow, grunting, air hungry, rate decreases as stages progresss
 Temp: increase heat loss, subnormal
 Mentation: altered cerebral function often first sigh of early stage
-anxiety
-restlessness
-agitation
-confusion, progresses to loss of consciousness
 Skin: cold/clammy, pale, mottled, increased cap refill, cyanosis as stages progress
 Urine output: oliguria, renal damage